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1 lux, which may be related to increased intra-abdominal pressure.
2 rred in transpulmonary pressure due to intra-abdominal pressure.
3  renal, and cardiac failure due to increased abdominal pressure.
4 o continence during rapid increases in intra-abdominal pressure.
5 s in spine anatomy, or external increases in abdominal pressure.
6 an dysfunction resulting from elevated intra-abdominal pressure.
7 stabilization by indirectly increasing intra-abdominal pressure.
8 phincteric closure during increases in intra-abdominal pressure.
9 ity, was filled with water to increase intra-abdominal pressure.
10 se in pleural pressure with increasing intra-abdominal pressure.
11 easured 30 mins after each increase in intra-abdominal pressure.
12                                  Above intra-abdominal pressure 5 mm Hg, plateau airway pressure incr
13    Addition of sustained continuous negative abdominal pressure (-5 cm H2O) to the positive end-expir
14 cle activity associated with increased intra-abdominal pressure and anal or vaginal EMG probes to obt
15 ntilation in the presence of increased intra-abdominal pressure and in calculating true transpulmonar
16 d cardiac output 5 mins after each new intra-abdominal pressure and positive end-expiratory pressure
17 casionally difficult, resulting in increased abdominal pressure and possible vascular compromise.
18  are necessary to decompress increased intra-abdominal pressure and prevent or treat persistent and r
19 nimize the risk of developing elevated intra-abdominal pressure and to aggressively treat intra-abdom
20 ure is the gold standard for measuring intra-abdominal pressure, and several nonsurgical methods can
21 evated intracranial pressure, elevated intra-abdominal pressure, and therapeutic hypothermia after ve
22                              Increased intra-abdominal pressure appears to produce this effect by aug
23 l as minimization of intrathoracic and intra-abdominal pressure as clinically possible.
24                      Indeed, increased intra-abdominal pressure, as an extreme marker of abdominal co
25                               The mean intra-abdominal pressure at admission day was an independent p
26 as significantly reduced by increasing intra-abdominal pressure at both positive end-expiratory press
27 injured lungs, sustained continuous negative abdominal pressure augmented lung recruitment and oxygen
28                         Four levels of intra-abdominal pressure (baseline, 12, 18, and 22 mm Hg) were
29  degrees of matching the corresponding intra-abdominal pressure: baseline positive end-expiratory pre
30 ominal pressure compared with baseline intra-abdominal pressure) but did not change cardiac output (p
31     We investigated the effect of increasing abdominal pressure by waist belt on reflux in patients w
32 e, urinary incontinence during elevations in abdominal pressure, can be divided into 2 systems: a sph
33                     Spontaneous respiration (abdominal pressure capsule) and ventilator inflations (a
34                      Acutely increased intra-abdominal pressure causes a significant increase in intr
35 g volume (+119% [p < .001] at 22 mm Hg intra-abdominal pressure compared with 5 cm H2O positive end-e
36 g volume (+233% [p < .001] at 22 mm Hg intra-abdominal pressure compared with 5 cm H2O positive end-e
37 % [p < .05], respectively, at 22 mm Hg intra-abdominal pressure compared with baseline intra-abdomina
38                                   High intra-abdominal pressure could enhance the penetration of chem
39 s muscle) activity that would increase intra-abdominal pressure during EAS contraction, and (4) reinf
40 and acute renal failure with regard to intra-abdominal pressure dynamics, preload limitation, and aft
41 otomy negated all effects of increased intra-abdominal pressure except the decreased cardiac index (1
42 01), and intra-abdominal hypertension (intra-abdominal pressure &gt;/= 12 mm Hg) occurred in 19.9%, 20.3
43  to the ICU was associated with higher intra-abdominal pressure, higher plasma C reactive protein con
44                               Elevated intra-abdominal pressure (IAP) is a frequent cause of morbidit
45                               Elevated intra-abdominal pressure (IAP) is associated with ICP elevatio
46 sidered the gold standard for indirect intra-abdominal pressure (IAP) measurements.
47 sought to determine whether changes in intra-abdominal pressure (IAP) with aggressive diuretic or vas
48 be present at relatively low levels of intra-abdominal pressure (IAP).
49 sitive end-expiratory pressure (= half intra-abdominal pressure in cm H2O + 5 cm H2O), and high posit
50 gh positive end-expiratory pressure (= intra-abdominal pressure in cm H2O).
51 in positive end-expiratory pressure or intra-abdominal pressures in both conditions.
52 tudied: a) group 1 (n = 9) animals had intra-abdominal pressure increased to 25 mm Hg above baseline,
53 to intra-abdominal pressure to prevent intra-abdominal pressure-induced end-expiratory lung volume de
54              When compared with normal intra-abdominal pressures, intra-abdominal hypertension increa
55 ive end-expiratory pressure matched to intra-abdominal pressure led to a preservation of end-expirato
56                          Continuous negative abdominal pressure may be a potential adjunct to positiv
57                                    Increased abdominal pressure may have a negative effect on intracr
58  into three subgroups according to the intra-abdominal pressure measurement policy in different years
59                                        Intra-abdominal pressure measurements in predefined at-risk pa
60 redictor of mortality in patients with intra-abdominal pressure measurements started within the first
61                       Whether limiting intra-abdominal pressure measurements to preselected at-risk p
62                                 Serial intra-abdominal pressure measurements, nonoperative pressure-r
63                       At each level of intra-abdominal pressure, moderate positive end-expiratory pre
64   We aimed to clarify whether expanded intra-abdominal pressure monitoring results in an increased de
65                       In patients with intra-abdominal pressure monitoring, the mean intra-abdominal
66 , and 1,241 patients (46.0%) underwent intra-abdominal pressure monitoring.
67     It is now appreciated that elevations of abdominal pressure occur in a wide variety of critically
68                         Application of intra-abdominal pressures of 0 and 20 cm H2O at positive end-e
69 ositive end-expiratory pressure to the intra-abdominal pressure on cardio-respiratory parameters.
70 bdominal pressure monitoring, the mean intra-abdominal pressure on the admission day is an independen
71     The detrimental impact of elevated intra-abdominal pressure, progressing to abdominal compartment
72 ; (4) leak-point pressure, the minimum intra-abdominal pressure required to cause incontinence, which
73 n of transient (15 mins) continuous negative abdominal pressure resulted in comparable and lasting (6
74 , and its resuscitation with increased intra-abdominal pressure results in many untoward hemodynamic
75 this condition, routine measurement of intra-abdominal pressure should be performed in high-risk pati
76 citation, an impedance threshold device, and abdominal pressure (sodium nitroprusside-enhanced cardio
77 why persons with chronically increased intra-abdominal pressure, such as the morbidly obese, suffer f
78 d, blood and protein losses, increased intra-abdominal pressure, systemic hyperthermia, and increased
79                       At each level of intra-abdominal pressure, three levels of positive end-expirat
80  (insertional force) and the transmission of abdominal pressure through the zone of apposition (appos
81                            Increase of intra-abdominal pressure to 25 mm Hg above baseline caused sig
82 he urethra is compressed during increases in abdominal pressure to maintain urethral closure pressure
83           Expanding the measurement of intra-abdominal pressure to more than 50% of intensive care ad
84 ive end-expiratory pressure matched to intra-abdominal pressure to prevent intra-abdominal pressure-i
85 y opening the abdomen, suggesting that intra-abdominal pressure transmission contributes little to co
86 fter PNT, and after SCI+PNT, suggesting that abdominal pressure transmission contributes to continenc
87 eased linearly by ~ 50% of the applied intra-abdominal pressure value, associated with commensurate c
88 y (n = 2) suggested that continuous negative abdominal pressure was an effective adjunct to positive
89 ined, but not transient, continuous negative abdominal pressure was associated with hemodynamic depre
90       Following baseline measurements, intra-abdominal pressure was increased by incrementally inflat
91                                    The intra-abdominal pressure was measured in 31.7%, 55.6%, and 41.
92 roposed is that central obesity raises intra-abdominal pressure, which increases pleural pressure and
93 aparoscopic surgery produces increased intra-abdominal pressure, which potentially influences hepatic
94 ography (OEP), (ii) intra-thoracic and intra-abdominal pressures with a balloon catheter in each comp

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