1 A screen for 1-TbAd mutants, complementation studies, an
2 We carried out
a screen for a potential link between triglyceride mobil
3 We performed
a screen for aberrant nodulation phenotypes using the Lo
4 In
a screen for abnormal dendrite development, we identifie
5 A screen for abnormal root architecture responses to hig
6 ene, ema (endosomal maturation defective) in
a screen for abnormal synaptic overgrowth and defective
7 We report here the isolation of SPPL3 in
a screen for activators of NFAT, a transcription factor
8 A screen for additional Dnf1 mutants capable of replacin
9 In
a screen for additional L. pneumophila proteins that ass
10 A screen for adjuvant activity of microbial products rev
11 A screen for adult ADHD was included in a probability su
12 A screen for agents to re-activate suppressed occludin g
13 A screen for alternative proinflammatory factors of F. t
14 In
a screen for antibiotic resistance determinants in Mycob
15 In
a screen for antiprion systems curing [PSI+] without pro
16 A screen for antiproliferative factors repressed after l
17 These features permit
a screen for antivirals for emerging viruses and select
18 The pbs3-1 mutant, identified in
a screen for Arabidopsis (Arabidopsis thaliana) mutants
19 A screen for Arabidopsis (Arabidopsis thaliana) mutants
20 We identified SUO in
a screen for Arabidopsis mutations that increase the acc
21 We identified MYB98 in
a screen for Arabidopsis thaliana genes expressed in the
22 ion of new cryptochrome1 (cry1) alleles from
a screen for Arabidopsis thaliana genomes uncoupled muta
23 In
a screen for Arabidopsis thaliana mutants affected in th
24 ning for phenotypic mutants was validated in
a screen for attachment to abiotic surfaces.
25 utation of TMEM184b, a protein identified in
a screen for axon degeneration mediators.
26 A screen for axonal cargo mislocalization in Caenorhabdi
27 A screen for axonal localization identifies a specific s
28 rhabditis elegans bre-1 gene was isolated in
a screen for Bacillus thuringiensis toxin-resistant (bre
29 al infections underwent standard culture and
a screen for bacteria by amplification and sequencing of
30 e mitochondrial ATP synthase beta subunit in
a screen for Bcl-x(L)-binding partners, we tested and fo
31 A screen for Bcr-Abl mutants emerging in the presence of
32 A screen for C. elegans mutants with a biofilm absent on
33 In
a screen for Caenorhabditis elegans mutant animals that
34 In
a screen for Caenorhabditis elegans mutants that display
35 or Neurogenic Differentiation 2 (NeuroD2) in
a screen for calcium-regulated transcription factors and
36 In
a screen for candidate genes stimulating cell death in G
37 A screen for candidate mediators of the PPARalpha-driven
38 We conducted
a screen for candidate mutations affecting DSB repair an
39 A screen for candidates revealed suppressor of cytokine
40 ved in postsynaptic exocytosis, we conducted
a screen for candidates that disrupted trafficking of a
41 Tropisetron was identified in
a screen for candidates that increase the ratio of the t
42 a regulator of sister chromatid cohesion in
a screen for cell cycle-controlled proteins.
43 We performed
a screen for cell types that have high levels of signali
44 In
a screen for cell-cycle regulators, we identified a Dros
45 In this study,
a screen for CER1 physical interaction partners was perf
46 By conducting
a screen for checkpoint genes in zebrafish, we found tha
47 We conducted
a screen for circadian-relevant neurons in the Drosophil
48 e, applying CLIK analysis retrospectively to
a screen for cisplatin sensitivity allowed us to identif
49 ule inhibitors of Cdks that we identified in
a screen for compounds that activate hPXR) leads to acti
50 A screen for compounds that downregulate Mcl-1 identifie
51 In
a screen for compounds that mimic CR effects in the live
52 In
a screen for conserved substrates of AMPK, we identified
53 A screen for corresponding substrates of the neprilysin
54 In
a screen for dauer regulatory genes that control the act
55 We identified elm1 mutants in
a screen for defects in Swe1p degradation and show that
56 We recently introduced
a screen for detecting protein-protein interactions base
57 Therefore, in a previous study we initiated
a screen for differential gene expression in the telence
58 A screen for differentially expressed genes in granuloma
59 echanisms enable this precision, we designed
a screen for disrupted sepal size and shape uniformity i
60 In
a screen for dominant enhancers of mutations in the home
61 solate additional proteasome-related mutants
a screen for dominant suppressors of Pros26(1) was carri
62 To study needle assembly, we performed
a screen for dominant-negative yscF alleles that prevent
63 A screen for downstream mediators revealed that Dkk-1 an
64 From
a screen for Drosophila mutants with aberrant synaptic d
65 A screen for Drosophila synaptic dysfunction mutants ide
66 is technique may potentially be exploited as
a screen for drug development and analysis.
67 measure gap junction mediated diffusion and
a screen for drugs that affect gap junction communicatio
68 We performed
a screen for drugs that upregulate the somatostatin rece
69 sovan and Neanderthal hominids, we conducted
a screen for endogenized retroviruses, identifying six n
70 In
a screen for endogenous tumor-associated T cell response
71 Here the authors develop
a screen for engineering new split FPs, and report a yel
72 In
a screen for enhancers of tir1-1 auxin resistance, we id
73 is a perinatal lethal mutation uncovered in
a screen for ENU-induced mutations on mouse chromosome 5
74 We conducted
a screen for ethyl methanesulfonate-induced suppressors
75 During
a screen for ethylnitrosourea-induced mutations in mice
76 ax group gene kismet (kis) was identified in
a screen for extragenic suppressors of Polycomb (Pc) and
77 A screen for F/P-dependent cytokines was performed in gr
78 Through
a screen for factors involved in kinetochore integrity i
79 entified an HMX homeodomain protein MLS-2 in
a screen for factors required for M lineage patterning.
80 In
a screen for factors required for the localization of th
81 Meanwhile, moody also has been identified in
a screen for fly mutants with altered sensitivity to coc
82 Herein, we identified leupaxin in
a screen for focal adhesion kinase binding partners in a
83 quired for biological activity, we conducted
a screen for functionally important MYC targets and iden
84 dation for the utility of our interactome as
a screen for functionally important novel V-ATPase-regul
85 From
a screen for functionally relevant ERG interactors, we i
86 A screen for fusion mutants in Chlamydomonas identified
87 Through
a screen for G2/M checkpoint regulators in zebrafish, we
88 In
a screen for gene copy number alterations in mouse mamma
89 In
a screen for gene copy-number changes in mouse mammary t
90 In
a screen for genes activated by Zic1, we identify severa
91 From
a screen for genes defective in caspase activation in th
92 In
a screen for genes directly activated by TGF-beta, we fo
93 r kinase (MELK) was previously identified in
a screen for genes enriched in neural progenitors.
94 In
a screen for genes involved in Slit-Robo repulsion, we h
95 In
a screen for genes involved with prostate cancer metasta
96 vered unexpectedly in healthy CNS neurons in
a screen for genes regulated by neural activity.
97 In
a screen for genes required for ISC proliferation in res
98 ave isolated an exocyst component, sec15, in
a screen for genes required for synaptic specificity.
99 several screens, including MAD2 (MAD2L1) in
a screen for genes required for the spindle assembly che
100 us, biclustering is a natural methodology as
a screen for genes that are functionally related, partic
101 We identified diaphanous in
a screen for genes that are necessary for the normal gro
102 A screen for genes that can ectopically activate a Rad3-
103 A screen for genes that control Drosophila heart develop
104 specific genes for regulation, we performed
a screen for genes that modify FOXO activation of TRAIL,
105 In
a screen for genes that modulate C. elegans insulin-like
106 In
a screen for genes that promote GCR, we identified MPH1,
107 In
a screen for genes that regulate cell lineage determinat
108 In
a screen for genes upregulated by taurine in developing
109 In
a screen for genes whose expression was altered in respo
110 new components of this pathway, we performed
a screen for genes whose loss-of-function debilitated TP
111 A (LCAA) antisense plants were obtained from
a screen for genes whose partial down-regulation results
112 me 1 (sxe1, cyp4d21), has been identified in
a screen for genes with sex-biased expression in the hea
113 d-dependent disease phenotypes, we performed
a screen for genetic modifiers of autoreactivity between
114 We performed
a screen for genetic suppressors of cobra, an Arabidopsi
115 A screen for genetic suppressors of the enhanced seed do
116 1 (ged1), which was previously discovered in
a screen for genomes uncoupled-like mutants and shows th
117 We conducted
a screen for glossy-eye flies that fail to incorporate B
118 A screen for growth on the beta-lactam antibiotic cephal
119 To investigate the basis for these defects,
a screen for growth suppressors was performed.
120 ew derivatives, which were then submitted to
a screening for HDAC inhibition as well as to a prelimin
121 A screen for HDAC6 inhibitors identified acyl derivative
122 A screen for hepatitis C virus (HCV) NS3 helicase inhibi
123 Here, we describe
a screen for host factors that influence transposition o
124 In
a screen for human signal transduction genes altered dur
125 In
a screen for hypermethylated CpG islands in cancer, bidi
126 We used this phenotype to carry out
a screen for identifying genes regulating Wingless distr
127 A screen for imprinted genes on mouse Chromosome 7 recen
128 hod is of high throughput, and it can act as
a screen for in vitro cancer stem cell response to drugs
129 A screen for increased longevity in Caenorhabditis elega
130 is a small molecule inhibitor discovered in
a screen for inhibitors of nonmuscle myosin IIA.
131 A screen for inhibitors of the protein binding domains o
132 To test this idea, we performed
a screen for inhibitors selective for Plasmodium Kinesin
133 In
a screen for inhibitors that block the association of TR
134 A screen for interdomain interactions by solution-state
135 We conducted
a screen for ISGs that inhibit HIV-1 virion production a
136 C. uda was identified in
a screening for its ethanologenic potential from AFEX-CS
137 In
a screen for kinases that promote survival of KRAS-depen
138 proteins, Ceg9, was previously identified in
a screen for L. pneumophila IDTS that manipulate secreto
139 A screen for LATS2-interacting proteins in liver-derived
140 During
a screen for liver defects from a zebrafish insertional
141 Using mariner mutagenesis and
a screen for loss of cytotoxicity, a genetic locus encod
142 re the utility of this method by undertaking
a screen for magnetic cells in the pigeon.
143 In
a screen for mediators of PhoPQ-regulated OM remodeling
144 Using
a screen for members of the Wnt pathway, we demonstrate
145 In
a screen for microRNAs (miRNAs) that modulate the DNA da
146 e amygdala from these mice was collected and
a screen for microRNAs that were recruited to the RNA-in
147 We conducted
a screen for miRNA function in primary T cells and ident
148 In
a screen for miRNAs regulated by myocardin-related trans
149 Here we report
a screen for miRNAs that affect the Wingless (Wg) pathwa
150 Using
a screen for mitophagy-deficient mutants, we found that
151 A screen for MMS-sensitive mutants identified a novel tr
152 Through
a screen for modified transport to the vacuole (mtv) mut
153 In this work, we performed
a screen for modifiers of dendritic growth in hippocampa
154 A screen for modifiers of Dpp adult phenotypes led to th
155 From
a screen for modifiers of Notch signaling in Drosophila
156 The turnip (tnp) mutant was identified in
a screen for modifiers of POLARIS expression, a gene req
157 In
a screen for modifiers of the oocyte DV patterning defec
158 We identified sqd in
a screen for modifiers of the Protein Kinase A (PKA) oog
159 A screen for modifiers of the six cycB phenotypes identi
160 r mutants with spindle formation defects and
a screen for molecules that antagonized EB1 function.
161 In
a screen for molecules that participate with XIAP in reg
162 cmpy was identified in
a screen for motoneuron-expressed genes and encodes a si
163 A screen for mRNAs whose polyadenylation is altered by G
164 ne containing mltG was initially isolated in
a screen for multicopy plasmids generating a lethal phen
165 We performed
a screen for multicopy suppressors of arp2-7 and identif
166 A screening for multicopy suppressors of these Vpr activ
167 In
a screen for mutagenesis-defective mutants, we isolated
168 ation to the length of the night, we devised
a screen for mutant Arabidopsis thaliana plants in which
169 In
a screen for mutants affecting gonad formation we identi
170 Snx4/Atg24 proteins have been identified in
a screen for mutants defective in a type of selective ma
171 The rev6-1 allele was isolated in
a screen for mutants deficient for UV-induced reversion
172 We have identified the new PAK gene max-2 in
a screen for mutants disrupted in UNC-6/netrin-mediated
173 A screen for mutants in which a winter-annual Arabidopsi
174 Dam methylase mutants were recovered in
a screen for mutants sensitive to UV irradiation or mild
175 In
a screen for mutants showing altered photomorphogenesis
176 A screen for mutants that affect this process identified
177 In
a screen for mutants that are defective in transitioning
178 omes 1 (brt1) mutant was first identified in
a screen for mutants that exhibit a reduced epidermal fl
179 In
a screen for mutants that no longer require vernalizatio
180 A screen for mutants that poorly expressed a comEA-lucif
181 In
a screen for mutants that suppress the expression of the
182 In
a screen for mutants with abnormal mitochondrial morphol
183 iated protein (CLASP) Peg1 was identified in
a screen for mutants with spindle formation defects and
184 Here we used
a screen for mutations affecting endodermal organ morpho
185 Five new alleles of dig-1 were isolated in
a screen for mutations affecting the morphology or funct
186 In
a screen for mutations modifying clv1 mutants, we have i
187 els of sHsp chaperone activity, we performed
a screen for mutations of Synechocystis Hsp16.6 that red
188 in short days6 (esd6) mutant was isolated in
a screen for mutations that accelerate flowering time.
189 A screen for mutations that affect habenular laterality
190 In
a screen for mutations that affect the biosynthesis of r
191 A screen for mutations that affect the recruitment of th
192 essential for organ abscission, we conducted
a screen for mutations that alter floral organ shedding
193 separate set of substitutions, identified in
a screen for mutations that alter the emission of a fluo
194 In
a screen for mutations that bypassed the critical requir
195 ed a dominant mutation in wapl (wapl(AG)) in
a screen for mutations that counteract silencing mediate
196 serine/threonine kinase PAR-1 (MARK/Kin1) in
a screen for mutations that disrupt border cell migratio
197 In
a screen for mutations that disrupted brain laterality,
198 A screen for mutations that enhance ROP1-overexpression-
199 characterize these mechanisms, we undertook
a screen for mutations that enhance sepA defects.
200 rabidopsis thaliana mutants were isolated by
a screen for mutations that impair cold-induced transcri
201 A screen for mutations that interact with CDC34(tm) unco
202 In
a screen for mutations that modify wg loss-of-function p
203 Through
a screen for mutations that restore organ separation in
204 A screen for mutations that suppress this precocious phe
205 seele was identified in
a screen for mutations that, when homozygous in ovarian
206 A screen for Mycobacterium tuberculosis (Mtb) mutants se
207 A screen for NAB-regulated genes identified several (inc
208 Following
a screen for neuromuscular mouse mutants, we identified
209 In
a screen for neuronal Ca(2+) sensors that respond to cha
210 In
a screen for new DNA repair mutants, we tested 6275 Dros
211 From
a screen for new memory mutants, we identified alleles o
212 uable tool that holds broad applicability in
a screen for non-ATP site binders.
213 Using a transposon library and
a screen for nonencapsulated mutants, we identified the
214 62 and MDV3100, two compounds optimized from
a screen for nonsteroidal antiandrogens that retain acti
215 tional co-activator mastermind, we conducted
a screen for Notch signal modifiers using the Exelixis c
216 In
a screen for novel candidate genes involved in human T(r
217 A screen for novel colonization factors by use of TnphoA
218 A screen for novel inhibitors of eCB hydrolysis identifi
219 he mouse, mhyp (mosaic hypopigmentation), in
a screen for novel proviral integration sites in a multi
220 From
a screen for novel targets of bun repression we have ide
221 ators of migration could be obtained through
a screen for overly adhesive mutants.
222 We performed
a screen for P2Y1 receptor-mediated receptor tyrosine ki
223 on GOLD36, an EMS mutant identified through
a screen for partial displacement of the Golgi marker, S
224 mutants, frd4-1 and frd4-2, were isolated in
a screen for plants that do not induce Fe(III) chelate r
225 fluorescence5 (ref5-1) mutant, identified in
a screen for plants with defects in soluble phenylpropan
226 From
a screen for plastid-to-nucleus signaling mutants in Ara
227 In
a screen for potential downstream effectors of spheroid
228 Using
a screen for potential interactions between telomere rep
229 A screen for potential regulators of the CLDN7 gene duri
230 In
a screening for potential monoamine oxidase A and B inhi
231 2+) and integrin-binding protein-1 (CIB1) in
a screen for previously unknown regulators of cardiomyoc
232 We propose that
a screen for progeny-permissive mutants of microorganism
233 In
a screen for proteins co-precipitating with CHP1 in quie
234 In
a screen for proteins relevant to this IFN-stimulatory D
235 We identified RBP2 in
a screen for proteins that bind to such pRB variants.
236 In
a screen for proteins that interact with the FLAGELIN-SE
237 In
a screen for proteins that interact with the Rsp5 C2 dom
238 A screen for proteins that interacted with the AAD of fu
239 A screen for proteins that preferentially bound wild-typ
240 otide exchange factor that was identified in
a screen for proteins whose overexpression cause deregul
241 Here, we performed
a screen for putative IKKepsilon substrates using an unb
242 In
a screen for PyMT variants that could not activate the A
243 Rad51-G103E was identified in
a screen for Rad51 interaction-deficient mutants and was
244 articipants in these responses, we undertook
a screen for regulators that, when present on a high-cop
245 Here, we report the results of
a screen for repair genes induced in cancer cells treate
246 solated catalase-deficient mutants (cat2) in
a screen for resistance to hydroxyurea-induced cell deat
247 A screen for resistance to the nucleotide analog 6-thiog
248 strain CFT073 (CFT073 fim L-ON), followed by
a screen for restoration of motility in soft agar.
249 Finally,
a screen for retinoid-induced genes identifies metabolic
250 To circumvent this problem, we performed
a screen for revertants and dominant suppressors of the
251 for mitochondrial integrity and function, in
a screen for RNAs that exit the nucleus through NE buddi
252 ture-sensitive allele (rsw12) of the gene in
a screen for root radial swelling mutants.
253 A screen for Saccharomyces cerevisiae temperature-sensit
254 embryonic chicken pancreatic cDNA library in
a screen for secreted factors.
255 We developed
a screen for small molecules that block Wnt secretion.
256 In
a screen for small molecules that protect cells from end
257 The compound Retro-1 was discovered in
a screen for small molecules that reduce the actions of
258 Here we design
a screen for small-molecule inhibitors of beta-catenin's
259 A screen for substrates of AMPK identified mitochondrial
260 In
a screen for substrates of these kinases involved in Sna
261 ypothesis, we have developed and carried out
a screen for such candidate proteins using an in vitro e
262 A screen for supernodulating Medicago truncatula mutants
263 A screen for suppressors of a fission yeast chk1 mutant
264 In
a screen for suppressors of activated GOA-1 (Galpha(o))
265 r to identify gp2.5 interactions we designed
a screen for suppressors of gp2.5 lacking the C-terminal
266 on intragenic mec-4 mutations identified in
a screen for suppressors of mec-4(d)-induced necrosis, w
267 esterase (PDE) gene cgs2+ were identified in
a screen for suppressors of mutant alleles of the adenyl
268 d regulation of wax production, we performed
a screen for suppressors of the cer7 mutant.
269 The sel-6 gene was previously identified in
a screen for suppressors of the egg-laying defect associ
270 he recovery of alleles of six new genes from
a screen for suppressors of the egg-laying defect associ
271 A screen for suppressors of the highwire synaptic overgr
272 In
a screen for suppressors of the SIN mutant cytokinesis c
273 A screen for suppressors of this low-temperature phenoty
274 A screen for suppressors of transgenic Killer of prune l
275 transmembrane helix 2 of Pdr5 identified in
a screen for suppressors that eliminated Pdr5-mediated c
276 -binding domains that is also highlighted in
a screen for synapsis mutants.
277 In
a screen for synaptic mutants in Drosophila, we identifi
278 Using
a screen for synthetic lethality, we found that the V. c
279 In
a screen for T cell-inhibitory molecules in cHL, we foun
280 em coupled with RNA sequencing analysis, and
a screen for tandem TTGAC cis-elements in the upstream s
281 In
a screen for temperature-sensitive behavioral mutants, w
282 In
a screen for temperature-sensitive conditional seizure m
283 tivation sensitizes IRS1 to degradation, and
a screen for the responsible E3 ligase identified Fbxo40
284 A screen for the systematic identification of cis-regula
285 A screening for the BsmI SNP may emphasize the importanc
286 We identified AGL61 in
a screen for transcription factor genes expressed in the
287 Here, we describe
a screen for transcription factors (TFs) that regulate t
288 In
a screen for transcription factors regulating the expres
289 brm was identified in
a screen for transcriptional activators of homeotic gene
290 A screen for transcripts robustly induced in cultured ne
291 In
a screen for trichome branching defects, we isolated a m
292 In
a screen for tumour suppressors that cooperate with onco
293 A screen for type III effector proteins from Pst for the
294 In
a screen for ubi4Delta suppressors, a hypomorphic allele
295 In
a screen for unexplained mutation events we identified a
296 We performed
a screen for unidentified actors in the cellulose-respon
297 and its linked gene, vciA, were isolated in
a screen for V. cholerae genes that permitted growth of
298 identified the RNA-binding protein Hermes in
a screen for vegetally localized RNAs in Xenopus oocytes
299 To that end,
a screen for visual system mutants was performed on 250
300 Fourth,
a screen for yeast mutants that enhance or suppress grow