ライフサイエンスコーパス: abdominal abscess

コーパス検索結果 (１語後でソート)

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1 ammation did LA significantly increase intra-abdominal abscesses.

2 h their ability to induce experimental intra-abdominal abscesses.

3 virulence factors in the formation of intra-abdominal abscesses.

4 e both able to induce the formation of intra-abdominal abscesses.

5 anaerobe most frequently isolated from intra-abdominal abscesses.

6 uded three anastomotic leaks and three intra-abdominal abscesses.

7 sis, intra-abdominal fluid collection, intra-abdominal abscess (10% vs 25%, P = 0.027), severe (>/=gr

8 was 10.3%, wound dehiscence 4.4%, and intra-abdominal abscess 5.8%, and did not significantly differ

9 The third complication was an intra-abdominal abscess after a TV appendectomy.

10 he enteric anastomosis site, including intra-abdominal abscess and enterocutaneous fistula formation,

11 l symptoms and signs of peritonitis or intra-abdominal abscess and isolation of Candida species eithe

12 postoperative complications including intra-abdominal abscess and pancreatic fistula (all P < 0.02).

13 ponse necessary for the development of intra-abdominal abscesses and underscore the role of IL-17 in

14 M101 contributes to persistence within intra-abdominal abscesses, at least in part through activation

15 .93; 95% CI, 0.38-2.32; P = .88) or on intra-abdominal abscess development (OR, 0.89; 95% CI, 0.34-2.

16 postoperative hemorrhage (P = 0.174), intra-abdominal abscess formation (P = 0.199), biliary leakage

17 Intra-abdominal abscess formation was more common following la

18 ative pyrexia, ileus, wound infection, intra-abdominal abscess formation, operative time, and postope

19 ment included CT-guided drainage of an intra-abdominal abscess in 23 patients, and total parenteral n

20 and in vivo they were attenuated in an intra-abdominal abscess infection model.

21 ssary for maximal virulence in a mouse intra-abdominal abscess model.

22 90 days included wound infections and intra-abdominal abscess (n = 75) and failure to thrive (n = 38

23 sary to repair the perforation to prevent an abdominal abscess or sepsis.

24 There were no intra-abdominal abscesses or other major complications associa

25 recurrence (n = 66, 12%) or because of intra-abdominal-abscess or fistula (n = 93, 16%).

26 nic procedures, OR = 1.5), presence of intra-abdominal abscess (OR = 5.0) or enteric fistula (OR = 4.

27 4.90; 95% CI, 1.41-17.06; P = .01) and intra-abdominal abscess (OR, 7.46; 95% CI, 1.65-33.66; P = .00

28 e IAI (peritonitis, biliary tract infection, abdominal abscess, or enteritis) with those who did not

29 omy closure (6.5%), trauma (2.7%), and intra-abdominal abscess/other peritonitis (1.9%).

30 With CPG-directed care, intra-abdominal abscess rate decreased from 0.24 to 0.10 (adju

31 ts were postoperative wound infection, intra-abdominal abscess, reoperation, length of hospital stay,

32 ts were postoperative wound infection, intra-abdominal abscess, reoperation, length of hospital stay,

33 ery and postoperative wound infection, intra-abdominal abscess, reoperation, or readmission.

34 ery and postoperative wound infection, intra-abdominal abscess, reoperation, or readmission.

35 s were fatal), 6 had viral infections, 2 had abdominal abscesses requiring surgery, one had arm cellu

36 gastrointestinal tract [12; colitis (seven), abdominal abscess (three), and diverticulitis and esopha

37 d collections, one pseudocyst, and one intra-abdominal abscess; two donors underwent reoperation.

38 ic antibiotics, and the development of intra-abdominal abscess was significantly lower in patients re

39 The risk of abdominal abscesses was higher for laparoscopic surgery

40 atients who had an anastomotic leak or intra-abdominal abscess were included in the infection group (

41 diseased terminal ileum longer than 40 cm or abdominal abscesses were excluded.

42 cteroides fragilis is the formation of intra-abdominal abscesses, which are induced by the capsular p

43 rtality and postoperative morbidities (intra-abdominal abscess, wound dehiscence, and intestinal stri

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