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1 nerve in DRS is secondary to absence of the abducens nerve.
2 slope of CR acquisition was recorded in the abducens nerve.
4 random sinusoidal cycles, we stimulated the abducens nerve and observed the resultant eye movements.
5 ysfunction of the oculomotor, trochlear, and abducens nerves and/or the muscles that they innervate.
6 on of motor columns, loss of the phrenic and abducens nerves, and intercostal nerve pathfinding defec
8 l correlate of this response recorded in the abducens nerve can be conditioned entirely in vitro usin
11 motor nerves were typically small, with the abducens nerve (cranial nerve [CN]6) often nondetectable
12 , we demonstrate that selectively disrupting abducens nerve development is sufficient to cause second
13 d that in vitro classical conditioning of an abducens nerve eye-blink response is generated by NMDA r
14 o imaging of Chn1KI/KI mice revealed stalled abducens nerve growth and selective trochlear and first
16 with subarachnoid CN3 hypoplasia, occasional abducens nerve hypoplasia, and subclinical ON hypoplasia
17 ted individuals demonstrated small or absent abducens nerves in all four, small oculomotor nerve in o
20 rded from pairs of oculomotor, trochlear, or abducens nerves of an in vitro turtle brainstem preparat
24 model of the classically conditioned turtle abducens nerve response, we investigated the effect of c
25 t the primary cause of DRS is failure of the abducens nerve to fully innervate the lateral rectus mus
26 erized by a failure of cranial nerve VI (the abducens nerve) to develop normally, resulting in restri
27 mice did not have DRS, and embryos displayed abducens nerve wandering distinct from the Chn1KI/KI phe
28 f left oculomotor, right trochlear and right abducens nerves were approximately aligned with leftward
29 nce of Listing's law, we microstimulated the abducens nerve with the eye at different initial vertica
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