ライフサイエンスコーパス: Vibrio

1 -type family of quorum-sensing regulators in vibrios.

2 olytic activity and its regulation in marine vibrios.

3 by the reference method was 1.2% ETEC, 0.1% Vibrio, 0% Y. enterocolitica, and 0% P. shigelloides Com

4 .8% (99.5 to 99.9), and 0.93 (0.87 to 0.99); Vibrio, 100% (96.4 to 100), 99.7% (99.4 to 99.8), and 0.

5 e models revealed that long-term increase in Vibrio abundance is promoted by increasing sea surface t

6 Here we examined the population growth of Vibrio after natural and simulated pulses of atmospheric

7 was recently observed in the marine bacteria Vibrio alginolyticus and is possibly exhibited by other

8 s to capture the motion dynamics of swarming Vibrio alginolyticus at cellular resolution over hundred

9 formance chemotaxis by tracking thousands of Vibrio alginolyticus cells in microfluidic gradients.

10 ntly that polar flagellated marine bacterium Vibrio alginolyticus is capable of exhibiting taxis towa

11 to the swarming architecture and dynamics of Vibrio alginolyticus isolate B522 on carrageenan agar th

12 of exposure, strains of Vibrio cholerae and Vibrio alginolyticus were able to directly use Saharan d

13 In this study we report a strain of Vibrio alginolyticus, a species that exhibits similar co

14 n inactive ADP-bound structure of KtrAB from Vibrio alginolyticus, determined by cryo-electron micros

15 terium Escherichia coli and marine bacterium Vibrio alginolyticus, respectively.

16 vators of flagellar gene transcription as in Vibrio and Pseudomonas species.

17 nas), Sphingobacteria (i.e., Microscilla and Vibrio) and Flavobacteria dominated the epibiont communi

18 , for example, in genomes of Enterobacteria, Vibrio, and Halomonas species, and in typical soil bacte

19 ca, enterotoxigenic Escherichia coli (ETEC), Vibrio, and Plesiomonas shigelloides The study included

20 ii, and Flavobacterium sp. and the pathogens Vibrio anguillarum and Edwardsiella ictaluri with coatin

21 he bacterial challenge test using pathogenic Vibrio anguillarum revealed that the larval disease resi

22 ne particular group of pathogenic bacteria - vibrios - are a globally important cause of diseases in

23 e for the first time the potential of marine vibrios as fast urea hydrolyzers for biotechnological ap

24 e specifically, how one symbiosis, the squid-vibrio association, provides insight into the persistent

25 The vibrio autoinducer molecules bind to transmembrane recep

26 Vibrios belonging to the Harveyi clade are major pathoge

27 stressosome from the Gram-negative bacterium Vibrio brasiliensis.

28 act of indole signalling on the virulence of Vibrio campbellii was investigated.

29 -worm' was immobilized with a probe DNA from Vibrio Cholera and duplexed with a target which was reve

30 osed with Phenylketonuria and total D-AAs in Vibrio cholera cultures are pioneer illustrated as relev

31 e determined structures of Escherichia coli, Vibrio cholera, and Klebsiella pneumonia SlmA-DNA-FtsZ C

32 Escherichia coli, Staphylococcus aureus and Vibrio cholera, identified a number of EWs as potential

33 nt bacterial diseases (Bacillus subtilis and Vibrio cholera, respectively).

34 nsferase found in non-O1/non-O139 strains of Vibrio cholera.

35 tal structure characterization of DHBPS from Vibrio cholerae (vDHBPS) with a competitive inhibitor 4-

36 Typhimurium)] and two extracellular (Vibrio cholerae and Staphylococcus aureus) bacterial pat

37 in vivo competition experiments between T6S+ Vibrio cholerae and T6S-sensitive Escherichia coli.

38 s of the non-invasive Gram-negative pathogen Vibrio cholerae and the invasive pathogen Salmonella ent

39 Within 24 h of exposure, strains of Vibrio cholerae and Vibrio alginolyticus were able to di

40 tems of enterotoxigenic Escherichia coli and Vibrio cholerae are among the simplest of Type IV pilus

41 enomic framework for identifying VAPs, using Vibrio cholerae as a model.

42 Here, using Vibrio cholerae as our model organism, we show that duri

43 s, including other diarrheagenic E. coli and Vibrio cholerae bacteria, suggesting that this mucin-deg

44 ble live single-cell resolution imaging of a Vibrio cholerae biofilm as it develops from one single f

45 microscopy to image all individual cells in Vibrio cholerae biofilms at different stages of developm

46 ructural switch controls the architecture of Vibrio cholerae biofilms by mediating the interactions b

47 Current pandemic O1 Vibrio cholerae biotype El Tor is resistant to polymyxin

48 ANR homologs of ETEC and Vibrio cholerae bound to AggR as well as to other member

49 DncV is associated with hyperinfectivity of Vibrio cholerae but has not been found in many bacteria,

50 rotein (anti-OmpW) in sensitive detection of Vibrio cholerae by developing an immunosensor based on S

51 Vibrio cholerae causes cholera and is the leading cause

52 Vibrio cholerae causes human infection through ingestion

53 Vibrio cholerae causes the human disease cholera by prod

54 lems arising from the circularity of the two Vibrio cholerae chromosomes, chrI and chrII, are resolve

55 nships between globally circulating pandemic Vibrio cholerae clones and local bacterial populations.

56 Vibrio cholerae colonizes the human terminal ileum to ca

57 Vibrio cholerae contains three chemotaxis clusters (I, I

58 The diarrheal pathogen Vibrio cholerae contains three gene clusters that encode

59 Vibrio cholerae cytolysin (VCC) is a potent membrane-dam

60 Vibrio cholerae cytolysin (VCC) is a toxin secreted by t

61 We recently incorporated a Vibrio cholerae diguanylate cyclase into an adenovirus v

62 nstrate detection of DNA coils formed from a Vibrio cholerae DNA target at picomolar concentrations u

63 Detection of a Vibrio cholerae DNA-sequence using an optomagnetic read-

64 -polymerase beta-like superfamily (including Vibrio cholerae DncV), a minimal version of the CRISPR p

65 Here we report the structure of Vibrio cholerae FadR (VcFadR) alone, bound to DNA, and i

66 igh-throughput sequencing to reconstruct the Vibrio cholerae genome from the preserved intestine of a

67 nstability of the Fe(II) form suggested that Vibrio cholerae H-NOX may act as a sensor of the redox s

68 We also present two structures of the Vibrio cholerae IMPDH in complex with IMP/NAD(+) and XMP

69 the MshE N-terminal domain (MshEN1-145) from Vibrio cholerae in complex with c-di-GMP at a 1.37 A res

70 The structures of SpeG from Vibrio cholerae in complexes with polyamines and cofacto

71 he energetics of drug extrusion by NorM from Vibrio cholerae in proteoliposomes in which purified Nor

72 e, Yan et al. show that matrix production in Vibrio cholerae increases the osmotic pressure within th

73 richia coli, Mycobacterium tuberculosis, and Vibrio cholerae is a dimer.

74 Vibrio cholerae is a Gram-negative pathogen that can use

75 Vibrio cholerae is a natural inhabitant of aquatic envir

76 Vibrio cholerae is a natural resident of the aquatic env

77 Vibrio cholerae is an intestinal pathogen that causes th

78 Vibrio cholerae is responsible for the diarrheal disease

79 Vibrio cholerae is the bacterium that causes the diarrhe

80 Vibrio cholerae is the causative agent of the severe dia

81 Vibrio cholerae is the causative bacteria of the diarrhe

82 Vibrio cholerae is ubiquitous in aquatic environments, w

83 In contrast, we report here that Vibrio cholerae lacking DacA-1, a PBP5 homologue, displa

84 The Vibrio cholerae MARTXVc toxin delivers three effector do

85 ing infection, the human intestinal pathogen Vibrio cholerae must overcome noxious compounds that dam

86 An occurrence of Vibrio cholerae non-O1/O139 gastroenteritis in the U.S.

87 From 2000 to 2012, Vibrio cholerae O1 and Shigella species isolates from ur

88 r advance.PXVX0200, based on live attenuated Vibrio cholerae O1 classical Inaba vaccine strain CVD 10

89 Vibrio cholerae O1 El Tor strains have been responsible

90 Vibrio cholerae O1 is a major cause of acute watery diar

91 We used genomic data from 1070 Vibrio cholerae O1 isolates, across 45 African countries

92 odulates biofilm development and motility in Vibrio cholerae O1 of the classical biotype.

93 ess of humans caused by toxigenic strains of Vibrio cholerae O1 or O139.

94 rapid, sensitive and selective detection of Vibrio cholerae O1 which converts the antibody-antigen b

95 dministration of the cholera vaccine (killed Vibrio cholerae O1 whole cells and recombinant cholera t

96 ulted in lower vibriocidal responses against Vibrio cholerae O1, and there was a positive relationshi

97 ed two novel non-toxigenic (ctxA/B-negative) Vibrio cholerae O1.

98 spected cases who tested positive to PCR for Vibrio cholerae O1.

99 haride fragment of the O-specific antigen of Vibrio cholerae O139 were synthesized by applying 1 + 1,

100 end in the O-specific polysaccharide of both Vibrio cholerae O22 and O139.

101 n acute, watery diarrhoeal disease caused by Vibrio cholerae of the O1 or O139 serogroups.

102 to an antigen of interest were purified from Vibrio cholerae or Escherichia coli and used for immuniz

103 Anti-OMV IgG renders Vibrio cholerae organisms immotile, thus they pass throu

104 Here, we show that the Vibrio cholerae pathogenicity factor DncV is a prokaryot

105 Pathogenic Vibrio cholerae produces a cholera toxin which is the ca

106 Pathogenic Vibrio cholerae remains a major human health concern.

107 Analyses of intraintestinal Vibrio cholerae revealed infection-stage and region-spec

108 Recent studies have linked Vibrio cholerae secreted toxins to inflammasome activati

109 demonstrated with the detection of toxigenic Vibrio cholerae serogroups O1 and O139, which are associ

110 -resolution structure of a native contracted Vibrio cholerae sheath determined by cryo-electron micro

111 mG is known to be involved in remodeling the Vibrio cholerae surface, but its specific role was not c

112 am-negative rod and human diarrheal pathogen Vibrio cholerae synthesizes a VPS exopolysaccharide-depe

113 tion X-ray structures of VcINDY, a DASS from Vibrio cholerae that catalyses the co-transport of Na(+)

114 ree PL genosensor for sensitive detection of Vibrio cholerae that is based on a DNA hybridization str

115 Vibrio cholerae the causative agent of cholera epidemics

116 Introduction of Vibrio cholerae to Haiti during the deployment of United

117 The specific machine we analyse is the Vibrio cholerae toxin-coregulated pilus machine (TCPM).

118 ToxR activates expression of T3SS2 resembles Vibrio cholerae ToxR regulation of distinct virulence el

119 Vibrio cholerae uses a multiprotein transcriptional regu

120 The major Vibrio cholerae virulence gene transcription activator,

121 idual bacterial species Aeromonas veronii or Vibrio cholerae was sufficient to block locomotor hypera

122 y to identify 90 proteins present in OMVs of Vibrio cholerae when grown under conditions that activat

123 Vibrio cholerae's CarRS two-component regulatory system

124 factors are known to modulate expression of Vibrio cholerae's principal virulence factors.

125 cteria (Escherichia coli, Salmonella spp and Vibrio cholerae), with 8 strains of each bacterium, and

126 In the cholera pathogen, Vibrio cholerae, and in related species, secondary chrom

127 oS is critical for natural transformation in Vibrio cholerae, and it was previously presumed to exert

128 terobactin hydrolysis products by C. jejuni, Vibrio cholerae, and other bacteria with homologous peri

129 egulatory circuit was recently identified in Vibrio cholerae, and the H-NOX protein has been spectros

130 as aeruginosa, Stenotrophomonas maltophilia, Vibrio cholerae, and Yersinia enterocolitica T2S-express

131 terium diphtheriae, Salmonella enterica, and Vibrio cholerae, are infected with lysogenic bacteriopha

132 secreted by the type II secretion system in Vibrio cholerae, belongs to this subfamily.

133 ortant environmentally transmitted pathogen, Vibrio cholerae, can modulate the evolutionary trajector

134 ANR homologs of Vibrio cholerae, Citrobacter rodentium, Salmonella enter

135 In Vibrio cholerae, ClpV binds the N terminus of TssC withi

136 The waterborne agent of cholera, Vibrio cholerae, encounters phosphate limitation in both

137 brio vulnificus, Vibrio parahaemolyticus and Vibrio cholerae, grow in warm, low-salinity waters, and

138 that a successful enteric pathogen, such as Vibrio cholerae, must circumvent.

139 Vibrio cholerae, responsible for acute gastroenteritis s

140 non-protein coding RNA (npcRNA) sequences of Vibrio cholerae, Salmonella sp. and Shigella sp., which

141 tic bacterium and human intestinal pathogen, Vibrio cholerae, senses and responds to a variety of env

142 In pathogenic vibrios, including Vibrio cholerae, the accumulation of autoinducers trigge

143 sequence-specific detection of the bacterium Vibrio cholerae, the causative agent of acute diarrheal

144 controls virulence and biofilm formation in Vibrio cholerae, the causative agent of cholera disease.

145 Vibrio cholerae, the causative agent of cholera, remains

146 For example, Vibrio cholerae, the causative agent of cholera, utilize

147 nc transport systems in the enteric pathogen Vibrio cholerae, the causative agent of cholera.

148 chanism of horizontal gene transfer (HGT) in Vibrio cholerae, the causative agent of cholera.

149 creases both the survival and infectivity of Vibrio cholerae, the causative agent of cholera.

150 Vibrio cholerae, the causative agent of the severe diarr

151 Vibrio cholerae, the causative agent of the severe diarr

152 The human pathogen Vibrio cholerae, the causative agent of the severe diarr

153 kinase/response regulator pair that enables Vibrio cholerae, the cholera pathogen, to survive exposu

154 Vibrio cholerae, the etiological agent of cholera, is kn

155 e environmental survival and transmission of Vibrio cholerae, the facultative human pathogen responsi

156 negative bacteria Haemophilus influenzae and Vibrio cholerae, the master regulator Sxy/TfoX controls

157 In Vibrio cholerae, there are 13 distinct PTS transporters.

158 In Vibrio cholerae, three chromosomal clusters each encode

159 Two of the primary virulence regulators of Vibrio cholerae, ToxR and TcpP, function together with c

160 In Vibrio cholerae, VgrG3 has a hydrolase extension domain

161 athogens, including life-threatening spp. of Vibrio cholerae, Vibrio vulnificus, and Aeromonas hydrop

162 mathematical modelling and experiments with Vibrio cholerae, we show how killing adjacent competitor

163 In Vibrio cholerae, we uncover that this requirement is due

164 shigelloides, Salmonella spp., Vibrio spp., Vibrio cholerae, Yersinia enterocolitica, enteroaggregat

165 ation is critical for the infection cycle of Vibrio cholerae.

166 common pathogens, Staphylococcus aureus and Vibrio cholerae.

167 cSiaPQM, a sialic acid TRAP transporter from Vibrio cholerae.

168 used by certain strains of serogroup O1/O139 Vibrio cholerae.

169 , and the opposite occurs for the pathogenic Vibrio cholerae.

170 peG enzyme from the important human pathogen Vibrio cholerae.

171 e (Na(+)-NQR) is the main ion transporter in Vibrio cholerae.

172 spectrometry (MS) for the identification of Vibrio cholerae.

173 a bEBP that controls flagellar synthesis in Vibrio cholerae.

174 s one of the major porins of human pathogen, Vibrio cholerae.

175 l residue of Ogawa O-polysaccharide (OPS) of Vibrio cholerae.

176 nd to control chemotaxis and colonization by Vibrio cholerae.

177 ra, a severely dehydrating disease caused by Vibrio cholerae.

178 kers involved in relevant diseases caused by Vibrio cholerae.

179 pathway within the cholera-causing microbe, Vibrio cholerae.

180 ce gene expression and human colonization by Vibrio cholerae.

181 We demonstrate that the coral pathogen Vibrio coralliilyticus causes a rapidly lethal disease i

182 Here we describe a strain of Vibrio coralliilyticus, OCN014, which was isolated from

183 dy, we investigated the adhesion dynamics of Vibrio crassostreae on polystyrene microparticles (micro

184 s for heatwaves, Lyme disease in Canada, and Vibrio emergence in northern Europe highlight evidence t

185 Vibrio exopolysaccharides (VPS) and the matrix proteins

186 n of organisms and toxicity end points using Vibrio fischeri (nonspecific), specific fish macrophage

187 t elevated intracellular citrate levels in a Vibrio fischeri aconitase mutant correlate with activati

188 The inhibition effect of OSPW on Vibrio fischeri and the toxicity effect on goldfish prim

189 a scolopes squid is colonized exclusively by Vibrio fischeri bacteria.

190 evolved ecologically distinct bioluminescent Vibrio fischeri by colonization and growth within the li

191 how that cAMP-CRP is active and important in Vibrio fischeri during colonization of its host squid Eu

192 both: (i) selection of the specific partner Vibrio fischeri from the bacterioplankton during symbios

193 itously discovered that the marine bacterium Vibrio fischeri induces sexual reproduction in one of th

194 The marine bacterium Vibrio fischeri is the monospecific symbiont of the Hawa

195 The squid symbiont Vibrio fischeri uses an elaborate TCS phosphorelay conta

196 Vibrio fischeri uses the AinS/AinR pheromone-signaling s

197 minescence inhibition of the marine bacteria Vibrio fischeri, have been used.

198 uprymna scolopes and its beneficial symbiont Vibrio fischeri, which form a highly specific binary mut

199 effect on the acute toxicity of OSPW toward Vibrio fischeri.

200 s and its specific, bioluminescent symbiont, Vibrio fischeri.

201 e association of multiple pathogenic genera (Vibrio, Flavobacterium, Tenacibaculum, Pseudomonas) with

202 diversity of microorganisms, including some Vibrio genus members, raising questions about the role o

203 is an essential micronutrient that can limit Vibrio growth.

204 The genome of Vibrio harveyi BAA-1116 contains a nonribosomal peptide

205 The quorum-sensing bacterium Vibrio harveyi exclusively detects the autoinducer N-((R

206 Previous studies with Vibrio harveyi have shown that LuxR, the master quorum-s

207 s), called the Qrr1-5 sRNAs, function in the Vibrio harveyi quorum-sensing cascade to drive its opera

208 mental analyses of the Bacillus subtilis and Vibrio harveyi quorum-sensing networks to show that accu

209 Rv1625c by the quorum-sensing receptor from Vibrio harveyi which has an identical 6TM design and obt

210 In Vibrio harveyi, the master quorum-sensing transcription

211 n the outer membrane of the marine bacterium Vibrio harveyi.

212 Chitoporin from the chitinolytic marine Vibrio has been characterized as a trimeric OmpC-like ch

213 In vibrios, homologous small RNAs called the Qrr sRNAs func

214 Because the growth of pathogenic vibrios in the natural environment is largely dictated b

215 ed retrospectively the relative abundance of vibrios, including human pathogens, in nine areas of the

216 In pathogenic vibrios, including Vibrio cholerae, the accumulation of

217 The relative abundance of Vibrio increased from approximately 1 to approximately 2

218 o ascertain the relationship between SST and Vibrio infection through a conditional logistic regressi

219 estimated exposure-response relationship for Vibrio infections at a threshold of 16 degrees C reveale

220 ented occurrence of environmentally acquired Vibrio infections in the human population of Northern Eu

221 early warning system as the risk of further Vibrio infections increases in the 21st century due to c

222 ect the suitability of marine conditions for Vibrio infections under climate change scenarios.

223 Climate change projections for Vibrio infections were developed for Representative Conc

224 Vibrio is a ubiquitous genus of marine bacteria, typical

225 s not, suggesting this ancestral habitat for Vibrios is a replete medium with metabolically redundant

226 The ECDC Vibrio Map Viewer detected environmentally suitable area

227 ontrol (ECDC) developed a platform (the ECDC Vibrio Map Viewer) to monitor the environmental suitabil

228 vealing three dominant strategies within the vibrio: mesophiles, psychrophiles and apparently general

229 gnificant impact on the interactions between vibrios, (micro)algae and higher organisms, with major e

230 Using the squid-vibrio model system, we provide a characterization of th

231 The bacterium Vibrio natriegens has the fastest growth rate of any kno

232 ed by up to a 30-fold increase of culturable Vibrio over background levels within 24 h.

233 y designed to have a complementary region in Vibrio parahaemolyticus (VP) genome and to make differen

234 ia, including the species Vibrio vulnificus, Vibrio parahaemolyticus and Vibrio cholerae, grow in war

235 Here we show that the Vibrio parahaemolyticus effector protein VopQ is a poten

236 Vibrio parahaemolyticus is an important human foodborne

237 Vibrio parahaemolyticus is the most common cause of seaf

238 Vibrio parahaemolyticus sequence type 36 (ST36) strains

239 ting technology with the cytotoxicity of two Vibrio parahaemolyticus T3SSs (T3SS1 and T3SS2) to ident

240 ly emergent penaeid shrimp disease caused by Vibrio parahaemolyticus that has already led to tremendo

241 We discovered that Vibrio parahaemolyticus VtrC, along with VtrA and VtrB,

242 In Vibrio parahaemolyticus, a significant enteric pathogen

243 vSGLT), a solute-sodium symporter (SSS) from Vibrio parahaemolyticus, shares a common structural fold

244 In Gram-negative enteric pathogen Vibrio parahaemolyticus, we found that polar flagella ca

245 odium hypochlorite (NaOCl) solutions against Vibrio parahaemolyticus.

246 Vibrio Pathogenicity Island (VPI)-1 was present; however

247 A genome-wide screen revealed that Vibrio polysaccharide (VPS) production was inversely cor

248 ow-temperature-induced biofilm formation and Vibrio polysaccharide gene expression.

249 stem negatively regulates expression of vps (Vibrio polysaccharide) genes and biofilm formation.

250 bacterial heterotrophs, we demonstrated that Vibrio proliferate in response to a broad range of dust-

251 e to respond rapidly to nutrient influx, yet Vibrio response to environmental pulses of Fe remains un

252 , to our knowledge, is the first to describe Vibrio response to Saharan dust nutrients, having implic

253 ain locus from two V. fischeri strains and a Vibrio salmonicida strain to explore ain regulation.

254 We discovered that Vibrio shilonii AK1, a well-studied coral pathogen, prod

255 nity on the physiological characteristics of Vibrio sp. B2 and biofilm formation on nanofiltration (N

256 f a generalist saprophytic marine bacterium (Vibrio sp. F13 9CS106) on complex resources derived from

257 vitroprocines A-J, from the marine bacterium Vibrio sp. QWI-06 by an integrated approach using imagin

258 ered, chemotactic signaling arrays, which in Vibrio species are found at the cell pole.

259 demonstrate EptA orthologs from non-cholerae Vibrio species are functional.

260 Vibrio species have robust metabolic capabilities and an

261 Vibrio species naturally reside in the aquatic environme

262 in actin assembly factors used by infectious Vibrio species to induce actin assembly in host cells.

263 In Vibrio species, at low cell density, the sigma 54-depend

264 dy revealed that toxR, an ancestral locus in Vibrio species, is required for V. parahaemolyticus fitn

265 rolled by oxygen-dependent signalling within Vibrio species.

266 ce different torques, from Campylobacter and Vibrio species.

267 ded no Escherichia coli and few instances of Vibrio species.

268 n inducing cue for natural transformation in Vibrio species.

269 sely related strains of the marine bacterium Vibrio splendidus One strain, V. splendidus 13B01, exhib

270 is conserved across some bacteria including Vibrio spp. and Pseudomonas aeruginosa.

271 ominant communities in starved copepods were Vibrio spp. and related Gammaproteobacteria, suggesting

272 Some Vibrio spp. are pathogenic and ubiquitous in marine wate

273 to either CFCF did not significantly affect Vibrio spp. growth.

274 detected environmentally suitable areas for Vibrio spp. in the Baltic Sea in July 2014 that were acc

275 bacterales in the laboratory experiment, and Vibrio spp. in the in situ experiment when corals were e

276 ironmental suitability of coastal waters for Vibrio spp. using remotely sensed SST and salinity.

277 enotypes) to putatively pathogenic bacteria (Vibrio spp.).

278 so induce natural genetic competence in many Vibrio spp., a physiological state in which bacteria tak

279 important gram negative pathogens, including Vibrio spp., Salmonella spp., Shigella spp., Yersinia sp

280 , Plesiomonas shigelloides, Salmonella spp., Vibrio spp., Vibrio cholerae, Yersinia enterocolitica, e

281 To answer this question, vibrio strains isolated at a coastal time series were ph

282 derstanding of the early stages of the squid-vibrio symbiosis, and more generally inform the transcri

283 ng, host immune responses in the model squid-vibrio symbiosis.

284 In El Tor biotype vibrios, transcription of vieSAB is repressed by the quo

285 , we established a conformational map of the Vibrio vulnificus add adenine riboswitch that reveals fi

286 ficient mice with the siderophilic bacterium Vibrio vulnificus and found that hepcidin deficiency res

287 Studies of Vibrio vulnificus growth ex vivo show that high iron ser

288 Vibrio vulnificus has the highest death rate (>35%) and

289 Vibrio vulnificus is a Gram-negative aquatic bacterium f

290 Vibrio vulnificus is a striking and enigmatic human path

291 Vibrio vulnificus is the leading cause of seafood-relate

292 ocessing repeats-in-toxin (MARTXVv) toxin of Vibrio vulnificus plays a significant role in the pathog

293 (MARTX) toxin-effector domain DUF5(Vv) from Vibrio vulnificus to be a site-specific endopeptidase th

294 nse to iron in the broad-range host pathogen Vibrio vulnificus under the hypothesis that iron is one

295 e and elucidated the structure of VvPL2 from Vibrio vulnificus YJ016, which represents a transitional

296 Vibrio vulnificus, a pervasive human pathogen, can cause

297 ng life-threatening spp. of Vibrio cholerae, Vibrio vulnificus, and Aeromonas hydrophila.

298 ram-negative bacteria, including the species Vibrio vulnificus, Vibrio parahaemolyticus and Vibrio ch

299 n important group of marine prokaryotes, the vibrios, which are responsible for several infections in

300 ventional methods for the detection of ETEC, Vibrio, Y. enterocolitica, and P. shigelloides in stool