ライフサイエンスコーパス: WBC

1 WBC count is also a complex trait that varies among indi

2 WBC count more than 50,000/microL was an adverse prognos

3 WBC count was independently associated with somatic depr

4 WBC count was measured at baseline in 160,117 postmenopa

5 WBC separated from whole blood was trapped by the paper

6 WBC trapped on the paper leads to the ion diffusion bloc

7 WBC, CRP, ESR and DNI were higher in APN than in lower U

8 ratio, 19.7 [95% CI, 4.4-87.4]; p < 0.0001), WBCs more than 12,000/mm3 (odds ratio, 11.6 [95% CI, 2.8

9 (hazard ratio [HR] = 1.67; P = .04], log(10)(WBC) (HR = 1.33; P = .02), and trisomy 22 (HR = 0.54; P

10 C count and of the mean of baseline + year 3 WBC count.

11 s, we show that, although leukopenia (< 4000 WBC/mm(3) during infection) was associated with an accel

12 times the number of RBCs; CNS3a to 3c, >/= 5 WBCs/muL plus blasts with/without >/= 10 RBCs/muL or cli

13 blasts with/without >/= 10 RBCs/muL or >/= 5 WBCs/muL plus blasts, with WBCs >/= 5 times the number o

14 ts in a nontraumatic sample) nor CNS-3 (>/=5 WBCs/muL with blasts in a nontraumatic sample or the pre

15 s follows: CNS1, no blasts; CNS2a to 2c, < 5 WBCs/muL and blasts with/without >/= 10 RBCs/muL or >/=

16 Neither CNS-2 (less than 5 WBCs/muL with blasts in a nontraumatic sample) nor CNS-3

17 Our results indicate that as little as 10(5) WBC and 100 mul of blood collected from asymptomatic scr

18 that the intravenous administration of 10(5) WBCs is sufficient to cause scrapie in recipient sheep.

19 When combined with MRD and a WBC count >/=200 x 10(9)/L, it identifies a significant

20 Fourteen patients (8.0%) had a WBC count of >50.0 x 10(9)/microL.

21 whereas animals injected with PYKK081 had a WBC count that resembled that of the uninfected control.

22 Patients harboring a WBC count >/=200 x 10(9)/L, gHiR classifier, and MRD >/=

23 of 46%, whereas the 58 patients (30%) with a WBC count <200 x 10(9)/L, gLoR classifier, and MRD <10(-

24 lbuminema, elevated creatinine, and abnormal WBC were all significant predictors (P < 0.0001) of incr

25 duction of mortality with bivalirudin across WBC tertiles was independent of major bleeding, and a si

26 how LtxA preferentially recognizes activated WBCs is not known.

27 LtxA preferentially targets activated WBCs and is being developed as a therapeutic agent for t

28 on between bivalirudin therapy and admission WBC count was apparent for 1-year mortality.

29 Age, WBC count, secondary AML, Eastern Cooperative Oncology G

30 ther known prognostic features, such as age, WBC, and karyotype, were included in a multivariate anal

31 d 8 other prognostic factors, including age, WBC count, cytogenetics, and gene mutations, into surviv

32 ivariable Cox regression analysis, sex, age, WBC count, and cytogenetic risk category were related to

33 compared with age >60 years, secondary AML, WBC >50 G/L, European LeukemiaNet risk groups, and Easte

34 blood samples from three studies to analyze WBC DNA methylation of two ATM intragenic loci (ATMmvp2a

35 2), hematocrit levels (P = 9.5 x 10(-7)) and WBC count (P = 3.1 x 10(-5)).

36 induced blood and organ cytokine content and WBC composition changes, including lymphocyte subsets in

37 n 5th POD, PCT has a better AUC than CRP and WBC (0.862 vs 0.806 vs 0.611).

38 correlation between appendiceal diameter and WBC was 80% (P=0.01 <0.05).

39 nt for WT1 and NPM1 mutations, FLT3-ITD, and WBC.

40 nce status, lactate dehydrogenase level, and WBC count, were confirmed as independent prognostic fact

41 clinical tumor growth rate, lymphocytes, and WBC.

42 fied the inverse association between MDS and WBC count and partially accounted for the association wi

43 PLT and WBC counts were both inversely related to MD adherence (

44 association between the MD and both PLT and WBC counts.

45 perative level for 1 year posttransplant and WBC counts were significantly lower for 3 years after tr

46 gnostic factors such as cytogenetic risk and WBC count, neither the presence of EMD nor the number of

47 8), C-reactive protein (odds ratio 1.3), and WBCs (odds ratio 1.4).

48 ian UrMis concentration in rectal mucosa and WBCs among individuals treated with folate was not signi

49 Mis concentrations in both rectal mucosa and WBCs did not correlate significantly with folate measure

50 9 deficiency is a common finding in RBCs and WBCs in patients with chronic hemolysis suffering from p

51 neously obtained myocardial core samples and WBCs were compared for SCN5A variants C (VC) and D (VD).

52 ly diagnosed, low- or intermediate-risk APL (WBC at diagnosis </= 10 x 10(9)/L).

53 combinations of temporal biomarkers, such as WBC, oxygen content, mean arterial pressure, and heart r

54 The genetic factors underlying basal WBC traits in Hispanics/Latinos are unknown.

55 Leptin increased basal WBCs, decreased basal and AMD3100-mobilized LSK cells, a

56 The associations between baseline WBC and MACE (composite of cardiac death, stent thrombos

57 After adjusting for baseline WBC and prognostic factors in a multivariate model, AA w

58 Children had significantly higher baseline WBC counts (P < .001).

59 hazards associated with deciles of baseline WBC count and of the mean of baseline + year 3 WBC count

60 gion and then tested the association between WBC and genetic ancestry at each locus.

61 The association between WBC and MACE was consistent in acute coronary syndrome a

62 The association between WBC count and MDS disappeared when further adjusted for

63 ence is increasing for a correlation between WBC count and thrombosis, but prospective data are lacki

64 ause mortality and cardiac mortality between WBC and bivalirudin therapy.

65 nes do not change >10% of rank range between WBC and OO; only 878 (3.9%) change rank >50%.

66 vention had higher expression levels of both WBC-derived SCN5A variants compared with patients with H

67 genotype and the cellular milieu defined by WBC counts may influence HIV disease course, and this ma

68 White blood cell (WBC) analysis provides rich information in rapid diagnos

69 ugh onset, and higher peak white blood cell (WBC) and lymphocyte counts.

70 tigated whether aortic and white blood cell (WBC) CoQ is programmed by suboptimal early nutrition and

71 io [HR] = 0.58, P = .002), white blood cell (WBC) count <10 x 10(9)/L (HR = 0.60, P = .005), and ETV6

72 n, including decreased CSF white blood cell (WBC) count (P < .001), interleukin (IL)-4 (P = .02), IL-

73 I, 8.26-10.49), and higher white blood cell (WBC) count (per 1000/muL: beta = 0.95; 95% CI, 0.74-1.16

74 effect of birth weight on white blood cell (WBC) count among blacks and whites was examined in 2,080

75 be an interaction between white blood cell (WBC) count and bivalirudin for the risk of mortality, an

76 atory responses [change in white blood cell (WBC) count and neutrophil activity], and that these resp

77 White blood cell (WBC) count appears to predict total mortality and corona

78 White blood cell (WBC) count is a common clinical measure used as a predic

79 Elevated white blood cell (WBC) count is associated with increased major adverse ca

80 Average white blood cell (WBC) count was 37.7 x 10(9)/microL.

81 and HIV disease markers or white blood cell (WBC) count were examined using mixed-effects and linear

82 otein (CRP) concentration, white blood cell (WBC) count, and absolute neutrophil cell (ANC) count for

83 hsCRP), lipid profile, and white blood cell (WBC) count, at baseline and 1, 3, and 6 months.

84 antly elevated circulating white blood cell (WBC) count, whereas animals injected with PYKK081 had a

85 , platelet count (PLT) and white blood cell (WBC) count.

86 netic classifier, MRD, and white blood cell (WBC) count.

87 30% bone marrow blasts and white blood cell (WBC) counts </=15 x 109/L (AZA-AML-001 study).

88 Circulating white blood cell (WBC) counts (neutrophils, monocytes, lymphocytes, eosino

89 ration, hematocrit levels, white blood cell (WBC) counts and platelet counts in 31,340 individuals ge

90 Platelet (PLT) and white blood cell (WBC) counts are 2 markers of inflammation and have been

91 c variation in circulating white blood cell (WBC) counts from large samples of otherwise healthy indi

92 greatly reduced peripheral white blood cell (WBC) counts in leukemia recipient mice and induced apopt

93 ry, on average, have lower white blood cell (WBC) counts than those of European descent (ethnic leuko

94 ith electrocardiogram, and white blood cell (WBC) counts with hematology analyzer.

95 ein (hsCRP) levels, higher white blood cell (WBC) counts, and lower serum albumin levels, are associa

96 f over one quarter-million white blood cell (WBC) nuclei together with CD15/CD16 protein expression f

97 he generalizability of the white blood cell (WBC) transcriptome to the general, multiorgan transcript

98 omparing recipients of non-white blood cell (WBC)-reduced versus WBC-reduced blood components in card

99 en BBB opening, pattern of white blood cell (WBCs) entry into the brain and seizure occurrence.

100 after ischemia and affects white blood cell (WBCs) function.

101 <30%) or first leukopenia (white blood cell [WBC] count <3000 cells/mm), which required clinical inte

102 onal inflammatory markers (white blood cell [WBC] count, erythrocyte sedimentation rate [ESR], C-reac

103 retic peptide [NT-proBNP], white blood cell [WBC], platelet counts) in 1265 AMI patients.

104 etween DNA methylation in white blood cells (WBC) and the risk of breast cancer.

105 avenous administration of white blood cells (WBC) resulted in efficient disease transmission.

106 rostate cancer cells from white blood cells (WBC) through forces generated by ultrasonic resonances i

107 omes latent in peripheral white blood cells (WBC).

108 s, more specifically from white blood cells (WBC).

109 dum 16S RNA in CSF or CSF white blood cells (WBCs) >20/uL or a reactive CSF-Venereal Disease Research

110 tion antigen-1 (LFA-1) on white blood cells (WBCs) and causes cell death.

111 White blood cells (WBCs) play an important role in host immune responses ea

112 cells (PBMCs) and of CSF white blood cells (WBCs) that were activated monocytes (CD14+CD16+) was det

113 DNA methylation in white blood cells (WBCs) was associated with total IgE levels in an epigeno

114 l mRNA splice variants in white blood cells (WBCs) with risk of arrhythmias in heart failure (HF).

115 ularity, peripheral blood white blood cells (WBCs), neutrophils, and platelets, and a 4.4-fold increa

116 crobial response in human white blood cells (WBCs).

117 that specifically targets white blood cells (WBCs).

118 ine oxime [HMPAO]-labeled white blood cells [WBC]).

119 for baseline and procedural characteristics, WBC remained independently associated with MACE (hazard

120 sex, blood pressure, serum HDL cholesterol, WBC count, and history of current cigarette smoking; and

121 genetic mechanisms that regulate circulating WBC counts and suggest a prominent shared genetic archit

122 levels strongly correlated with circulating WBC samples for both VC and VD variants (r = 0.78 and 0.

123 Because circulating WBCs demonstrate similar SCN5A splicing patterns, we hyp

124 t the sepsis-induced decrease in circulating WBCs, augment the early (6 hr postcecal ligation and pun

125 The circulating WBCs and LSKs, and CFUs were reduced in both models with

126 itory to define two datasets for comparison, WBC and OO (Other Organ) sets.

127 quantification to achieve rapid and low-cost WBC analysis at the point-of-care under resource limited

128 White blood count (WBC), C-reactive protein (CRP) and PCT levels were measu

129 r in patients with a white blood cell count (WBC) <50 Giga per liter (G/L) (P < .0001) and in older p

130 individual's maximum white blood cell count (WBC) as a continuous measure.

131 White blood cell count (WBC) is an important clinical marker that varies among d

132 appendicoliths, and white blood cell count (WBC) were significantly correlated with the inflammation

133 interleukin (IL)-6, white blood cell count (WBC), vascular cell adhesion molecule (VCAM)-1, and inte

134 62P) concentrations; white blood cell count (WBC); heart rate; and blood pressure.

135 umin ratio, and CSF white blood cell counts (WBC), neopterin levels, and concentrations of chemokines

136 defined as undetectable CSF T. pallidum, CSF WBCs </=5/uL and nonreactive CSF-VDRL.

137 Current WBC counting method relies on bulky instrument and train

138 up by the investigators because of decreased WBC counts, mostly outside of study protocol criteria, d

139 WBC HIF-1alpha was associated with decreased WBC CCR1, -2, and -4 expression.

140 en race and treatment discontinuation/delay, WBC counts, and survival in women enrolled onto breast c

141 or high risk (HR) on the basis of diagnostic WBC count.

142 time of BBB-induced epileptiform discharges, WBCs populated the perivascular space of a leaky BBB.

143 label-free smartphone based electrochemical WBC counting device on microporous paper with patterned

144 Elevated WBC count during follow-up was correlated with thrombosi

145 underlying pathophysiologic role of elevated WBC across a spectrum of coronary artery disease present

146 pigenome-wide association study adjusted for WBC types (including eosinophils), methylation changes i

147 However, adjustment for WBC types resulted in markedly fewer significant sites.

148 mission rate (P = .005) after adjustment for WBC; shorter disease-free survival (P = .01) after adjus

149 rs CCR1, -2, and -4, which are necessary for WBC mobilization and recruitment to inflammatory cytokin

150 nd the MD was not affected by adjustment for WBCs.

151 No frank WBCs extravasation in the brain parenchyma was observed.

152 to discriminate and divert tumor cells from WBCs using erythrocyte-lysed blood from healthy voluntee

153 rich both viable and fixed cancer cells from WBCs with very high recovery and purity.

154 Whole-genome methylation of DNA from WBCs was measured by using the Illumina Infinium HumanMe

155 of cancer cells from cell culture lines from WBCs with a recovery rate better than 83%.

156 fluorescent protein (GFP)-labeled WBCs (GFP-WBCs) suspended in Evans Blue we found that, at time of

157 In PR-GOAL WBC types (ie, neutrophils, eosinophils, basophils, lymp

158 ore including ASXL1 status, age, hemoglobin, WBC, and platelet counts defines three groups of CMML pa

159 Her WBC count was 10,370/muL, with a differential showing 5%

160 A high WBC count, raised serum ALT, raised serum total bilirubi

161 e levels among patients with NSCLC, and high WBC levels among patients with advanced disease.

162 Higher WBC counts are associated with higher mortality in succe

163 Higher WBC counts were significantly associated with positive b

164 Higher WBC, KIT, and/or FLT3-ITD/TKD gene mutations, and a less

165 Lower albumin and %LYM and a higher WBC count were significantly associated with outcomes.

166 positively associated with older age, higher WBC and platelet counts, intermediate-risk and normal cy

167 re each independently associated with higher WBC counts in adjusted models (P < .01); the highest qua

168 Patients with higher WBC were more often younger, smokers, and with less como

169 st of the added value from the (99m)Tc-HMPAO-WBC scan for decision making was seen in patients in who

170 We assessed the value of (99m)Tc-HMPAO-WBC scintigraphy including SPECT/CT acquisitions in a se

171 (99m)Tc-HMPAO-WBC scintigraphy results were correlated with transthora

172 lts demonstrate the ability of (99m)Tc-HMPAO-WBC scintigraphy to reduce the rate of misdiagnosed case

173 aphy but correctly negative at (99m)Tc-HMPAO-WBC scintigraphy: these patients had marantic vegetation

174 hsCRP, WBC count, and serum albumin were measured at baseline i

175 oducts, adopting strategies to prevent HTRs, WBC-reducing components transfused to patients undergoin

176 expression of antimicrobial factors in human WBCs and reveal a distinctive role, not shared with ET,

177 s found to be abundantly expressed in IgM(+) WBC from CyHV-3 latently infected koi.

178 e terminal repeat was investigated in IgM(+) WBC from koi with latent CyHV-3 infection.

179 The presence of the CyHV-3 genome in IgM(+) WBC was about 20-fold greater than in IgM(-) WBC.

180 3 latency was further investigated in IgM(+) WBC.

181 WBC was about 20-fold greater than in IgM(-) WBC.

182 enome for loci underlying this difference in WBC by using admixture mapping.

183 attributable to a secular downward trend in WBC.

184 approximately 20% of population variation in WBC.

185 l8, Il10, Pghs2, Tnfa, and Crp expression in WBCs, elevated plasma levels of IL-1beta, IL-6, and IL-8

186 UrMis measurements in WBCs are not a robust surrogate for UrMis measurements i

187 ation between genome-wide DNA methylation in WBCs and total IgE levels in 2 studies of Hispanic child

188 T/ MR), recruitment of immune cells ((111)In-WBC SPECT), or enhanced glycolytic flux seen in inflamma

189 Increased WBC is an independent predictor of MACE after percutaneo

190 acteremia, altered liver function, increased WBC counts, pathogen-specific Ab (IgM and IgG), and cell

191 nted with lower hemoglobin levels, increased WBC and platelet counts, and were more likely to have DN

192 (WT) or Tet2(+/-) BM cells, led to increased WBC counts, monocytosis, and splenomegaly in WT recipien

193 lly increases HR, reduces HRV, and increases WBC.

194 Infected WBCs can also function as 'Trojan horses' and carry viru

195 Age, initial WBC count, genetic aberrations, and minimal residual dis

196 s that included age, race/ethnicity, initial WBC, and day-29 minimal residual disease < 0.1%, CSF bla

197 We demonstrate that several previously known WBC-associated genetic loci (e.g. the African Duffy anti

198 sing green fluorescent protein (GFP)-labeled WBCs (GFP-WBCs) suspended in Evans Blue we found that, a

199 patients with immune deficiency or leukemia WBC should be persistently monitored.

200 yocardial inflammation in the setting of low WBC HIF-1alpha expression through bone marrow transplant

201 African Americans are known to have a lower WBC than European Americans.

202 gher body-surface area (P < .0001) and lower WBC (P = .0009).

203 EVI1(-) t(9;11) AMLs had lower WBC counts, more commonly FAB M5 morphology, and frequen

204 Women of African ancestry (AA) have lower WBC counts and are more likely to have treatment delays

205 but not in patients in the mid-WBC or lower WBC tertiles.

206 pronounced in those with progressively lower WBC counts.

207 risk factors compared with those with lower WBC.

208 0) and increased odds of being in the lowest WBC-count group (IMI: odds ratio = 1.41; 95% confidence

209 of the common West African allele had a mean WBC of 4.9 (SD 1.3); participants who had both common Eu

210 had both common European alleles had a mean WBC of 7.1 (SD 1.3).

211 High deciles of both baseline and mean WBC count were positively associated with total mortalit

212 ered from MLD(-) patients only by lower mean WBC counts, not by biologic characteristics, cytogenetic

213 ly; P<0.0001) but not in patients in the mid-WBC or lower WBC tertiles.

214 coding variants) associated with one or more WBC traits, the majority of which are pleiotropically as

215 mortality in association with the use of non-WBC-reduced ABT.

216 Notably, WBC CoQ levels correlated with aortic telomere-length (P

217 e, confirmed that high MRD values (> 0.1% of WBC) were associated with a higher risk of relapse after

218 The association of WBC count with mortality was independent of smoking and

219 th Initiative to examine the associations of WBC count with total mortality, CHD mortality, and cance

220 arm and baseline risk group (on the basis of WBC, performance status, number of sites of disease, and

221 studies demonstrated that downregulation of WBC HIF-1alpha was associated with decreased WBC CCR1, -

222 The evaluation of WBC DNA methylation as a biomarker of cancer risk is of

223 el tracks the evolution of the morphology of WBC subpopulations as a patient transitions from a healt

224 Our PheWAS of WBC found expected results, including associations with

225 ed models (P < .01); the highest quartile of WBC counts (>/=6500 cells/microL) was associated with in

226 ression resulted in decreased recruitment of WBC to the sites of inflammation and improvement in card

227 as a therapeutic agent for the treatment of WBC diseases such as hematologic malignancies and autoim

228 approximately 4000 with retention of 98% of WBCs.

229 increased G-CSF- or AMD3100-mobilization of WBCs and LSKs, compared to the untreated.

230 (myeloperoxidase activity), total number of WBCs, and neutrophils in bronchoalveolar lavage fluids c

231 severe (>or=90%) and prolonged reduction of WBCs was observed at the maximum dose of (90)Y-anti-CD20

232 The effect of HIF-1alpha on WBC function and inflammation following myocardial infar

233 of downregulation of leukocyte HIF-1alpha on WBC migration are attributable, at least in part, to the

234 ctively) and were not associated with age or WBC count.

235 symptoms were not associated with hs-CRP or WBC count.

236 igh-risk categories were defined as hsCRP or WBC levels above the 75th percentile (5.1 mg/L and 6.9 x

237 with reduced UrMis in rectal mucosa cells or WBCs.

238 differentiation, or clearance of particular WBC lineages (myeloid, lymphoid) and also potentially in

239 High-risk patients (WBCs >or= 10 x 10(9)/L) received GO on the first day.

240 In multivariate analyses, peak WBC count, birth weight, intubation, and receipt of nitr

241 spital lengths of stay) and incremental peak WBC counts (hospital length of stay only).

242 (7) Preoperative WBC >11,000/mm in elective operations was associated wit

243 esponses occurred in AML with low presenting WBC count.

244 RD monitoring was far superior to presenting WBC (hazard ratio, 1.02; 95% CI, 1.00 to 1.03; P = .02),

245 older age (P < .001) and higher pretreatment WBC (P = .04) compared with wild-type TET2 (TET2-wt).

246 ningfully associated with hs-CRP, NT-proBNP, WBC, or platelet counts 1 month after AMI, suggesting th

247 We are able to rapidly quantify WBC concentrations covering the common physiological and

248 Recuperated WBCs also had reduced CoQ (74+/-5.8%; P<0.05).

249 se variants in CXCR2 associated with reduced WBC count (gene-based P = 2.6 x 10(-13)).

250 Laboratory work revealed WBC 30.4 k/muL, hemoglobin 7.9 g/dL, and platelets 16 k/

251 77 days post transmission) had CSF HIV RNA, WBC, neopterin, and CXCL10 concentrations similar to the

252 duals and hospitalized patients with similar WBC counts can be robustly classified based on their WBC

253 RL21A did not stain WBCs (total WBCs) or normal tissues from deceased HLA-A2

254 range, 17 to 85 years), performance status, WBC count, and mutation status of NPM1, CEBPA, and FLT3-

255 operative Oncology Group performance status, WBC count, creatinine clearance, albumin, AST, number of

256 In the GALA II study WBC types were imputed.

257 Downregulation of HIF-1alpha suppressed WBC cytokine receptors CCR1, -2, and -4, which are neces

258 ue Index (PI), Gingival Index (GI), systemic WBC counts, and peripheral neutrophil oxidative activity

259 rve (AUC) (0.775 vs 0.772), both better than WBC (0.601); in 5th POD, PCT has a better AUC than CRP a

260 These results demonstrate that WBC infiltration into the newly injured myocardium plays

261 Together, our findings indicate that WBC DNA methylation levels at ATM could be a marker of b

262 The WBC count is a clinical marker of inflammation and a str

263 The WBC count was nonlinearly associated with all-cause mort

264 The WBC differential showed 64% blasts and 24% promyelocytes

265 king status (in adolescents and adults), the WBC count decreased across quartiles of increasing birth

266 to keep the platelet count, and arguably the WBC count, within the normal range.

267 sed systemic inflammation as depicted by the WBC count in childhood and adulthood, thereby potentiall

268 pression rank and correlation profile in the WBC dataset.

269 We show that the WBC nucleus images alone can be used to replicate CD exp

270 ived GO on day 1, if high risk, and if their WBC increased to more than 30 x 10(9)/L during induction

271 ts can be robustly classified based on their WBC population dynamics.

272 plicing patterns, we hypothesized that these WBC-derived splice variants might further stratify patie

273 ith HIV disease course differed according to WBC but not CD4(+) T-cell counts, such that leukopenic b

274 Patients were stratified according to WBC tertiles.

275 avoiding prospective donors alloimmunized to WBC antigens from donating plasma products, adopting str

276 s and suggests that the toxin contributes to WBC depletion.

277 t for neutrophil count) are generalizable to WBC traits in Hispanics/Latinos.

278 Total WBC and neutrophil counts did not change.

279 low-frequency power by 90% (P = 0.01), total WBC count by 139% (P = 0.006), and lymphocyte count by 1

280 med a genome-wide association study of total WBC and differential counts in a large, ethnically diver

281 an exome array-based meta-analysis of total WBC and subtype counts (neutrophils, monocytes, lymphocy

282 erform a trans-ethnic meta-analysis of total WBC, neutrophil and monocyte counts.

283 onse to blood neutrophils (rather than total WBC) was also not well correlated between humans and mic

284 Reduction in triglyceride, VLDL, total WBC, lymphocyte, and neutrophil counts and increase in h

285 RL21A did not stain WBCs (total WBCs) or normal tissues from deceased HLA-A2 donors, sub

286 001)] and reduction of therapeutic toxicity [WBC decrease (P = 0.04); gastrointestinal adverse reacti

287 patients enrolled in the HORIZONS-AMI trial, WBC count was available in 3433 (95.3%) patients.

288 ptional and signaling pathways that underlie WBC development and lineage specification can contribute

289 IIb/IIIa inhibitors in patients in the upper WBC tertile (all-cause death: 4.1% versus 9.3%, respecti

290 of non-white blood cell (WBC)-reduced versus WBC-reduced blood components in cardiac surgery have doc

291 Therefore, defining which WBCs are permissive to Zika virus (ZIKV) is critical.

292 us on chromosome 1q strongly associated with WBC (p < 10(-12)).

293 have identified genomic loci associated with WBC and its subtypes, but much of the heritability of th

294 , birth weight was inversely associated with WBC count in children (beta coefficients (unit, cells/mi

295 egion that was significantly associated with WBC on chromosome 1q.

296 gnals within loci previously associated with WBC or its subtypes were identified.

297 -induced nor chronic seizures correlate with WBC brain parenchymal migration while albumin and IgG br

298 RBCs/muL or >/= 5 WBCs/muL plus blasts, with WBCs >/= 5 times the number of RBCs; CNS3a to 3c, >/= 5

299 iAMP21 was associated with age >/= 10 years, WBC less than 50,000/muL, female sex, and detectable MRD

300 ed ASXL1 mutations, age older than 65 years, WBC count greater than 15 x10(9)/L, platelet count less