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1 s and granulomatosis with polyangiitis (GPA, Wegener's granulomatosis).
2 lyangiitis and glomerulonephritis (excluding Wegener's granulomatosis).
3 matosis with polyangiitis, formerly known as Wegener's granulomatosis.
4 se and Henoch-Schonlein purpura, and A1AT in Wegener's granulomatosis.
5 damage and its relevance to the diagnosis of Wegener's granulomatosis.
6  on specific forms of damage associated with Wegener's granulomatosis.
7 s an important concern for all patients with Wegener's granulomatosis.
8 tant source of morbidity among patients with Wegener's granulomatosis.
9                               One of them is Wegener's granulomatosis.
10 phil cytoplasmic antibodies in patients with Wegener's granulomatosis.
11 itis led to an initial mistaken diagnosis of Wegener's granulomatosis.
12  have been implicated in the pathogenesis of Wegener's granulomatosis.
13 gham Vasculitis Activity Score, modified for Wegener's granulomatosis.
14 ission induction agent for severe refractory Wegener's granulomatosis.
15 y may play a role in the etiopathogenesis of Wegener's granulomatosis.
16 ical diagnosis was suggested to be a type of Wegener's granulomatosis.
17  45 transplant recipients and 1 patient with Wegener's granulomatosis.
18 e in selected patients with limited forms of Wegener's granulomatosis.
19 es, including aortitis, atherosclerosis, and Wegener's granulomatosis.
20 s in whom leptomeningeal biopsy demonstrated Wegener's granulomatosis.
21  hospitalizations among 571 individuals with Wegener's granulomatosis.
22 rate efficacy in severe vasculitis or severe Wegener's granulomatosis.
23 n estimated 10,771 hospitalizations included Wegener's granulomatosis among the discharge diagnoses.
24         A 62-year-old male with a history of Wegener's granulomatosis and immunosuppressive therapy p
25 ologists are often in a position to diagnose Wegener's granulomatosis and prevent significant morbidi
26 data suggest a familial link between risk of Wegener's granulomatosis and rheumatoid arthritis.
27 d previously unexpected associations between Wegener's granulomatosis and the incidence of malignancy
28 matosis with polyangiitis (formerly known as Wegener's granulomatosis) and microscopic polyangiitis.
29 ligand for PECAM-1 and it may have a role in Wegener's granulomatosis, and antibodies to HNA-2a frequ
30 pears to be useful in preventing relapses in Wegener's granulomatosis, and patients develop fewer inf
31 rcoidosis, systemic lupus erythematosus, and Wegener's granulomatosis are also reported.
32 ifferences in the geographic distribution of Wegener's granulomatosis are apparent when analysis cons
33 logic imaging patterns of orbital disease in Wegener's granulomatosis are presented.
34             Granulomatous syndromes, such as Wegener's granulomatosis, are defined according to compl
35 ath certificates in the United States listed Wegener's granulomatosis as a cause of death.
36     We describe a patient who presented with Wegener's granulomatosis associated with antineutrophil
37                             New diagnosis of Wegener's granulomatosis at baseline was an independent
38       Controversy has arisen over the eponym Wegener's granulomatosis because of alleged involvement
39                       The syndrome resembles Wegener's granulomatosis both clinically and histologica
40 ined as Birmingham Vasculitis Activity Score/Wegener's Granulomatosis (BVAS/WG)=0 off prednisone.
41 on (Birmingham Vasculitis Activity Score for Wegener's granulomatosis [BVAS/WG] score of 0) at month
42                   This case illustrates that Wegener's granulomatosis can cause chronic meningitis.
43 icate certain systemic conditions, including Wegener's granulomatosis, Cogan's syndrome, polyarteriti
44 se 3, a biomarker for the autoimmune disease Wegener's granulomatosis, diluted up to 10(7)-fold in 1%
45                                   During the Wegener's Granulomatosis Etanercept Trial (WGET), a plac
46 d solid malignancies was observed during the Wegener's Granulomatosis Etanercept Trial (WGET), which
47                   We evaluated data from the Wegener's Granulomatosis Etanercept Trial.
48    This study used the BVAS/WG data from the Wegener's Granulomatosis Etanercept Trial.
49                          Although studies of Wegener's granulomatosis frequently focus on controlling
50 teritis nodosa, hypersensitivity vasculitis, Wegener's granulomatosis, giant cell arteritis and Takay
51 tain forms of systematic vasculitis, such as Wegener's granulomatosis, have circulating antineutrophi
52 specificity; for example, most patients with Wegener's granulomatosis in China have myeloperoxidase-a
53                  Other genes associated with Wegener's granulomatosis in replicated candidate gene st
54                            The prevalence of Wegener's granulomatosis in the United States is approxi
55 roscopic polyangiitis (P = 2.9 x 10(-4)) and Wegener's granulomatosis in two independent cohorts from
56 e, and to have systemic lupus erythematosus, Wegener's granulomatosis, inflammatory myopathy, or poly
57                                              Wegener's granulomatosis is a multisystem disease charac
58 ase 3 (PR3), the major target autoantigen in Wegener's granulomatosis is a serine proteinase that is
59  amount of evidence supports the notion that Wegener's granulomatosis is an autoimmune disease.
60                         Standard therapy for Wegener's granulomatosis is fraught with substantial tox
61 th the characteristic granuloma formation of Wegener's granulomatosis is unclear.
62 of granulomatosis with polyangiitis (GPA; or Wegener's granulomatosis) is the granulomatous inflammat
63 hic arthritis, systemic lupus erythematosis, Wegener's granulomatosis, juvenile dermatomyositis, juve
64 ic of the subset of vasculitis that includes Wegener's granulomatosis, microscopic polyangiitis (micr
65 ondary to microscopic polyangiitis (n=43) or Wegener's granulomatosis (n=42).
66 olled 197 ANCA-positive patients with either Wegener's granulomatosis or microscopic polyangiitis.
67 flammatory diseases such as Crohn's disease, Wegener's granulomatosis, or sarcoidosis.
68 erity may vary considerably in patients with Wegener's granulomatosis, polyarteritis nodosa, microsco
69 oid arthritis, systemic lupus erythematosus, Wegener's granulomatosis, polymyositis, dermatomyositis,
70 omatosis with polyangiitis (GPA), previously Wegener's granulomatosis, requires prompt diagnosis and
71                                   Within the Wegener's granulomatosis subgroup, the median VDI score
72 y to proteinase 3 and neutrophil elastase in Wegener's granulomatosis, support the concept that there
73                In particular, in contrast to Wegener's granulomatosis, the need for routine cyclophos
74 0,000 hospitalizations/year in patients with Wegener's granulomatosis to 2 cases/10,000 hospitalizati
75                                Patients with Wegener's granulomatosis usually seek medical care for r
76                               A diagnosis of Wegener's granulomatosis was considered in all of them,
77                                              Wegener's granulomatosis was initially considered to be
78                                Patients with Wegener's granulomatosis were excluded.
79 ic markers for the small vessel vasculitides Wegener's granulomatosis (WG) and microscopic polyangiit
80                                              Wegener's granulomatosis (WG) and microscopic polyangiit
81                                              Wegener's granulomatosis (WG) is a granulomatous vasculi
82                                              Wegener's granulomatosis (WG) is a systemic inflammatory
83                                              Wegener's granulomatosis (WG) is a systemic vasculitis o
84     The precipitating event(s) that triggers Wegener's granulomatosis (WG) is unknown.
85 is report describes a case of severe limited Wegener's granulomatosis (WG) presenting in the third tr
86 nfection, chronic hepatitis C infection, and Wegener's granulomatosis (WG), an inflammatory, granulom
87 ccompany the neutrophilic vasculitis seen in Wegener's granulomatosis (WG), are directed against prot
88 T) may be a determinant of susceptibility to Wegener's granulomatosis (WG).
89            She was initially thought to have Wegener's granulomatosis (WG).
90 tients with CIMDL with that in patients with Wegener's granulomatosis (WG).
91 neutrophil cytoplasmic antibodies (ANCAs) in Wegener's granulomatosis (WG).
92 phil cytoplasmic antibody (cANCA)-associated Wegener's granulomatosis (WG).
93 bodies in approximately 60% of patients with Wegener's granulomatosis (WG).
94                     Total PSV, subgroups (47 Wegener's granulomatosis [WG], 12 microscopic polyangiit

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