ライフサイエンスコーパス: abdominal obesity

1 s associations with body mass index (BMI) or abdominal obesity.

2 of the subjects were obese, and 94 (76%) had abdominal obesity.

3 eural networks that may lead toward ultimate abdominal obesity.

4 ors for coronary heart disease in women with abdominal obesity.

5 randomly assigned participants with moderate abdominal obesity.

6 glyceride level, elevated glucose level, and abdominal obesity.

7 nt of the metabolic syndrome associated with abdominal obesity.

8 the metabolic abnormalities associated with abdominal obesity.

9 uring early fasting is blunted in women with abdominal obesity.

10 xpenditure, altered lipolysis, and increased abdominal obesity.

11 cardiovascular risk factors associated with abdominal obesity.

12 dditional insight on genetic contribution to abdominal obesity.

13 intensity resulted in similar reductions in abdominal obesity.

14 postprandial lipid metabolism in humans with abdominal obesity.

15 d 2.24 (95%CI, 1.78-2.82), respectively, for abdominal obesity.

16 tion effect did not differ by general versus abdominal obesity.

17 overweight or obese and less likely to have abdominal obesity.

18 itions of equal energy deficit in women with abdominal obesity.

19 e models of genes predicted to be causal for abdominal obesity.

20 ngly influenced by fasting hyperglycemia and abdominal obesity.

21 fat and systemic inflammation in people with abdominal obesity.

22 ions, as well as coexistent hypertension and abdominal obesity.

23 all obesity (16.0 vs 11.0, respectively) and abdominal obesity (16.7 vs 11.0).

24 5.2) greater WC, and 3-fold greater odds of abdominal obesity (2.9; 1.6, 5.1) compared with women wh

25 valence of components in MetS was 57.75% for abdominal obesity, 44.05% for elevated blood pressure, 4

26 rolled trial among 278 sedentary adults with abdominal obesity (75%) or dyslipidemia in an isolated w

27 For abdominal obesity, a dichotomous variable was created: 1

28 ve assessed the association between nuts and abdominal obesity, although an inverse association with

29 ferentiation of new adipocytes, resulting in abdominal obesity and a metabolic syndrome-like conditio

30 Metabolic syndrome, abdominal obesity and dyslipidaemia, are strongly associ

31 Abdominal obesity and exaggerated postprandial lipemia a

32 2.59) in subjects in the lowest quintiles of abdominal obesity and fasting hyperglycemia, respectivel

33 Abdominal obesity and hyperglycemia were responsible for

34 drome, men on ADT had a higher prevalence of abdominal obesity and hyperglycemia.

35 ioprotective system becomes dysfunctional in abdominal obesity and hyperglycemia.

36 ndicates that women tended to have onsets of abdominal obesity and hypo-alpha-lipoproteinemia in youn

37 l design in which HIV-infected subjects with abdominal obesity and insulin resistance were randomized

38 which hsCRP is likely to be elevated, namely abdominal obesity and insulin resistance, provides a fra

39 it may play a role in the pathophysiology of abdominal obesity and insulin resistance.

40 lity exists, a negative relationship between abdominal obesity and insulin sensitivity was confirmed

41 s a potential interaction (P = 0.08) between abdominal obesity and low 25(OH)D concentrations that sh

42 giogenic and anti-adipogenic, while reducing abdominal obesity and metabolic abnormalities.

43 ed adipogenesis and angiogenesis, leading to abdominal obesity and metabolic syndrome which were inhi

44 nutritional risk predicts the development of abdominal obesity and MetS during long-term follow-up in

45 ted prevalences of overweight or obesity and abdominal obesity and odds ratios with SUDAAN software (

46 efect in muscle aPKC is sufficient to induce abdominal obesity and other lipid abnormalities of the m

47 ad a 2- to 3-fold risk of the development of abdominal obesity and overall MetS during 12 y of follow

48 abolic pathways and networks contributing to abdominal obesity and overlapped with a macrophage-enric

49 id economic development and urban migration, abdominal obesity and related chronic diseases are likel

50 ized controlled trial, 92 men and women with abdominal obesity and relatively low HDL-cholesterol con

51 optimal exercise modality for reductions of abdominal obesity and risk factors for type 2 diabetes i

52 iple cardiometabolic risk factors, including abdominal obesity and smoking.

53 iple cardiometabolic risk factors, including abdominal obesity and smoking.

54 changes in body composition, involving both abdominal obesity and stavudine-induced peripheral lipoa

55 have been identified, chief among which are abdominal obesity and the metabolic syndrome.

56 omic status, higher systolic blood pressure, abdominal obesity, and a complex medical history.

57 Waist:hip ratio was used to assess abdominal obesity, and forced expiratory volume in 1 sec

58 conditions (diabetes, hypertension, obesity, abdominal obesity, and hypercholesterolemia), and access

59 include advanced age, male sex, white race, abdominal obesity, and tobacco use.

60 In a younger population, overall and abdominal obesity are associated with increased prevalen

61 Effects on cardiorespiratory fitness and abdominal obesity are both likely to contribute to the i

62 G variant contributes to overall fatness and abdominal obesity are confirmed.

63 General overweight and general and abdominal obesity are independently associated with an i

64 adults, highlights the urgency of addressing abdominal obesity as a healthcare priority.

65 We defined abdominal obesity as a waist circumference > or =90th pe

66 We used fasting hyperglycemia and abdominal obesity as surrogates for insulin sensitivity.

67 r aim was to examine the association between abdominal obesity, as measured by the waist-to-hip ratio

68 as measured by body mass index, and that of abdominal obesity, as measured by waist-to-hip ratio, ha

69 Total and abdominal obesity, as well as metabolic factors such as

70 the metabolic syndrome: insulin resistance, abdominal obesity based on waist circumference, hypertri

71 umference and standard and new indicators of abdominal obesity based on waist circumference.

72 studies, significant improvements in weight, abdominal obesity, blood pressure, and lipid profile wer

73 ssociations of BPA exposure with general and abdominal obesity, BPF or BPS, at current exposure level

74 The results suggest that abdominal obesity, but not elevated body mass index, pre

75 Volunteers with abdominal obesity consumed each of 5 identical weight-ma

76 the epidemic has leveled off, prevalence of abdominal obesity continues to rise, especially among ad

77 vide partial support for the hypothesis that abdominal obesity contributes to GERD, which may in turn

78 In patients with type 2 diabetes with abdominal obesity, CR ameliorates glomerular hyperfiltra

79 e prevalence of overweight or obesity and of abdominal obesity decreased with increased snacking freq

80 Limited data suggest that abdominal obesity decreases the sensitivity of palpation

81 Comfort food ingestion that produces abdominal obesity, decreases CRF mRNA in the hypothalamu

82 ages at onset of 5 cardiometabolic diseases: abdominal obesity, diabetes, hypertension, hypertriglyce

83 cludes insulin resistance, hyperinsulinemia, abdominal obesity, dyslipidemia with high triglyceride a

84 risk factors, including insulin resistance, abdominal obesity, dyslipidemia, and hypertension, and i

85 were significant differences in measures of abdominal obesity, dyslipidemia, hyperinsulinemia, and t

86 Metabolic syndrome and its components-abdominal obesity, elevated fasting blood glucose concen

87 g, and Blood Institute criteria and included abdominal obesity, elevated triglycerides, low high-dens

88 , being divorced/widowed, alcohol intake and abdominal obesity had higher odds of HEPHA; higher educa

89 Abdominal obesity has a direct effect on unfavorable per

90 Increased abdominal obesity has been related to lower insulin sens

91 The prevalence of abdominal obesity has increased, while those in the low-

92 children and adults, particularly those with abdominal obesity, have an elevated serum triacylglycero

93 molecule biochemicals, dramatically improves abdominal obesity, hepatosteatosis, hypertriglyceridemia

94 The influences of age, whole-body/abdominal obesity, homeostasis model of insulin resistan

95 mference but no increased risk in women with abdominal obesity (HR: 0.96; 95% CI: 0.52, 1.76).

96 In individuals with type 2 diabetes with abdominal obesity, hyperfiltration is a risk factor for

97 3 or more of the following characteristics: abdominal obesity, hyperglycemia, hypertension, hypertri

98 diabetes later; men tended to have onsets of abdominal obesity, hypo-alpha-lipoproteinemia, and hyper

99 Different onset patterns of abdominal obesity, hypo-alpha-lipoproteinemia, and male

100 ociations of snacking with weight status and abdominal obesity in adolescents 12-18 y of age (n = 581

101 , but it is not known whether DHEA decreases abdominal obesity in humans.

102 We prospectively studied measures of abdominal obesity in relation to the incidence of sympto

103 s considered a poor indicator of overall and abdominal obesity in the elderly.

104 Overweight and abdominal obesity increase mortality risk, although the

105 Total effect of abdominal obesity increased risk of AL and BOP in differ

106 Data on the role of abdominal obesity, insulin resistance, and metabolic syn

107 Abdominal obesity is associated with metabolic abnormali

108 evealed that even though a high incidence of abdominal obesity is observed in females with SMS, they

109 Whether abdominal obesity is related to coronary artery calcific

110 a, low HDL cholesterol, hypertension, and/or abdominal obesity, is a risk factor for the development

111 Obesity, particularly abdominal obesity, is associated with Barrett's esophagu

112 strong interrelation between generalized and abdominal obesity leading to a mutually confounding effe

113 ts, independently, were associated with with abdominal obesity, low energy expenditure, and muscle we

114 However, abdominal obesity may be more closely related to stroke

115 The insulin resistance syndrome, including abdominal obesity, may constitute the intermediate link

116 Abdominal obesity measured by waist girth or WHR is asso

117 Sixty genetic loci associated with abdominal obesity, measured by waist circumference (WC)

118 o have CAD risk factors, but neither BMI nor abdominal obesity measures were significantly associated

119 dence of an interaction between genotype and abdominal obesity on atherosclerosis and cardiovascular

120 those with 4 births had the highest odds of abdominal obesity (OR, 2.0; 95% confidence interval, 1.5

121 related to the 20-y cumulative prevalence of abdominal obesity (P = 0.05) and high glucose (P = 0.02)

122 In multivariate analyses, abdominal obesity (PAR, 29.3%), smoking (PAR, 25.6%), no

123 The increasing prevalence of abdominal obesity, particularly among female adults, hig

124 Abdominal obesity, particularly excess intraperitoneal f

125 In a MSM, those with abdominal obesity presented greater risk of AL and BOP i

126 defined as the presence of three or more of: abdominal obesity, raised blood pressure, high triglycer

127 IGF-1, and metabolic syndrome abnormalities (abdominal obesity; raised A1C, blood pressure, and trigl

128 (95% CIs) for overweight or obesity and for abdominal obesity ranged from 0.63 (0.48, 0.85) to 0.40

129 hysical inactivity compared with overall and abdominal obesity remains unclear.

130 those with more than 20 years of overall and abdominal obesity, respectively, experienced progression

131 circumference to define general obesity and abdominal obesity, respectively.

132 ow-up, 40.4% and 41.0% developed overall and abdominal obesity, respectively.

133 ental disorder with the cardinal features of abdominal obesity, retinopathy, polydactyly, cognitive i

134 /=35 kg/m(2)), long-lasting (>30 years), and abdominal obesity stratified for metabolic status.

135 Hispanics/Latinas with a high prevalence of abdominal obesity suggests high risk for metabolic dysre

136 2 weeks developed the characteristics of the abdominal obesity syndrome, including insulin resistance

137 tors, a complication develops that resembles abdominal obesity syndrome, with insulin resistance and

138 a high-fat diet may be a useful model of the abdominal obesity syndrome.

139 s with insulin resistance, dyslipidaemia and abdominal obesity, the identification of genes for defec

140 esponse compared with casein in persons with abdominal obesity, thereby indicating a beneficial impac

141 , dyslipidaemia, diabetes or prediabetes, or abdominal obesity) to placebo, once-daily phentermine 7.

142 The prevalence of abdominal obesity varies considerably with new and stand

143 We found that abdominal obesity (waist circumference) was the stronges

144 measures, change in overall body weight and abdominal obesity (waist circumference), and weight and

145 The association of body mass index and abdominal obesity (waist/hip ratio) with stroke incidenc

146 Longer duration of overall and abdominal obesity was associated with subclinical corona

147 Duration of overall and abdominal obesity was calculated using repeat measuremen

148 Abdominal obesity was defined as waist circumference (WC

149 Abdominal obesity was higher in those aged 70-79 compare

150 ver, these associations were attenuated when abdominal obesity was included in the statistical model.

151 men (body mass index, 18.5 to < 25 kg/m(2)), abdominal obesity was significantly associated with elev

152 Abdominal obesity (WC) correlated with inflammation (r =

153 r CAC for each additional year of overall or abdominal obesity were 1.02 (95% CI, 1.01-1.03) and 1.03

154 Reduced risks of overweight or obesity and abdominal obesity were associated with snacking.

155 Indices of abdominal obesity were more consistently and strongly pr

156 in resistance, increased blood pressure, and abdominal obesity, which together markedly increase the

157 Efforts to reduce abdominal obesity will not only reduce the risk of chron

158 conducted a meta-analysis of associations of abdominal obesity with approximately 2.5 million single

159 High-risk obesity is characterized by abdominal obesity with evidence of abnormal glucose and