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1 tions were performed between Dark Agouti and Wistar Furth rats.
2 heterotopically into the abdominal aorta of Wistar-Furth rats.
3 , remnant nephropathy would be attenuated in Wistar-Furth rats.
4 ctal carcinomas in situ by 50 days of age in Wistar-Furth rats.
6 upon 5/6 nephrectomy was markedly reduced in Wistar-Furth rats, a finding not attributable to reduced
9 ic nephropathy depends on aldosterone and if Wistar-Furth rats are resistant to aldosterone, remnant
10 he study was repeated, with Wistar rats (not Wistar-Furth rats) being treated with a hydralazine-rese
13 The resulting mammary carcinomas in intact Wistar-Furth rats exhibit a mixed hormonal response in t
16 major histocompatibility complex-mismatched Wistar Furth rat or C57 mouse recipients with a clinical
19 ontrol group into the spleen of two diabetic Wistar Furth rats resulted in the death of the recipient
20 diabetic BB/Wor rats, and normal nondiabetic Wistar-Furth rats showed that the responses of those wit
22 erformed from dark agouti rat strain (DA) to Wistar furth rat strain rats and syngenic DA-DA grafts w
24 opic abdominal transplantation from Lewis to Wistar-Furth rats was used as a model of cardiac allogra
25 ant Copenhagen (COP) and carcinoma-sensitive Wistar-Furth rats, we have confirmed the identification
29 g. kg(-1). d(-1) PO; n=14), (4) normotensive Wistar-Furth rats (WF) at 9 months (n=12), and (5) WF at
30 responses, kidneys were transplanted between Wistar-Furth rats, with and without antioxidant 21-amino
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