ライフサイエンスコーパス: Wiskott-Aldrich syndrome protein

1 actin-nucleating machine activated by WASp (Wiskott Aldrich syndrome protein).

2 n with the nucleation-promoting factor Wasp (Wiskott-Aldrich syndrome protein).

3 fining mechanisms that control activation of Wiskott-Aldrich syndrome protein.

4 se 1, Grb2-associated protein 2 (Grap2), and Wiskott-Aldrich syndrome protein.

5 ft microdomain along with Arp2/3 complex and Wiskott-Aldrich syndrome protein.

6 ing edges of lamellipodia, Arp3 and neuronal Wiskott-Aldrich syndrome protein.

7 Cdc42 using the GTPase-binding domain of the Wiskott-Aldrich syndrome protein.

8 adhesion kinase, Rho GTPase Rac1, and neural Wiskott-Aldrich syndrome protein.

9 e cytoskeleton by binding and activating the Wiskott-Aldrich syndrome protein.

10 suring vector copy numbers and expression of Wiskott-Aldrich syndrome protein.

11 ngagement increases actin polymerization and Wiskott-Aldrich syndrome protein activation in a Btk-dep

12 achment but inhibits ingestion by decreasing Wiskott-Aldrich syndrome protein activation, and hence a

13 as the levels of F-actin and phosphorylated Wiskott Aldrich syndrome protein, an actin nucleation pr

14 r binding to the SH3 domains with the neural Wiskott-Aldrich Syndrome protein, an actin filament nucl

15 exists in a macromolecular complex with the Wiskott-Aldrich syndrome protein, an actin nucleation-pr

16 rmore, syndapin I associates with the neural Wiskott-Aldrich syndrome protein, an actin-depolymerizin

17 e Cdc42-binding domains of the CRIB motif of Wiskott-Aldrich Syndrome protein and p21(cdc42/rac)-acti

18 contractility, independent of its effects on Wiskott-Aldrich syndrome protein and p21-activated kinas

19 t complex and the endosomal Arp2/3 activator Wiskott-Aldrich syndrome protein and Scar homolog (WASH)

20 l. and Gomez and Billadeau reveal that WASH (Wiskott-Aldrich syndrome protein and SCAR homolog) activ

21 WASH (Wiskott-Aldrich Syndrome Protein and SCAR Homolog) is an

22 FAM21, which also binds retromer, within the Wiskott-Aldrich syndrome protein and SCAR homologue (WAS

23 w that the COMMD/CCDC22/CCDC93 (CCC) and the Wiskott-Aldrich syndrome protein and SCAR homologue (WAS

24 JIPs are recruited by Wiskott-Aldrich syndrome protein and scar homologue (WAS

25 Wiskott-Aldrich syndrome protein and SCAR homologue (WAS

26 The Arp2/3-activator Wiskott-Aldrich syndrome protein and Scar homologue (WAS

27 dc42 may regulate the activation of neuronal Wiskott-Aldrich syndrome protein and the actin related p

28 ptor that replaces toca-1 to mobilize neural Wiskott-Aldrich syndrome protein and the Arp2/3 complex.

29 hat drive actin polymerization such as WASp (Wiskott-Aldrich syndrome protein) and HS1 (hematopoietic

30 mbly protein Las17 (a yeast homolog of human Wiskott-Aldrich syndrome protein) and participate in the

31 ns of increased detergent stringency Sam 68, Wiskott-Aldrich Syndrome protein, and hnRNP-K, but not C

32 owever was slower and required intact actin, Wiskott-Aldrich syndrome protein, and microtubule functi

33 kott-Aldrich syndrome protein (WASP), neural Wiskott-Aldrich syndrome protein, and WASP-interacting p

34 The Src family kinase Hck, Wiskott-Aldrich-syndrome protein, and phospholipase Cgam

35 ctin dynamics through the Nck/N-WASp (neural Wiskott-Aldrich syndrome protein)/Arp2/3 pathway is esse

36 to which we apply these ideas is that of the Wiskott-Aldrich Syndrome Proteins as activators of actin

37 previously that the yeast orthologue of the Wiskott-Aldrich Syndrome protein, Bee1/Las17p, and the t

38 The constitutively active mutant of the Wiskott-Aldrich Syndrome protein (CA-WASp) is the cause

39 Mutations of the Wiskott-Aldrich syndrome protein can result in highly va

40 cell division cycle 42, which, together with Wiskott-Aldrich syndrome protein, coordinates F-actin re

41 for actin binding (profilin or the WH2 from Wiskott-Aldrich syndrome protein) decrease full-length I

42 Studies of Wiskott-Aldrich syndrome protein-deficient cell lines an

43 Wiskott-Aldrich syndrome protein-deficient neutrophils a

44 ition, Arp3-silenced cells expressing neural Wiskott-Aldrich syndrome protein-derived peptides that i

45 surface receptors was rapid and depended on Wiskott-Aldrich syndrome protein-driven actin polymeriza

46 ely in hematopoietic stem cells, and because Wiskott-Aldrich syndrome protein exerts a strong selecti

47 Recombinant Arc40 bound the VCA domain of Wiskott-Aldrich syndrome protein family activators at a

48 The scaffolding protein WAVE-1 (Wiskott-Aldrich syndrome protein family member 1) direct

49 Wiskott-Aldrich syndrome protein family verprolin homolo

50 binding regions of TOCA1 and a member of the Wiskott-Aldrich syndrome protein family, N-WASP.

51 EspF(U) potently activates the host WASP (Wiskott-Aldrich syndrome protein) family of actin-nuclea

52 proteins 2/3) complex is activated by WASP (Wiskott-Aldrich syndrome protein) family proteins to nuc

53 be mediated by the protein Scar/WAVE (WASP (Wiskott-Aldrich syndrome protein)-family verprolin homol

54 Further, CYFIP2 is part of the Wiskott-Aldrich syndrome protein-family verprolin-homolo

55 NK cell function relies on Wiskott-Aldrich syndrome protein for filamentous actin (

56 We purified native WASp (Wiskott-Aldrich Syndrome protein) from bovine thymus and

57 Wiskott-Aldrich syndrome protein gene mutations result i

58 drich syndrome is caused by mutations of the Wiskott-Aldrich syndrome protein gene, which codes for a

59 By fusing the Wiskott-Aldrich syndrome protein/GTPase-binding domain t

60 he discovery of unique functional domains of Wiskott-Aldrich syndrome protein has been instrumental i

61 ress made in dissecting the functions of the Wiskott-Aldrich syndrome protein has direct implications

62 g a proline-rich domain and an actin-binding Wiskott-Aldrich syndrome protein homology 2 (WH2) domain

63 length short (SALS) is a recently identified Wiskott-Aldrich syndrome protein homology 2 (WH2) domain

64 despite having only a single G-actin-binding Wiskott-Aldrich syndrome protein Homology 2 (WH2) domain

65 y relies on a cluster of three actin-binding Wiskott-Aldrich syndrome protein homology 2 (WH2) domain

66 autoregulatory domain (DAD) that resembles a Wiskott-Aldrich syndrome protein homology 2 (WH2) sequen

67 tant manner, bound actin monomer via a WASP (Wiskott-Aldrich syndrome protein) homology 2 domain, bou

68 variable was the presence or absence of the Wiskott-Aldrich syndrome protein in the lymphoid cells f

69 he actin regulatory protein N-WASP (neuronal Wiskott-Aldrich syndrome protein) in which the "output"

70 Wiskott-Aldrich syndrome protein-interacting protein (WI

71 s includes Wiskott-Aldrich syndrome protein, Wiskott-Aldrich syndrome protein-interacting protein, co

72 WIP, the Wiskott-Aldrich syndrome protein-interacting protein, is

73 Wiskott-Aldrich syndrome protein is a signaling molecule

74 ed to aspartate or glutamate, the binding to Wiskott-Aldrich Syndrome protein is abrogated.

75 Since Wiskott-Aldrich syndrome protein is expressed exclusivel

76 In addition, the Wiskott-Aldrich syndrome protein is required for natural

77 nucleation-promoting protein N-WASP (Neural Wiskott-Aldrich syndrome protein) is up-regulated in bre

78 toskeletal protein with sequence homology to Wiskott-Aldrich syndrome protein, is required for the fi

79 mphiphysin-RVS-domain protein Rvs167 and the Wiskott-Aldrich syndrome protein Las17 at the point of p

80 In contrast, Wiskott-Aldrich syndrome protein localized exclusively a

81 such as integrin beta1, cortactin, neuronal Wiskott-Aldrich syndrome protein, membrane type 1 metall

82 sion required actin polymerization, neuronal Wiskott-Aldrich syndrome protein, myosin II and Rho GTPa

83 stimulated phosphoprotein (VASP), and neural Wiskott Aldrich syndrome protein (N-WASP) are present at

84 tein c-Abl interactor 1 (Abi1) with neuronal Wiskott-Aldrich syndrome protein (N-WASP) (an actin-regu

85 complex was necessary for cdc42 and neuronal Wiskott-Aldrich syndrome protein (N-WASP) activation, ac

86 spatial and temporal regulation of neuronal Wiskott-Aldrich syndrome protein (N-WASP) activity in li

87 localization of the actin-regulatory neural Wiskott-Aldrich syndrome protein (N-WASP) and actin-rela

88 nts in the actin regulatory protein neuronal Wiskott-Aldrich syndrome protein (N-WASP) and an SH2 dom

89 Neural Wiskott-Aldrich syndrome protein (N-WASP) and p34-Arc, a

90 474/1 leads to recruitment of Nck and neural Wiskott-Aldrich syndrome protein (N-WASP) and strong act

91 c42), the nucleation-promoting factor neural Wiskott-Aldrich syndrome protein (N-WASP) and the actin

92 Neuronal Wiskott-Aldrich syndrome protein (N-WASP) and the actin-

93 zation is mediated by activation of neuronal Wiskott-Aldrich syndrome protein (N-WASp) and the Arp (a

94 iprotein compound containing CrkII, neuronal Wiskott-Aldrich Syndrome Protein (N-WASP) and the Arp2/3

95 gulators of actin cytoskeleton dynamics, the Wiskott-Aldrich syndrome protein (N-WASP) and the Arp2/3

96 ole of the actin nucleation promoters neural Wiskott-Aldrich syndrome protein (N-WASP) and WAVE2 in c

97 and Src family tyrosine kinases, and neural Wiskott-Aldrich syndrome protein (N-WASP) but not the Ar

98 indicates that the nuclear localized neural Wiskott-Aldrich syndrome protein (N-WASP) can induce de

99 Neuronal Wiskott-Aldrich syndrome protein (N-WASP) has an essenti

100 The Neuronal Wiskott-Aldrich syndrome protein (N-WASP) has emerged as

101 we showed that S. flexneri relies on neural Wiskott-Aldrich Syndrome protein (N-WASP) in HT-29 cells

102 f hyaluronan (HA) and CD44 with the neuronal Wiskott-Aldrich syndrome protein (N-WASP) in regulating

103 f the actin-regulatory protein called neural Wiskott-Aldrich syndrome protein (N-WASP) interacting wi

104 Neuronal Wiskott-Aldrich syndrome protein (N-WASP) is a member of

105 The VCA domain of the neuronal Wiskott-Aldrich syndrome protein (N-WASP) is a potent ac

106 Co et al. now show that the neural Wiskott-Aldrich syndrome protein (N-WASP) mediates dynam

107 er suggests that the actin-regulatory neural Wiskott-Aldrich syndrome protein (N-WASP) mediates the e

108 In this study, we show neural Wiskott-Aldrich syndrome protein (N-WASP) regulates the

109 actin nucleating endocytic protein neuronal Wiskott-Aldrich syndrome protein (N-WASP) to facilitate

110 Neuronal Wiskott-Aldrich Syndrome protein (N-WASP) transmits sign

111 n activation of the Arp2/3 complex by neural Wiskott-Aldrich Syndrome protein (N-WASP) via Grb2 and N

112 I, synaptojanin, synapsin I, and the neural Wiskott-Aldrich syndrome protein (N-WASP), a stimulator

113 Neural Wiskott-Aldrich syndrome protein (N-WASP), a ubiquitous

114 -mediated EGFR signaling up-regulated neural Wiskott-Aldrich syndrome protein (N-WASP), an actin nucl

115 acted with lacrimal acinar dynamin, neuronal Wiskott-Aldrich Syndrome protein (N-WASP), and synaptoja

116 ched in actin-related protein 3 and neuronal Wiskott-Aldrich syndrome protein (N-WASP), and their ass

117 in-binding and -polymerizing proteins neural Wiskott-Aldrich syndrome protein (N-WASP), cortactin, an

118 ockout approach to assess the role of neural Wiskott-Aldrich syndrome protein (N-WASP), the ubiquitou

119 s, we have analysed the dynamics of neuronal Wiskott-Aldrich syndrome protein (N-WASP), WASP-interact

120 We altered the function of neural Wiskott-Aldrich syndrome protein (N-WASP), which induces

121 nce microscopy, we demonstrate that neuronal Wiskott-Aldrich syndrome protein (N-WASP), which is coex

122 Here, we demonstrate that neuronal Wiskott-Aldrich syndrome protein (N-WASP), which promote

123 M. avium led to the recruitment of neuronal Wiskott-Aldrich syndrome protein (N-WASp), which was not

124 Neuronal Wiskott-Aldrich syndrome protein (N-WASP)-activated acti

125 karyotic sorting nexin 9 (SNX9) and neuronal Wiskott-Aldrich syndrome protein (N-WASP).

126 the actin polymerization catalyst, neuronal Wiskott-Aldrich syndrome protein (N-WASP).

127 nd like IcsA, YapV recruits mammalian neural Wiskott-Aldrich syndrome protein (N-WASP).

128 Nck and the actin nucleation promoter neural Wiskott-Aldrich syndrome protein (N-WASP).

129 onse to signals that locally activate neural Wiskott-Aldrich-syndrome protein (N-WASP) and the Arp2/3

130 ed that C. parvum activates the Cdc42/neural Wiskott-Aldrich syndrome protein network in host cells r

131 the microtubule-organizing center, F-actin, Wiskott-Aldrich syndrome protein, nor proline rich tyros

132 the actin regulatory proteins Las17p (yeast Wiskott-Aldrich syndrome protein) or Arp2/3, or deletion

133 became more tightly associated with neuronal Wiskott-Aldrich syndrome protein, promoting actin-relate

134 Neuronal Wiskott-Aldrich syndrome protein regulates TGF-beta1-med

135 WASH is an Arp2/3 activator of the Wiskott-Aldrich syndrome protein superfamily that functi

136 ve studies of patients with mutations of the Wiskott-Aldrich syndrome protein unequivocally demonstra

137 The actin-modulating protein Wiskott-Aldrich syndrome protein verprolin homologous-1

138 The direct interaction of Skap2 with the Wiskott-Aldrich syndrome protein via its SH3 domain is c

139 ctin dynamics and Ag transport by activating Wiskott-Aldrich syndrome protein via Vav and phosphatidy

140 pe, a gene encoding a protein related to the Wiskott-Aldrich Syndrome protein was discovered.

141 Finally, the Wiskott-Aldrich syndrome protein was shown to play an im

142 to mutations of the X-chromosome gene WASP (Wiskott-Aldrich syndrome protein), was characterized ori

143 The 70 C-terminal amino acids of Wiskott-Aldrich syndrome protein (WASp WA) activate the

144 3 complex also interacts with members of the Wiskott-Aldrich syndrome protein (WASP) [8] family - Sca

145 We have shown previously that Wiskott-Aldrich syndrome protein (WASP) activation at th

146 The Wiskott-Aldrich syndrome protein (WASP) and its homolog

147 The Wiskott-Aldrich syndrome protein (WASP) and its relative

148 The Wiskott-Aldrich syndrome protein (WASP) and neural WASP

149 The WIP C-terminal domain binds to Wiskott-Aldrich syndrome protein (WASp) and regulates it

150 ctivation, when it is able to associate with Wiskott-Aldrich syndrome protein (WASp) and the actin fi

151 Actin polymerization mediated by Wiskott-Aldrich syndrome protein (WASp) and the actin-re

152 tion by activating the pathway involving the Wiskott-Aldrich syndrome protein (WASP) and the actin-re

153 Nodule formation is dependent on Wiskott-Aldrich syndrome protein (WASp) and the ARP2/3 c

154 namics are controlled by Arp2/3 complex, the Wiskott-Aldrich syndrome protein (WASp) and the related

155 in polymerization through Arp2/3 nucleation, Wiskott-Aldrich syndrome protein (WASP) and WASP family

156 ing proteins were the actin binding proteins Wiskott-Aldrich syndrome protein (WASP) and WASP-interac

157 actin polymerization in pseudopods, whereas Wiskott-Aldrich syndrome protein (WASP) assembles actin

158 s) induces localized activation of Cdc42 and Wiskott-Aldrich Syndrome protein (WASP) at the immune sy

159 the SRC homology 3 (SH3) domain and impairs Wiskott-Aldrich syndrome protein (WASP) binding, but it

160 tative model of allosteric regulation of the Wiskott-Aldrich syndrome protein (WASP) by the Rho GTPas

161 the formation of endogenous Lck-Dlgh1-Zap70-Wiskott-Aldrich syndrome protein (WASp) complexes in whi

162 Wiskott-Aldrich syndrome protein (WASP) deficiency in mi

163 which are caused by WAS mutations affecting Wiskott-Aldrich syndrome protein (WASp) expression or ac

164 rimary immunodeficiency caused by absence of Wiskott-Aldrich syndrome protein (WASP) expression, resu

165 The proteins of the Wiskott-Aldrich syndrome protein (WASP) family are activ

166 Members of the Wiskott-Aldrich syndrome protein (WASP) family control a

167 Members of the Wiskott-Aldrich syndrome protein (WASP) family control c

168 We recently showed that the Wiskott-Aldrich syndrome protein (WASP) family member, W

169 d by different human proteins, including the Wiskott-Aldrich syndrome protein (WASp) family members.

170 the actin cytoskeleton through activation of Wiskott-Aldrich syndrome protein (WASP) family members.

171 The binding of Wiskott-Aldrich syndrome protein (WASP) family NPFs indu

172 w that mycolactone operates by hijacking the Wiskott-Aldrich syndrome protein (WASP) family of actin-

173 WAVE proteins are members of the Wiskott-Aldrich syndrome protein (WASP) family of scaffo

174 everal recent studies have demonstrated that Wiskott-Aldrich syndrome protein (WASP) family proteins

175 esponse to upstream signals, proteins in the Wiskott-Aldrich Syndrome protein (WASP) family regulate

176 Wiskott-Aldrich syndrome protein (WASP) family verprolin

177 , and acidic (VCA) region of proteins in the Wiskott-Aldrich syndrome protein (WASp) family, Arp2/3 c

178 controls actin by activating members of the Wiskott-Aldrich syndrome protein (WASP) family.

179 the actin machinery, such as members of the Wiskott-Aldrich syndrome protein (WASp) family.

180 in response to signals from proteins in the Wiskott-Aldrich syndrome protein (WASP) family.

181 WAS and XLT are caused by mutations of the Wiskott-Aldrich syndrome protein (WASP) gene which encod

182 Wiskott-Aldrich syndrome protein (WASP) has emerged as a

183 No defects related to deficiency of the Wiskott-Aldrich Syndrome protein (WASp) have been descri

184 Mutations in the Wiskott-Aldrich syndrome protein (WASP) have been hypoth

185 f formins, known filament nucleators use the Wiskott-Aldrich syndrome protein (WASP) homology 2 (WH2

186 A role for Wiskott-Aldrich syndrome protein (WASP) in chemotaxis to

187 The role of the Wiskott-Aldrich syndrome protein (WASp) in platelet func

188 Wiskott-Aldrich Syndrome protein (WASp) is a hematopoiet

189 The Wiskott-Aldrich syndrome protein (WASP) is a key cytoske

190 The Wiskott-Aldrich syndrome protein (WASp) is a key regulat

191 The Wiskott-Aldrich syndrome protein (WASP) is a multi-domai

192 The Wiskott-Aldrich syndrome protein (WASP) is a product of

193 The Wiskott-Aldrich syndrome protein (WASP) is an effector o

194 In this study, we demonstrate that the Wiskott-Aldrich syndrome protein (WASp) is an essential

195 We demonstrate that the Wiskott-Aldrich syndrome protein (WASp) is an important

196 Wiskott-Aldrich syndrome protein (WASp) is essential for

197 The Wiskott-Aldrich syndrome protein (WASp) is important for

198 previously that tyrosine phosphorylation of Wiskott-Aldrich syndrome protein (WASP) is important for

199 Wiskott-Aldrich syndrome protein (WASP) is in a complex

200 Here, we report that the Wiskott-Aldrich syndrome protein (WASp) is necessary for

201 ucleation by Arp2/3 complex activated by the Wiskott-Aldrich syndrome protein (WASP) or Scar protein;

202 Wiskott-Aldrich syndrome protein (WASP) plays a key role

203 The Wiskott-Aldrich syndrome protein (WASp) regulates actin

204 ed individuals reveals that mutations in the Wiskott-Aldrich syndrome protein (WASP) result in struct

205 utation (Leu270Pro) in the gene encoding the Wiskott-Aldrich syndrome protein (WASp) resulting in an

206 The Wiskott-Aldrich Syndrome protein (WASp) serves as a cruc

207 fic mutations in the human gene encoding the Wiskott-Aldrich syndrome protein (WASp) that compromise

208 LN) is caused by activating mutations in the Wiskott-Aldrich syndrome protein (WASP) that result in a

209 Here we have used cells deficient in the Wiskott-Aldrich syndrome protein (WASp) to demonstrate t

210 etal dysfunction caused by deficiency of the Wiskott-Aldrich syndrome protein (WASp) to explore the c

211 r receptor-bound protein 2 (Grb2) and to the Wiskott-Aldrich syndrome protein (WASp) to form a hetero

212 Whereas Cdc42 must activate Wiskott-Aldrich Syndrome protein (WASP) to stimulate nuc

213 Defects in Wiskott-Aldrich Syndrome protein (WASp) underlie develop

214 Mutations of Wiskott-Aldrich syndrome protein (WASP) underlie the sev

215 ds through its BAR domain and interacts with Wiskott-Aldrich Syndrome Protein (WASP) via its SRC homo

216 roteins involved in actin dynamics including Wiskott-Aldrich syndrome protein (WASp) were regulated b

217 ed via the phagocyte-specific kinase Hck and Wiskott-Aldrich syndrome protein (WASP), 2 major regulat

218 we demonstrated that ACK1 phosphorylates the Wiskott-Aldrich syndrome protein (WASP), a Cdc42 effecto

219 hat the PSTPIP SH3 domain interacts with the Wiskott-Aldrich syndrome protein (WASP), a cytoskeletal

220 s, develop in patients and mice deficient in Wiskott-Aldrich syndrome protein (WASP), a hematopoietic

221 protein, Bee1, exhibits sequence homology to Wiskott-Aldrich syndrome protein (WASP), a human protein

222 tients bearing inactivating mutations in the Wiskott-Aldrich syndrome protein (WASP), a key regulator

223 onsisting of WASp-interacting protein (WIP), Wiskott-Aldrich syndrome protein (WASp), actin, and myos

224 o the cytoskeleton through its effector, the Wiskott-Aldrich syndrome protein (WASP), activation of w

225 f, required an activating factor such as the Wiskott-Aldrich syndrome protein (WASP), and might exhib

226 rcent of natural killer (NK) cells expressed Wiskott-Aldrich syndrome protein (WASP), and NK cells co

227 HS1 interacts with Wiskott-Aldrich syndrome protein (WASp), another key act

228 The Wiskott-Aldrich syndrome protein (WASP), encoded by the

229 proline-rich protein that interacts with the Wiskott-Aldrich syndrome protein (WASP), from BJAB cell

230 teractions with a wide network of molecules: Wiskott-Aldrich syndrome protein (WASp), Grb2, ribosomal

231 One such effector, the Wiskott-Aldrich syndrome protein (WASP), is postulated t

232 se pulldown analyses show Robo4 binding to a Wiskott-Aldrich syndrome protein (WASP), neural Wiskott-

233 When activated by Wiskott-Aldrich Syndrome protein (WASp), the Arp2/3 comp

234 we provide evidence that Kit signals through Wiskott-Aldrich syndrome protein (WASP), the central hem

235 e B (PhyB) and fused the Cdc42 effector, the Wiskott-Aldrich Syndrome Protein (WASP), to the light-de

236 Two such adaptor proteins, Nck and the Wiskott-Aldrich syndrome protein (WASp), were recruited

237 Podosome formation requires the Wiskott-Aldrich syndrome protein (WASP), which is a prod

238 hat branching occurs when Arp2/3 is bound to Wiskott-Aldrich syndrome protein (WASP), which is in tur

239 racts through its SH3 domains with the human Wiskott-Aldrich syndrome protein (WASp), which plays a r

240 Here we show that deficiency of Wiskott-Aldrich syndrome protein (WASp), which signals t

241 edly reduced in macrophages deficient in the Wiskott-Aldrich syndrome protein (WASP), which still con

242 WAVE1--the Wiskott-Aldrich syndrome protein (WASP)--family verproli

243 for the assembly of filopodia-like bundles: Wiskott-Aldrich syndrome protein (WASP)-coated beads, ac

244 tients with the Wiskott-Aldrich syndrome and Wiskott-Aldrich syndrome protein (WASP)-deficient mice,

245 Wiskott-Aldrich syndrome protein (WASP)-deficient T cell

246 Here, we show that Wiskott-Aldrich syndrome protein (WASP)-family verprolin

247 P) intracellular domain (AICD) downregulates Wiskott-Aldrich syndrome protein (WASP)-family verprolin

248 Wiskott-Aldrich syndrome protein (WASP)-homology domain

249 Here, we demonstrate that Wiskott-Aldrich syndrome protein (WASp)-interacting prot

250 Wiskott-Aldrich syndrome protein (WASP)-interacting prot

251 s, where it was activated by p78/83, a viral Wiskott-Aldrich syndrome protein (WASP)-like protein.

252 e, Sos-activated Ras signaling and the human Wiskott-Aldrich Syndrome protein (WASp)-mediated actin c

253 inase (ACK), p21-activated kinase (PAK), and Wiskott-Aldrich syndrome protein (WASP).

254 r of the actin cytoskeleton and activator of Wiskott-Aldrich syndrome protein (WASP).

255 The gene defective in WAS encodes Wiskott-Aldrich syndrome protein (WASP).

256 ematopoietic-specific cytoskeletal regulator Wiskott-Aldrich syndrome protein (WASP).

257 lex by the actin nucleation-promoting factor Wiskott-Aldrich Syndrome protein (WASp).

258 olled by signaling pathways that include the Wiskott-Aldrich syndrome protein (WASP).

259 ional change in its downstream effector, the Wiskott-Aldrich syndrome protein (WASP).

260 A related switch has been seen in the Wiskott-Aldrich syndrome protein (WASP).

261 recognize other motility proteins, like the Wiskott-Aldrich syndrome protein (WASP).

262 inhibit the ability of Nwk-SH3a to activate Wiskott-Aldrich syndrome protein (WASp)/actin related pr

263 mparable to podosomes in the localization of Wiskott-Aldrich syndrome protein (WASP)/matrix metallopr

264 Nucleation-promoting factors (NPFs) of the Wiskott-Aldrich syndrome protein (WASP)/Scar family are

265 In animal cells and yeasts, Wiskott-Aldrich Syndrome protein (WASP)/suppressor of cA

266 The protein N-WASP [a homolog to the Wiskott-Aldrich syndrome protein (WASP)] regulates actin

267 lex must bind ATP, protein activators [e.g., Wiskott-Aldrich syndrome protein (WASp)], and the side o

268 Two molecules were isolated, the Wiskott-Aldrich syndrome protein (WASP, a putative CDC42

269 Wiskott-Aldrich syndrome proteins (WASP) are a family of

270 ycolactone-mediated activation of neural (N) Wiskott-Aldrich syndrome proteins (WASP) induces defects

271 /3 complex activation domain (WCA) of Las17 (Wiskott-Aldrich syndrome protein [WASp] homologue) fused

272 complex and in Bee1p/Las17p, a member of the Wiskott-Aldrich syndrome protein(WASP) family, lead to a

273 The Wiskott-Aldrich syndrome protein, WASP, is an adaptor pr

274 Wiskott-Aldrich syndrome protein (WASPs) control actin d

275 the DSH3PX1 SH3 domain interaction with the Wiskott-Aldrich Syndrome protein while enabling DSH3PX1

276 This includes Wiskott-Aldrich syndrome protein, Wiskott-Aldrich syndro

277 lation increased the association of neuronal Wiskott-Aldrich syndrome protein with Cdc42 and the Arp2