1 t Plectropomus leopardus--has partner-choice abilities comparable to those of chimpanzees in the cont
2 To date, no H7 viruses have acquired the ability for sustained transmission among humans.
3 s study demonstrates an array of interfacing abilities of the CLEAR device and its utility for neural
4 n the growth medium selectively inhibits the ability of bacteriophage T7 to infect Escherichia coli b
5 alpha-actinin-binding activity abolished the ability of CLP36 to promote breast cancer cell migration
6 ibody potency and was used to anticipate the ability of each antibody to effectively bind and neutral
7 Based on the ability of F. tularensis to resist high ROS/RNS levels,
8 At ~10 microM all four APs impaired the ability of HBMECs to reduce MTT which was followed by de
9 4 and MERS-CoV RBD interaction predicted the ability of MERS-CoV to bind the DPP4s of camel, goat, co
10 rvations in human brain, we believe that the ability of microglia to provide neuroprotection deterior
11 have been a number of articles reporting the ability of microRNAs to help evaluate patients with nonm
12 Inbreeding reduced the ability of plants to up-regulate resistance traits follo
13 The ability of Salmonella to cause disease depends on two ty
14 The ability of sensory systems to adapt to static inputs all
15 Determining if the ability of SGHBV envelope proteins to cause the formatio
16 The ability of the 4th-generation Determine combo to detect
17 sion of CD1d on B cells is suggestive of the ability of these cells to present Ag to, and form cognat
18 This commentary focuses on the ability of Toll-like receptors (TLRs), specifically TLR4
19 asingly confined to this state, with limited ability to adapt perfusion in response to a challenged e
20 These include improved ability to analyze highly rearranged genomes using a var
21 Cancer cells with extra centrosomes have the ability to cluster the extra centrosomes and divide in a
22 locks is of particular interest due to their ability to confine visible light.
23 nding of RDS-associated pathobiology and our ability to design rational treatment strategies.
24 There is a major deficit in our ability to detect and predict the clinical behavior of p
25 hown that this propensity translates into an ability to detect one's own relatives, one of the key pr
26 is shown in simulation studies to affect our ability to discover true correlations to a small extent.
27 nto damaged mitochondria and blocks parkin's ability to drive mitophagy, whereas reducing USP30 activ
28 cing (HTS) technologies have transformed our ability to examine antigen receptor repertoires at singl
29 Attracted by the ability to functionalize molecules directly at ostensibl
30 but not Hut78 cells, demonstrating selective ability to induce apoptosis in macrophages.
31 rast, pre-silencing of Ash2L abrogated P53's ability to induce the expression of these transcriptiona
32 I propose that closing the gap in our ability to interpret variation responsible for Mendelian
33 The ability to link systems together such that they behave a
34 Here we show the ability to move this strain soliton with a perpendicular
35 mability of transcriptional switches and the ability to obtain predictive dynamical systems in a cell
36 that incorporate biomarkers may improve our ability to predict the occurrence of thromboembolism in
37 ir intracellular distributions, and in their ability to promote formation of the [PSI(+)] prion.
38 neficial HNO effects depend, in part, on its ability to release calcitonin gene-related peptide (CGRP
39 rion activities in yeast also abrogate their ability to signal in mammalian cells.
40 Their ability to stabilize highly abundant histone proteins in
41 This memory confers upon a plant the ability to subsequently activate defenses more robustly
42 f virions containing ssDNA is related to its ability to support high levels of virion production or i
43 This is due to its ability to survive and multiply in the bloodstream and i
44 tions and soft, active devices for which the ability to sustain large deformations is required.
45 e of LGP2 interference is suppression of its ability to synergize with MDA5.
46 , methodological challenges have limited our ability to test the degree to which this assumption hold
47 in gene therapy, not least because of their ability to transduce neurons following their distal axon
48 eins as noise suppressors and demonstrate an ability to uncouple signal and noise in a prototypical s
49 tion, progressor cells displayed a decreased ability to upregulate membrane-associated caspase-8 acti
50 The ability to visualize and manipulate cell fate and gene e
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