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1 cterial lipopeptide, and polymicrobial intra-abdominal sepsis.
2  outcome of experimental polymicrobial intra-abdominal sepsis.
3 is graft secondary to bowel injury and intra-abdominal sepsis.
4 Forty animals were randomized, and 1 died of abdominal sepsis.
5 e cecum was ligated and punctured to produce abdominal sepsis.
6 echanism underlying its protective effect in abdominal sepsis.
7 ional activities during the host response to abdominal sepsis.
8 mice underwent a cecal slurry model of intra-abdominal sepsis.
9 ion for an open abdomen (n = 398, 68.9%) was abdominal sepsis.
10 dults with abdominal injury (46.7%) or intra-abdominal sepsis (52.3%) were randomly allocated to the
11 , enteroatmospheric fistula (EAF), and intra-abdominal sepsis/abscess (IAS) are major challenges for
12 led among a multitude of problems, including abdominal sepsis and battlefield surgery.
13  well-accepted model of murine polymicrobial abdominal sepsis and begin characterizing (in the parlan
14 -3, and CIS was transiently increased during abdominal sepsis and temporally associated with the deve
15 vival of mice subjected to various models of abdominal sepsis because of an overwhelming innate immun
16                  Induction of polymicrobial, abdominal sepsis by cecal ligation and puncture.
17      Abscess formation associated with intra-abdominal sepsis causes severe morbidity and can be fata
18 al ligation and puncture (CLP) as a model of abdominal sepsis, followed 24 h later by intratracheal (
19 ncture (CLP), a well-accepted model of intra-abdominal sepsis, followed by daily subcutaneous injecti
20 formation of abscesses associated with intra-abdominal sepsis in humans.
21 inflammation after abdominal injury or intra-abdominal sepsis is associated with poor outcomes.
22                          Using the rat intra-abdominal sepsis model, we found that administration of
23 riction for 3 weeks followed by induction of abdominal sepsis or endotoxemia by intraperitoneal injec
24 is an option following laparotomy for severe abdominal sepsis or trauma.
25 hematologic, and biochemical consequences of abdominal sepsis produced by intraperitoneal implantatio
26                              In murine intra-abdominal sepsis, pulmonary injury cannot be considered
27  severe (n = 21) or non-severe (n = 8) intra-abdominal sepsis; severe (n = 23) or non-severe (n = 21)
28 on of sophorolipids after induction of intra-abdominal sepsis significantly decreases mortality in th
29              These observations suggest that abdominal sepsis syndrome results in significant impairm
30 ponses in the context of pneumonia and intra-abdominal sepsis than wild-type animals.
31 iate abscess formation associated with intra-abdominal sepsis, the role of T-cell activation and the
32 e function aggravates the clinical course of abdominal sepsis via HIF-1alpha- and NF-kappaB-mediated
33                                        Intra-abdominal sepsis was induced in vivo in 166 rats via cec
34                                              Abdominal sepsis was produced in rats by cecal ligation
35                       Using a mouse model of abdominal sepsis, we showed that innate response activat

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