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1 deficiency and describe how rodent models of Wernicke-Korsakoff Syndrome aid in developing a more det
2 nitive impairment, early Alzheimer dementia, Wernicke-Korsakoff syndrome, and "alcoholic dementia," a
3 aware of the risk factors for cancer-related Wernicke-Korsakoff syndrome, and that they are vigilant
4 the development of the neurological disorder Wernicke-Korsakoff Syndrome as well as the potentially f
5 ited the entire triad of classic features of Wernicke-Korsakoff syndrome: confusion, ataxia, and opht
9 cy and associated features of cancer-related Wernicke-Korsakoff syndrome in the published literature.
10 (PTD) was used to produce a rodent model of Wernicke-Korsakoff syndrome that results in acute neurol
11 ere observed in the brain from patients with Wernicke-Korsakoff syndrome, the clinical manifestation
12 ogy in cases of diencephalic amnesia such as Wernicke Korsakoff Syndrome (WKS), there is also functio
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