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1 s and granulomatosis with polyangiitis (GPA, Wegener's granulomatosis).
2 lyangiitis and glomerulonephritis (excluding Wegener's granulomatosis).
3 matosis with polyangiitis, formerly known as Wegener's granulomatosis.
4 se and Henoch-Schonlein purpura, and A1AT in Wegener's granulomatosis.
5 damage and its relevance to the diagnosis of Wegener's granulomatosis.
6 on specific forms of damage associated with Wegener's granulomatosis.
7 s an important concern for all patients with Wegener's granulomatosis.
8 tant source of morbidity among patients with Wegener's granulomatosis.
9 One of them is Wegener's granulomatosis.
10 phil cytoplasmic antibodies in patients with Wegener's granulomatosis.
11 itis led to an initial mistaken diagnosis of Wegener's granulomatosis.
12 have been implicated in the pathogenesis of Wegener's granulomatosis.
13 gham Vasculitis Activity Score, modified for Wegener's granulomatosis.
14 ission induction agent for severe refractory Wegener's granulomatosis.
15 y may play a role in the etiopathogenesis of Wegener's granulomatosis.
16 ical diagnosis was suggested to be a type of Wegener's granulomatosis.
17 45 transplant recipients and 1 patient with Wegener's granulomatosis.
18 e in selected patients with limited forms of Wegener's granulomatosis.
19 es, including aortitis, atherosclerosis, and Wegener's granulomatosis.
20 s in whom leptomeningeal biopsy demonstrated Wegener's granulomatosis.
21 hospitalizations among 571 individuals with Wegener's granulomatosis.
22 rate efficacy in severe vasculitis or severe Wegener's granulomatosis.
23 n estimated 10,771 hospitalizations included Wegener's granulomatosis among the discharge diagnoses.
25 ologists are often in a position to diagnose Wegener's granulomatosis and prevent significant morbidi
27 d previously unexpected associations between Wegener's granulomatosis and the incidence of malignancy
28 matosis with polyangiitis (formerly known as Wegener's granulomatosis) and microscopic polyangiitis.
29 ligand for PECAM-1 and it may have a role in Wegener's granulomatosis, and antibodies to HNA-2a frequ
30 pears to be useful in preventing relapses in Wegener's granulomatosis, and patients develop fewer inf
32 ifferences in the geographic distribution of Wegener's granulomatosis are apparent when analysis cons
40 ined as Birmingham Vasculitis Activity Score/Wegener's Granulomatosis (BVAS/WG)=0 off prednisone.
41 on (Birmingham Vasculitis Activity Score for Wegener's granulomatosis [BVAS/WG] score of 0) at month
43 icate certain systemic conditions, including Wegener's granulomatosis, Cogan's syndrome, polyarteriti
44 se 3, a biomarker for the autoimmune disease Wegener's granulomatosis, diluted up to 10(7)-fold in 1%
46 d solid malignancies was observed during the Wegener's Granulomatosis Etanercept Trial (WGET), which
50 teritis nodosa, hypersensitivity vasculitis, Wegener's granulomatosis, giant cell arteritis and Takay
51 tain forms of systematic vasculitis, such as Wegener's granulomatosis, have circulating antineutrophi
52 specificity; for example, most patients with Wegener's granulomatosis in China have myeloperoxidase-a
55 roscopic polyangiitis (P = 2.9 x 10(-4)) and Wegener's granulomatosis in two independent cohorts from
56 e, and to have systemic lupus erythematosus, Wegener's granulomatosis, inflammatory myopathy, or poly
58 ase 3 (PR3), the major target autoantigen in Wegener's granulomatosis is a serine proteinase that is
62 of granulomatosis with polyangiitis (GPA; or Wegener's granulomatosis) is the granulomatous inflammat
63 hic arthritis, systemic lupus erythematosis, Wegener's granulomatosis, juvenile dermatomyositis, juve
64 ic of the subset of vasculitis that includes Wegener's granulomatosis, microscopic polyangiitis (micr
66 olled 197 ANCA-positive patients with either Wegener's granulomatosis or microscopic polyangiitis.
68 erity may vary considerably in patients with Wegener's granulomatosis, polyarteritis nodosa, microsco
69 oid arthritis, systemic lupus erythematosus, Wegener's granulomatosis, polymyositis, dermatomyositis,
70 omatosis with polyangiitis (GPA), previously Wegener's granulomatosis, requires prompt diagnosis and
72 y to proteinase 3 and neutrophil elastase in Wegener's granulomatosis, support the concept that there
74 0,000 hospitalizations/year in patients with Wegener's granulomatosis to 2 cases/10,000 hospitalizati
79 ic markers for the small vessel vasculitides Wegener's granulomatosis (WG) and microscopic polyangiit
85 is report describes a case of severe limited Wegener's granulomatosis (WG) presenting in the third tr
86 nfection, chronic hepatitis C infection, and Wegener's granulomatosis (WG), an inflammatory, granulom
87 ccompany the neutrophilic vasculitis seen in Wegener's granulomatosis (WG), are directed against prot
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