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1 A role for BCGM in conditioning expression divergence between
2 Future larger studies may determine whether there might be a role for losartan in specific vulnerable subgroups.
3 ng of motility, and actin polymerization studies to confirm a role for CD13 in impaired MO-MDSC transmigration.
7 variants cause down-regulation of autophagy, demonstrating a role for SDHD in autophagy-associated pathogenesis of diffe
12 We explore potential regulators and find a role for HSF1 in the induction of a subset of heat shock-in
13 ain maintains its ability to bind mitochondria, arguing for a role for Actr10 in dynactin-mitochondria interaction.
20 ent-dependent regulation of glucagon secretion and identify a role for mTORC1 in controlling alpha cell-mass maintenance.
22 Thus, our genetic, in vivo, and biochemical data indicate a role for Coy1 in regulating COG complex-dependent fusion of
26 antly lower postprandial serum triglycerides, suggestive of a role for TM6SF2 in the small intestine.
29 mes from concatemeric viral DNA.IMPORTANCE This paper shows a role for pUL33 in one of the two DNA cleavage events requir
30 te CDK19 as a regulator of p53 stress responses and suggest a role for CDK19 in cellular resistance to nutlin-3.
33 ith the c-src inhibitor PP2 rescued this effect, suggesting a role for GCs in promoting c-src-mediated proteosomal degrad
35 agments of these proteins not found in controls, suggesting a role for PHEX in SIBLING protein degradation.
36 of empty capsids were observed in the cytoplasm, suggesting a role for UL21 in preventing their exit from the nucleus.
39 tered cellular responses to cisplatin treatment, supporting a role for SLC16A5 in the development of cisplatin-induced ot
40 ith hypertension in humans, whereas the evidence supporting a role for variants in the genes that alter levels of epoxyei
44 e host response to bacterial infection, and they underscore a role for Sts in regulating functionally relevant immune res
45 the Nef alphaB-helix from deuterium uptake, consistent with a role for alphaB in dimer formation.
46 AMPs are hCVAM antimicrobial agents and are consistent with a role for AMPs in mediating antimicrobial properties of the
48 m anemic stress and persistent cell cycling consistent with a role for KLF3 in dampening KLF1-driven proliferation.
50 was downregulated in the mutant forebrain, consistent with a role for Vax1 in mediating transduction of this pathway.
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