ライフサイエンスコーパス: acellular

1 llularization of whole organs for generating acellular 3D scaffolds with preserved ECM protein conten

2 High levels of free acellular adult hemoglobin (free HbA) are associated wit

3 sport due to loading using a model system of acellular agarose disks and dextran in phosphate-buffere

4 from negative pressure dressing therapies to acellular allograft meshes.

5 oot and periodontal apparatus, including the acellular and cellular cementum, periodontal ligament (P

6 ues can be grown from banked cells, rendered acellular, and then used for tissue regeneration in vivo

7 osols as measured by the dichlorofluorescein acellular assay but not by the uric acid, ascorbic acid,

8 nicotinamide were evaluated using a panel of acellular assays and lipopolysaccharide-stimulated RAW 2

9 Additionally, in acellular assays, peptides were cross-linked in the pres

10 Acellular B. pertussis vaccines were not efficiently pro

11 d, lymphocyte phenotyping was performed, and acellular BAL fluid, plasma HIV RNA load, and BAL cell a

12 Acellular biological scaffolds, or decellularized extrac

13 Injectable, acellular biomaterials hold promise to limit left ventri

14 16s RNA gene sequencing was performed on acellular bronchoalveolar lavage (BAL) fluid from 30 sub

15 mass spectrometry based proteome analysis of acellular bronchoalveolar lavage (BAL) fluid samples on

16 By evaluating both upper airway and acellular bronchoalveolar lavage samples from 49 subject

17 We have previously demonstrated that acellular bronchoalveolar lavage samples from half of th

18 dard for OCS); (2) whole blood (WB); and (3) acellular buffered dextran-albumin solution (analogous t

19 ssive normal cell destruction, leading to an acellular (but matrix filled) gap between the tumour and

20 E35, the optic chiasm and optic tract remain acellular, but the latter contains radial processes with

21 and morphological characteristics including acellular capillaries (AC) and pericyte loss (PL), vesse

22 d Schiff to determine pericyte loss (PL) and acellular capillaries (AC), and TUNEL assays were perfor

23 e effect of reduced Cx30.2 on development of acellular capillaries (ACs) and pericyte loss (PL) was s

24 (-/-), and LXRalpha/beta(-/-) mice developed acellular capillaries and EPC dysfunction similar to the

25 Retinas were evaluated for number of acellular capillaries and glial fibrillary acidic protei

26 cell activation were analyzed by quantifying acellular capillaries and glial fibrillary acidic protei

27 Acellular capillaries and pericytes were counted in reti

28 TZ/WD mice, GW3965 treated mice showed fewer acellular capillaries and reduced GFAP expression.

29 r mm(2)) the diabetes-associated increase of acellular capillaries and the increase of infiltrating i

30 Eyes were enumerated for acellular capillaries and were stained for oxidative dam

31 al digestion using trypsin was performed and acellular capillaries enumerated.

32 ta activity and an increase in the number of acellular capillaries in diabetic mouse retinas, which w

33 The number of acellular capillaries increased threefold, and nitrotyro

34 apoptosis, formation of pericyte ghosts, and acellular capillaries were measured.

35 , including vascular tortuosity, obliterated acellular capillaries, and pericyte ghosts.

36 how earlier onset and an increased number of acellular capillaries, sustained gliosis, and increased

37 ina, pericyte apoptosis and the formation of acellular capillaries, the most specific vascular pathol

38 d retinal vascular preparations, to quantify acellular capillaries.

39 gnaling in vascular cells or the presence of acellular capillaries.

40 oss but no difference in pericyte density or acellular capillaries.

41 icant increases in the number of degenerate (acellular) capillaries and pericyte ghosts were measured

42 ficant increase in the number of degenerate (acellular) capillaries in diabetic animals.

43 subnormal retinal thickness and supernormal acellular capillary density.

44 ovascular cell apoptosis; pericyte ghost and acellular capillary development was inhibited by FOXO1 s

45 k-end labeling positivity and also prevented acellular capillary formation (P < 0.05).

46 r inhibition of ASM activity by DHA prevents acellular capillary formation.

47 romised retinal vasculature, as indicated by acellular-capillary formation and pericyte loss.

48 human scale and obtained biocompatible human acellular cardiac scaffolds with preserved extracellular

49 ecrease in eruption rate is due to a lack of acellular cementum and associated defective periodontal

50 Nonsignificant changes in acellular cementum did not appear to affect periodontal

51 In Phospho1(-/-) mice, acellular cementum formation and mineralization were una

52 nd cellular cementum, further revealing that acellular cementum formation is not substantially regula

53 and SEM revealed a significant reduction in acellular cementum formation on Bsp (-/-) mouse molar an

54 ggest that BSP plays a non-redundant role in acellular cementum formation, likely involved in initiat

55 periodontal tissue breakdown, with a lack of acellular cementum leading to periodontal ligament detac

56 utant mouse molars revealed 4-fold increased acellular cementum thickness ( P = 0.002) and 5-fold inc

57 short molar roots with thin dentin, lack of acellular cementum, and osteoid accumulation in alveolar

58 phosphate (PP(i)) and a severe deficiency in acellular cementum.

59 Our method yields highly aligned, acellular collagen constructs with predictable microstru

60 -1beta; 100 ng) was control released into an acellular collagen sponge cube with underlying ASCs, MSC

61 gh all cell types randomly migrated into the acellular collagen sponge cube, TGF-beta3 and/or SDF-1be

62 Human PMN transmigration across acellular collagen-coated filters toward the bacterial c

63 biogeochemical cycling of both cellular and acellular community components.

64 ructs was significantly greater than that of acellular constructs after 1 and 3 months.

65 Cellular and acellular constructs were implanted subcutaneously in th

66 Of these 37, 30 (81%) had implantation of acellular dermal allograft (ADA) and 7 (19%) implantatio

67 Acellular dermal matrices have many current and potentia

68 tained with a coronally positioned flap plus acellular dermal matrix (ADM) allograft to that of a tun

69 controlled clinical trial was to compare two acellular dermal matrix (ADM) materials produced by diff

70 can be treated by various methods, including acellular dermal matrix (ADM) or coronally advanced flap

71 ntation using bone allograft covered with an acellular dermal matrix (ADM) or polytetrafluoroethylene

72 of bilateral recession defects treated with acellular dermal matrix (ADM) with and without recombina

73 a coronally positioned tunnel (CPT) plus an acellular dermal matrix allograft (ADM) to that of a CPT

74 Acellular dermal matrix graft (ADMG) or enamel matrix de

75 amounts of keratinized tissue (KT) with the acellular dermal matrix graft (ADMG).

76 procedures and coronally advanced flap plus acellular dermal matrix grafts, enamel matrix derivative

77 Three of the 4 commonly used acellular dermal matrix materials are resistant to in vi

78 aps for root coverage with either of the two acellular dermal matrix materials.

79 A non-cross-linked porcine acellular dermal mesh was sutured to the pelvic floor re

80 plicate implants were used in this study: 1) acellular dTBs; 2) recellularized dTBs seeded with porci

81 al systems composed of distinct cellular and acellular elements that collectively dictate glioblastom

82 which still undergoes apical constriction in acellular embryos as in wildtype.

83 tem without polarized cell intercalation, in acellular embryos.

84 hat fail to form cells before gastrulation ('acellular' embryos), such that the global redistribution

85 on of the seminiferous tubules, which become acellular, empty spaces among the extant Leydig cells.

86 d4-deficient endocardium was associated with acellular endocardial cushions, absent epithelial-to-mes

87 e various concentrations and combinations of acellular extracellular matrix (ECM) components that may

88 Our study aims at producing acellular extracellular matrix scaffolds from the human

89 ies, delamination defects, and deposition of acellular extracellular matrix.

90 Complex acellular facial scaffolds were obtained, preserving sim

91 dhesion interface, allowing formation of the acellular fissure that defines the somite boundary.

92 jection of biomaterials, containing cells or acellular, following myocardial infarction (MI) to influ

93 lavage fluid was separated into cellular and acellular fractions by centrifugation.

94 G2 RSs injected with acellular GelMA alone, and G3 empty RSs were used as con

95 In contrast, acellular GelMA and empty RS constructs supported the fo

96 ived pulp-like tissue was observed in the G2 acellular GelMA and G3 empty RS groups.

97 The acellular grafts were then seeded with cells that were d

98 cnemius muscle recovery at 5 months than the acellular group, but the NCSC-SC group didn't.

99 myofibres beyond a certain threshold through acellular growth causes a reduction in the specific tens

100 tant did not exhibit any growth defect under acellular growth conditions; however, it was impaired fo

101 r in Anopheles gambiae, we show here that an acellular gut barrier, resulting from the tyrosine cross

102 We present the first experience using an acellular hemoglobin-based oxygen carrier (HBOC) Hemopur

103 ts using perfluorocarbon (PFC) emulsions and acellular hemoglobin-based oxygen carriers (HBOCs).

104 Gross inspection revealed that acellular implants had significantly decreased in size b

105 rvation of an intact liver capsule, a porous acellular lattice structure with intact vessels and stri

106 liver progenitor cells self-assembled inside acellular liver extracellular matrix scaffolds to form t

107 Moreover, acellular liver scaffolds seeded with hepatocytes produc

108 r the rapid and accurate production of human acellular liver tissue cubes (ALTCs) using normal liver

109 onstrate that lung fibroblasts reseeded into acellular lung matrices can be subsequently assayed usin

110 Acellular lung matrices were prepared from whole rat or

111 y epithelium and vascular endothelium on the acellular lung matrix.

112 Here, we tested whether acellular mammary extracellular matrix (mECM) preparatio

113 Enamel is an acellular material formed by the intricate process of am

114 Acellular materials of xenogenic origin are used worldwi

115 ed environment of supporting cells (SCs) and acellular matrices.

116 Furthermore, comprehensive analysis of acellular matrix ECM details significant compositional d

117 plastic support, MatrigelTM and human liver acellular matrix.

118 This layer was acellular, measured 10.15 +/- 3.6 microns composed of 5

119 Otogelin-like is localized to the acellular membranes of the cochlea and the vestibular sy

120 A complementary in vitro acellular mineralization study revealed that the hydroge

121 Enamel, the outermost layer of teeth, is an acellular mineralized tissue that cannot regenerate; the

122 to infiltrate and grow in CSF, a remarkably acellular, mitogen-poor metastasis microenvironment.

123 acellular mucin and 92% for patients without acellular mucin (P = 0.954).

124 ll survival rates were 85% for patients with acellular mucin and 92% for patients without acellular m

125 However, the presence of acellular mucin has no prognostic significance.

126 Acellular mucin is present within 27% of resected specim

127 The presence of acellular mucin pools in the resected specimens of patie

128 Acellular mucin was present in 27 (27%) of the 100 patie

129 The clinical significance of the presence of acellular mucin with respect to local recurrence and sur

130 he prevalence and prognostic significance of acellular mucin within resected specimens of rectal canc

131 The presence or absence of acellular mucin within the specimen was identified by th

132 t failures occurred in 0 and 2 patients with acellular mucin, and in 1 and 6 patients without acellul

133 lular mucin, and in 1 and 6 patients without acellular mucin, respectively.

134 91% (P = 0.881) in patients with and without acellular mucin, respectively.

135 en implanted into rat osteochondral defects, acellular nanofiber scaffolds supported enhanced chondro

136 Here, we developed an acellular, nanofiber-based preparation of self-assembled

137 Because of their acellular nature, iPSC-EVs represent a safer alternative

138 Though acellular, nonvital, and without capacity for turnover o

139 nic components, have been mostly replaced by acellular or subunit vaccines composed of well-defined,

140 rlying extracellular matrix, and produced an acellular, perfusable vascular architecture, competent a

141 alyzed comparative responses to cellular and acellular perfusates to identify these benefits.

142 Acellular perfusion was limited to 6 hours on the OCS sy

143 Acellular pertussis (aP) and whole-cell (wP) pertussis v

144 e efficacy and duration of protection of the acellular pertussis (aP) vaccine is lower than that of t

145 A maternal monovalent acellular pertussis (aP) vaccine, in development, could

146 nia in an cohort vaccinated exclusively with acellular pertussis (aP) vaccine.

147 tely understood, we hypothesize that current acellular pertussis (aP) vaccines fail to prevent coloni

148 ing immunity induced after immunization with acellular pertussis (aP) vaccines.

149 Maternal vaccination with an acellular pertussis (aP)-containing vaccine is a recomme

150 ay in vaccines containing diphtheria-tetanus-acellular pertussis (DTaP) is associated with reduced ri

151 ceive five doses of diphtheria, tetanus, and acellular pertussis (DTaP) vaccine before 7 years of age

152 ion for the diphtheria, tetanus toxoids, and acellular pertussis (DTaP) vaccine.

153 omen immunized with tetanus, diphtheria, and acellular pertussis (Tdap) after the 20th week of their

154 sly, uptake of both tetanus, diphtheria, and acellular pertussis (Tdap) and influenza vaccines is ver

155 hird-trimester (>/=GW 26) tetanus-diphtheria-acellular pertussis (Tdap) immunization in pregnant wome

156 mended to receive a tetanus, diphtheria, and acellular pertussis (Tdap) vaccine at 27-36 weeks gestat

157 ommended to receive tetanus, diphtheria, and acellular pertussis (Tdap) vaccine at the start of the t

158 The effect a maternal tetanus, diphtheria, acellular pertussis (Tdap) vaccine booster between 2 con

159 tanus toxoid, reduced diphtheria toxoid, and acellular pertussis (Tdap) vaccine could prevent infant

160 recommend tetanus and diphtheria toxoids and acellular pertussis (Tdap) vaccine for postpartum women

161 CIP) recommends the tetanus, diphtheria, and acellular pertussis (Tdap) vaccine for pregnant women du

162 gh-income countries using tetanus-diphtheria-acellular pertussis (Tdap) vaccines in their maternal an

163 mbination diphtheria and tetanus toxoids and acellular pertussis adsorbed (DTaP), inactivated poliovi

164 heduled administration of tetanus/diphtheria/acellular pertussis and meningococcal vaccines, respecti

165 For acellular pertussis antigens, 2-fold higher maternal ant

166 oduction and persistence of antibodies after acellular pertussis booster vaccination during adolescen

167 veness of US-licensed vaccines containing an acellular pertussis component.

168 and after they received a priming series of acellular pertussis containing vaccines.

169 commended universal tetanus, diphtheria, and acellular pertussis immunisation during pregnancy.

170 priming dose of wP vaccine into the current acellular pertussis vaccination schedule.

171 Tetanus, diphtheria, and acellular pertussis vaccination was required by 66%, 70%

172 superior protection contrasted with a solely acellular pertussis vaccine (aP) series.

173 group despite high diphtheria, tetanus, and acellular pertussis vaccine (DTaP) coverage, indicating

174 tanus toxoid, reduced diphtheria toxoid, and acellular pertussis vaccine (Tdap) be administered durin

175 n with tetanus, reduced-dose diphtheria, and acellular pertussis vaccine (Tdap) could be an effective

176 ine effectiveness (VE) of tetanus-diphtheria-acellular pertussis vaccine (Tdap) for preventing pertus

177 hed data on the safety of tetanus-diphtheria-acellular pertussis vaccine (Tdap) in persons aged >/=65

178 n healthcare personnel (HCP) vaccinated with acellular pertussis vaccine (Tdap).

179 e dose of diphtheria and tetanus toxoids and acellular pertussis vaccine 10 years ago, during adolesc

180 Baboons receiving acellular pertussis vaccine and infants born to mothers

181 Baboons were vaccinated with acellular pertussis vaccine at 2 days of age or at 2 and

182 ate the genetics of antibody responses to an acellular pertussis vaccine by a genome-wide association

183 eived at least 3 doses of diphtheria-tetanus-acellular pertussis vaccine by the end of 15 months of a

184 r data suggests that the current schedule of acellular pertussis vaccine doses is insufficient to pre

185 of earlier or more numerous booster doses of acellular pertussis vaccine either as part of routine im

186 theria booster with tetanus, diphtheria, and acellular pertussis vaccine for adolescents and adults)

187 with diphtheria toxoid, tetanus toxoid, and acellular pertussis vaccine has been associated with som

188 In Denmark, acellular pertussis vaccine has been included in the com

189 izures, but whether this risk applies to the acellular pertussis vaccine is not known.

190 The population-level safety benefits of the acellular pertussis vaccine may have been underestimated

191 Pertussis toxoid, an acellular pertussis vaccine prepared by hydrogen peroxid

192 It appears that a wholly acellular pertussis vaccine series is significantly less

193 accination, adult female baboons primed with acellular pertussis vaccine were boosted in the third tr

194 1994 and March 1996 (before introduction of acellular pertussis vaccine) and between April 1998 and

195 l 1998 and March 2000 (after introduction of acellular pertussis vaccine) in Ontario, Canada.

196 tanus toxoid, reduced diphtheria toxoid, and acellular pertussis vaccine).

197 There is accumulating literature on waning acellular pertussis vaccine-induced immunity, confirming

198 eceipt of diphtheria and tetanus toxoids and acellular pertussis vaccine.

199 tand the effect of vaccination in the era of acellular pertussis vaccines (DTaP and Tdap), we assesse

200 s that received different diphtheria-tetanus-acellular pertussis vaccines (DTaP) during childhood and

201 ssis has been linked to switch to the use of acellular pertussis vaccines and the evolution of Bordet

202 easing evidence that the currently available acellular pertussis vaccines are not providing optimal c

203 y inactivated influenza, tetanus toxoid, and acellular pertussis vaccines are recommended during preg

204 ssis, and tetanus and diphtheria toxoids and acellular pertussis vaccines are reviewed in this second

205 ciated adverse events led the development of acellular pertussis vaccines containing 1 or more purifi

206 y in mice, it is not included in any current acellular pertussis vaccines due to protein stability is

207 Current acellular pertussis vaccines may not protect against tra

208 Childhood acellular pertussis vaccines were licensed and implement

209 d whole-cell pertussis vaccines, and OPV and acellular pertussis vaccines were similar.

210 her vaccines (most commonly pneumococcal and acellular pertussis vaccines), whereas standalone IPV va

211 It is a primary component of acellular pertussis vaccines, and anti-PRN antibody tite

212 and diphtheria; and tetanus, diphtheria, and acellular pertussis vaccines.

213 gate, and tetanus and diphtheria toxoids and acellular pertussis vaccines.

214 ategic development of the next generation of acellular pertussis vaccines.

215 at 10 years after high childhood coverage of acellular pertussis vaccines.

216 rogram and the transition from whole-cell to acellular pertussis vaccines.

217 tanus toxoid, reduced diphtheria toxoid, and acellular pertussis) vaccine was recommended for women d

218 scent and adult tetanus, reduced diphtheria, acellular pertussis) vaccine.

219 ty infants of Tdap (tetanus, diphtheria, and acellular pertussis)-vaccinated pregnant women and 37 in

220 omavirus, diphtheria and tetanus toxoids and acellular pertussis, and tetanus and diphtheria toxoids

221 ent in reduced responses to booster doses of acellular pertussis, inactivated polio, and diphtheria v

222 eumococcal and combined diphtheria, tetanus, acellular pertussis, inactivated polio, hepatitis B, Hae

223 oxyfen or vaccines (tetanus, diphtheria, and acellular pertussis, measles and rubella, or measles, mu

224 ations of diphtheria and tetanus toxoids and acellular pertussis, meningococcal conjugate and pneumoc

225 ations of diphtheria and tetanus toxoids and acellular pertussis, pneumococcal conjugate, and human p

226 eceive 3 doses of diphtheria-tetanus toxoids-acellular pertussis-hepatitis B virus-inactivated poliov

227 program using diphtheria-tetanus-5-component acellular pertussis-inactivated polio vaccine (dT5aP-IPV

228 d in the combined diphtheria-tetanus toxoids-acellular pertussis-inactivated poliovirus-Haemophilus i

229 ation diphtheria vaccine (diphtheria-tetanus-acellular pertussis-inactivated poliovirus/Haemophilus i

230 t limb group received the diphtheria-tetanus-acellular pertussis-inactived polio-Haemophilus influenz

231 2010, corresponding directly to the aging of acellular pertussis-vaccinated cohorts.

232 xoid, reduced diphtheria toxoid, and reduced acellular pertussis; and human papillomavirus vaccines)

233 ng a tetanus/low-dose diphtheria/5-component acellular-pertussis/inactivated-polio (TdaP5/IPV) vaccin

234 ammation, the long-term safety of living and acellular pig tissue implants in recipients warrants fur

235 om the cellular (circulating leukocytes) and acellular (plasma cell-free DNA) compartments of periphe

236 evidence for the existence of a distinctive acellular pre-Descemet's stromal layer in the human corn

237 , these data indicate that both cellular and acellular presentation of T-cadherin can be used to modu

238 rating host cells make contact only with the acellular protective coat of the parasite, called lamina

239 observed at contralateral sites treated with acellular QD-free scaffolds.

240 enchyme around the aortic sac also developed acellular regions, and the distal aortic sac became gros

241 ation grade 2R or higher cellular rejection, acellular rejection, or allograft dysfunction of uncerta

242 n protocol enabled us to obtain a completely acellular renal scaffold while maintaining the extracell

243 uitment of diabetic CD34(+) cells to injured acellular retinal capillaries was greater after TGF-beta

244 he hallmark feature of diabetic retinopathy, acellular retinal capillaries.

245 One approach combines cells with acellular scaffolds derived from animal tissue.

246 Acellular scaffolds obtained via decellularization are a

247 n all facial grafts within 12 days revealing acellular scaffolds with full preservation of innate mor

248 Cardiac implantation of acellular scaffolds with pore diameters of 30-40 microm

249 uctural and biochemical properties of native acellular scaffolds with subsequent recellularization te

250 man kidneys by detergent perfusion, yielding acellular scaffolds with vascular, cortical and medullar

251 rsional strength compared to those receiving acellular scaffolds, although there were no significant

252 Decellularized (acellular) scaffolds, composed of natural extracellular

253 dge with scar tissue); 1 eye showed a dense, acellular scar overlying a portion of the DMEK graft tha

254 This is a plasmodial, vegetative stage of acellular slime mould.

255 There exists a novel, well-defined, acellular, strong layer in the pre-Descemet's cornea.

256 ons affect the production and/or function of acellular structures of the inner ear, which ultimately

257 ounds, especially those containing permanent acellular structures, such as scar tissue.

258 This is an acellular system that reproduces the assembly of part of

259 ibited a high radical scavenging activity in acellular systems.

260 y cell migration, buttons were nested within acellular uncompressed outer collagen matrices before gr

261 llularization of porcine urethras to produce acellular urethra bioscaffolds for future tissue enginee

262 sed to evaluate the recellularization of the acellular urethra bioscaffolds.

263 ghlight discrepancies between whole-cell and acellular vaccination that could contribute to the incre

264 infection since the transition to the use of acellular vaccination.

265 LPS drastically improved the efficacy of the acellular vaccine Adacel against B. parapertussis.

266 Importantly, we demonstrate that acellular vaccine antigen-encoding genes are evolving at

267 pertussis without the pertactin protein, an acellular vaccine immunogen, has been reported in the Un

268 It has been suggested that acellular vaccine may be less effective than previously

269 ion was able to prevent transmission, but an acellular vaccine that effectively controls disease fail

270 ents taking place before introduction of the acellular vaccine versus after introduction by calculati

271 ts introduction, despite changing to another acellular vaccine with different antigen composition.

272 aring event rates before the introduction of acellular vaccine with those after introduction.

273 s per month were avoided by switching to the acellular vaccine, which is a 38-fold higher impact than

274 vaccines; (7) memory B cells persist in both acellular vaccine- and whole cell vaccine-primed childre

275 arrying prn2 and ptxP3 under the pressure of acellular vaccine-induced immunity.

276 ole cell vaccine-primed children; and (8) in acellular vaccine-primed children, T-cell responses rema

277 relative incidence after introduction of the acellular vaccine.

278 ted Kingdom switched from a whole-cell to an acellular vaccine.

279 ate the potential utility of an OMP subunit (acellular) vaccine for protecting mammals against type A

280 It has been proposed that current acellular vaccines (Pa) composed of only a few bacterial

281 Current acellular vaccines against Bordetella pertussis are effe

282 n this article we discuss the following: (1) acellular vaccines are immunogenic, but responses vary b

283 vaccination, inferior long-term efficacy of acellular vaccines compared with whole-cell vaccines, ci

284 minantly T-helper 1 (Th1) responses, whereas acellular vaccines generate mixed Th1/Th2 responses; (5)

285 Though acellular vaccines have been in use for 20 years, new da

286 The efficacy of acellular vaccines is inferior to that of WCVs, however,

287 However, waning of immunity from acellular vaccines may be driving the recent resurgence

288 In countries using acellular vaccines, waning immunity is at least part of

289 ounced since the introduction of the current acellular vaccines.

290 immunogenicity, and/or efficacy of different acellular vaccines.

291 ut suppression has been seen less often with acellular vaccines; (7) memory B cells persist in both a

292 perfusable vascular architecture, competent acellular valves and intact chamber geometry.

293 tergent perfusion and yielded scaffolds with acellular vasculature, airways and alveoli.

294 e developed and tested a bioengineered human acellular vessel as a potential solution to these limita

295 A novel bioengineered human acellular vessel was implanted into the arms of patients

296 Bioengineered human acellular vessels seem to provide safe and functional ha

297 Human acellular vessels were implanted into 60 patients.

298 wounding on tumor growth can be mimicked by acellular wound fluid, suggesting that T cells secrete o

299 In MMC-treated eyes, an anterior acellular zone overlaid a sparsely populated stroma cont

300 ore penetrating keratoplasty demonstrated an acellular zone with a hyperintense signal consistent wit