ライフサイエンスコーパス: Zika virus infection

1 imaging findings associated with congenital Zika virus infection.

2 ine the association between microcephaly and Zika virus infection.

3 ay be the only initial finding in congenital Zika virus infection.

4 this neuronal population as a consequence of Zika virus infection.

5 ociation between microcephaly and congenital Zika virus infection.

6 studies supported a causal association with Zika virus infection.

7 resumed or laboratory-confirmed intrauterine Zika virus infection.

8 il, which was later attributed to congenital Zika virus infection.

9 rocephaly epidemic is a result of congenital Zika virus infection.

10 on the incidence and prevalence of prenatal Zika virus infection.

11 ogic correlation in a patient with the acute Zika virus infection.

12 of neurological symptoms, suggesting recent Zika virus infection.

13 nd two spontaneous abortions associated with Zika virus infection.

14 boratory tests and no controls had confirmed Zika virus infections.

15 nital infections or are unique to congenital Zika virus infection: (1) severe microcephaly with parti

16 (95% CI 13.0-infinity) for microcephaly and Zika virus infection after adjustments.

17 id and systematic review about links between Zika virus infection and (a) congenital brain abnormalit

18 Due to associations between Zika virus infection and a range of fetal maladies(1,2),

19 tion of available evidence from outbreaks of Zika virus infection and clusters of microcephaly is tha

20 gs provide strong evidence of a link between Zika virus infection and different congenital central ne

21 ned questions about the relationship between Zika virus infection and each of the two clinical outcom

22 ing the spread of emerging diseases, such as Zika virus infection and Ebola virus disease.

23 ded both for appraisal of the evidence about Zika virus infection and for the next public health thre

24 tion of available evidence from outbreaks of Zika virus infection and GBS is that Zika virus infectio

25 nfant laboratory evidence of possible recent Zika virus infection and outcomes reported in the contin

26 The ocular manifestations and sequelae of Zika virus infection are not well known.

27 imaging findings associated with congenital Zika virus infection as found in the Instituto de Pesqui

28 trated to aid the radiologist in identifying Zika virus infection at imaging.

29 with laboratory evidence of possible recent Zika virus infection, birth defects potentially related

30 ts with microcephaly or laboratory-confirmed Zika virus infection but not for all infants potentially

31 or infants with birth defects after maternal Zika virus infection by trimester of infection and mater

32 Zika virus infection can be prenatally passed from a pre

33 irst in vivo evidence that prior exposure to Zika virus infection can enhance Dengue infection, which

34 nt of ZIKV-3'UTR-LAV is warranted for humans.Zika virus infection can result in congenital disorders

35 is is the first study providing evidence for Zika virus infection causing Guillain-Barre syndrome.

36 24 (80%) of 30 mothers of cases had Zika virus infection compared with 39 (64%) of 61 mother

37 Zika virus infection could also cause Guillain-Barre syn

38 none of 62 controls had laboratory-confirmed Zika virus infection; crude overall OR 55.5 (95% CI 8.6-

39 Zika virus infection during pregnancy can cause congenit

40 fection and clusters of microcephaly is that Zika virus infection during pregnancy is a cause of cong

41 ng the risk of birth defects associated with Zika virus infection during pregnancy may help guide com

42 Laboratory-confirmed Zika virus infection during pregnancy was defined as det

43 aneiro, Brazil, who presented with suspected Zika virus infection during pregnancy were referred to o

44 hers had polymerase chain reaction-confirmed Zika virus infection during pregnancy.

45 ing the association between microcephaly and Zika virus infection during pregnancy.

46 lete protection, which suggests that primary Zika virus infection elicits protective immunity.

47 Congenital Zika virus infection has stimulated great international

48 tiveness of this policy in reducing prenatal Zika virus infection has yet to be quantified.

49 Definite cases had laboratory evidence of Zika virus infection; highly probable cases presented sp

50 Laboratory evidence of possible recent Zika virus infection in a maternal, placental, fetal, or

51 d to epidemiologic data from 2015 to 2016 on Zika virus infection in Colombia.

52 e included, as well as those with documented Zika virus infection in fluid or tissue (n = 17, confirm

53 112 with polymerase chain reaction-confirmed Zika virus infection in maternal specimens, 24 infants (

54 Exposure: Zika virus infection in pregnant women.

55 Background: Because of the risk for Zika virus infection in the Americas and the links betwe

56 The control of primary Zika virus infection in the blood was correlated with ra

57 ctrum of findings associated with congenital Zika virus infection in the IPESQ in northeastern Brazil

58 ers would be useful for medical diagnosis of Zika virus infection in various aptamer-based diagnostic

59 large populations at risk of mosquito-borne Zika virus infection include India (67 422 travellers ar

60 Zika virus infection induced detectable Dengue cross-rea

61 cient evidence to conclude that intrauterine Zika virus infection is a cause of microcephaly and seri

62 eaks of Zika virus infection and GBS is that Zika virus infection is a trigger of GBS.

63 ns from individuals with a typical course of Zika virus infection is low.Zika virus (ZIKV) is present

64 ective, and rapidly scalable vaccine against Zika virus infection is needed.

65 m of adverse reproductive outcomes caused by Zika virus infection is not yet determined, a distinctiv

66 months, the cumulative incidence of prenatal Zika virus infections is likely to decrease by 17% to 44

67 The public health implications of Zika virus infection lend special importance to identify

68 cross-reactive antibodies induced following Zika virus infection might enhance subsequent Dengue inf

69 fetuses or infants with prenatal exposure to Zika virus infection only in the second or third trimest

70 ective, and durable protection against human Zika virus infections or syndromes.

71 potential causes of microcephaly: congenital Zika virus infection, vaccines, and larvicides.

72 and the risk of microcephaly associated with Zika virus infection was 95 cases (34-191) per 10,000 wo

73 In 32 (35%) cases, congenital Zika virus infection was confirmed by laboratory tests a

74 ociation between microcephaly and congenital Zika virus infection was confirmed.

75 ed (n = 17) and presumed (n = 28) congenital Zika virus infections were similar, with ventriculomegal

76 ss of mucocutaneous findings associated with Zika virus infection will support its early recognition

77 or those with brain findings suspicious for Zika virus infection, with intracranial calcifications (