1 For differences in variability,
we calculated a random-effects model for measures of variance
2 Using results for both tests,
we calculated a seroprevalence range maximising either specif
3 We calculated a stimulation map using voxel-wise statistical
4 We calculated adjusted effect estimates for antibiotic type a
5 We calculated adjusted hazard ratios (aHRs) with 95% CIs for
6 We calculated adjusted risk differences (ARDs) following logi
7 To document HCV-associated deaths sub-nationally,
we calculated age-adjusted, HCV-associated death rates and co
8 We calculated and experimentally verified the excitation puls
9 We calculated annual percentages of men with >=1 gonorrhea te
10 We calculated between-group differences in CSF concentrations
11 nalysed (n = 8.694). Based on indirect population formulas,
we calculated body fat percentage (%BF) and skeletal muscle m
12 data inferred from mobile-phone calling records in Namibia,
we calculated both the number of trips between districts and
13 We calculated CAAP, NA-LRI, and CXR examinations annual incid
14 Prepregnancy diet was assessed every 4 y, from which
we calculated dietary pattern scores from the DASH diet (8 co
15 We calculated each hospital's average annual volume for total
16 We calculated enrichment scores (ES) across severity and prev
17 We calculated GA area growth rate for each eye based on the f
18 We calculated GL, total carbohydrate intake, total dietary fi
19 In the observational cohort study,
we calculated hazard ratios (HRs) adjusted for likelihood of
20 We calculated incremental cost-effectiveness ratios (ICERs) a
21 t (HCP) and the Developing Human Connectome Project (dHCP),
we calculated intrinsic functional connectivity during restin
22 We calculated Kaplan-Meier curves and used adjusted Cox propo
23 We calculated likelihood ratios of result intervals and cutof
24 We calculated mean rintSO2 and rcSO2 for 60-120 minutes for e
25 Finally,
we calculated medicinal chemistry properties of benzenoid con
26 To investigate longitudinal consistency of the model,
we calculated model deviations in 46 neonates who were scanne
27 Using marginal structural models,
we calculated model-adjusted prevalence rates and ratios to d
28 We calculated neonatal (age 0-27 days), post-neonatal (age 28
29 We calculated population-level validity ratios (measured cove
30 We calculated proportions of NHIS participants with and witho
31 We calculated reaction free energies and demonstrate the ther
32 We calculated risk scores for 6 previously published tools (t
33 We calculated shifts in phenology using quantile regression a
34 For each of these populations,
we calculated TDD and PDD with reference to a DNA reference l
35 In addition,
we calculated that unaffected carriers of mutations are numer
36 le logistic regression models (one for each delivery mode),
we calculated the adjusted rates of PCS and PPCS among women
37 For each parameter,
we calculated the area under the receiver operating character
38 of interest placed in the dentate nucleus and the pons, and
we calculated the dentate nucleus-to-pons signal intensity ra
39 We calculated the effect of parenteral anticoagulant administ
40 We calculated the effect of tagging on apparent survival, con
41 We calculated the incidence of clinically significant CA-AKI
42 We calculated the linear weighted kappa coefficients for inte
43 We calculated the overall and quarterly HCC incidence rates.
44 For each approach,
we calculated the paired percentage difference to the "true"
45 We calculated the previously unidentified fraction, defined a
46 For each center,
we calculated the probability of deceased donor kidney transp
47 We calculated the sensitivity and specificity for detection o
48 tic variation of postprandial TG independent of fasting TG,
we calculated the TG response at 150 minutes by the residuals
49 To assess for dose response,
we calculated the total days of PPS prescriptions filled and
50 of furosemide and famciclovir (metabolized to penciclovir),
we calculated their kidney clearances on the basis of sequent