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1 Further discs were alumina-air-abraded.
2 Two hundred forty porcelain discs were air-abraded.
3 LR3(-/-), TRIF(-/-), and MyD88(-/-) mice was abraded and stimulated with the synthetic TLR3 ligand po
5 LR2(-/-), TLR9(-/-), and MyD88(-/-) mice was abraded and treated with Pam(3)Cys, LPS, or CpG DNA, whi
7 helium of C57BL/6 and gene knockout mice was abraded, and 1 x 10(7) S. marcescens were added in the p
8 ium of BALB/c, C3H/HeJ, and C3H/HeN mice was abraded, and Pseudomonas aeruginosa endotoxin (10 microg
12 fected PA-III cells were inoculated over the abraded calvaria of nude mice, large tumors formed with
13 and a 2-mm diameter punch was placed on the abraded corneal epithelium of either untreated or cyclop
15 Using a murine model of keratitis in which abraded corneas are infected with P. aeruginosa parent a
20 ined decussating enamel rods but was rapidly abraded following weaning, despite the mice being mainta
25 spores or vegetative bacilli onto intact or abraded mouse flank skin, followed by evaluation of the
29 ealed that in the respirable fraction of the abraded particles, approximately 4000 ppm of the MWCNTs
36 layers of approximately 10 microns each were abraded simultaneously with reference slabs of sound ena
38 mas as early as 3 weeks after application to abraded skin on the muzzles and tails of athymic NCr nud
39 1 X 10(7) B. anthracis (Sterne) spores onto abraded skin or injected with the spores intradermally o
41 curs through contact of mucous membranes and abraded skin with freshwater contaminated by pathogenic
46 R4(-/-), TLR9(-/-), and MyD88(-/-) mice were abraded using a trephine and epithelial brush and were e
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