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1 d the antiapoptotic factors Bcl-xL and XIAP (X-linked Inhibitor of Apoptosis Protein).
2 IR3 (baculovirus IAP repeat) domain of XIAP (X-linked inhibitor of apoptosis protein).
3 aspase activation by proteasome activity and X-linked inhibitor of apoptosis protein.
4 promoter of PDA chemotherapy resistance, the X-linked inhibitor of apoptosis protein.
5 include inhibitors of apoptosis proteins and X-linked inhibitor of apoptosis protein.
6 radation, or anti-apoptotic factors, such as x-linked inhibitor of apoptosis protein.
7 tivation by facilitating association between X-linked inhibitor of apoptosis protein, an inhibitor of
8 cIAP-1, cIAP-2, Bcl-2, Bcl-xL, survivin, and X-linked inhibitor of apoptosis protein, and up-regulate
9 caspase) mimetics to their target, the XIAP (X-linked inhibitor of apoptosis) protein, and their bind
10 CrmA), a suppressor of caspase-8, but not by X-linked inhibitor of apoptosis protein-baculovirus IAP
11 (L), or deletion of Smac) with expression of X-linked inhibitor of apoptosis protein decreases the se
12 t with ucf-101 before reperfusion attenuated X-linked inhibitor of apoptosis protein degradation and
13 ation, apoptosis-inducing factor, as well as X-linked inhibitor of apoptosis protein degradation, but
14 IL-6 increases expression of the endogenous X-linked inhibitor of apoptosis protein expression by tr
16 findings that IL-6 translationally regulates X-linked inhibitor of apoptosis protein expression revea
17 sine kinase localization at the membrane and X-linked inhibitor of apoptosis protein expression, resp
18 ellular inhibitor of apoptosis protein-1 and X-linked inhibitor of apoptosis protein, Fas-induced apo
19 c-Myc, matrix metalloproteinase-9, survivin, X-linked inhibitor-of-apoptosis protein (IAP), IAP1, Bcl
20 combinant adenovirus was used to express the X-linked inhibitor of apoptosis protein in these cells.
22 rtantly, GM-CSF prevented GC-induced loss of X-linked inhibitor of apoptosis protein, late activation
23 1 (copper metabolism MURR1 domain) and XIAP (X-linked inhibitor of apoptosis protein) may also be req
24 is a mitochondrial protein that binds XIAP (X-linked inhibitor of apoptosis protein) upon entering t
25 osis (IAP) family members (cIAP1, cIAP2, and X-linked inhibitor of apoptosis protein), was overexpres
26 the antiapoptotic proteins bcl-2, mcl-1, and X-linked inhibitor of apoptosis protein which occurs as
27 pase-3 and significantly increased levels of X-linked inhibitor of apoptosis protein, which also corr
28 3/1524A), the inhibitory interaction between X-linked inhibitor of apoptosis protein (XIAP) and caspa
29 d found to bind to the antiapoptotic protein X-linked inhibitor of apoptosis protein (XIAP) and showe
31 gned to mimic the binding of Smac protein to X-linked inhibitor of apoptosis protein (XIAP) are being
34 and AKT2, interacts with and phosphorylates X-linked inhibitor of apoptosis protein (XIAP) at residu
35 s may be due to the high-level expression of X-linked inhibitor of apoptosis protein (XIAP) by CLL ce
36 mk or by expressing the BIR1/BIR2 domains of X-linked inhibitor of apoptosis protein (XIAP) decreased
37 s involved down-regulation of active AKT and X-linked inhibitor of apoptosis protein (XIAP) expressio
38 al. report on the prognostic significance of X-linked inhibitor of apoptosis protein (XIAP) in melano
39 e prognostic and therapeutic significance of X-linked inhibitor of apoptosis protein (XIAP) in melano
42 bition of activated CASP7 (p19/p12-CASP7) by X-linked inhibitor of apoptosis protein (XIAP) is a pote
50 as well as cells overexpressing full-length X-linked inhibitor of apoptosis protein (XIAP) or the BI
51 lls stably overexpressing either full-length X-linked inhibitor of apoptosis protein (XIAP) or the BI
53 TNFalpha-dependent apoptosis induction, and X-linked inhibitor of apoptosis protein (XIAP) plays a c
54 ptibility to apoptosis and determines if the X-linked inhibitor of apoptosis protein (XIAP) regulates
55 monstrate that the E3 ubiquitin ligase named x-linked inhibitor of apoptosis protein (XIAP) restricts
56 along with negative regulation of NF-kappaB- X-linked inhibitor of apoptosis protein (XIAP) signaling
57 ytosol, which activate apoptosis and inhibit X-linked inhibitor of apoptosis protein (XIAP) suppressi
61 om mitochondria and subsequent inhibition of X-linked inhibitor of apoptosis protein (XIAP) was requi
63 sely, biochemical interactions occur between x-linked inhibitor of apoptosis protein (XIAP), an E3 ub
64 B-cell leukemia/lymphoma-2 protein (Bcl-2), X-linked inhibitor of apoptosis protein (XIAP), and myel
65 intracellular SLAM-associated protein (SAP), X-linked inhibitor of apoptosis protein (XIAP), and perf
66 of apoptosis proteins, including c-IAP1 and X-linked inhibitor of apoptosis protein (XIAP), are obli
68 a number of antiapoptotic proteins including X-linked inhibitor of apoptosis protein (XIAP), c-IAP1,
69 ral NF-kappaB-dependent antiapoptotic genes: X-linked inhibitor of apoptosis protein (XIAP), cIAP1, c
70 revealed a strong decrease of expression of X-linked inhibitor of apoptosis protein (XIAP), compared
71 Overexpression of the apoptotic regulator, X-linked inhibitor of apoptosis protein (XIAP), has been
73 his study, we found that CSR1 interacts with X-linked Inhibitor of Apoptosis Protein (XIAP), using ye
74 s, antiapoptotic proteins, such as Mcl-1 and X-linked inhibitor of apoptosis protein (XIAP), were rap
75 e-2 (XLP-2) is associated with deficiency in X-linked inhibitor of apoptosis protein (XIAP), with poo
76 ow that resveratrol induced p53-independent, X-linked inhibitor of apoptosis protein (XIAP)-mediated
77 lines LoVo and HCT-116 Smac(-/-), similar to X-linked inhibitor of apoptosis protein (XIAP)-overexpre
84 spase-8 and caspase-3 activation and reduced X-linked inhibitor-of-apoptosis protein (XIAP) levels in
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