ライフサイエンスコーパス: abruption

1 l-for-gestational-age infants, and placental abruption).

2 e pregnancy may benefit from reduced risk of abruption.

3 ng may influence the recurrence of placental abruption.

4 of the fetal membranes (PPROM) to placental abruption.

5 lowest) had a 25-fold higher mortality with abruption.

6 y that is punctuated by microbial population abruptions.

7 mbranes (23%), preeclampsia (18%), placental abruption (11%), cervical incompetence (5%), and fetal i

8 Risk factors for abruption among singleton and twin births, respectively,

9 contrast risk factor profiles for placental abruption among singleton and twin gestations.

10 tion between cigarette smoking and placental abruption and a weak association with placenta previa bu

11 ed women were at increased risk of placental abruption and cesarean delivery, and their infants were

12 Placental abruption and excess thrombin generation elicit preterm

13 We examined the association between abruption and newborn outcomes.

14 Risks of mortality among abruption and nonabruption births were 102.7 and 6.2 per

15 and our knowledge of the association between abruption and perinatal and neonatal outcomes.

16 The individual effects of placental abruption and placenta previa on the risk of SIDS did no

17 n and pregnancy complications (eg, placental abruption and preeclampsia), which increase the risk of

18 m labor, preterm membrane rupture, placental abruption, and cervical insufficiency) and abnormal plac

19 or premature rupture of membranes, placental abruption, and cervical insufficiency.

20 outcomes of preterm birth, PPROM, placental abruption, and pre-eclampsia aggregate in families, whic

21 7.5-8.8) for preterm birth, PPROM, placental abruption, and pre-eclampsia, respectively).

22 .4-4.8), for preterm birth, PPROM, placental abruption, and pre-eclampsia, respectively).

23 ture rupture of membranes (PPROM), placental abruption, and pre-eclampsia.

24 as preterm birth, preeclampsia and placental abruption, are common, with acute and long-term complica

25 on (aRR, 1.85; 95% CI, 1.43-2.29), placental abruption (aRR, 1.68; 95% CI, 1.18-2.38), induction (aRR

26 ry was substantially increased even for mild abruptions (aRR for 25% separation, 5.5; 95% CI, 4.2-7.3

27 ivity analyses, there was a direct effect of abruption associated with increased neonatal risks.

28 ssion in term decidual cells may explain how abruption-associated PPROM promotes decidual neutrophil

29 ses that can degrade extracellular matrix in abruption-associated PPROM, we examined whether decidual

30 decidual neutrophil infiltration complicates abruption-associated PPROM.

31 Thus, abruption-associated PTD is initiated by functional prog

32 assessed via immunohistochemical staining in abruption-associated PTD versus gestational-age matched

33 p-ERK1/2, and is thus one pathway initiating abruption-associated PTD.

34 atal mortality was 119 per 1,000 births with abruption compared with 8.2 per 1,000 among all other bi

35 performed in placentas obtained after overt abruption (decidual hemorrhage) with or without PPROM an

36 Placental abruption (early separation of the placenta) is associat

37 Because abruptions elicit intense decidua-enhanced thrombin prod

38 hese pathways collectively lead to placental abruption, fetal demise, and female sterility, thereby p

39 In the abruption group, 14.3% of neonates were growth restricte

40 In this cohort, placental abruption had a profound impact on stillbirth, preterm d

41 emoval of women with HCA-positive placentas, abruption, hypertensive disorders, or obesity.

42 pertension (RR = 2.34) were risk factors for abruption in singleton births but not in twin births.

43 rs found that, among women without placental abruption in the first pregnancy, smoking was associated

44 and second pregnancies and risk of placental abruption in the second pregnancy.

45 moking was associated with increased risk of abruption in the second pregnancy; however, this effect

46 This suggests that abruption in twins may result from different pathophysio

47 in production, we examined the regulation of abruption-induced neutrophil infiltration.

48 xaminations, including evidence of placental abruption, infarction, hypoxia, decidual vasculopathy, o

49 Placental abruption is an uncommon obstetric complication associat

50 The observation that the recurrence of abruption is substantially increased regardless of chang

51 The authors conclude that abruption is twice as likely to occur in twins as in sin

52 n and abruption suggests that the origins of abruption lie at least in midpregnancy and perhaps even

53 ot related to stillbirth caused by placental abruption, obstetric conditions, or infection.

54 Following blunt trauma abruption of the placenta is the more common cause of fe

55 irect (preterm delivery-mediated) effects of abruption on mortality were 10.18 (95% confidence interv

56 to examine the extent to which the effect of abruption on perinatal mortality is mediated through pre

57 of mothers of controls had either placental abruption or placenta previa during the index pregnancy.

58 ine whether placental abnormality (placental abruption or placental previa) during pregnancy predispo

59 rth due to placental dysfunction, defined as abruption or unexplained stillbirth associated with grow

60 ), in the absence of preeclampsia, placental abruption, or fetal growth restriction.

61 (SGA) newborn (<10th percentile), placental abruption, or pregnancy loss >20 weeks.

62 high risk of perinatal death associated with abruption persisted.

63 he distinct pattern of results for placental abruption, placenta previa, and uterine bleeding of unkn

64 ing as a potential risk factor for placental abruption, placenta previa, and uterine bleeding of unkn

65 m ITs; GR was higher in IT than DCs, with no abruption-related changes in either cell type; p-ERK1/2

66 smoked had a twofold increase in the risk of abruption (relative risk = 2.05, 95% confidence interval

67 confidence interval: 6.4, 9.8) and placental abruption (relative risk = 6.6, 95% confidence interval:

68 Pregnancies complicated by abruption result in increased frequency of perinatal dea

69 daily demonstrated a dose-response trend for abruption risk in singletons and twins.

70 on with number of cigarettes smoked daily on abruption risk.

71 he link between fetal growth restriction and abruption suggests that the origins of abruption lie at

72 her cell type; p-ERK1/2 was higher in DCs in abruption than control decidua, with total ERK 1/2 uncha

73 almost nine times as likely to be born with abruption than those in the heaviest (> or =90%) birth w

74 Among women with a prior abruption, the risk of repeating abruption was increased

75 unostaining for PR was lower in DC nuclei in abruption versus control decidua and was absent from ITs

76 The incidence of abruption was 1 % (n = 530).

77 Incidence of abruption was 1.6% (n = 3,619).

78 Abruption was associated with an 8.9-fold (95% confidenc

79 Abruption was associated with an elevated risk of newbor

80 The high mortality with abruption was due, in part, to its strong association wi

81 ith a prior abruption, the risk of repeating abruption was increased irrespective of smoking habits.

82 Placental abruption was indicated in 9.9 per 1,000 pregnancies, wh

83 Abruption was more likely to occur among smokers with ch

84 Abruption was recorded in 5.9 per 1,000 singleton births

85 Abruption was recorded in 6.5 per 1,000 births.

86 rth proportions among women with and without abruption were 39.6% and 9.1 %, respectively, yielding a

87 ery; 55% of the excess perinatal deaths with abruption were due to early delivery.

88 Direct effects attributable to abruption were examined by conditioning on intermediates

89 Abruptions were associated with a marked decidual neutro

90 eath, fetal growth restriction, or placental abruption who had been referred within the 12th gestatio

91 The authors explored the associations of abruption with fetal growth restriction, preterm deliver

92 tocytes, vacuolar changes, and mitochondrial abruption, with absence of anoikic nuclei.