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1              It seems to be driven mainly by a delay in achieving viral suppression.
2  exposure to pathway agonists, hESCs exhibit a delay in activation of beta-catenin signaling, which l
3 ranged from 3-fold to 30-fold and was due to a delay in activation of early sporulation genes.
4 vation upon adenoviral infection, leading to a delay in adenoviral clearance in vivo.
5 xpression of PtAIL1 expression, which led to a delay in adventitious root formation.
6 FAS possibly due to the higher flow rate and a delay in aerobic bacterial degradation.
7  murine GC-B cells results in larger GCs and a delay in affinity maturation, demonstrating the import
8 minant-negative ATO1(G53D) allele results in a delay in alkalinization, a defect in hyphal formation,
9 mic control post-transplant and demonstrated a delay in allograft rejection.
10 oesin clearance from cell poles at anaphase, a delay in anaphase elongation, together with defects in
11 ably, we find that pk2 knockdown larvae show a delay in anterograde transport.
12 ouse embryo is significantly extended due to a delay in APC/C activation.
13 of B6-MRL Fas lpr/j-RAGE(-/-) mice exhibited a delay in apoptosis and expressed significantly less ac
14                                              A delay in application of the label of multiple myeloma
15 duced overexpression of HO-1 in cells led to a delay in autophagy progression, generated significantl
16 ults in exuberant VSN axonal projections and a delay in axonal coalescence into well defined glomerul
17 beta-catenin loss-of-function mutations show a delay in axonal sorting; conversely, gain-of-function
18                     The photopic ERGs showed a delay in b-wave time to peak, but the photopic hill, i
19 e phenotype, and neither genotypes exhibited a delay in behavioural timing in responses to dLAN.
20      Cells without centrioles exhibited both a delay in bipolar spindle assembly and a high rate of c
21 r ALAT2 caused unexpected phenotypes such as a delay in blood digestion, a massive accumulation of ur
22 entirely unclear how a lack of Ano6 leads to a delay in bone mineralization by osteoblasts.
23 lysis, with virion-associated CD55 providing a delay in both aggregation and lysis more substantial t
24 pared to diploid embryos, haploids exhibited a delay in both zygotic gene expression and cell cycle l
25 poptotic nuclear morphology correlating with a delay in caspase-3 activation.
26 PC maturation in the vaginal mucosa leads to a delay in CD8 T cell activation in the draining lymph n
27            This phenotype is associated with a delay in cell cycle progression and ectopic DNA elemen
28  mediates reversion of T(EM) into T(CM), via a delay in cell cycle progression at the G2/M stage.
29 hese mice were miRNA-deficient and exhibited a delay in cell cycle progression involving the G(1) to
30  the precocious inhibition of mitosis due to a delay in cell cycle progression.
31 nificant reduction in cell proliferation and a delay in cell cycle progression.
32 eplication terminus bound Noc-YFP and caused a delay in cell division.
33 binase-mediated inactivation of Hdac3 led to a delay in cell-cycle progression, cell-cycle-dependent
34 e had increased basal cell proliferation and a delay in cellular maturation; these mice developed epi
35  accumulation of Aurora A at centrosomes and a delay in centrosome separation.
36 e mechanism underlying these observations is a delay in channel opening after application of protons,
37 at cells without functional centrosomes have a delay in chromosome congression and anaphase onset, wh
38 e delay in fruit ripening is associated with a delay in climacteric respiration and reduced synthesis
39 n the alpha tocopherol group translates into a delay in clinical progression of 19% per year compared
40            A multidomain intervention showed a delay in cognitive decline (low-strength evidence).
41     Furthermore, low-dose combination caused a delay in colonic adenocarcinoma progression.
42 ults in reduction of the fracture callus and a delay in conversion of cartilage to bone.
43              The absence of SleB also causes a delay in cortex fragment release.
44                  These findings may indicate a delay in cortical maturation in several brain areas im
45 ing proteins at the CR, which in turn led to a delay in CR formation and sensitivity to other perturb
46 ic division and failure in cytokinesis, with a delay in daughter cell abscission revealed by a fluore
47 red patients were least likely to experience a delay in definitive surgery, followed by Medicare and
48 tions were acquired prenatally and linked to a delay in dentate gyrus maturation in the ventral hippo
49 + complement-mediated cell lysis resulted in a delay in development of MBP cells and myelination.
50 , Caspase-3 and Caspase-6-knockout mice show a delay in developmental pruning of retinocollicular axo
51 oms and multiple hospitalizations leading to a delay in diagnosis caused by incomplete initial workup
52         Results Tumor stage did not indicate a delay in diagnosis of breast cancer in women previousl
53 scribed a problem with medical care, such as a delay in diagnosis or treatment; 47% described a commu
54 tation in this age group sometimes can cause a delay in diagnosis.
55 d found that mutant mice had smaller testes, a delay in differentiation of pre-meiotic germ cells, de
56 eed, miR-223(-/-) eosinophil progenitors had a delay in differentiation.
57                       SATB1 knockdown caused a delay in DNA repair following exposure to H2O2, an inc
58 o show that the absence of seipin results in a delay in droplet appearance with concomitant accumulat
59 A to attenuate p54(nrb) expression exhibited a delay in DSB repair in a gamma-H2AX focus assay.
60            Global deletion of Twsg1 leads to a delay in ductal elongation, reduced secondary branchin
61 IE1-p72 and IE2-p86 proteins correlated with a delay in early and late viral gene expression and move
62  in Rab7-positive late endosomes, suggesting a delay in early-to-late endosome maturation.
63                           The results showed a delay in elongation and disorganization of the HERS in
64 ndosperm development that are accompanied by a delay in embryo development followed by embryo arrest
65                                     However, a delay in EMG induction was observed, which became more
66                      The loss of Pan1 caused a delay in endocytic progression and weakened connection
67 ay early developmental phenotypes, including a delay in epiboly, depleted S1P levels, elevated levels
68 n with an "eyes open at birth" defect due to a delay in epithelial sheet extension.
69                          Coated plums showed a delay in ethylene production and respiration rate at 2
70      We show this cell death correlates with a delay in expression of Fgf8 in branchial arch ectoderm
71 lay increased sensitivity to mitomycin C and a delay in FANCD2 foci formation compared with their wil
72 gat2(-/-) mice, Mogat2(IKO) mice also showed a delay in fat absorption, a decrease in food intake, an
73 Col12a1 gene showed decreased grip strength, a delay in fiber-type transition and a deficiency in pas
74 season length were associated primarily with a delay in first frost of the fall season and lengthenin
75 s in unmanipulated plots showed no change or a delay in flowering over the 21-year period, despite mo
76 d lines and show that tropical lines exhibit a delay in flowering transition of more than 3 weeks und
77 ic Arabidopsis plants led to reduced growth, a delay in flowering, and strongly attenuated senescence
78  plants, noninductive photoperiods result in a delay in flowering, but such plants eventually flower,
79 collagen in affected horse fibroblasts shows a delay in folding and secretion and a decrease in hydro
80  the first hair follicles is responsible for a delay in follicular destruction, which results in lowe
81 he Ras-related protein R-Ras2/TC21 displayed a delay in formation of neurofibromas but an acceleratio
82                                              A delay in functional maturity of repopulating HLA-speci
83                                              A delay in G(1) to S phase of cell cycle was also seen t
84 roblasts led to a proliferative block due to a delay in G(1)-S cell cycle progression; this defect wa
85  many different aneuploid strains exhibiting a delay in G1, a cell cycle stage governed by extracellu
86 ks Tom1-mediated turnover of Dia2 and causes a delay in G1-to-S phase progression.
87 olonged repression of housekeeping genes and a delay in gene activation at inducible loci.
88 er progenitor proliferation rate, along with a delay in gliogenesis, is also observed in Gdf11(-/-) s
89 utamate receptor subunit 1 (GluA1), there is a delay in GluA1 increase in the trained hemisphere of t
90                      MeJA application led to a delay in grape technological maturity and a significan
91 es, hmp and nor hmp mutants both experienced a delay in growth initiation, whereas the nor mutant's a
92 less acm deletion mutant and did not observe a delay in growth.
93 stocyst cultures, Hinfp-null embryos exhibit a delay in hatching, abnormal growth, and loss of histon
94 mall fiber neuropathy and is associated with a delay in healing of shallow, but not deep wounds.
95 paired erythroid maturation characterized by a delay in hemoglobin accumulation, larger mean cell are
96 ificantly increased hepatocyte apoptosis and a delay in hepatocyte regeneration after injury, which w
97                 Deletion of AIM44 results in a delay in Hof1p phosphorylation and altered Hof1p local
98 ermined that mild disease is associated with a delay in host expression of genes linked to virulence,
99                       Here, we test for such a delay in impact by relating numbers of threatened spec
100 e show that at CB1 1, 10, 13, and 18 display a delay in inhibiting CP55,940-mediated cAMP inhibition,
101 er, larger droplets that are consistent with a delay in initiation but are otherwise normal in morpho
102 s waiting for laboratory tests and therefore a delay in initiation of chemotherapy.
103 ctive recovery from hematological stress and a delay in initiation of HSPC proliferation.
104 susceptibility in diabetic mice results from a delay in innate immune response to inhaled Mycobacteri
105 ntly different in GF and SPF mice, there was a delay in intestinal epithelial repair to DSS-induced i
106      Stat5-expressing mammary glands exhibit a delay in involution despite induction of proapoptotic
107 ts of the cholinergic-evoked inhibition, and a delay in IPSC latency.
108 itude of the apical Ca(2+) signal and caused a delay in its initiation.
109            The observed hyperplasia involved a delay in keratinocyte differentiation toward a more un
110 Zmers1b or Zmetr2b were larger and exhibited a delay in leaf senescence characteristic of ethylene in
111 n the Barnes maze test, mSOD1 mice displayed a delay in learning, outperformed wild-type mice during
112  that myeloid-specific Foxm1 deletion caused a delay in liver repair.
113  with cyclin A2-deficient hepatocytes, where a delay in liver tumor formation was observed.
114 inatal hypoxia alters development is through a delay in maturation of affected cell types, including
115 els, less severe histologic alterations, and a delay in mean time to death, compared with MARV-Ang in
116                                              A delay in mesophyll differentiation apparent both in th
117 aptor protein complex 2 binding site, caused a delay in MHC I recycling to the plasma membrane withou
118 grouped PV-KO and wild-type mice, suggesting a delay in microglial activation when PV is absent from
119                                This requires a delay in mitosis that is mediated by the spindle assem
120                Shp2-depleted cells exhibited a delay in mitotic entry and an earlier mitotic exit.
121 nation revealed that these filaments exhibit a delay in mitotic exit mediated by the checkpoint prote
122 cts in spindle assembly, pole splitting, and a delay in mitotic progression.
123 e marrow microenvironmental cell can lead to a delay in MM tumor progression.
124 wn Sema5A, which resulted in two phenotypes: a delay in motor axon extension into the ventral myotome
125 h(-/-);Gli3(-/-) mutant spinal cord exhibits a delay in motor neuron differentiation and an accumulat
126 (lox/lox) mice heterozygous for Agr2 exhibit a delay in mPanIN initiation and progression to PDAC.
127 uch that loss of integrin signaling leads to a delay in myelination of small-diameter axons.
128 elopmental pattern seen in wild type matches a delay in myelination of the superficial tracts of the
129 ic D2 inactivation (ALB-D2KO) is followed by a delay in neonatal expression of key lipid-related gene
130            One SD below the mean established a delay in neurodevelopment (score <85).
131 a reduced postmitotic layer, consistent with a delay in neurogenesis.
132 he intermediate forms of SMA presenting with a delay in neuromuscular junction maturation and a decre
133 ants lacking only 2-O-methylfucose exhibited a delay in nodule development during symbiosis.
134           Furthermore, A2BAR KO mice display a delay in normal fracture physiology with lower express
135                   Trailing chromatids induce a delay in nuclear envelope reassembly concomitant with
136 gs from observational studies have suggested a delay in nursing home placement with dementia drug tre
137 e association between nutritional status and a delay in nutrition initiation was independent of poten
138 ns in pacman result in small imaginal discs, a delay in onset of pupariation and lethality during the
139 POPs measurements (to our knowledge) reveals a delay in onset with higher concentrations.
140 aused by mating in blood-fed females, causes a delay in oocyte development, and impairs the function
141 ype, germinating spores without CwlJ1 suffer a delay in optical density loss and cortex peptidoglycan
142 losure of the palatal shelves accompanied by a delay in ossification along the fusion area of seconda
143 tly reduced RANKL expression, accounting for a delay in osteoclast formation.
144 ficiency than wild-type spores, and they had a delay in outgrowth.
145 ults in a more reduced Mia40 redox state and a delay in oxidative folding and mitochondrial import of
146  is delayed by 48 hours; this corresponds to a delay in p38MAPK activation.
147 B4049/emb30-1) (gnom(B)(/E)) mutant revealed a delay in papilla formation and reduced penetration res
148  Resistance in CD73 knockout mice was due to a delay in parasite differentiation in the central nervo
149 r regrowth in hepFXR-KO mice was unaffected, a delay in peak hepatocyte proliferation from day 2 to d
150                (Epid)CaR(-/-) mice exhibited a delay in permeability barrier formation during embryon
151 ffects may depend on age at vaccination, and a delay in pertussis vaccination has been linked to redu
152                   Lrp4 mutant mice displayed a delay in placode initiation and changes in distributio
153                           Our results showed a delay in plant senescence with an increase in the expr
154 e lacking the S1P receptor S1PR2 also showed a delay in plasma histamine clearance and a poor recover
155 dothelial cell (EC) migration contributes to a delay in postnatal development of the retinal vasculat
156 When CXCR4 is lacking, KikGR(+) B cells show a delay in PP egress.
157 ease or worse at diagnosis), likely owing to a delay in presentation, nonspecific presenting symptoms
158 s showed that corpora allata ablation caused a delay in production of the major female-specific sex p
159 phase III studies and phase II studies where a delay in progression is expected in the absence of rad
160 assembled with the mutant molecules revealed a delay in prothrombin activation with persistence of me
161  extension reduced lytic activity and led to a delay in rapid egress, but did not significantly decre
162                     Absence of DraRnl elicts a delay in reconstitution of the 10 kGy IR-shattered D.
163 nsuspected and likely harmful consequence of a delay in recovering from acidification in boreal lakes
164 ficant increase in hepatocellular injury and a delay in recovery compared to control-treated mice.
165                    Both mutations also cause a delay in recovery following bleaching.
166 epcidin rapidly after hemorrhage and exhibit a delay in recovery from blood loss.
167 ation resulted in increased facilitation and a delay in recovery from synaptic depression, indicating
168      Interestingly, APE2-deficient mice show a delay in recovery of B lymphocyte progenitors followin
169      When B cells return to the MZ, there is a delay in recovery of SIGN-R1-expressing macrophages.
170       Analysis of muscle regeneration showed a delay in recovery, probably as a result of decreased a
171                     All 4 of these cases had a delay in referral for surgical intervention.
172 clear activation, findings that characterize a delay in regenerative reprogramming.
173 creases CCL2-induced chemotaxis and mediates a delay in reinsertion of the CCL2 receptor, CCR2, into
174 nt of reovirus transit to late endosomes and a delay in reovirus disassembly.
175                             We also observed a delay in reprogramming of the maternal allele of the i
176                Here, we directly demonstrate a delay in restriction endonuclease synthesis after tran
177 ectopic Foxp3 expression and correlated with a delay in retinoic acid-related orphan receptor gammat
178 ward their targets is impaired, resulting in a delay in RGC axons reaching the dorsal thalamus compar
179                             First, there was a delay in rod recovery that has remained relatively con
180 omatin association of CDC6 and cyclin E, and a delay in S phase entry.
181 n chromatin condensation is not secondary to a delay in S-phase completion.
182                           In S phases 11-13, a delay in satellite replication emerged in sync with mo
183 e absence of p38 activity is directly due to a delay in satisfying the mitotic checkpoint.
184                 A deficiency of SIRT1 led to a delay in SC activation that could also be partially re
185 creased plasmodesmal permeability and led to a delay in senescence and flowering time.
186                      We recently showed that a delay in sigma(E) activation resulted in the novel phe
187 nctional relevance as Tlr3-/- mice displayed a delay in skin barrier repair following UVB damage.
188 onditional ablation of AP-2 gamma results in a delay in skin development and abnormal expression of p
189 ickness skin wounds to beryllium only causes a delay in skin regeneration.
190                                              A delay in SMC differentiation was observed in Six1(-/-)
191          In the Fmr1 knockout mouse, we find a delay in somatosensory map formation, alterations in t
192 ed levels of spore-specific gray pigment and a delay in spore formation.
193 fect infectious particle production, causing a delay in spread to presynaptic neurons and amplificati
194 e-free and particle-modified surfaces reveal a delay in spreading rate after an elapsed time of about
195                                     Finally, a delay in stomatal conductance recovery during the peri
196 tting of suspected macular holes may lead to a delay in surgical treatment, with attendant worse anat
197 nction, as suggested by echocardiography and a delay in switching off the slow skeletal troponin I.
198 in-1 gene have hair bundle dysmorphology and a delay in synapse maturation.
199 n of synapse elimination was associated with a delay in synapse maturation.
200                            At the human NMJ, a delay in synaptic maturation and an altered maintenanc
201 e retinas in postnatal development indicated a delay in synaptogenesis in Slitrk6-deficient animals.
202 period of T cell proliferation, resulting in a delay in T cell contraction.
203              This elimination process caused a delay in TEB outgrowth, after which the gland develope
204  approximately 20-hour lag, concomitant with a delay in the activation of a pan-neuronal differentiat
205 The loss of endothelial IRs also resulted in a delay in the acute hypoglycemic effect of systemic ins
206 s of the liver because DeltaEGFR mice showed a delay in the appearance of diethyl-nitrosamine-induced
207 nregulated VEGFR expression, which may cause a delay in the bone repair/remodeling process.
208 to approximately 50% of normal levels causes a delay in the cell cycle and accumulation of cells in e
209                                              A delay in the completion of metaphase induces a stress
210      Finally, we demonstrated that there was a delay in the development and severity of inflammation
211   Furthermore, mice lacking CX3CR1 exhibited a delay in the development of allodynia following VCR ad
212  junction barrier formation, as indicated by a delay in the development of peak transepithelial elect
213                   These results thus suggest a delay in the differentiation process of Klhl6-deficien
214                                              A delay in the DOC mobilization became apparent as the a
215 inding of the virus to the cell, we detected a delay in the entry and subsequent delivery of virion D
216 diminished KGF expression in GFs, leading to a delay in the epithelial wound-healing process that was
217 e epitopes, and that this is associated with a delay in the epitope-specific CD8(+) T cells respondin
218         These phenotypes are correlated with a delay in the eviction of nucleosomes surrounding the D
219        Furthermore, loss of Aplnr results in a delay in the expression of the cardiogenic transcripti
220                                     In vivo, a delay in the expression of the myelin protein MBP was
221 tilage was strongly suppressed, resulting in a delay in the formation of the secondary ossification c
222 tion that each intermediate hospital conveys a delay in the further spread of the pathogen.
223                     Deletion of DIA2 rescues a delay in the G1-to-S phase transition in the tom1Delta
224 o 30 days post-primary infection, suggesting a delay in the generation of memory.
225                 Phenotypic analysis revealed a delay in the germination of atlig6 mutants compared wi
226  different particle concentrations did cause a delay in the growth of P. aeruginosa, whereas impressi
227                       JAM-C knockdown caused a delay in the hfRPE cell polarization, as shown by redu
228                     Mutations in lep-2 cause a delay in the juvenile-to-adult transition, with adult
229 K20 methylase, mono- and dimethyl H4K20, and a delay in the kinetics of 53BP1 foci formation at sites
230 hereas lower concentrations resulted in only a delay in the lag phase.
231  catalytic reaction slows down, resulting in a delay in the lasing onset time, which is used as the s
232 on of the palatal mesenchyme, resulting from a delay in the maturation of osteoblasts.
233            Here we demonstrate that there is a delay in the maturation of the intrinsic properties of
234                          Here we demonstrate a delay in the maturation of the properties and synaptic
235                     ant ail6 mutants display a delay in the meristem identity transition and in LFY i
236 characterized by an advance in the onset and a delay in the offset of daily activity, thus revealing
237 lopes slowly to reach their target, there is a delay in the onset of action that may be reduced by in
238    With compound 2 treatment, there was only a delay in the onset of assembly, with no effect on the
239              Computer modeling predicts that a delay in the onset of base generation can lead to impr
240 develop blood-stage parasitemia or exhibited a delay in the onset of blood-stage patency.
241 activity alters this ROS balance, leading to a delay in the onset of differentiation.
242 e qoxAB mutants are attenuated in mice, with a delay in the onset of disease signs and with increased
243 derm development, that end-3(-) embryos have a delay in the onset of E lineage cell fate and that end
244 ncrease in the proportion of nonlearners and a delay in the onset of learning in both FX and conditio
245 s for development later in life and to cause a delay in the onset of menopause as measured by the num
246 ions,in a subset of genes, that likely cause a delay in the onset of stationary phase, which appears
247 nts using a single bacterial species yielded a delay in the onset of transmission, which we hypothesi
248                                     There is a delay in the phasing of H3K4me3 relative to the peak i
249  problematic owing to its infrequency and/or a delay in the positivity of a cerebrospinal fluid (CSF)
250 virus to produce immune antagonists leads to a delay in the production of the most effective immune a
251                 AS and NOS inhibition caused a delay in the progress of neural differentiation, as su
252 d levels of O-GlcNAc modification as well as a delay in the rate of wound closure in vitro.
253                          For flucloxacillin, a delay in the reaction onset and identification of huma
254                      In the presence of NAC, a delay in the recovery rate of the hemocyte number was
255  indicate that morphine treatment results in a delay in the recruitment of cellular events following
256 endent reduction of Mon1a levels resulted in a delay in the reformation of the Golgi apparatus after
257              These observations suggest that a delay in the release of cytochrome c or delay in ATP i
258 lanes relax under prolonged load that causes a delay in the reload curve which ultimately catches up
259 eage cell expansion and survival, leading to a delay in the remyelination process.
260 leting cells of HP1 caused genotoxic stress, a delay in the repair of DSBs and elevated levels of apo
261 hrin adaptin protein complex-1 (AP-1) causes a delay in the return to intracellular retention after i
262  high-grade bacteremia and is accompanied by a delay in the rise of the peripheral polymorphonuclear
263                  ERG recordings demonstrated a delay in the rising phase of the ERG b-wave, larger ph
264 esthetic treatment, time-trained bees showed a delay in the start of foraging of 3.3 h, and whole-hiv
265                     These findings suggested a delay in the structural and functional maturation of R
266  with high antibody levels to PvMSP1(19) and a delay in the time to P. vivax reinfection.
267           The conjunctival route resulted in a delay in the time to peak organ burden in comparison t
268 e surface protein 1 (PvMSP1(19)), would have a delay in the time to reinfection following treatment t
269 lent PvDBPII allele (O) were associated with a delay in the time to reinfection with the same variant
270           SCs deficient in miRNAs manifested a delay in the transition between the distinct different
271 b INs results in hindlimb hyperextension and a delay in the transition from stance phase to swing pha
272 nd larger cell size and is not simply due to a delay in the transition to flowering.
273       Mechanistically, this corresponds with a delay in the transition to PI(3,4,5)P3 and phagocytic
274 on of stress waves to the face, resulting in a delay in the transmission of stresses to the intracran
275 s subject to a circadian gate that generates a delay in the TTFL, and this delay is thought to be cri
276 e subsequent S and G(2)/M phases, exhibiting a delay in their cell cycle, accumulation at G(2)/M, mas
277 l precursors at decreased efficiency, caused a delay in their terminal maturation, and did not invoke
278               The second zen(RNAi) defect is a delay in this activity, suggesting that a temporal win
279 lium, partial absence of the thymic capsule, a delay in thymus and parathyroid separation, and failed
280 E-cadherin and ZO-1 to junctions, as well as a delay in tight junction barrier formation, as indicate
281              Thus, acute stress coupled with a delay in time from a negative experience may be a stro
282 (P < 0.05) in statin myalgic subjects due to a delay in time to reach peak power output.
283 nt by a medicine service was associated with a delay in time to surgical intervention (IRR = 1.84, 95
284 %) receiving ChAd63-MVA ME-TRAP demonstrated a delay in time to treatment, compared with unvaccinated
285 ch as the prion protein, and correlated with a delay in translocation initiation.
286                      Our results demonstrate a delay in transmission of action potentials by the gang
287 odel of diffusive pathogen transfer predicts a delay in transmission that depends both on the distanc
288 health care settings, because there is often a delay in treatment initiation.
289                                Patients with a delay in treatment of 21 weeks or more compared to a d
290                    Both interruptions led to a delay in triple-helix folding, with the 15-residue int
291 y, comparisons with other phyla suggest that a delay in trunk development is a feature of indirect de
292 lysis of normal melanocytes, associated with a delay in tumor progression.
293 bese diabetic (NOD.Ncf1(m1J)) mice exhibited a delay in type 1 diabetes (T1D) partially due to blunte
294  the offender population is a consequence of a delay in typical development, rather than a distinct a
295 n to lung tight junctions in situ along with a delay in up-regulation of claudin-4.
296 tinal volume due to increased apoptosis, and a delay in ventral optic vesicle invagination leading to
297 ups of New World begomoviruses, resulting in a delay in viral DNA accumulation and symptom appearance
298 bility of astrocytes to WNV-MAD78 was due to a delay in viral genome replication and an interferon-in
299 ction, reduced the HIV reservoir, and caused a delay in viral rebound after ART interruption.
300 ice with the MEK inhibitor U0126 resulted in a delay in wound healing suggesting that aberrant ERK ac

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