ライフサイエンスコーパス: ability to proliferate

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1 ssociated with liver dysfunction and reduced ability to proliferate.

2 ese NK cells were severely impaired in their ability to proliferate.

3 ced functional properties, they retained the ability to proliferate.

4 s suppressed, and some cells recovered their ability to proliferate.

5 roblasts from Id2-null mice display impaired ability to proliferate.

6 tissue culture and was more crippled in its ability to proliferate after invasion.

7 ity of memory T cell pools in terms of their ability to proliferate and accumulate at effector sites

8 veloping or regenerating organs, such as the ability to proliferate and alter tissue organization.

9 e comparable in number to the control, their ability to proliferate and become activated to form a ha

10 To identify cells that have the ability to proliferate and differentiate into all epithe

11 an neoblasts morphologically and share their ability to proliferate and differentiate into derivative

12 In addition, their ability to proliferate and differentiate into plasma cel

13 o these aberrant stem cells, impairing their ability to proliferate and differentiate.

14 opreserved and, when recovered, retain their ability to proliferate and differentiate.

15 o, class Ib-restricted memory CTL retain the ability to proliferate and expand when provided with Ag

16 e subset of CD4(+) T cells that retained the ability to proliferate and express IL-2.

17 earlier stages of somite maturation than the ability to proliferate and form muscle tissue.

18 vitro revealed that mSOD1 SVZa cells had the ability to proliferate and form neurospheres but had an

19 f cancer cells that differ markedly in their ability to proliferate and form new tumors.

20 (ARF) induction, but irreversibly lost their ability to proliferate and initiate follicle growth.

21 rom dedifferentiated cells have acquired the ability to proliferate and lost the NE-like cell propert

22 ptors, as found in primary RGCs, this line's ability to proliferate and non-neuronal appearance diffe

23 CHB have more HBV-specific T cells with the ability to proliferate and produce cytokines than adult

24 evel, the anergic splenic T cells regain the ability to proliferate and produce IFN-gamma when stimul

25 .AND and B10.AND mice were impaired in their ability to proliferate and produce IL-2 after challenge

26 spite the absence of full lactation or their ability to proliferate and produce progeny with diverse

27 mAPL:S-palmAPL-primed cells show an enhanced ability to proliferate and produce the anti-inflammatory

28 reserved donor cells were compared for their ability to proliferate and replace damaged parenchyma.

29 sses not only progenitor-like qualities (the ability to proliferate and repopulate a mammary gland) a

30 other contaminating cells, they regained the ability to proliferate and secrete cytokines.

31 tudy also suggests that SsPV1 has a vigorous ability to proliferate and spread via hyphal contact.

32 gardless of age, KLRG1(+) cells retained the ability to proliferate and survive in response to homeos

33 of the hallmarks of leukemic cells is their ability to proliferate and survive in the absence of exo

34 ls recovered from septic mice retained their ability to proliferate and synthesize cytokines albeit a

35 marks of stem and progenitor cells are their ability to proliferate and to give rise to functional pr

36 nto FACS-sorted TLX-null cells rescues their ability to proliferate and to self-renew.

37 that the surface-modified hMSCs retain their ability to proliferate and to undergo multilineage diffe

38 facultative progenitor cells based on their ability to proliferate and trans-differentiate into type

39 f signal transduction proteins, a diminished ability to proliferate, and a decreased production of cy

40 ells that are larger in size, have a limited ability to proliferate, and do not produce MT-1-MMP, the

41 ein are unable to differentiate, acquire the ability to proliferate, and invade the extracellular mat

42 double-positive (DP) cells have a diminished ability to proliferate, and that these DP thymocytes up-

43 tivated solely through TCR ligation lose the ability to proliferate as a result of autocrine IL-2 pro

44 t, cortical cells from the H-Tx rat have the ability to proliferate as normal.

45 Old CD8(+) T cells showed reduced ability to proliferate, but blockade of B7-H1 restored t

46 restimulation, these T cells showed reduced ability to proliferate, confirming a state of T cell ane

47 Due to their ability to proliferate, deregulated NSCs or their progen

48 ys of embryonic stem cells and thus gain the ability to proliferate, differentiate and alter cell-cel

49 Because of their ability to proliferate, differentiate into endothelial c

50 r complexes, are partially impaired in their ability to proliferate during MCMV infection, display di

51 these cells are functional in terms of their ability to proliferate, express cytolytic activity, and

52 op a preactivated phenotype and an intrinsic ability to proliferate faster upon stimulation, allowing

53 er n-butyrate in primary cultures lose their ability to proliferate in Ag-stimulated secondary cultur

54 affords E(2)-dependent MCF-7 cells with the ability to proliferate in E(2)-depleted media.

55 TCL cell line (HUT78R), characterized by its ability to proliferate in high concentration of recombin

56 nii clinical isolates were evaluated for the ability to proliferate in human serum.

57 ones demonstrated a dose-dependent, enhanced ability to proliferate in low serum conditions, compared

58 ong-lasting, but reversible defects in their ability to proliferate in lymph nodes and secrete IL-2 a

59 nt, the ability to produce IFN-gamma, or the ability to proliferate in response to Ag in vitro.

60 xic effector function, are impaired in their ability to proliferate in response to Ag-specific stimul

61 with the donor or the intrinsically reduced ability to proliferate in response to alloantigens.

62 Finally, pleural cells were examined for the ability to proliferate in response to concanavalin A and

63 ate antigen become activated and acquire the ability to proliferate in response to cytokines.

64 and reduced in number, and show a decreased ability to proliferate in response to different growth f

65 When tested for their ability to proliferate in response to GM-CSF, only chime

66 The cells' ability to proliferate in response to growth factor stim

67 II-specific T cells demonstrated a decreased ability to proliferate in response to the CII immunodomi

68 t cells were functionally selected for their ability to proliferate in serum-free, EGF-free medium.

69 in an undifferentiated state, based on their ability to proliferate in suspension, as nonadherent mam

70 yme itself, is responsible for the increased ability to proliferate in the absence of growth factors.

71 peptides, murine T cells were tested for the ability to proliferate in vitro and antibody responses t

72 Here we identified ccRCC lines whose ability to proliferate in vitro and in vivo is sensitive

73 l mutants in L. donovani and evaluated their ability to proliferate in vitro and trigger infections i

74 n IL-2/CD25 and CD40-CD40L pathways, and the ability to proliferate in vitro in response to anti-CD3

75 The long-time proliferating cells' ability to proliferate in vivo for an extended period of

76 vivo, but Uqcrfs1(-/-) T cells retained the ability to proliferate in vivo under lymphopenic conditi

77 Immortalization, the acquisition of the ability to proliferate indefinitely, was also affected b

78 A hallmark of cancer cells is the ability to proliferate indefinitely.

79 The ability to proliferate independently of signals from oth

80 apability of tumor cells to invade and their ability to proliferate indicate an emergent behavior.

81 d from human cervical cancers inhibits their ability to proliferate, indicating that the expression o

82 inally differentiated myocytes have lost the ability to proliferate, indicating the existence of a do

83 istics of MCF7F-B5 cells by increasing their abilities to proliferate, migrate, and invade in vitro.

84 progenitors can respond to injury, but their ability to proliferate, migrate, differentiate, and surv

85 nts' cells transduced with JAK3 acquired the ability to proliferate normally in response to IL-2.

86 ytes must replicate the complex function and ability to proliferate of primary human hepatocytes.

87 ta-catenin in IPF fibroblasts inhibits their ability to proliferate on collagen.

88 on the intestinal stem cells regulates their ability to proliferate or to undergo apoptosis.

89 n naive CD8+ T cells without affecting their ability to proliferate or up-regulate activation markers

90 intestinal and respiratory tracts retain the ability to proliferate postnatally, which enables adapti

91 ndothelial cells are viable and retain their ability to proliferate, produce collagen VIII, and expre

92 onal exhaustion of T cells, during which the ability to proliferate, secrete cytokines, and mediate l

93 They retained their ability to proliferate, secrete IFN-gamma, and lyse targ

94 B knockout (p50-/-) mice to determine their ability to proliferate, secrete Ig, express germ-line CH

95 Mammalian cardiomyocytes lose the ability to proliferate shortly after birth, and further

96 are terminally differentiated and lose their ability to proliferate shortly after birth; however, in

97 ature T cell phenotypes are present, had the ability to proliferate to GAD peptides.

98 n anergic, Lyn deficiency does restore their ability to proliferate to LPS.

99 promoter were generated and tested for their ability to proliferate under conditions where SUAP expre

100 nding mutant in cancer cells decreased their ability to proliferate under magnesium-deprived situatio

101 F-alpha/c-myc hepatocytes rapidly lose their ability to proliferate upon mitogenic stimulation follow

102 hat the responding clonotypes have a similar ability to proliferate, which is independent of TCR beta

103 D2 in diploid cells strongly potentiated the ability to proliferate with increased DNA content despit

104 , Smad2P, as well as Smad4, indicating their ability to proliferate within a microenvironment that co

105 As an opportunistic pathogen, the ability to proliferate within and disseminate from subgi

106 On the other hand, ATL cells acquire the ability to proliferate without Tax by intracellular gene

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