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1 an be restored to almost 50% of wild-type by X irradiation.
2 m E. coli organisms subjected to both UV and X irradiation.
3 uced following early gestational exposure to x-irradiation.
4 l, respectively, before their co-exposure to X-irradiation.
5 res chronic administration and is blocked by x-irradiation.
6 g NES mutants exhibit reduced survival after X-irradiation.
7 chemotherapeutic agents, UV irradiation, and X-irradiation.
8 nd to colocalize spatially after UV- but not x-irradiation.
9 was blocked by ablation of neurogenesis with X-irradiation.
10 of the cell cycle and following exposure to X-irradiation.
11 ((60)Co) gamma-radiation or 600 cGy, 250 kVp x-irradiation.
12 ons had been produced using ethidium bromide/X-irradiation.
13 PSA, after infantile (PND 2-15) exposure to x-irradiation.
14 y exposed to O, 12, or 21-Gy single doses of x-irradiation.
15 e progenitor cells several generations after X-irradiation.
16 erved in normal syncytial embryos exposed to X-irradiation.
17 d by exposure of the spinal cord to 40 Gy of X-irradiation.
18 ming growth factor-beta1 protected them from x-irradiation.
19 ked in developing chick limb buds exposed to X-irradiation.
20 cellular components of the lesion site with x-irradiation.
22 viable cell than that observed after 0.5 Gy x-irradiation, a dose that causes the same level of toxi
24 results, showing substantial deficits after x-irradiation alone, encourage the hypothesis that hippo
25 elated with an increased hypersensitivity to X-irradiation and a DNA DSB repair deficit in synchroniz
26 hypoxia, both protect cells from killing by X-irradiation and are present in solid tumors but not in
27 Using the taiep rat and a combination of X-irradiation and cell transplantation, it has been poss
28 adult, or aged adult rat sciatic nerves into X-irradiation and ethidium bromide-induced demyelinated
29 of 128 WG-RH hybrid cell lines generated by X-irradiation and fusion has been tested for the retenti
31 e adult rat spinal cord were demyelinated by x-irradiation and intraspinal injections of ethidium bro
33 that this increase was a consequence of the X-irradiation and to highlight the significance of remye
34 s have been described for cells that survive x-irradiation, and suggest random genetic damage is a co
35 as arrested in mice using low-dose, targeted x-irradiation, and the effects of irradiation were assay
36 hippocampal lesions, and infant exposure to X-irradiation, and they add strongly to these earlier de
39 the cytotoxicity of the DNA damaging agents X-irradiation, cisplatin or bleomycin was p53-independen
40 luoxetine, and ablation of neurogenesis with x-irradiation completely blocked the effects of chronic
41 lity to rapidly phosphorylate HDM2 following X-irradiation, confirming a role for ATM in its phosphor
42 to three subsequent cell divisions following X-irradiation, confirming that X-rays induce cell death
43 ellular constituents at the lesion site with x-irradiation delivered within a critical time window af
45 proximodistal patterning is unaffected after X-irradiation does not support the predictions of the pr
48 constituted cells were transplanted into the X-irradiation/ethidium bromide lesioned dorsal columns o
50 s to 6-aminonicotinamide prior to 3- or 6-Gy x-irradiation induced a supra-additive cell kill, indica
52 etaphase/anaphase transition after extensive X-irradiation-induced DNA damage, whereas Grp/Chk1, but
55 s and lineage tracing in chick, we show that X-irradiation-induced phocomelia is fundamentally not a
56 ert culture dish method is used to show that X-irradiation induces medium-mediated bystander effects
57 on between radiation inducible TNF-alpha and X-irradiation occurs selectively within the tumor vessel
61 e technique of ionizing neutral compounds by X-irradiation of cryogenic Ar matrices to radicals embed
66 mbiguously distinguished DNA SSBs induced by X-irradiation of mouse leukemia L1210 cells from DNA DSB
67 We have reported in a separate article that x-irradiation of sectioned adult rat spinal cord resulte
68 by either total body X-irradiation (TBI) or X-irradiation of the chest (CI) with 62.5, 125, 250, or
73 d intervention in certain cellular events by x-irradiation prevents the onset of degeneration and thu
74 inhibited by exposing the brain to 40 Gy of X-irradiation prior to cuprizone intoxication and this r
75 to show that cells die at a fixed time after X-irradiation rather than from a specific cell cycle poi
77 istration of analog II prior to fractionated X-irradiation significantly diminished the number and de
78 ll cycle, and their differential response to X-irradiation suggest that they may perform different fu
79 g progenitors, followed by either total body X-irradiation (TBI) or X-irradiation of the chest (CI) w
81 l populations, we used low-dose, whole-brain x-irradiation to eliminate proliferating dentate granule
82 the effects of random DNA breaks induced by X-irradiation to site-specific I-CreI endonuclease-induc
84 is regulated by a variety of factors such as x-irradiation, ultraviolet radiation, steroids, growth f
87 mocytes, as well as their toxicities without X-irradiation, was successfully evaluated using this met
88 e if the occlusive effects of Ad.Egr-TNF and X-irradiation were specific for tumor vessels, non-tumor
89 asts sharply to the situation observed after x-irradiation, where peak levels of gamma-H2AX foci were
90 Da protein was not significantly affected by X-irradiation, while the abundance of the 73 and the 62
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