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1 XCR4(WHIM) mutations are highly prevalent in Waldenstrom's macroglobulinemia.
2 at we previously analyzed from patients with Waldenstrom's macroglobulinemia.
3 thy of uncertain significance, and 1 case of Waldenstrom's macroglobulinemia.
4 ponses, and safe in pretreated patients with Waldenstrom's macroglobulinemia.
7 in tumor samples from 49 of 54 patients with Waldenstrom's macroglobulinemia and in 3 of 3 patients w
8 t(9;14)(p13;q32), previously associated with Waldenstrom's macroglobulinemia and lymphoplasmacytoid l
9 inemia that can be useful in differentiating Waldenstrom's macroglobulinemia and non-IgM LPL from B-c
11 utation in benign monoclonal IgM gammopathy, Waldenstrom's macroglobulinemia, and diffuse large B cel
13 n, as well as NF-kappaB nuclear staining, in Waldenstrom's macroglobulinemia cells expressing MYD88 L
17 lly with thymoma, a myopathy associated with Waldenstrom's macroglobulinemia, Lambert-Eaton myastheni
18 (n = 4, 22%), multiple myeloma (n = 2, 11%), Waldenstrom's macroglobulinemia (n = 2, 11%), extranodal
19 red normal tissue samples from patients with Waldenstrom's macroglobulinemia or non-IgM LPL and in B
20 commonly recurring mutation in patients with Waldenstrom's macroglobulinemia that can be useful in di
21 e of a 77-year-old white man with history of Waldenstrom's macroglobulinemia transforming to large B-
22 found among RFs from HID or individuals with Waldenstrom's macroglobulinemia who do not have joint di
23 of ibrutinib in 63 symptomatic patients with Waldenstrom's macroglobulinemia who had received at leas
25 of bone marrow LPL cells in 30 patients with Waldenstrom's macroglobulinemia, with paired normal-tiss
26 ples from patients with multiple myeloma and Waldenstrom's macroglobulinemia without invasive fungal
33 ificance (MGUS), multiple myelomas (MM), and Waldenstrom's macroglobulinemias (WM) using protein macr
34 four small lymphocytic lymphomas (SLL), two Waldenstrom's macroglobulinemias (WM), and one marginal
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