1 These results are quantitatively explained
by a model in which partially dissociated bound proteins are sus
2 In most
conditions,
a model in which the recent history of past samples determine
3 We
discuss a model in which an "epigenetic addiction" to the HJURP chape
4 Thus, we
present a model in which macrophage/tumor cell contact allows for the
5 We
present a model in which sheath formation depends on the coordinated
6 We thus
propose a model in which kinetochores mature through Ska complex recr
7 Based on these results, we
propose a model in which a conformational change in BTN3A1 is a key e
8 We
propose a model in which during re-synthesis of resected DNA, the rep
9 Based on these findings, we
propose a model in which ER Ca(2+) depletion promotes the activation
10 We
propose a model in which NK-cell education prevents or delays downreg
11 We
propose a model in which the Astrin-SKAP complex acts together with t
12 We
propose a model in which the C-terminus of ATP8A2 consists of an auto
13 We
propose a model in which the central regulatory region promotes ASK1
14 We
propose a model in which the local variance in tension between juncti
15 On the basis of these findings, we
propose a model in which Treg cell responses at peripheral sites conv
16 Our results
reveal a model in which the GtCCR2 retinylidene SB chromophore rapid
17 Together, our data
suggest a model in which sustained FGF signaling acts to suppress car
18 Our data
suggest a model in which the CTD serves primarily to anchor Tim44 to
19 Our data
suggest a model in which the majority of NK cells in the human lung d
20 We
suggest a model in which interaction of KinG with SHR allows for the
21 Gene expression and mutant analyses
support a model in which DnaA and Rok cooperate to repress transcript
22 These data
support a model in which a single neuronal stem cell can produce a la
23 Together, our data
support a model in which DACH1 stimulates coronary artery growth by a
24 The data
support a model in which EAP deficits lead to poor functional outcome
25 Taken together, these data
support a model in which FGFs, possibly from axons, activate FGFR2 in
26 Altogether, our data
support a model in which small neutral hydrophobic residues facilitat
27 These data
support a model in which the MBD2/3 methylation-dependent functional
28 Our findings
support a model in which initial inhomogeneities in inputs are amplif
29 Taken together, our findings
support a model in which neighboring binding sites interact competiti
30 Our findings
support a model in which repression of activin signaling by FST enabl
31 neddylates cullins in vivo Combined, these findings
support a model in which the SCCRO, substrate, and substrate adaptors
32 Together, our results
support a model in which acute inflammation after injury initiates im
33 Our results
support a model in which MW deteriorates performance in the task at h
34 Our results
support a model in which NMC is dependent on ectopic NUT-mediated int
35 Taken together, these results
support a model in which this IDR of Med13 plays a key role in contro
36 Our results
support a model in which TOPBP1(Dpb11) plays a conserved role in medi
37 e discussed in relation to O-O cleavage in HCOs,
supporting a model in which a peroxo intermediate serves as the active H
38 linear progression through these subpopulations,
supporting a model in which these three states correspond to consecutive
39 atures in XPF-deficient Caenorhabditis elegans and
supports a model in which translesion synthesis polymerases perform a
40 Such a transition state
supports a model in which the rate-limiting step of the hybridization
41 Our work
supports a model in which AdamTS-A anchors cells in place and preserve
42 HD-ZIPIII and KAN gene expression in the shoot, leading
to a model in which dorsoventral genes coordinate to regulate pl
43 This leads
to a model in which Vpu monomers, Vpu homooligomers, and Vpu-tar
44 ) to be equal across all families in which it segregates
to a model in which the effect of a SNP can vary across families
45 Collectively, our investigations are consistent
with a model in which PhoPQ-dependent Mg(2+) homeostasis protects
46 n PMS abundance and microtubule regulation, consistent
with a model in which PMS-dependent microtubule polymerization con
47 These findings are consistent
with a model in which RVs, as persistent MPs, prevent fusion betwe
48 These unexpected in vivo data are most consistent
with a model in which synthesis and degradation are both increased
49 ard the fully spliced isoform, our data are consistent
with a model in which unproductive splicing complexes assembled at
50 Our results are consistent
with a model in which, upon the onset of heat stress, translation