ライフサイエンスコーパス: Vibrio cholerae

1 spectrometry (MS) for the identification of Vibrio cholerae.

2 a bEBP that controls flagellar synthesis in Vibrio cholerae.

3 s one of the major porins of human pathogen, Vibrio cholerae.

4 ra, a severely dehydrating disease caused by Vibrio cholerae.

5 l residue of Ogawa O-polysaccharide (OPS) of Vibrio cholerae.

6 nd to control chemotaxis and colonization by Vibrio cholerae.

7 kers involved in relevant diseases caused by Vibrio cholerae.

8 pathway within the cholera-causing microbe, Vibrio cholerae.

9 ring by optimizing natural transformation in Vibrio cholerae.

10 urface of the Gram-negative aquatic pathogen Vibrio cholerae.

11 eptor cluster in Rhodobacter sphaeroides and Vibrio cholerae.

12 nd nucleoside drug selectivity of a CNT from Vibrio cholerae.

13 een protein secretion and pilus formation in Vibrio cholerae.

14 of the Vibrionaceae, including the pathogen Vibrio cholerae.

15 induced pilus in the Gram-negative bacterium Vibrio cholerae.

16 material containing the toxigenic bacterium Vibrio cholerae.

17 variations in coastal aquatic reservoirs of Vibrio cholerae.

18 for in vitro growth of the cholera pathogen, Vibrio cholerae.

19 o T7 bacteriophage and T7-like virus JSF7 of Vibrio cholerae.

20 n-seq) to identify T6SS immunity proteins in Vibrio cholerae.

21 obacter jejuni, Clostridium perfringens, and Vibrio cholerae.

22 hogenic non-O1/non-O139 serogroup strains of Vibrio cholerae.

23 AphB, a LysR-type regulator of virulence in Vibrio cholerae.

24 toxin gene from diarrheal-causing toxigenic Vibrio cholerae.

25 to a single high-resolution example, NorM of Vibrio cholerae.

26 system (VC1777-1779) from the human pathogen Vibrio cholerae.

27 he mannose-sensitive hemagglutinin (MSHA) of Vibrio cholerae.

28 of RtxA and in secretion of this toxin from Vibrio cholerae.

29 S) apparatus in the gram-negative bacterium, Vibrio cholerae.

30 low cell density (LCD) in Vibrio harveyi and Vibrio cholerae.

31 ce gene expression and human colonization by Vibrio cholerae.

32 ation is critical for the infection cycle of Vibrio cholerae.

33 common pathogens, Staphylococcus aureus and Vibrio cholerae.

34 cSiaPQM, a sialic acid TRAP transporter from Vibrio cholerae.

35 used by certain strains of serogroup O1/O139 Vibrio cholerae.

36 , and the opposite occurs for the pathogenic Vibrio cholerae.

37 peG enzyme from the important human pathogen Vibrio cholerae.

38 e (Na(+)-NQR) is the main ion transporter in Vibrio cholerae.

39 formation and virulence factor production in Vibrio cholerae, a human pathogen that causes the diseas

40 em mediates the export of >/= 20 proteins in Vibrio cholerae, a human pathogen that is indigenous to

41 rs of evolutionary fitness of pathogens like Vibrio cholerae, a mounting threat to global heath.

42 d to insect pathogens, as the human pathogen Vibrio cholerae also encodes rhabduscin's aglycone, and

43 Vibrio cholerae, an etiological agent of cholera, circul

44 mmatory response during early infection with Vibrio cholerae and a strong proinflammatory reaction to

45 ediated secretion and target cell killing by Vibrio cholerae and Acinetobacter baylyi.

46 s, RbmA, Bap1, and RbmC, which are unique to Vibrio cholerae and appear to support biofilm formation

47 RpoN is an essential colonization factor of Vibrio cholerae and controls important cellular function

48 roaches to the modeling of spatial spread of Vibrio cholerae and mechanisms of cholera transmission,

49 (+)-pumping NADH:quinone complex is found in Vibrio cholerae and other marine and pathogenic bacteria

50 Typhimurium)] and two extracellular (Vibrio cholerae and Staphylococcus aureus) bacterial pat

51 in vivo competition experiments between T6S+ Vibrio cholerae and T6S-sensitive Escherichia coli.

52 s of the non-invasive Gram-negative pathogen Vibrio cholerae and the invasive pathogen Salmonella ent

53 Within 24 h of exposure, strains of Vibrio cholerae and Vibrio alginolyticus were able to di

54 In the cholera pathogen, Vibrio cholerae, and in related species, secondary chrom

55 oS is critical for natural transformation in Vibrio cholerae, and it was previously presumed to exert

56 terobactin hydrolysis products by C. jejuni, Vibrio cholerae, and other bacteria with homologous peri

57 ion types of lipid A from Escherichia coli, Vibrio cholerae, and Pseudomonas aeruginosa using an Orb

58 egulatory circuit was recently identified in Vibrio cholerae, and the H-NOX protein has been spectros

59 as aeruginosa, Stenotrophomonas maltophilia, Vibrio cholerae, and Yersinia enterocolitica T2S-express

60 tems of enterotoxigenic Escherichia coli and Vibrio cholerae are among the simplest of Type IV pilus

61 terium diphtheriae, Salmonella enterica, and Vibrio cholerae, are infected with lysogenic bacteriopha

62 enomic framework for identifying VAPs, using Vibrio cholerae as a model.

63 Here, using Vibrio cholerae as our model organism, we show that duri

64 Here we demonstrate that the Vibrio cholerae autoinducer (S)-3-hydroxytridecan-4-one,

65 s, including other diarrheagenic E. coli and Vibrio cholerae bacteria, suggesting that this mucin-deg

66 taminated food or water by a human host, the Vibrio cholerae bacterium produces virulence factors, in

67 The toxigenic bacterium Vibrio cholerae belonging to the O1 and O139 serogroups

68 secreted by the type II secretion system in Vibrio cholerae, belongs to this subfamily.

69 ble live single-cell resolution imaging of a Vibrio cholerae biofilm as it develops from one single f

70 The Vibrio cholerae biofilm matrix contains three major prot

71 microscopy to image all individual cells in Vibrio cholerae biofilms at different stages of developm

72 ructural switch controls the architecture of Vibrio cholerae biofilms by mediating the interactions b

73 Vibrio cholerae biofilms displayed three distinct levels

74 By studying Vibrio cholerae biofilms in microfluidic devices, we sho

75 d by B. subtilis using the equivalent of the Vibrio cholerae biosynthetic pathway, (2) exogenous nors

76 Current pandemic O1 Vibrio cholerae biotype El Tor is resistant to polymyxin

77 ANR homologs of ETEC and Vibrio cholerae bound to AggR as well as to other member

78 The Vibrio cholerae BreR protein is a transcriptional repres

79 DncV is associated with hyperinfectivity of Vibrio cholerae but has not been found in many bacteria,

80 tic competence and natural transformation in Vibrio cholerae by co-ordinating expression of the regul

81 rotein (anti-OmpW) in sensitive detection of Vibrio cholerae by developing an immunosensor based on S

82 ortant environmentally transmitted pathogen, Vibrio cholerae, can modulate the evolutionary trajector

83 bset of non-O1/non-O139 serogroup strains of Vibrio cholerae cause disease using type 3 secretion sys

84 Vibrio cholerae causes cholera and is the leading cause

85 Vibrio cholerae causes human infection through ingestion

86 Vibrio cholerae causes the human disease cholera by prod

87 Mutations of Y241(Vc) (to A/F/H/S) in the Vibrio cholerae cbb3 eliminate catalytic activity, but a

88 erified bacterial human pathogens, including Vibrio cholerae (cholera) in a 19th century intestinal s

89 RctB, the initiator of replication of Vibrio cholerae chromosome 2 (chr2), binds to the origin

90 The origin region of Vibrio cholerae chromosome II (chrII) resembles plasmid

91 lems arising from the circularity of the two Vibrio cholerae chromosomes, chrI and chrII, are resolve

92 ANR homologs of Vibrio cholerae, Citrobacter rodentium, Salmonella enter

93 nships between globally circulating pandemic Vibrio cholerae clones and local bacterial populations.

94 In Vibrio cholerae, ClpV binds the N terminus of TssC withi

95 Vibrio cholerae colonize the small intestine where they

96 Vibrio cholerae colonizes the human terminal ileum to ca

97 Here we report that an HD-GYP PDE from Vibrio cholerae contains a non-heme diiron-carboxylate a

98 Vibrio cholerae contains three chemotaxis clusters (I, I

99 The diarrheal pathogen Vibrio cholerae contains three gene clusters that encode

100 The Vibrio cholerae Cpx system was previously found to respo

101 Vibrio cholerae CqsA/S synthesizes and detects (S)-3-hyd

102 Vibrio cholerae cytolysin (VCC) is a potent membrane-dam

103 Vibrio cholerae cytolysin (VCC) is a prominent member in

104 Vibrio cholerae cytolysin (VCC) is a toxin secreted by t

105 rm of the related, but inactive, lectin from Vibrio cholerae cytolysin.

106 Vibrio cholerae cytolysin/hemolysin (VCC) is an amphipat

107 d glass electrode and have been utilized for Vibrio cholerae detection.

108 /C. coli, Clostridium difficile, Salmonella, Vibrio cholerae, diarrheagenic Escherichia coli strains

109 We recently incorporated a Vibrio cholerae diguanylate cyclase into an adenovirus v

110 nstrate detection of DNA coils formed from a Vibrio cholerae DNA target at picomolar concentrations u

111 Detection of a Vibrio cholerae DNA-sequence using an optomagnetic read-

112 -polymerase beta-like superfamily (including Vibrio cholerae DncV), a minimal version of the CRISPR p

113 Vibrio genus, including the enteric pathogen Vibrio cholerae, encode only a single PG amidase, AmiB.

114 The waterborne agent of cholera, Vibrio cholerae, encounters phosphate limitation in both

115 es of vaccine to areas with high exposure to Vibrio cholerae, enough for two doses for 5% of the popu

116 Biofilm formation is a key factor in Vibrio cholerae environmental survival and host coloniza

117 Vibrio cholerae excreted by cholera patients is "hyperin

118 oxidoreductase (Na(+)-NQR) from the pathogen Vibrio cholerae exploits the free energy liberated durin

119 Here we report the structure of Vibrio cholerae FadR (VcFadR) alone, bound to DNA, and i

120 to identify the undermethylated sites in the Vibrio cholerae genome for the two DNA methyltransferase

121 igh-throughput sequencing to reconstruct the Vibrio cholerae genome from the preserved intestine of a

122 brio vulnificus, Vibrio parahaemolyticus and Vibrio cholerae, grow in warm, low-salinity waters, and

123 nstability of the Fe(II) form suggested that Vibrio cholerae H-NOX may act as a sensor of the redox s

124 ingle stranded probe DNA (ssDNA) sequence of Vibrio cholerae has been covalently functionalized onto

125 How c-di-GMP inhibits the motility of Vibrio cholerae has not been determined.

126 lly, the O1 El Tor and classical biotypes of Vibrio cholerae have been differentiated by their resist

127 In lakes and rivers, aquatic reservoirs of Vibrio cholerae have been evocated.

128 Vibrio harveyi and Vibrio cholerae have quorum sensing pathways with simila

129 In Vibrio cholerae, Hfq and four Hfq-dependent small RNAs a

130 We investigated the roles of the Vibrio cholerae high-molecular-weight bifunctional penic

131 f NaDC3 on the basis of the structure of the Vibrio cholerae homolog vcINDY.

132 We also present two structures of the Vibrio cholerae IMPDH in complex with IMP/NAD(+) and XMP

133 Biofilms promote attachment of Vibrio cholerae in aquatic ecosystems and aid in transmi

134 the MshE N-terminal domain (MshEN1-145) from Vibrio cholerae in complex with c-di-GMP at a 1.37 A res

135 a concentrative nucleoside transporter from Vibrio cholerae in complex with uridine at 2.4 A.

136 The structures of SpeG from Vibrio cholerae in complexes with polyamines and cofacto

137 he energetics of drug extrusion by NorM from Vibrio cholerae in proteoliposomes in which purified Nor

138 e, Yan et al. show that matrix production in Vibrio cholerae increases the osmotic pressure within th

139 ransposon insertion mutant with tolerance to Vibrio cholerae infection and markedly decreased transcr

140 The innate immune response to Vibrio cholerae infection is poorly understood, but this

141 Vibrio cholerae inhabits aquatic environments and coloni

142 -mediated activation of the toxT promoter in Vibrio cholerae, initiating a regulatory cascade that cu

143 richia coli, Mycobacterium tuberculosis, and Vibrio cholerae is a dimer.

144 Vibrio cholerae is a globally important pathogen that is

145 Vibrio cholerae is a Gram-negative bacterial pathogen th

146 Vibrio cholerae is a Gram-negative bacterium that persis

147 Vibrio cholerae is a Gram-negative pathogen that can use

148 Vibrio cholerae is a human pathogen that causes mild to

149 Vibrio cholerae is a natural inhabitant of aquatic envir

150 Vibrio cholerae is a natural resident of the aquatic env

151 Colonization of the human small intestine by Vibrio cholerae is an essential step in pathogenesis tha

152 Vibrio cholerae is an estuarine bacterium and the human

153 Vibrio cholerae is an inhabitant of aquatic systems and

154 Vibrio cholerae is an intestinal pathogen that causes th

155 , the outer leaflet of the outer membrane of Vibrio cholerae is comprised of lipopolysaccharide.

156 Vibrio cholerae is lethal to the model host Drosophila m

157 Vibrio cholerae is responsible for the diarrheal disease

158 Vibrio cholerae is the bacterium that causes the diarrhe

159 Vibrio cholerae is the causative agent of the acute diar

160 Vibrio cholerae is the causative agent of the deadly dia

161 Vibrio cholerae is the causative agent of the severe dia

162 Vibrio cholerae is the causative agent of the severe dia

163 Vibrio cholerae is the causative bacteria of the diarrhe

164 Vibrio cholerae is the cause of the diarrheal disease ch

165 Competence for genetic transformation in Vibrio cholerae is triggered by chitin-induced transcrip

166 Vibrio cholerae is ubiquitous in aquatic environments, w

167 toxin (CT), a virulence factor elaborated by Vibrio cholerae, is sufficient to induce the severe diar

168 In Vibrio cholerae it was proposed that VipA and VipB assem

169 In contrast, we report here that Vibrio cholerae lacking DacA-1, a PBP5 homologue, displa

170 The Vibrio cholerae MARTXVc toxin delivers three effector do

171 Very little is known about Vibrio cholerae Mlc.

172 ing infection, the human intestinal pathogen Vibrio cholerae must overcome noxious compounds that dam

173 that a successful enteric pathogen, such as Vibrio cholerae, must circumvent.

174 h the core regions in a crystal structure of Vibrio cholerae Na(+)-dicarboxylate transporter VcINDY,

175 An occurrence of Vibrio cholerae non-O1/O139 gastroenteritis in the U.S.

176 eal infection with certain pathogens such as Vibrio cholerae O1 and norovirus.

177 From 2000 to 2012, Vibrio cholerae O1 and Shigella species isolates from ur

178 cid sequence encoded by the VC1216 gene from Vibrio cholerae O1 biovar El Tor str.

179 r advance.PXVX0200, based on live attenuated Vibrio cholerae O1 classical Inaba vaccine strain CVD 10

180 phage VP5, one of the typing phages for the Vibrio cholerae O1 El Tor biotype.

181 Vibrio cholerae O1 El Tor strains have been responsible

182 ing in an area in which at least one case of Vibrio cholerae O1 infection had been confirmed by cultu

183 Vibrio cholerae O1 is a major cause of acute watery diar

184 Isolation of Vibrio cholerae O1 is necessary for cholera outbreak con

185 We used genomic data from 1070 Vibrio cholerae O1 isolates, across 45 African countries

186 odulates biofilm development and motility in Vibrio cholerae O1 of the classical biotype.

187 ess of humans caused by toxigenic strains of Vibrio cholerae O1 or O139.

188 A total of 720 Vibrio cholerae O1 strains were recovered for investigat

189 rapid, sensitive and selective detection of Vibrio cholerae O1 which converts the antibody-antigen b

190 dministration of the cholera vaccine (killed Vibrio cholerae O1 whole cells and recombinant cholera t

191 ulted in lower vibriocidal responses against Vibrio cholerae O1, and there was a positive relationshi

192 important in selected sites (eg, Aeromonas, Vibrio cholerae O1, Campylobacter jejuni).

193 Volunteer challenges with Vibrio cholerae O1, enterotoxigenic Escherichia coli (ET

194 Vibrio cholerae O1, serotype Inaba, was the predominant

195 ed two novel non-toxigenic (ctxA/B-negative) Vibrio cholerae O1.

196 spected cases who tested positive to PCR for Vibrio cholerae O1.

197 A strip test for the detection of Vibrio cholerae O139 was developed using two monoclonal

198 haride fragment of the O-specific antigen of Vibrio cholerae O139 were synthesized by applying 1 + 1,

199 end in the O-specific polysaccharide of both Vibrio cholerae O22 and O139.

200 secretory diarrhoea caused by infection with Vibrio cholerae of the O1 or O139 serogroup.

201 n acute, watery diarrhoeal disease caused by Vibrio cholerae of the O1 or O139 serogroups.

202 aeruginosa, T6SS-dependent killing of either Vibrio cholerae or Acinetobacter baylyi is greatly stimu

203 to an antigen of interest were purified from Vibrio cholerae or Escherichia coli and used for immuniz

204 Anti-OMV IgG renders Vibrio cholerae organisms immotile, thus they pass throu

205 ay use intercellular communication to stymie Vibrio cholerae pathogenesis, indicating how the microbi

206 Here, we show that the Vibrio cholerae pathogenicity factor DncV is a prokaryot

207 Pathogenic Vibrio cholerae produces a cholera toxin which is the ca

208 e mutualist Vibrio fischeri and the pathogen Vibrio cholerae promotes release of a potent bacteria-de

209 In the Vibrio harveyi and Vibrio cholerae quorum-sensing circuits, multiple non-co

210 Vibrio harveyi and Vibrio cholerae regulate their virulence factors accordi

211 Pathogenic Vibrio cholerae remains a major human health concern.

212 The acute, voluminous diarrhoea caused by Vibrio cholerae represents a dramatic example of enterop

213 secretion by the type VI secretion system of Vibrio cholerae requires the action of a dynamic intrace

214 Production of NCDAAs in Vibrio cholerae requires the stress response sigma facto

215 onal component, our data suggest a potential Vibrio cholerae reservoir in the Rift Valley lakes and t

216 Vibrio cholerae responds to environmental changes by alt

217 Vibrio cholerae, responsible for acute gastroenteritis s

218 Although toxigenic Vibrio cholerae, responsible for the disease cholera, ca

219 Analyses of intraintestinal Vibrio cholerae revealed infection-stage and region-spec

220 the prototypic MATE family member, NorM from Vibrio cholerae, reveals a protein fold composed of 12 t

221 Vibrio cholerae's CarRS two-component regulatory system

222 factors are known to modulate expression of Vibrio cholerae's principal virulence factors.

223 non-protein coding RNA (npcRNA) sequences of Vibrio cholerae, Salmonella sp. and Shigella sp., which

224 Recent studies have linked Vibrio cholerae secreted toxins to inflammasome activati

225 The human intestinal pathogen Vibrio cholerae secretes a pore-forming toxin, V.cholera

226 Vibrio cholerae secretes the Zn-dependent metalloproteas

227 tic bacterium and human intestinal pathogen, Vibrio cholerae, senses and responds to a variety of env

228 Vibrio cholerae serogroup O1, the causative agent of the

229 demonstrated with the detection of toxigenic Vibrio cholerae serogroups O1 and O139, which are associ

230 -resolution structure of a native contracted Vibrio cholerae sheath determined by cryo-electron micro

231 nts, the glycosylation transition states for Vibrio cholerae sialidase-catalyzed hydrolysis reactions

232 protein unfolding conditions, we made use of Vibrio cholerae strain O395 lacking the Hsp33 gene hslO.

233 is a waterborne diarrheal disease caused by Vibrio cholerae strains of serogroups O1 and O139.

234 Certain Vibrio cholerae strains produce cholix, a potent protein

235 Vibrio cholerae strains unable to produce CT show severe

236 mG is known to be involved in remodeling the Vibrio cholerae surface, but its specific role was not c

237 am-negative rod and human diarrheal pathogen Vibrio cholerae synthesizes a VPS exopolysaccharide-depe

238 Here, we present evidence that VgrG-3 of the Vibrio cholerae T6SS has both structural and toxin activ

239 tion X-ray structures of VcINDY, a DASS from Vibrio cholerae that catalyses the co-transport of Na(+)

240 Here, we identify several genes in Vibrio cholerae that collectively are required for growt

241 Vibrio cholerae that had colonized the naive host was HI

242 ree PL genosensor for sensitive detection of Vibrio cholerae that is based on a DNA hybridization str

243 Vibrio cholerae the causative agent of cholera epidemics

244 In pathogenic vibrios, including Vibrio cholerae, the accumulation of autoinducers trigge

245 sequence-specific detection of the bacterium Vibrio cholerae, the causative agent of acute diarrheal

246 controls virulence and biofilm formation in Vibrio cholerae, the causative agent of cholera disease.

247 Vibrio cholerae, the causative agent of cholera, remains

248 By directly applying a reporter strain of Vibrio cholerae, the causative agent of cholera, to a th

249 We show that Vibrio cholerae, the causative agent of cholera, use the

250 For example, Vibrio cholerae, the causative agent of cholera, utilize

251 chanism of horizontal gene transfer (HGT) in Vibrio cholerae, the causative agent of cholera.

252 creases both the survival and infectivity of Vibrio cholerae, the causative agent of cholera.

253 nc transport systems in the enteric pathogen Vibrio cholerae, the causative agent of cholera.

254 Like other Gram-negative pathogens, Vibrio cholerae, the causative agent of the diarrheal di

255 Toxigenic Vibrio cholerae, the causative agent of the disease chol

256 The human pathogen Vibrio cholerae, the causative agent of the severe diarr

257 Vibrio cholerae, the causative agent of the severe diarr

258 Vibrio cholerae, the causative agent of the severe diarr

259 kinase/response regulator pair that enables Vibrio cholerae, the cholera pathogen, to survive exposu

260 In El Tor biotype strains of toxigenic Vibrio cholerae, the CTXvarphi prophage often resides ad

261 since October 2010, are grim reminders that Vibrio cholerae, the etiologic agent of cholera, remains

262 Vibrio cholerae, the etiological agent of cholera, is kn

263 In Vibrio cholerae, the etiological agent of cholera, the T

264 e environmental survival and transmission of Vibrio cholerae, the facultative human pathogen responsi

265 In Vibrio cholerae, the feo operon consists of three genes,

266 negative bacteria Haemophilus influenzae and Vibrio cholerae, the master regulator Sxy/TfoX controls

267 In Vibrio cholerae, the RNA binding protein and chaperone H

268 Shigella spp and enteroinvasive E coli, and Vibrio cholerae-the strength of association with diarrho

269 In Vibrio cholerae, there are 13 distinct PTS transporters.

270 In Vibrio cholerae, three chromosomal clusters each encode

271 Introduction of Vibrio cholerae to Haiti during the deployment of United

272 Type VI secretion is critical for Vibrio cholerae to successfully combat phagocytic eukary

273 The specific machine we analyse is the Vibrio cholerae toxin-coregulated pilus machine (TCPM).

274 ion of the two critical virulence factors of Vibrio cholerae, toxin-coregulated pilus and cholera tox

275 ToxR activates expression of T3SS2 resembles Vibrio cholerae ToxR regulation of distinct virulence el

276 Two of the primary virulence regulators of Vibrio cholerae, ToxR and TcpP, function together with c

277 We describe a Vibrio cholerae Type 3 secretion system effector VopE th

278 t & Microbe, Suzuki et al. (2014) describe a Vibrio cholerae Type-III-secreted effector that targets

279 Vibrio cholerae uses a multiprotein transcriptional regu

280 ry) technique was developed for detection of Vibrio cholerae ( V. cholerae ).

281 4.2.1.1) from the human pathogenic bacterium Vibrio cholerae, VchCA.

282 tal structure characterization of DHBPS from Vibrio cholerae (vDHBPS) with a competitive inhibitor 4-

283 In Vibrio cholerae, VgrG3 has a hydrolase extension domain

284 eats-in-toxin) family toxins are produced by Vibrio cholerae, Vibrio vulnificus, Aeromonas hydrophila

285 athogens, including life-threatening spp. of Vibrio cholerae, Vibrio vulnificus, and Aeromonas hydrop

286 HsiB1 is homologous to the Vibrio cholerae VipA component, which contributes to the

287 The major Vibrio cholerae virulence gene transcription activator,

288 Vibrio cholerae was identified in 525 stool specimens, a

289 idual bacterial species Aeromonas veronii or Vibrio cholerae was sufficient to block locomotor hypera

290 GMP functions via high or low specificity in Vibrio cholerae, we correlated the in vivo c-di-GMP conc

291 Here, in Vibrio cholerae, we identified HubP (hub of the pole), a

292 mathematical modelling and experiments with Vibrio cholerae, we show how killing adjacent competitor

293 In Vibrio cholerae, we uncover that this requirement is due

294 y to identify 90 proteins present in OMVs of Vibrio cholerae when grown under conditions that activat

295 Using experiments with Vibrio cholerae, which secretes extracellular enzymes to

296 cteria (Escherichia coli, Salmonella spp and Vibrio cholerae), with 8 strains of each bacterium, and

297 the Mop protein involved in the virulence of Vibrio cholerae, with conservation in both overall struc

298 n-producing E. coli [STEC], E. coli O157:H7, Vibrio cholerae, Yersinia enterocolitica, and toxigenic

299 or adenovirus 40/41, norovirus, rotavirus A, Vibrio cholerae, Yersinia enterocolitica, Entamoeba hist

300 shigelloides, Salmonella spp., Vibrio spp., Vibrio cholerae, Yersinia enterocolitica, enteroaggregat