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1 rom pacing or preexcitation in patients with Wolff-Parkinson-White syndrome).
2 some affected individuals (pre-excitation or Wolff-Parkinson-White syndrome).
3 pre-excitation, a characteristic feature of Wolff-Parkinson-White syndrome.
4 luate its outcome in pediatric patients with Wolff-Parkinson-White syndrome.
5 for the asymptomatic pediatric patient with Wolff-Parkinson-White syndrome.
6 d to be responsible for an inherited form of Wolff-Parkinson-White syndrome.
7 ing ventricular arrhythmias in patients with Wolff-Parkinson-White syndrome.
8 3 from Family 1 and 8 from Family 2) had the Wolff-Parkinson-White syndrome.
9 979 and 1989 before undergoing operation for Wolff-Parkinson-White syndrome.
10 reatment of the clinical arrhythmias seen in Wolff-Parkinson-White syndrome.
11 e for re-entrant cardiac arrhythmias such as Wolff-Parkinson-White syndrome.
13 ow report a novel mutation in PRKAG2 causing Wolff-Parkinson-White syndrome and conduction system dis
15 c abnormalities, particularly preexcitation (Wolff-Parkinson-White syndrome) and atrioventricular con
16 ial flutter, atrioventricular nodal reentry, Wolff-Parkinson-White syndrome, and atrial tachycardia.
17 biochemical properties reminiscent of human Wolff-Parkinson-White syndrome, arising from mutations i
18 eloped in adults also identify children with Wolff-Parkinson-White syndrome at risk for sudden death.
22 he largest reported to date of children with Wolff-Parkinson-White syndrome having a cardiac arrest,
23 ent retrospective and prospective studies of Wolff-Parkinson-White syndrome in asymptomatic pediatric
24 of detailed long-term data in patients with Wolff-Parkinson-White syndrome is limited, and no prospe
25 ntricular preexcitation, which characterizes Wolff-Parkinson-White syndrome, is caused by the presenc
26 athology of many diseases, including cancer, Wolff-Parkinson-White syndrome, neurodegenerative disord
27 This enabled detection of such phenomena as Wolff-Parkinson-White syndrome, QRS aberrancy, and multi
30 ophy and electrophysiological abnormalities: Wolff-Parkinson-White syndrome (WPW) and progressive deg
31 about the long-term natural history of adult Wolff-Parkinson-White syndrome (WPW) patients in regard
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