ライフサイエンスコーパス: Wolff

1 Wolff often used the human proximal femur as an example

2 Wolff rearrangements to 1,8-naphthyleneketenes (15a-d) a

3 Wolff's "law" of the functional adaptation of bone is ro

4 Wolff-Kishner conditions (KOH/ethylene glycol, 200 degre

5 Wolff-Parkinson-White is associated with high ARI disper

6 nm it results in efficient (phi(254) = 0.34) Wolff rearrangement, while irradiation with 355 nm light

7 ext of ventricular tachycardia (VT) (n = 9), Wolff-Parkinson-White (WPW) syndrome (n = 2), atrial fib

8 rovided for the use of TBSH derivatives in a Wolff-Kishner-type reduction protocol that proceeds at l

9 The key steps involved a Wolff rearrangement, followed by a stereoselective dihyd

10 c management of asymptomatic patients with a Wolff-Parkinson-White (WPW) pattern is controversial.

11 ve consecutive patients aged<18 years with a Wolff-Parkinson-White pattern and persistent preexcitati

12 ophy and electrophysiological abnormalities: Wolff-Parkinson-White syndrome (WPW) and progressive deg

13 about the long-term natural history of adult Wolff-Parkinson-White syndrome (WPW) patients in regard

14 erstanding of the risk of sudden death among Wolff-Parkinson-White patients were published.

15 egrees C results in decarbonylation of 1 and Wolff rearrangement to fulven-6-one (13) either concerte

16 ar arrhythmia, ventricular fibrillation, and Wolff-Parkinson-White.

17 ophy also present with skeletal myopathy and Wolff-Parkinson-White (WPW) syndrome; mutations in the g

18 te that leads to elimination of nitrogen and Wolff rearrangement is one of the highest singlet excite

19 fluorescence-plus-Giemsa method of Perry and Wolff to produce harlequin endoreduplicated chromosomes

20 This enabled detection of such phenomena as Wolff-Parkinson-White syndrome, QRS aberrancy, and multi

21 e for re-entrant cardiac arrhythmias such as Wolff-Parkinson-White syndrome.

22 the evaluated patients with an asymptomatic Wolff-Parkinson-White preexcitation persisting at peak e

23 study of either symptomatic or asymptomatic Wolff-Parkinson-White patients referred to our Arrhythmo

24 ar tachycardia in patients with asymptomatic Wolff-Parkinson-White ECG pattern.

25 profile analyses reveal that the barrierless Wolff rearrangement proceeds via an out-of-plane carbene

26 LH, Biro FM, Valentin-Blasini L, Blount BC, Wolff MS, for the Breast Cancer and Environment Research

27 thyloxirene to diacetylcarbene and thence by Wolff rearrangement to acetylmethylketene.

28 athology of many diseases, including cancer, Wolff-Parkinson-White syndrome, neurodegenerative disord

29 ow report a novel mutation in PRKAG2 causing Wolff-Parkinson-White syndrome and conduction system dis

30 ntricular preexcitation, which characterizes Wolff-Parkinson-White syndrome, is caused by the presenc

31 e diazodicarbonyl moiety, results in a clean Wolff rearrangement.

32 Thermal decomposition of 1 leads to clean Wolff rearrangement, while heating of 2 causes quantitat

33 cy than the ketone analogue due to competing Wolff rearrangement (WR) in the excited state of the dia

34 One pathway involves concerted Wolff rearrangement and nitrogen extrusion, most likely

35 In order to directly consider Wolff's observations, measurements were also made on two

36 edox design, on the basis of dehydrogenation/Wolff-Kishner (WK) reduction, to simultaneously tackle t

37 ular nodal reentrant tachycardia), excluding Wolff-Parkinson-White, comparing digoxin with propranolo

38 N,N-diethyl diazoamides) or almost exclusive Wolff rearrangement to ketenes (in the case of the cycli

39 s shown to be more effective in facilitating Wolff rearrangement than copper(I), although both are mo

40 utation (Arg302Gln) responsible for familial Wolff-Parkinson-White (WPW) syndrome.

41 979 and 1989 before undergoing operation for Wolff-Parkinson-White syndrome.

42 eactions, cyclopropanation, ylide formation, Wolff rearrangement, and cycloaddition reactions.

43 JG, Chen J, Holland NT, Barr DB, Perera FP, Wolff MS.

44 re was more neurology taught under Harold G. Wolff at Cornell University Medical College in New York

45 ine, or tetrahydrofurfuryl alcohol generates Wolff-rearranged, pyrrole ring-contracted azeteoporphyri

46 biochemical properties reminiscent of human Wolff-Parkinson-White syndrome, arising from mutations i

47 uperior alternatives to simple hydrazones in Wolff-Kishner-type reduction reactions, in the Barton vi

48 reatment of the clinical arrhythmias seen in Wolff-Parkinson-White syndrome.

49 Two-photon induced Wolff rearrangement of a terphenyl diazoketone 1 was ach

50 onding EP beta-lactams via an intramolecular Wolff rearrangement.

51 idt, Egon Macher, Gerd Steigleder, and Klaus Wolff.

52 rise to clean C-H insertion with only minor Wolff rearrangement to ketenes.

53 (1) In 6 cases of Wolff-Parkinson-White syndrome, ECM accurately identifie

54 he adjacent phenyl group, and no evidence of Wolff rearrangement.

55 pre-excitation, a characteristic feature of Wolff-Parkinson-White syndrome.

56 d to be responsible for an inherited form of Wolff-Parkinson-White syndrome.

57 The management of Wolff-Parkinson-White is based on the distinction betwee

58 ent retrospective and prospective studies of Wolff-Parkinson-White syndrome in asymptomatic pediatric

59 inception in the eighteenth century works of Wolff and Goethe, through the mid nineteenth century dis

60 some affected individuals (pre-excitation or Wolff-Parkinson-White syndrome).

61 s, hypothyroidism is related to a persistent Wolff-Chaikoff effect and often has a vague presentation

62 ults in an efficient (Phi(254) = 0.34) photo-Wolff reaction, while at 355 nm, the formation of diazir

63 hanism triggered by thermal or photochemical Wolff rearrangement of a diazo ketone.

64 becomes electron-rich, and the photoinduced Wolff rearrangement produces a highly emissive rhodol dy

65 c abnormalities, particularly preexcitation (Wolff-Parkinson-White syndrome) and atrioventricular con

66 ial flutter, atrioventricular nodal reentry, Wolff-Parkinson-White syndrome, and atrial tachycardia.

67 This method is inspired by the Semmler-Wolff reaction, a classic method that exhibits limited s

68 Among 8575 symptomatic Wolff-Parkinson-White patients with atrioventricular ree

69 f a key cyclobutane intermediate by a tandem Wolff rearrangement/asymmetric ketene addition, (2) a di

70 Subsequent tandem Wolff rearrangement/lactonization of these alpha-diazo i

71 ntal and computational results indicate that Wolff rearrangement of the diacetylcarbene occurs with a

72 nusual wavelength selectivity indicates that Wolff rearrangement and isomerization originate from dif

73 The Wolff-Parkinson-White registry was an 8-year prospective

74 The Wolff-Parkinson-White syndrome, with a prevalence in Wes

75 rine 2 leads to the loss of nitrogen and the Wolff rearrangement, apparently via a carbene intermedia

76 processes: loss of nitrogen followed by the Wolff rearrangement and isomerization into diazo compoun

77 fies this intermediate as resulting from the Wolff rearrangement of the diazochlorins upon N(2) loss.

78 3 from Family 1 and 8 from Family 2) had the Wolff-Parkinson-White syndrome.

79 H insertion product 6, while products of the Wolff rearrangement were not detected in both cases.

80 ions yields ketoester 3a, the product of the Wolff rearrangement, while products produced from the si

81 ures a previously unknown application of the Wolff-Kishner reduction of a nonresonance stabilized or

82 ought to characterize an animal model of the Wolff-Parkinson-White (WPW) syndrome to help elucidate t

83 The prognosis of the Wolff-Parkinson-White syndrome essentially depends on in

84 Under these conditions, formation of the Wolff-rearranged product is inhibited and the phenyl add

85 as accomplished by a new modification of the Wolff-Semmler rearrangement.

86 Laser power dependence studies show that the Wolff rearrangement is induced by two-photon absorption

87 Taken together, the results suggest that the Wolff rearrangement is subject to the same kind of nonst

88 calculations, allows us to conclude that the Wolff rearrangement of 1 is a concerted process.

89 owing that azeteoporphyrin formation via the Wolff rearrangement is dependent upon the structural dis

90 We identified two families in which the Wolff-Parkinson-White syndrome segregated as an autosoma

91 ement in most asymptomatic patients with the Wolff-Parkinson-White ECG pattern.

92 tegy in asymptomatic young patients with the Wolff-Parkinson-White electrocardiographic pattern are s

93 ) acetate leads to a remarkably facile "thia-Wolff rearrangement", producing thio-substituted ketenes

94 represents the lower limit to the barrier to Wolff rearrangement of the carbene.

95 in nascent carbenes results in the ultrafast Wolff rearrangement of the hot species.

96 n-poor core and, upon irradiation, undergoes Wolff rearrangement to give a ring-expanded xanthene cor

97 ischer carbene, which subsequently undergoes Wolff rearrangement and loss of CO.

98 igh yields from amino acids in two steps via Wolff rearrangement.

99 In adults with Wolff-Parkinson-White syndrome, a shortest RR interval <

100 R531G (AMPKgamma2(RG)), are associated with Wolff-Parkinson-White (WPW) syndrome, a cardiac disorder

101 eloped in adults also identify children with Wolff-Parkinson-White syndrome at risk for sudden death.

102 he largest reported to date of children with Wolff-Parkinson-White syndrome having a cardiac arrest,

103 w York, a teaching hospital of Cornell, with Wolff as my Director of Training.

104 for the asymptomatic pediatric patient with Wolff-Parkinson-White syndrome.

105 Fourteen pediatric patients with Wolff-Parkinson-White syndrome and no other congenital d

106 Symptomatic patients with Wolff-Parkinson-White syndrome generally have a good out

107 of detailed long-term data in patients with Wolff-Parkinson-White syndrome is limited, and no prospe

108 rom pacing or preexcitation in patients with Wolff-Parkinson-White syndrome).

109 luate its outcome in pediatric patients with Wolff-Parkinson-White syndrome.

110 ing ventricular arrhythmias in patients with Wolff-Parkinson-White syndrome.