ライフサイエンスコーパス: aberrancy

1 tempt to differentiate it from RBBB and LAHB aberrancy.

2 t for IRE1alpha's responsiveness to membrane aberrancy.

3 ey also exhibit morphological and functional aberrancies.

4 tion near the N terminus results in multiple aberrancies.

5 rrest and exhibited 1 to 11 immunophenotypic aberrancies.

6 M) that results in oncogenic transcriptional aberrancies.

7 e investigated the frequency of EGFR pathway aberrancies among African American patients with NSCLC,

8 may arise from transcriptional or chromatin aberrancies and engage prostate tumorigenic mechanisms.

9 d upregulation of aurora kinases, centrosome aberrancies, and aneuploidy.

10 o metaphase chromosome misalignment, spindle aberrancy, and eventual cell death.

11 omena as Wolff-Parkinson-White syndrome, QRS aberrancy, and multiple reentrant pathways that were cru

12 ins unknown, however, whether such molecular aberrancies are causally related to arrhythmogenesis in

13 lones of cells that have any type of somatic aberrancy associated with an increased propensity to bec

14 ation, among others, are among the principal aberrancies driving CRPC growth.

15 al cell populations and recognize phenotypic aberrancies, even when present in a small proportion of

16 Furthermore, tissue aberrancies generated from oncogenic Hras, and even muta

17 Aberrancies in AMPAR-mediated signaling are associated w

18 Aberrancies in IgA1 glycosylation have been linked to th

19 studies have addressed intestinal microbiota aberrancies in pediatric CD, but the results are inconsi

20 and hence no indication of nonsense-mediated aberrancies in splicing.

21 such diseases is to identify characteristic aberrancies in the biological pathways and molecular net

22 The majority of human malignancies have aberrancies in the Retinoblastoma (Rb) pathway.

23 Our studies further suggest that aberrancies in this delicate degradation pathway might c

24 rtant role of DNA methylation in DLBCL where aberrancies in transcription factors were frequently obs

25 We confirmed the same aberrancy in IgA1 secreted by the human DAKIKI B cell li

26 nous IRE1alpha protein to recognize membrane aberrancy in mammalian cells.

27 systemic lupus erythematosus (SLE) involves aberrancy in multiple components of the immune system in

28 on-mediated PCR) assay and found significant aberrancy in promoter methylation patterns compared with

29 Waf1/Cip1) showed normal development with no aberrancy in their cell cycles, and antisense p21(Waf1/C

30 ation that isolated the response to membrane aberrancy induced by palmitate from unfolded protein str

31 omes the multitude of genetic and epigenetic aberrancies inherent in human cancers to restore normal

32 The most frequent aberrancy is the "common deletion" that involves a 4,977

33 review of the mechanics of the UPS, describe aberrancies leading to cancer, and give an overview of c

34 is unknown, but is critical, as hemodynamic aberrancies may cause severe clinical deterioration.

35 ms and insights into ways in which molecular aberrancies occur are becoming better understood through

36 ic lethal, whereas haploinsufficiency caused aberrancies of adult skin, but not a frank cEDS-like phe

37 Unlike in primary MDS, aberrancies of chromosome 5 and 20 were infrequent.

38 ndrogen synthesis as well as the most common aberrancies of the AR confirm that prostate cancer retai

39 ptor (EGFR) -targeting agents and in genetic aberrancies of the EGFR pathway according to ethnic back

40 thesis and biological methylation reactions, aberrancies of which have all been implicated in carcino

41 Certainly, epigenetic aberrancies play a fundamental role in propagation of th

42 Aberrancy produced via tryptophan depletion results in a

43 N-glycan content and the timing of N-glycan aberrancy relative to disease onset were compared in hea

44 alous arterial patterning may cause vascular aberrancies that often require surgical treatment.

45 Glycosylation aberrancy was comparable in patients with and those with

46 ly differentiating LPF-VT from RBBB and LAHB aberrancy was developed.

47 The growth aberrancy was normalized after restoring the distal enha

48 QT duration, were unchanged, although minor aberrancies were noted in 20% (16/80) of the G628S mice

49 cell-cycle control can lead to oncogenesis, aberrancies within the UPS pathway can result in a malig

50 E1alpha also directly recognize the membrane aberrancy wrought by lipid perturbation.