1 ing alterations of PrPM: 1) expression of an
aberrant form lacking the glycosylphosphatidylinositol a
2 The nature of these particles and of other
aberrant forms made in the absence of scaffold demonstra
3 in reduced acetylation of ER proteins and an
aberrant form of autophagy.
4 th BRAF(V600E) coupled with expression of an
aberrant form of BRAF(V600E) that retains RAF pathway de
5 r the concept that these tumors represent an
aberrant form of follicle organogenesis, and uncover pot
6 Herein, an
aberrant form of the hepatic secretory protein alpha1-an
7 To examine the fate of
aberrant forms of a well characterized, disulfide-bonded
8 is review, we present emerging evidence that
aberrant forms of condensates are associated with many h
9 Alcohol abuse leads to
aberrant forms of emotionally salient memory, i.e., limb
10 d Ig-like transcript receptors may recognize
aberrant forms of HLA-B27.
11 We also confirm the inability of
aberrant forms of LOS to activate Toll-like receptor 2 (
12 neurodegenerative diseases characterized by
aberrant forms of tau protein accumulation leading to ne
13 Aberrant forms of the anaplastic lymphoma kinase (ALK) h
14 that high-dose IFN-gamma treatment elicited
aberrant forms of the bacterium.
15 h harbors truncated proviruses and expresses
aberrant forms of the Gag protein.
16 nal MATa1 product and to avoid production of
aberrant forms of the repressor.
17 In vitro,
aberrant forms of the WNT1 protein showed impaired capac
18 e pathways contribute to the surveillance of
aberrant forms of these transcripts.
19 Our results support a hypothesis that
aberrant forms of U1-70K in AD can directly sequester so
20 esult, HLA-A2 transcripts are spliced to two
aberrant forms,
one with exon 2 skipping and the other w
21 Aberrant forms with reduced infectivity were also induce