ライフサイエンスコーパス: Western blot analysis

1 tor, and CTGF protein levels were assayed by Western blot analysis.

2 ines and human normal breast cell line using Western blot analysis.

3 these neurons using immunohistochemistry and western blot analysis.

4 Protein expression changes were confirmed by Western blot analysis.

5 ns junction protein using immunostaining and Western blot analysis.

6 se results by quantitative PCR, in situ, and Western blot analysis.

7 ts, and secretion is observed within 10 d by Western Blot analysis.

8 titative polymerase chain reaction (qPCR)and western blot analysis.

9 oscopy, flow cytometry, Rac1 activation, and Western blot analysis.

10 priate time after injury were assessed using Western blot analysis.

11 easurement of PDE-associated metabolites and Western blot analysis.

12 ation of the selective inhibitor Y-27632 and Western blot analysis.

13 assays, polysome fractionation studies, and Western blot analysis.

14 ion were evaluated by cell fractionation and Western blot analysis.

15 slated region luciferase activity assay, and Western blot analysis.

16 ion 48 h post-treatment, assessed by in-cell Western blot analysis.

17 y immunofluorescence confocal microscopy and Western blot analysis.

18 LC26A6 in duodenum by immunofluorescence and Western blot analysis.

19 by RT-PCR, immunofluorescence microscopy and Western blot analysis.

20 showed SMA expression, as evidenced by using Western blot analysis.

21 n the culture supernatants was determined by Western blot analysis.

22 ssed by surface biotinylation and subsequent Western blot analysis.

23 tive real-time PCR and protein expression by Western blot analysis.

24 inant proteins were measured by quantitative Western blot analysis.

25 was confirmed by co-immunoprecipitation and Western blot analysis.

26 tial involvement of DCPIB in this pathway by western blot analysis.

27 yruvate metabolism) was further validated by western blot analysis.

28 growth factor levels in skin, as revealed by Western blot analysis.

29 DeltaHS, and loss of occludin as detected by Western blot analysis.

30 lamide gel electrophoresis and corresponding Western blot analysis.

31 backbone protein of PI-2b) by whole cell and Western blot analysis.

32 nstream targets were examined by qRT-PCR and Western blot analysis.

33 mples from human and mouse models of DCM via Western blot analysis.

34 kinase (ERK)-1/2 activity was determined by Western blot analysis.

35 /WAF/Cip1 levels were determined by means of Western blot analysis.

36 n of the Smad pathway were evaluated through Western blot analysis.

37 or the presence of antiretinal antibodies by Western blot analysis.

38 fully decreased ASK1, which was confirmed by Western blot analysis.

39 markers was determined by real-time PCR and Western blot analysis.

40 d tissues were analyzed by real-time PCR and Western blot analysis.

41 lin expression was analyzed using RT-PCR and Western blot analysis.

42 R-224, as shown by both luciferase assay and Western blot analysis.

43 bility compared with WT ZnT-2 as revealed by Western blot analysis.

44 protein of the expected molecular weight in western blot analysis.

45 oxygen consumption, mitochondrial mass, and Western blot analysis.

46 sults in a stable colony, as demonstrated by Western blot analysis.

47 amino acid transporter 3) were validated by Western blot analysis.

48 ibition of EGFR downstream signalling in the western blot analysis.

49 ations, as measured by two-dimensional (2-D) Western blot analysis.

50 tative RT-PCR, and proteins were detected by Western blot analysis.

51 GluA1-S845A mutant mice were compared using western blot analysis.

52 ase can be isolated for mass spectrometry or western blot analysis.

53 from 5 healthy adults (24 +/- 3 years) using western blot analysis.

54 ronal marker protein expression, as shown by Western blot analysis.

55 um of rats by immunofluorescent staining and Western blot analysis.

56 -transcriptase polymerase chain reaction and Western blotting analysis.

57 s, real-time quantitative PCR (RT-qPCR), and Western blotting analysis.

58 Sixty patient samples underwent Western blot analysis (15 young, 24 aged, and 21 with AD

59 i1, Ngn1, and BMP4 proteins were measured by western-blot analysis 28 days after transplantation.

60 t target of miR-106b, which was confirmed by western blot analysis and a 3'-untranslated region repor

61 ent cohorts for the presence of THSD7A-Ab by Western blot analysis and a newly developed indirect imm

62 Phospho-p70 S6K was detected by Western blot analysis and activity blocked by rapamycin.

63 Complementary endpoint assays: western blot analysis and atomic force microscopy experi

64 (SMCs) under resting conditions, revealed by Western blot analysis and confocal microscopy of SMCs tr

65 Western blot analysis and confocal microscopy revealed t

66 lone JFH1 (HCV genotype 2) by real-time PCR, Western blot analysis and confocal microscopy.

67 Proteins were detected by immunostaining or Western blot analysis and cytokines by immunoassay.

68 Western blot analysis and electrophoretic mobility shift

69 mice and patients with COPD were examined by Western blot analysis and ELISA, respectively.

70 evel in lysates and MSC-conditioned media by Western blot analysis and ELISA.

71 CL21 mediates angiogenesis was determined by Western blot analysis and endothelial cell chemotaxis an

72 lar mediators of autophagy was measured with Western blot analysis and fluorescence microscopy.

73 when compared to WTMI animals as revealed by Western blot analysis and Gel-shift analysis, respective

74 Western blot analysis and gene expression profiling show

75 reased synthesis of tropoelastin detected by Western blot analysis and histology.

76 ng transfections of U-2 OS and HEK293 cells, Western blot analysis and immunocytochemistry data demon

77 decreased at day 7, 14, and 21, as shown by western blot analysis and immunofluorescence staining.

78 e PCR were used to evaluate gene expression, western blot analysis and immunohistochemistry for prote

79 Brain LCN2 expression was determined by Western blot analysis and immunohistochemistry.

80 xed brain specimens were analyzed by RT-PCR, Western blot analysis and immunohistochemistry.

81 ession was confirmed at the protein level by western blot analysis and immunohistochemistry.

82 Western blot analysis and immunostaining were performed

83 Western blot analysis and in situ immunolocalization sho

84 human plasma and Lp(a) uptake studied using Western blot analysis and intracellular localization of

85 human plasma and Lp(a) uptake studied using Western blot analysis and intracellular localization of

86 o eliminating the labor-intensive efforts of Western blot analysis and is not affected by the interfe

87 bsequent confirmation as mir-130a targets by western blot analysis and knockdown experiments validate

88 Targets of miRNA-31-3p were confirmed by Western blot analysis and luciferase reporter assay.

89 Here we used far-Western blot analysis and microscale thermophoresis to s

90 Western blot analysis and palmitate labeling indicated t

91 Western blot analysis and quantitative PCR confirmed tha

92 Western blot analysis and real-time PCR were performed t

93 trometry (LC-MS/MS) followed by quantitative Western blot analysis and retinal tissue immunolabeling

94 Western blot analysis and RNA supershift identified Musa

95 SARM1 protein levels were determined by Western blot analysis and SARM1 tissue localization was

96 ased the expression of CTGF as determined by Western blot analysis and the expression of its 3'-UTR f

97 tein and integrin ligand fibronectin (FN) by Western blot analysis and transmission electron microsco

98 Analysis of protein abundance using western-blot analysis and mass spectrometry revealed tha

99 was assessed by immunofluorescence staining, Western blot analysis, and ELISA.

100 Cleavage of MoMsb2 was further confirmed by western blot analysis, and five putative cleavage sites

101 our studies using ELISAs for cytokines, PCR, Western blot analysis, and flow cytometry.

102 xed mice, confirmed by immunohistochemistry, Western blot analysis, and fundus fluorescein angiograph

103 n of Lin28 by quantitative real-time RT-PCR, Western blot analysis, and IHC.

104 Results from RT-PCR, Western blot analysis, and immunocytochemistry demonstra

105 RT-PCR, Western blot analysis, and immunohistochemistry (IHC) co

106 were assessed by quantitative real-time PCR, Western blot analysis, and immunohistochemistry, respect

107 ly transfected cells was examined by RT-PCR, western blot analysis, and immunohistochemistry.

108 sts from one individual by real-time PCR and western blot analysis, and in mouse tissues by immunohis

109 ted by quantitative immunohistochemistry and Western blot analysis, and the collective differences we

110 the regenerative process, by real time PCR, Western blotting analysis, and immunohistochemistry.

111 quantified using both immunofluorescence and Western blotting analysis, and nuclear functional TDP-43

112 PKCalpha, but normal PKCzeta expression, by Western blotting analysis, and vice versa.

113 Western blot analysis, as well as characterization of th

114 Western blot analysis assessed the presence or absence o

115 C-HA-PTX3 complex was formed, as analyzed by Western blot analysis, by AM cells but not human skin fi

116 munoprecipitation experiments and reciprocal Western blot analysis confirm that there are two differe

117 Western blot analysis confirmed increased expression of

118 Quantitative PCR and western blot analysis confirmed loss of Bach2 in c-Rel m

119 Western blot analysis confirmed loss WIF1 expression and

120 Western blot analysis confirmed NOX5 expression in isola

121 Western blot analysis confirmed that IGF-II inhibits apo

122 Immunofluorescence microscopy and Western blot analysis confirmed that TbMCP5 is exclusive

123 Western blot analysis confirmed the IHC data.

124 Western blot analysis confirmed the increase in GluA1 ex

125 Western blot analysis confirmed the presence of nsp2 in

126 Results of Western blot analysis confirmed the reduced levels of th

127 Western blot analysis confirmed the reduction in TRAF2,

128 3'UTR luciferase reporter assays and Western blot analysis confirmed the targeting of MYD88 b

129 PCR and Western blotting analysis confirmed gene and protein exp

130 , respectively, at 2 h postadministration by Western blot analysis, confirming the bioavailability an

131 Western blot analysis consistently exhibited an increase

132 Western blot analysis data indicate that improved TJ int

133 Immunohistochemical staining and western blot analysis demonstrated a decreased expressio

134 Western blot analysis demonstrated expression of the ful

135 Western blot analysis demonstrated that agLDL reduced VS

136 Signaling study with specific inhibitors and Western blot analysis demonstrated that CTGF-induced exp

137 Furthermore, western blot analysis demonstrated that LCN stimulations

138 Mass spectrometry and Western blot analysis demonstrated that RAGE binds selec

139 Western blot analysis demonstrated UT-A1 in the inner me

140 Immunocytochemistry and Western blot analysis determining the presence of alpha-

141 Western blot analysis failed to show FAN1 protein in the

142 Western blot analysis, flow cytometry, and immunostainin

143 scence for alpha-smooth muscle actin; and by Western blot analysis for alpha-smooth muscle actin, SNA

144 Western blot analysis for comparison of protein abundanc

145 nt based on electron microscopy analysis and Western blot analysis for some mitochondrial proteins, a

146 Further, western blot analysis for STAT1 and STAT3 showed that th

147 l subjects from a U.S. cohort, and performed Western blot analysis from 6 patients with IPF and 5 con

148 Western blot analysis has revealed significant differenc

149 CD4(+) and CD8(+) T cells was documented by western blot analysis; however, differences in protein e

150 mLIFR), microarray expression profiling and Western blotting analysis identified preferential phosph

151 tors and anti-apoptotic molecules by RT-PCR, Western blot analysis, IHC, and cytotoxicity assays usin

152 ar and cellular changes were investigated by Western blot analysis, immunohistochemistry, and electro

153 We detected AqpB by Western blot analysis in a glycosylated and a non-glycos

154 expression profiling, using microarrays and Western blot analysis in B6J TMA-resistant and 129Sv TMA

155 carinic M2 receptors, which was confirmed by Western blot analysis in oxaliplatin-treated rats and th

156 real-time RT-PCR, high-content analysis, or Western blot analysis in the fibrotic liver, human bone

157 imately 45% relative to controls measured by western blot analysis in the putamen of active vector re

158 uantification of GFAP-positive cells nor the Western blot analysis indicated statistical significant

159 Western blot analysis indicated that several autophagy-r

160 The Annexin-V staining and Western blot analysis indicated that t-DARPP effectively

161 Western blot analysis indicated that the protein express

162 Western blot analysis indicated that Wnt7a induced both

163 Western blotting analysis indicated that pituitary tumor

164 The Western blot analysis indicates the downstream effectors

165 Western blotting analysis indicates that mGluR1 was coup

166 e harvested for immunohistochemical studies, Western blot analysis, laser capture microdissections an

167 rse transcription polymerase chain reaction, Western blot analysis, laser scanning cytometry, and con

168 Using a redox Western blot analysis, no change was observed in redox s

169 CTR1 expression levels were detected by Western blot analysis of a group of tumor cell lines.

170 Western blot analysis of atherosclerotic plaque lysates

171 Isoelectric focusing followed by Western blot analysis of bleached isolated retinae showe

172 titative polymerase chain reaction (PCR) and western blot analysis of C/EBPalpha-saRNA-transfected ce

173 Western blot analysis of cell lysates revealed that APOE

174 Western blot analysis of cells infected with adenovirus

175 iodic acid-Schiff staining of lung sections, Western blot analysis of chloride channel calcium activa

176 Western blot analysis of CL-L1 showed the presence of se

177 n high RI placental tissue, as determined by Western blot analysis of cleaved poly (ADP-ribose) polym

178 Western blot analysis of CNS extract showed that proteol

179 Immunoprecipitation and Western blot analysis of human umbilical cord plasma ena

180 tested regions, from both species, based on Western blot analysis of isolates using beta2 subunit-sp

181 Galphai proteins expression was measured by western blot analysis of LC punches.

182 Western blot analysis of llama and bull seminal plasma c

183 MCs using XMRV-specific gag and env primers, Western blot analysis of monkey plasma up to 31 to 32 we

184 er gene assay, immunofluorescence assay, and Western blot analysis of nuclear and cytoplasmic fractio

185 downregulation of mTOR pathway activation by Western blot analysis of p-mTOR, p70 ribosomal S6 kinase

186 ssociated beta-Galactosidase staining and by Western blot analysis of p16.

187 peaked by 72 h on immunohistochemistry, and Western blot analysis of P2X7R for saline- and lipopolys

188 On Western blot analysis of pathway checkpoints, p-mTOR (p=

189 tes, as demonstrated by confocal microscopy, Western blot analysis of podocyte-specific proteins, and

190 RT-PCR and Western blot analysis of SDHB mRNA and protein expressio

191 global quantitative proteomics approach and western blot analysis of selected proteins.

192 poVAD in wild-type spores were determined by Western blot analysis of spore fractions or total disrup

193 Western blot analysis of the 31 cases showed that severa

194 Western blot analysis of the basolateral amygdala (BLA)

195 Western blot analysis of total cell lysate obtained from

196 For this, we performed western blot analysis of total ubiquitination, free ubiq

197 n of intracellular signaling was measured by Western blot analysis of treated and untreated cells.

198 l staining, indirect immunofluorescence, and Western blotting analysis of human aggressive OCSCC spec

199 Western blotting analysis of PELP1-cyto HMECs showed up-

200 Western-blot analysis of frozen brain tissue displayed a

201 Western blot analysis on heart lysates from Snrk cmcKO a

202 ostimulation mapping and tissue sampling for Western blot analysis or contributed tissue for 3D elect

203 amples collected were either homogenized for Western blot analysis or fixed and embedded in paraffin

204 st, no correlation was observed with ex vivo Western blot analysis or in vitro flow-cytometry assays.

205 Western blot analysis, performed on phloem sap collected

206 y, the level of target protein expression by Western blot analysis, platelet aggregation using an agg

207 llected January 2007 to March 2014) included Western blot analysis, pulsed-field gel electrophoresis

208 mouse hepatic cell populations were used for Western blot analysis, quantitative real-time RT-PCR, an

209 vels were determined by quantitative PCR and Western blot analysis, respectively, in the fetus, its a

210 protein were assessed with real-time PCR and Western blot analysis, respectively.

211 immunofluorescence and confocal analysis and Western blot analysis, respectively.

212 levels were measured by quantitative PCR and Western blot analysis, respectively.

213 pts and proteins with the use of qRT-PCR and Western blot analysis, respectively.

214 Likewise, western blot analysis revealed a 41% clearance of solubl

215 FA pull-down assays with Western blot analysis revealed a FA-binding ability of A

216 Myocardial histochemistry and Western blot analysis revealed a significant influx of m

217 Western blot analysis revealed absence of the STAT2 prot

218 Western blot analysis revealed corresponding levels of A

219 increased levels of EDN2 and EDNRA mRNA, and Western blot analysis revealed increased EDN2 expression

220 Western blot analysis revealed NCAM was polysialylated a

221 Although Western blot analysis revealed no difference in opsin ex

222 Western blot analysis revealed reduced expression of sma

223 Western blot analysis revealed significantly increased S

224 Western blot analysis revealed that both purified Apo A-

225 erase reporter assay, mass spectrometry, and Western blot analysis revealed that caspase-2 and sirtui

226 Western blot analysis revealed that Cnm from the Deltapg

227 Mass spectrometric analysis and Western blot analysis revealed that Hsp70 was associated

228 Western blot analysis revealed that levels of GR protein

229 Western blot analysis revealed that mTOR deletion led to

230 Western blot analysis revealed that Notch2HCS mutant spl

231 Flow cytometry and Western blot analysis revealed that Rap2B elevates the i

232 Western blot analysis revealed that silencing of SlP4H7

233 graphy-elevated energy mass spectrometry and Western blot analysis revealed that the SNAT2 ER-alpha-E

234 Western blot analysis revealed that there was no effect

235 cles with a significant protein content, and Western blot analysis revealed that they contain, as exp

236 A Western blotting analysis revealed that biotin-labeled b

237 Western blotting analysis revealed that ligation of the

238 Western blot analysis reveals lower hepatic CEACAM1 expr

239 tion, as determined by luciferase assays and Western blot analysis, reversion-inducing cysteine-rich

240 ays regulating actin turnover were explored; western blot analysis showed a reduction in both Src and

241 Western blot analysis showed absence of Fhl1 in muscle a

242 Western blot analysis showed both increased phospho-AMPK

243 On-cell Western blot analysis showed decreased Kir2.1 cell surfa

244 Western blot analysis showed MYO5A and MYO5B expression

245 Western blot analysis showed reduced phosphorylation of

246 Western blot analysis showed significantly increased lev

247 Western blot analysis showed that both OTUB1 and FOXM1 e

248 Western blot analysis showed that both phosphorylated ST

249 Western Blot analysis showed that CjapOBP1 is predominan

250 Western blot analysis showed that hippocampal parvalbumi

251 Western blot analysis showed that IL28B TT genotype hepa

252 Western blot analysis showed that ketotifen restored imm

253 Moreover, western blot analysis showed that lipogenesis pathway en

254 Western blot analysis showed that SRSF2 expression was l

255 Western blot analysis showed that Tbeta4 treatment suppr

256 Immunohistochemistry and Western blot analysis showed that the mutation affects n

257 Immunohistochemistry and western blot analysis showed that the protein expression

258 In particular, live cell Ca(2+) imaging and Western blot analysis showed that TRAM-34 pretreatment d

259 Western blot analysis showed the virtual absence of RNas

260 le-cell variant of the patch-clamp technique.Western blotting analysis showed that Gbeta1, Gbeta2 and

261 Western blot analysis shows IL-1R3 is preferentially exp

262 Western blot analysis shows that the CD147-98 complex is

263 Mass spectrometry data confirmed Western blot analysis that ubiquitination of alpha4 occu

264 Compared with Western blot analysis, the IFT had a 92% sensitivity and

265 In particular, we examined, using Western blot analysis, the levels of H3K36me3 and H4K16a

266 Compared with western blot analysis, this ultrasensitive assay require

267 we employed immunofluorescence staining and Western blot analysis to assess the expression of autoph

268 s shown by pathway inhibitor experiments and Western blot analysis to be mediated via MEK-ERK signali

269 This study used western blot analysis to compare protein levels of tyros

270 macologic inhibitors, phosphoproteomics, and Western blot analysis to define the necessity of various

271 lasticity, in vivo place cell recording, and western blot analysis to determine protein levels relate

272 w conditions coupled with flow cytometry and Western blot analysis to elucidate the function and stru

273 (ERAI) mice and used quantitative RT-PCR and Western blot analysis to evaluate relative gene and prot

274 We also used Western blot analysis to quantify levels of PPK and PPX

275 Real-time PCR and Western blot analysis, together with immunofluorescence

276 iction enzyme digestion, DNA sequencing, and western blot analysis using HPA-1b-specific human matern

277 Almost no binding was found in western blot analysis using mouse monoclonal mAbs and se

278 re also processed for immunocytochemical and Western blot analysis using phosphorylated extracellular

279 agged Cx43 followed by confocal imaging, and Western blot analysis using protein isolated from mitoch

280 Western blot analysis utilizing this novel antibody, ter

281 vidity, and competition for epitope binding; Western blot analysis was also performed.

282 Western blot analysis was performed using soluble and in

283 Western blot analysis was utilized to assess the activat

284 secretory form of clusterin, as evaluated by Western blot analysis, was low or absent in cystinosis c

285 Using Western blot analysis, we confirmed that treatment of HS

286 Using Western blot analysis, we confirmed that treatment of HS

287 Utilizing Western blot analysis, we demonstrate that similar to ra

288 Using Western blot analysis, we demonstrated that eosinophils

289 Using western blot analysis, we found that the level of protei

290 Through western blot analysis, we identified that human glioma c

291 Using real time PCR and Western blot analysis, we observed that IL-2 treatment i

292 Here, using whole-cell patch clamp and Western blotting analysis, we demonstrate that the hERG/

293 rase chain reaction, immunofluorescence, and Western blot analysis were performed on muscle biopsy sp

294 time polymerase chain reaction (QRT-PCR) and Western blot analysis were used to assess matrix metallo

295 ophysiology, immunohistochemistry, qPCR, and Western blot analysis were used to determine that AA-ORS

296 Stereological methods and western blot analysis were used to estimate the total nu

297 Cellular fractionation and Western blot analysis were used to measure the levels of

298 n using an antibody against pTyr followed by Western blot analysis with an antibody against EGFR.

299 Western blot analysis with MAGE-A3 antibodies recognized

300 tor phosphorylation was evaluated by IHC and Western blot analysis with the new Ra-1124 antibody spec