1 bsorption of PV sera with rPX-His eliminated acantholytic activity, and eluted antibody immunoprecipi

2  to the preabsorbed PV IgG fraction restored acantholytic activity, indicating that acantholysis in P

3         Caspase activation may contribute to acantholytic blister formation in PF.

4  after passive transfer of PV mAbs; however, acantholytic cells retain cell surface Dsg3 compared wit

5 uding scabies, bullous pemphigoid, transient acantholytic dermatosis, and mycosis fungoides, and shou

6                             We conclude that acantholytic dyskeratotic naevi can arise from a somatic

7 we investigated the involvement of ATP2A2 in acantholytic dyskeratotic naevi following Blaschko's lin

8                             As patients with acantholytic dyskeratotic naevi often give a history of

9  reflect genetic mosaicism and that the term acantholytic dyskeratotic naevus might be replaced in th

10 mmunosuppression, MP may exhibit direct anti-acantholytic effects in epidermis, and we compared the e

11                         Therefore, the early acantholytic events are triggered by non-Dsg antibodies.

12                 Rarely, epidermal naevi show acantholytic histology similar to Darier's disease, a do

13       On biopsy, this papule was an invasive acantholytic squamous cell carcinoma.