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1 he medial vestibular nucleus and the nucleus abducens).
2 nce of the target nucleus from the accessory abducens.
5 g., oculomotor, trochlear, trigeminal motor, abducens, and vagal motor nuclei) contain protocadherin-
6 fibers travel independent of the trochlear, abducens, and Vidian nerves, but, otherwise, they use th
7 ent allows for the possibility that in vitro abducens conditioning is generated by coincident CS-US d
8 cleus prepositus hypoglossi, all vestibular, abducens, cuneate, and lateral reticular nuclei, labeled
9 s in the physiologic signals conveyed by the abducens internuclear (eye velocity and eye position) an
12 orphology and soma-dendritic distribution of abducens internuclear and ATD synaptic endings are corre
13 pre/postsynaptic membrane profile, both the abducens internuclear and ATD synaptic endings are label
15 of which were VEGF immunopositive, and that abducens internuclear neurons expressed the VEGF recepto
17 haracteristics and synaptology of axotomized abducens internuclear neurons, which mediate gaze conjug
18 motoneurons receive two main pontine inputs: abducens internuclear neurons, whose axons course throug
19 entials in comparison to those evoked by the abducens internuclear pathway as determined electrophysi
20 a synaptogenic compensatory mechanism of the abducens internuclear pathway that could lead to the obs
22 rograde horseradish peroxidase labeling, the abducens internuclear projection is predominantly, if no
25 the excitatory neurotransmitters utilized by abducens internuclear synaptic endings whose burst-tonic
28 had ipsilateral projections terminating near abducens motoneurons and collateralized extensively with
29 tudy investigates whether different types of abducens motoneurons exist that become active during dif
31 re, we studied a possible differentiation of abducens motoneurons into subtypes by evaluating their m
32 equencies and velocities allowed subdividing abducens motoneurons into two subgroups, one encoding th
34 e-stabilizing eye movements are commanded by abducens motoneurons that combine different sensory inpu
35 neurons in turtle respond in the same way as abducens motoneurons to horizontal rotations, an unusual
36 shes inhibitory postsynaptic potentials onto abducens motoneurons within 2 days postinjection, and tr
37 ntralateral projections collateralizing near abducens motoneurons, consistent with a role in disconju
39 d contralateral projections terminating near abducens motoneurons; these cells collateralized extensi
40 f several transcription factors critical for abducens motor neuron identity, including MAFB, or by he
41 l and contralateral eye movements across the abducens motor neuron pool may provide a basis for learn
42 cell type specific antibodies, that somatic abducens motor neurons and a small subset of OPCs arise
43 ry and trigeminal nerve synaptic inputs onto abducens motor neurons are in spatial proximity because
45 ing experiments demonstrated that individual abducens motor neurons receive inputs from both nerves a
46 remain in the cell cycle and fail to produce abducens motor neurons, and OPCs are entirely lacking in
53 eptor 4 (GluR4) AMPA receptor subunit in the abducens motor nuclei, but not with NMDAR1 or GluR1.
56 motor nerves were typically small, with the abducens nerve (cranial nerve [CN]6) often nondetectable
58 random sinusoidal cycles, we stimulated the abducens nerve and observed the resultant eye movements.
60 l correlate of this response recorded in the abducens nerve can be conditioned entirely in vitro usin
61 , we demonstrate that selectively disrupting abducens nerve development is sufficient to cause second
62 d that in vitro classical conditioning of an abducens nerve eye-blink response is generated by NMDA r
63 o imaging of Chn1KI/KI mice revealed stalled abducens nerve growth and selective trochlear and first
65 with subarachnoid CN3 hypoplasia, occasional abducens nerve hypoplasia, and subclinical ON hypoplasia
70 model of the classically conditioned turtle abducens nerve response, we investigated the effect of c
71 t the primary cause of DRS is failure of the abducens nerve to fully innervate the lateral rectus mus
72 mice did not have DRS, and embryos displayed abducens nerve wandering distinct from the Chn1KI/KI phe
73 nce of Listing's law, we microstimulated the abducens nerve with the eye at different initial vertica
74 erized by a failure of cranial nerve VI (the abducens nerve) to develop normally, resulting in restri
79 ysfunction of the oculomotor, trochlear, and abducens nerves and/or the muscles that they innervate.
80 ted individuals demonstrated small or absent abducens nerves in all four, small oculomotor nerve in o
81 rded from pairs of oculomotor, trochlear, or abducens nerves of an in vitro turtle brainstem preparat
82 f left oculomotor, right trochlear and right abducens nerves were approximately aligned with leftward
83 on of motor columns, loss of the phrenic and abducens nerves, and intercostal nerve pathfinding defec
84 interactions in distinct motor neuron pools: abducens neurons use bidirectional ephrin signaling via
85 bilateral horizontal gaze palsy from pontine abducens nuclear defects, rather than abducens nerve inv
86 tion from contralateral facial and accessory abducens nuclei and by their synaptic activation from th
87 an anterograde tracer into the oculomotor or abducens nuclei and combined tracer visualization with i
88 rom the Hoxa1 domain, such as the facial and abducens nuclei and nerves as well as r4 neural crest ce
89 and motoneurons in oculomotor, trochlear and abducens nuclei that dictate eye rotations in terms of t
91 e eyeblink conditioned while their accessory abducens nucleus (ACC), facial nucleus (FN), and surroun
92 anterogradely transported biocytin from the abducens nucleus and the ventral lateral vestibular nucl
95 F neurons located in a region ventral to the abducens nucleus produced 42 significant SpikeTA effects
96 pontine nuclei, vagus nerve, inferior olive, abducens nucleus, and motor trigeminal nucleus; protein
97 ead burst neuron, tonic neuron, interneuron, abducens nucleus, and oculomotor nucleus, is developed t
100 s revealed lateral incomitance suggestive of abducens palsy not detected by clinical examination.
102 ial postoperative follow-up in patients with abducens palsy undergoing IRT shows a significant improv
104 dren, unlike the adults, likely had a subtle abducens paresis rather than divergence insufficiency.
105 ojected to the contralateral and ipsilateral abducens, respectively, and GABAergic neurons projected
107 in corticospinal neurons, exhibited similar abducens wandering that paralleled previously reported g
109 sion of the CR (facial nucleus and accessory abducens) were reversibly inactivated with microinjectio
110 le or muscle unit contraction in response to abducens whole-nerve stimulation or stimulation of singl
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