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1 immunity against bacteriophages by inducing abortive infection.
2 as reverse transcription (RT), resulting in abortive infection.
3 s induces substantial cell death, leading to abortive infection.
4 on were evident in infected pDCs, indicating abortive infection.
5 DNA injection, restriction/modification, and abortive infection.
6 oci and resistance to bacteriophages through abortive infection.
7 inefficiently completed, which results in an abortive infection.
8 on with the viral capsid protein, leading to abortive infection.
9 ial sites of infection, potentially limiting abortive infections.
10 ication systems (R-M) (Tock & Dryden, 2005), abortive infection (Abi) (Chopin et al, 2005), Argonaute
11 iction and modification (R/M) system and the abortive infection (Abi) mechanism, AbiR, that impedes b
13 ding restriction-modification systems (R-M), abortive infection (Abi), Argonaute-based interference,
14 rabbit cells tested, vMyxM062-KO conducts an abortive infection, although it initiates viral DNA repl
15 arge fraction of infected cells dies through abortive infection and has a half-life of approximately
16 the productive growth cycle, resulting in an abortive infection and radically restricting viral repli
17 ative estimates of parameters characterizing abortive infection and support the notion that abortive
18 pression to establish quiescence and prevent abortive infection and that the virus usurps a Daxx-medi
21 of RabA2 resulted in an increased number of abortive infection events, including bursting of ITs and
23 e tissue die through pyroptosis triggered by abortive infection, i.e., infection of resting T cells i
28 replication-competent MCMV vector underwent abortive infection in human DC; this was accompanied by
29 ecimens from atypical lesions may produce an abortive infection in limited cell lines and a cytopathi
30 us mutants and other poxviruses that undergo abortive infection in mammalian cells are receiving spec
33 ubules near masses of dense viroplasm during abortive infection in the absence of the A17 or A14 prot
38 we analyzed the role of both proteins in the abortive infection of human HeLa cells with the poxvirus
39 by replication in hepatocytes and not by the abortive infection of Kupffer cells and the following cy
41 levels, and that SAMHD1 expression promotes abortive infection of this important memory cell subset.
42 defense strategy: that both restriction and abortive infections operate during coevolution with phag
43 ves the infected cell but rather enforces an abortive infection pathway leading to infected cell deat
44 ortive infection and support the notion that abortive infection represents an important mechanism und
46 e system is a genetically engineered form of abortive infection that traps and eliminates phages pote
49 e adsorption inhibition, injection blocking, abortive infection, toxin-antitoxin, and CRISPR-Cas syst
51 rity of CD4(+) T cells in tissue die through abortive infection, where the accumulation of incomplete
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