1 milliliter (range, 31 to 2335) in those with X-linked hypophosphatemia.

2 ated in those with oncogenic osteomalacia or X-linked hypophosphatemia, an immunometric assay was dev

3 opeptidases on the X chromosome (Phex) cause X-linked hypophosphatemia, characterized by rickets, ost

4 mutation homologous to that in patients with X-linked hypophosphatemia, have a 2-fold greater urinary

5 nant mutation of the normal gene Pex causing X-linked hypophosphatemia in the mouse.

6                                              X-linked hypophosphatemia is the most prevalent inherite

7 age, 43.0+/-13.3 years) and 21 patients with X-linked hypophosphatemia (mean age, 34.9+/-17.2 years)

8 ated in those with oncogenic osteomalacia or X-linked hypophosphatemia, suggesting that this growth f

9 xpress FGF-23 messenger RNA, and to those in X-linked hypophosphatemia, which is caused by inactivati

10 ophosphatemia and impaired mineralization in X-linked hypophosphatemia (XLH) and its mouse homologue,

11                                Patients with X-linked hypophosphatemia (XLH) and the hyp-mouse, a mod

12         Inactivating mutations of Phex cause X-linked hypophosphatemia (XLH) by increasing levels of

13 ed in mineral balance that are implicated in X-linked hypophosphatemia (XLH) in humans, its murine ho

14                                              X-linked hypophosphatemia (XLH) is a skeletal disease ca

15                                              X-linked hypophosphatemia (XLH) is an X-linked dominant

16                                              X-linked hypophosphatemia (XLH) is the most common herit

17                                              X-linked hypophosphatemia (XLH) is the most frequent for

18                                              X-linked hypophosphatemia (XLH), a disorder characterize