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1 erior and the other exterior to the orbit of Venus.
2 e movement of the Moon, and perhaps Mars and Venus.
3 mation of sulfuric acid in the atmosphere of Venus.
4 nomeric Teal Fluorescent Protein (mTFP)1 and venus.
5 evidence for lightning in the atmosphere of Venus.
6 ning), no similar signals were detected from Venus.
7 s using an in vivo clock reporter, her1:her1-venus.
8 ly identified exclusively on Earth, Mars and Venus.
9 the existence of a dust ring at the orbit of Venus.
10 ently recalculated HZ boundaries are: recent Venus--1.78; runaway greenhouse--1.04; moist greenhouse-
11 3 amino acids of the SsrA degradation tag to Venus, a rapidly folding yellow fluorescent protein, we
13 lular trafficking of ASTN1-Venus, with ASTN1-Venus accumulating in the forward aspect of the leading
14 hotter surface conditions, such as those on Venus, accumulation and inheritance of damage is negligi
16 er drives the monocistronic transcription of Venus and a puromycin-resistant gene via the foot-and-mo
17 with Mars is in many ways similar to that at Venus and at an active comet, that is, primarily an iono
22 tion of spectral ratiometric imaging of ECFP/Venus and high-speed FLIM-FRET of TagRFP/mPlum can thus
23 t across the root tip as quantified with DII-VENUS and is synergistically enhanced by hypoxia and the
25 N-terminal fragment containing residues from Venus and yellow fluorescent protein produced either con
26 here variants of yellow fluorescent protein (Venus) and cyan fluorescent protein (Cerulean) flank eit
28 e impact cratering record of the Moon, Mars, Venus, and Mercury and from the size distributions of as
30 ces in the epic: to Bootes and the Pleiades, Venus, and the New Moon; we supplement them with a conje
31 ation is greater for Venus-FMRP RNA than for Venus-ARC RNA and is increased in Fmr1-knockout neurons
34 fused combinations of Cerulean as donor (D), Venus as acceptor (A), and a photo-insensitive molecule
36 he core of smaller planets such as Earth and Venus as well as exoplanets: as planets cool off, the su
37 We tested model predictions using the DII-VENUS auxin sensor in conjunction with state-of-the-art
38 In addition, illumination of Mrgprd-ChR2-Venus(+) axon terminals in spinal cord slices evoked EPS
40 d acceptor, respectively, we have combined a Venus-based BiFC system with Cerulean to develop a BiFC-
42 dermal growth factor stimulation and an ECFP/Venus Cameleon FRET sensor for monitoring calcium transi
43 osis, pathogenesis, and treatment of central venus catheter-related thromboses are critical in the tr
47 om mice expressing one or two copies of ChR2-Venus could be activated in vitro as evidenced by light-
50 the possibility of liquid water existing on Venus early in its history, and extends the size of the
51 2), an important planetary material found in Venus, Earth, and Mars, is vital to the study of the evo
52 sensor with a circularly permuted version of Venus enhanced sensor dynamic range nearly 2.5-fold.
53 that RGS14-Luciferase and active H-Ras(G/V)-Venus exhibit a robust BRET signal at the plasma membran
55 Spectrometer on the European Space Agency's Venus Express spacecraft to identify compositional diffe
56 al replication and virulence in mice of this Venus-expressing influenza virus are primarily conferred
57 n enriched environment led to enhancement of Venus expression in dendritic segments of somatosensory
58 uronal activity using the coding sequence of Venus, flanked by short stretches of the 5'- and 3'-untr
59 nts of diffusion and molecular brightness of Venus fluorescent protein (FP) can be performed in solut
60 e found that an influenza virus encoding the Venus fluorescent protein acquired two mutations in its
61 rons with the mutant alleles tagged with the Venus fluorescent protein also revealed excess PM locali
62 ructed MYXV and VACV virions tagged with the Venus fluorescent protein and compared their characteris
64 yze ERAD based on mutants of split or intact Venus fluorescent protein for which fluorescence depends
67 N1) reporter virus that stably expresses the Venus fluorescent protein to identify antigen-bearing ce
68 rescuing transgene, VNS::SYS-1, which fuses VENUS fluorescent protein to SYS-1, we find more VNS::SY
70 on enables macrophages to serve as cellular "Venus fly traps", with the capacity to capture phagocyti
73 endopeptidases in the digestive fluid of the Venus flytrap (Dionaea muscipula) are cysteine proteases
75 nsor kinase BvgS is composed of four bilobed Venus flytrap (VFT) perception domains followed by alpha
76 ectrical properties of the upper leaf of the Venus flytrap and proposed the equivalent electrical cir
81 d receptor that has an extracellular bilobed venus flytrap domain (VFTD) predicted to contain five ca
82 ative ligand binding model that involved the Venus flytrap domain of T1R1 in which l-glutamate binds
83 operative ligand-binding model involving the Venus flytrap domain of T1R1, where L-glutamate binds cl
84 G protein-coupled receptors, the N-terminal Venus flytrap domain of T1R2 is required for recognizing
85 e hinge region and induce the closure of the Venus flytrap domain of T1R2, the enhancers bind close t
86 ng pocket localized in mGlu7's extracellular Venus flytrap domain, a region generally known for ortho
87 ctive interaction between XAP044 and mGlu7's Venus flytrap domain, whose three-dimensional structure
88 gth TAS1R2/TAS1R3 heterodimer, including the Venus Flytrap Domains (VFDs) [in the closed-open (co) ac
91 ation, providing compelling evidence for the Venus flytrap mechanism of glutamate receptor domain clo
93 teracts with both T1R2 and T1R3 and that the Venus flytrap module of T1R2 is important for brazzein a
96 mulus between a midrib and a lobe closes the Venus flytrap upper leaf without mechanical stimulation
97 ike type I and II PBPs, which undergo large "Venus flytrap" conformational changes upon ligand bindin
98 embers of the MBP cluster, differs from the "Venus flytrap" mechanism utilized by bacterial nonmetal
102 loses its holes in a manner reminiscent of a Venus flytrap, which prevents the Cs(+) ions from leachi
106 cated mGlu4 receptors lacking the N-terminal Venus-flytrap domain, as opposed to the mGlu4 receptor e
107 r of magnitude faster than the snap traps of Venus flytraps and catapulting tentacles of the sundew D
108 he ingenious snapping mechanism of predatory Venus flytraps that rely on concave-to-convex reconfigur
110 ability of bursty translation is greater for Venus-FMRP RNA than for Venus-ARC RNA and is increased i
112 luding Cerulean, Dendra2, DRONPA, TagRFP and Venus, for the expression of protein fusions in plant ce
117 The efficiency of FRET in a zero-length LUMP-Venus fusion is 62% compared to approximately 31% in a r
118 rons express a dendritically-targeted PSD-95:Venus fusion protein under the control of a c-fos promot
120 We found that reassortants with PB2 from MA-Venus-H5N1 (MA-PB2), MA-PA, or MA-NS expressed Venus mor
123 PA (R443K) increased the virulence of the WT-Venus-H5N1 virus in mice and that the presence of both o
124 lence and reporter stability by comparing WT-Venus-H5N1 virus with a mouse-adapted Venus-H5N1 (MA-Ven
125 t from the A/Puerto Rico/8/1934(H1N1) virus (Venus-H5N1 virus), became more lethal to mice, and the r
130 from the Magellan spacecraft in orbit around Venus, has established that the surface materials viewed
131 viewed at low and intermediate altitudes on Venus have a relative dielectric permittivity of 4.0 &pl
135 ression in wild-type HSV-1 infection, but NC-Venus-ICP27 was abundantly expressed at late times of in
137 eveloping quantitative reporters such as DII-VENUS in conjunction with parameterized mathematical mod
138 tio of the fluorescent proteins Cerulean and Venus in mammalian cells expressing a series of fusion p
139 tor, Tsr, labeled by the fluorescent protein Venus in the inner membrane of live Escherichia coli cel
140 tailed study of the stability and folding of Venus in the pH range from 6.0 to 8.0 using chemical den
141 d an Aux/IAA-based reporter, domain II (DII)-VENUS, in conjunction with a mathematical model to quant
148 n found in space exploration missions (i.e., Venus &Jupiter planetary exploration, and heliophysics m
150 y after photobleaching in slices from VGLUT1(Venus) knock-in mice reveal 75% of VGLUT1-containing ves
157 a covalent bonding of the two halves of the Venus molecule, the head-to-tail interaction of NC-Venus
159 vector BiFC system which utilizes monomeric Venus (mVenus) split at residue 210, and contains an int
161 radation of the N-end rule substrate, LR-GFP(Venus), occurs with a single ClpS bound per ClpA(6); one
162 -insensitive yellow fluorescent protein [NPY-Venus] or NPY-monomeric red fluorescent protein), while
163 mTagBFP, cyan mCerulean, green CrGFP, yellow Venus, orange tdTomato and red mCherry) in the popular m
164 with spectral ratiometric imaging of an ECFP/Venus pair we were thus able to maximize the spectral se
165 tually codependent as balboa-RNAi eliminates Venus::PPK from the sensory dendrites of nociceptors.
167 (S)-3,5-dihydroxyphenylglycine, the rate of Venus-PSD-95 mRNA translation increased rapidly in dendr
169 e-molecule imaging of a diffusion-restricted Venus-PSD-95 reporter under control of the PSD-95 3'UTR.
171 ned RET efficiencies and with known Cerulean/Venus ratios were constructed and used to test sRET.
173 iled to completely immobilize coexpressed D2-venus receptors (D2R-Vs), suggesting that the two did no
174 , we reported that an H5N1 virus bearing the Venus reporter gene became more pathogenic to mice and t
175 sly reported that an H5N1 virus carrying the Venus reporter gene, which was inserted into the NS gene
179 PER2 and, importantly, the behavior of PER2::VENUS runs counter to the Drosophila model: it does not
181 e human Ca(2+) receptor (hCaR) consists of a Venus's-flytrap (VFT) domain and a cysteine-rich (Cys-ri
182 r (hCaR) has been speculated to consist of a Venus's-flytrap domain (VFT) and a cysteine-rich domain.
183 associated virus encoding channelrhodopsin-2-Venus showed similar fiber labeling and association with
184 occasionally detect small aggregates of the Venus signal in nuclei, but these were likely to be imag
185 evidence has been reported for lightning on Venus, some searches have been negative and the existenc
188 shares properties with the polar vortices on Venus, such as polar location, cyclonic circulation, war
189 with thrombotic disorders of arterial and/or venus systems, spontaneous abortion(s) or thrombocytopen
191 his pattern was recreated upon expression of VENUS-tagged barley (Hordeum vulgare) CSLF6 and CSLH1 in
192 was used to characterize assembly mutants of Venus-tagged CaMKIIalpha to identify a dimeric CaMKII.
193 fluorescence anisotropy and FRET imaging of Venus-tagged CaMKIIalpha to test the hypothesis that neu
195 by inserting a 7-kb fragment consisting of a venus-tagged lac repressor gene along with a target lacZ
196 olysis was required, as was visualized using Venus-tagged lysenin probe, which specifically binds SM.
197 red for the normal polarized localization of Venus-tagged neuropeptides to axons of cholinergic motor
199 llular localization of Venus-tagged VirE2 or Venus-tagged VIP1, in the presence or absence of the oth
200 y to examine the subcellular localization of Venus-tagged VirE2 or Venus-tagged VIP1, in the presence
201 also been used to study interplanetary dust, Venus' tail and the interstellar medium.) Here we report
202 ared images and spectra of the night side of Venus taken at the Anglo-Australian Telescope during Feb
204 scribe a JA perception biosensor termed Jas9-VENUS that allows the quantification of dynamic changes
205 rescent fragments of the fluorescent protein Venus that can associate to reform the fluorescent compl
206 pothalamic neurons were transfected with NPY-Venus, the distribution of the fluorescent puncta replic
207 stics of deformation in the ridged plains of Venus, the most widely preserved volcanic terrain on the
208 ss and dynamical parameters of the Earth and Venus, they fall short of explaining the small size of M
211 developing quantitative sensors such as Jas9-VENUS to provide high-resolution spatiotemporal data abo
214 el and the fluorescent proteins Cerulean and Venus, two mutant proteins of CFP and YFP with better fo
216 mice expressing yellow fluorescent protein (Venus) under the control of the PPG promoter were used t
217 Under mildly acidic conditions, we show that Venus undergoes a drastic decrease in yellow fluorescenc
218 hydrogen-deuterium exchange of (15)N-labeled Venus using NMR spectroscopy over 13 months, residue-spe
219 ovincialis, Ostrea edulis, Chlamis varia and Venus verrucosa) were collected during the autumn 2011 a
220 ion, warm center, and long lifetime, but the Venus vortices have cold collars and are not associated
222 While essentially complete release of NPY-Venus was observed in 24 +/- 1% of glucose-stimulated ex
224 RNA sequences and an unrelated control gene, Venus, we have identified many toxic sequences - most of
225 omatographs have been flown to both Mars and Venus where detailed compositional measurements were mad
226 r AGS4-RLuc and alpha(2)-adrenergic receptor-Venus, which were Galpha(i)-dependent and reduced by ago
228 oxygen has been observed on the nightside of Venus with HIRES, the echelle spectrograph on the W. M.
229 veals the intracellular trafficking of ASTN1-Venus, with ASTN1-Venus accumulating in the forward aspe
230 ductance in the stem of the channel, we used Venus yellow fluorescent protein as a molecular stopper
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