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1       N-Terminal acetylome analyses revealed a decreased acetylation of a subset of NatA and NatE sub
2 el of DMD, mdx, dystrophin deficiency causes a decrease and mislocalization of muscle-specific neuron
3 ses emerged, with some variation by antigen: a decrease at 6 months (IFN-gamma and IL-5) or at both 6
4 response increase at the activation site but a decrease at nearby locations (center-surround modulati
5 ng reliance on mitochondrial respiration and a decreased dependence on glycolysis for cellular energe
6 cantly modify the film morphology but causes a decreased electronic coupling, the formation of a char
7  Warburg effect wherein cancer cells oxidize a decreased fraction of the pyruvate generated from glyc
8 odies generated against PhtD and PcpA caused a decrease in adherence to A549 cells (P < 0.05).
9 lyses of RARdn-expressing pancreata revealed a decrease in beta-cell mass and insulin per beta-cell 1
10 stent with the protective mechanism in which a decrease in CCR5 molecules on CD4(+) T cells decreases
11 in some genes caused an increase, and others a decrease in conjugation efficiency.
12                                              A decrease in efficiency and fidelity of these pathways
13                            Here we show that a decrease in ephrinB2 protein causes neural tube closur
14                                         Such a decrease in ephrinB2 protein levels is observed on the
15 quiring evolutionary steps that would entail a decrease in fitness.
16 mprovement in glycaemic control-specified as a decrease in glycosylated haemoglobin A1C (HbA1c) of >/
17  ADP-ribosylating activity, ES cells exhibit a decrease in ground state pluripotency as they cannot m
18                             Individuals with a decrease in HbA1c had a much greater risk of developin
19 eatment-induced neuropathy in diabetes, with a decrease in HbA1c of >4% points over 3 months the abso
20                                         With a decrease in HbA1c of 2-3% points over 3 months there w
21 icant disruption of the platform and, hence, a decrease in HCV genome replication.
22  micrococcal nuclease (MNase) digestion, and a decrease in histones at silent ES promoters but not te
23 no-6-O-sulfated disaccharides accompanied by a decrease in iduronic acid 2-O-sulfated disaccharide st
24 totelangiectatic rosacea subjects who showed a decrease in matrix metalloproteinase activity (P<0.05)
25                          Following training, a decrease in MyHC hybrid type IIa/IIx fibre frequency o
26                  The lesion also resulted in a decrease in nucleolar volume that was similar in all t
27 crobiota, and this shift was associated with a decrease in Paneth-cell-mediated antimicrobial host de
28 d the PI3K signaling pathway and resulted in a decrease in phosphocholine (PC), total choline (tCho)
29                      This is concurrent with a decrease in phosphorylation of signaling targets of mT
30 ation expansion, whereas IYSVother displayed a decrease in population size.
31 s and proliferation in the maternal decidua, a decrease in proliferation and increase in apoptosis in
32 ariation of productivity in combination with a decrease in species asynchrony in diverse communities.
33 d improvement with the effect solely through a decrease in test amplitude.
34 e protection against disease correlated with a decrease in Th1 cells that produced the cytokines IFN-
35 e translocation rate which is accompanied by a decrease in the ATP hydrolysis rate, such that the cou
36 lones from 0.93% to 83.22%, corresponding to a decrease in the error frequency of synthetic gene from
37                   Suppression of NF90 caused a decrease in the half-life of cyclin E1 mRNA, which was
38          This phenomenon was associated with a decrease in the number of cell colonies capable of gro
39  RNA lariat debranching enzyme (DBR1) led to a decrease in the production of HIV-1 cDNA.
40                         This could result in a decrease in the protein folding capacity of old hearts
41 ially disables the septal junctions leads to a decrease in the range of a signal dependent on the Het
42 uantal size, which occurs concomitantly with a decrease in the size of synaptic vesicles.
43  in a more severe phenotype characterized by a decrease in valves, failure of Prox1(high) cells to cl
44                 This was largely a result of a decreased likelihood of the channel opening and remain
45 ticipated, a reduced calpain activity led to a decreased number of alphaSyn-positive aggregates, wher
46 AT/enhancer-binding protein-beta, leading to a decreased production of interleukin-6 (IL-6) and low e
47 ular and cellular phenotypes associated with a decrease rather than overactivation of Wnt signaling.
48 d with successive passages, corresponding to a decreased replication in primary swine macrophages cul
49 of transcript levels at 18 and 24 h p.i. but a decrease thereafter.
50 aevis oocytes and human HEK293T cells showed a decreased threshold of activation and delayed deactiva

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