ライフサイエンスコーパス: acetaldehyde

1 organic compounds, VOCs (e.g., methanol and acetaldehyde).

2 2-knockdown human keratinocytes treated with acetaldehyde.

3 ing concentrations of free and sulfite-bound acetaldehyde.

4 tly decreased with the addition of exogenous acetaldehyde.

5 molecule that enables ALDH3A1 to metabolize acetaldehyde.

6 poised toward the production of ethanol from acetaldehyde.

7 es is detailed using the example of indole-3-acetaldehyde.

8 o our great surprise) restricts diffusion of acetaldehyde.

9 or acetone but a net source for methanol and acetaldehyde.

10 ting it to ammonia, inorganic phosphate, and acetaldehyde.

11 ic effects of ethanol-an exogenous source of acetaldehyde.

12 hough still with a 3-fold underestimation in acetaldehyde.

13 ct CH(2) horizontal lineCHOH tautomerizes to acetaldehyde.

14 oducts were 1-hexene, CO, vinyl alcohol, and acetaldehyde.

15 dative deamination of histamine to imidazole acetaldehyde.

16 boligation reaction between benzaldehyde and acetaldehyde.

17 ic mechanisms of ethanol and its metabolite, acetaldehyde.

18 thanol metabolism and was likely mediated by acetaldehyde.

19 ndicating the effect was in part mediated by acetaldehyde.

20 catalyst) or the aldol process starting from acetaldehyde.

21 thanol metabolism and was likely mediated by acetaldehyde.

22 lyzes the reversible oxidation of alcohol to acetaldehyde.

23 oacetaldehyde was oxidized more rapidly than acetaldehyde.

24 ction between L-cysteine and 5-hydroxyindole acetaldehyde.

25 uld be reproduced by histamine and imidazole acetaldehyde.

26 1) surface is intrinsically selective toward acetaldehyde.

27 ological conditions, preventing a buildup of acetaldehyde.

28 c agent recruited another ALDH to metabolize acetaldehyde.

29 d us to describe reaction of epicatechin and acetaldehyde.

30 s of nitrogen oxides (NOx), formaldehyde, or acetaldehyde.

31 5, usually interpreted as solely coming from acetaldehyde.

32 s, particularly nonaromatic amino acids, and acetaldehyde.

33 choline to produce trimethylamine (TMA) and acetaldehyde.

34 In this process, (tert-butyldimethylsilyloxy)acetaldehyde 1 was successfully utilized in two ways: as

35 y of the Julia-Kocienski olefination between acetaldehyde (1) and ethyl 1-phenyl-1H-tetrazol-5-yl sul

36 the wine content in 1-propanol, isobutanol, acetaldehyde, 1,1-diethoxiethane and ethyl lactate.

37 Intra-RVLM acetaldehyde (2 mug), the main metabolic product of etha

38 d VOCs such as methanol (5.39 pptv/ppbv CO), acetaldehyde (3.93 pptv/ppbv CO), acetone (3.59 pptv/ppb

39 sual AAAD enzyme products including indole-3-acetaldehyde, 4-hydroxyphenylacetaldehyde, and phenyleth

40 Monochloramine reacts with acetaldehyde, a common ozone and free chlorine disinfect

41 Acetaldehyde, a major product of ethanol metabolism, lik

42 ate, and rapid analysis of ethanol (Eth) and acetaldehyde (AA) in a wide variety of beverages and foo

43 ed the effect of ethanol and its metabolite, acetaldehyde (AcAld), on total and ureagenic respiration

44 ans carry an inactive ALDH2 gene and exhibit acetaldehyde accumulation after alcohol consumption.

45 ic site-catalyzed ethanol dehydrogenation to acetaldehyde, acetaldehyde to acetone conversion via a c

46 as alpha-aryl-alpha,alpha-difluoroketones, -acetaldehydes, -acetates, and acetic acids, and difluoro

47 The concentrations of seven VOCs-acetaldehyde, acetone, acetic acid, hexanoic acid, hydro

48 The concentrations of acetaldehyde, acetone, acetic acid, hexanoic acid, hydro

49 s was applied for a combination of six VOCs (acetaldehyde, acetone, acetic acid, hexanoic acid, hydro

50 Ethanol, acetaldehyde, acetone, and acetate increase the total in

51 e enhancement of HCV replication by ethanol, acetaldehyde, acetone, as well as acetate, whereas inhib

52 etermined over selected spectral regions for acetaldehyde, acetonitrile, ethanol, water, methanol, am

53 Formaldehyde, acetaldehyde, acrolein, and several other carbonyls were

54 2, CO, total particulate mass, formaldehyde, acetaldehyde, acrolein, and several polycyclic aromatic

55 Acetaldehyde acts as a bridging compound to form modifie

56 nant OSE adducts termed MAA (malondialdehyde-acetaldehyde-adducts), which are found on apoptotic cell

57 ent with previously reported TPD results for acetaldehyde adsorbed on reduced CeO(2)(111).

58 On the fully oxidized surface, acetaldehyde adsorbs weakly through its carbonyl O inter

59 Asians and results in accumulation of toxic acetaldehyde after consumption of ethanol.

60 Hansenula sp. which quantitatively produces acetaldehyde after reaction for 120 min at 40 degrees C

61 n of a Clostridium thermocellum bifunctional acetaldehyde/alcohol dehydrogenase.

62 erefore speculate that the second pathway in acetaldehyde also occurs via a roaming mechanism in the

63 Acetaldehyde, an alcohol catabolite detoxified by ALDH2,

64 Acetaldehyde, an I/R product (300 microM), elicited an 8

65 TAML/H2O2 slowly degrades metaldehyde to acetaldehyde and acetic acid.

66 The model predicted summer minima in acetaldehyde and acetone, which were not apparent in the

67 Acetaldehyde and acrolein coeluted with other wine compo

68 oduce toxic byproducts such as formaldehyde, acetaldehyde and acrolein.

69 f CO2 increased the release of formaldehyde, acetaldehyde and antimony (Sb).

70 is 2-aminothiazole formed from beta-mercapto-acetaldehyde and cyanamide in water at neutral pH.

71 ag phase of formation, with the exception of acetaldehyde and diacetyl formation.

72 surfaces has been derived from the study of acetaldehyde and dimethylamine in combination with previ

73 At 20 degrees C, acetaldehyde and ethanol increased significantly with th

74 Exposure of the acini to acetaldehyde and ethyl oleate followed by CCK-8 stimulat

75 Acetaldehyde and ethyl oleate redirected CCK-8-stimulate

76 The presence of propionaldehyde, acetaldehyde and formaldehyde were correlated, corrobora

77 lyze the decarboxylation of pyruvate to form acetaldehyde and formate, respectively.

78 icting the alcoholic strength, the methanol, acetaldehyde and fusel alcohols content of grape-derived

79 ese findings suggest that alcohol, by way of acetaldehyde and its associated adducts, stimulates hepa

80 ence of ethanol, S. pneumoniae AdhE produced acetaldehyde and NADH, which subsequently led Rex (redox

81 site and oxidative denitrification to yield acetaldehyde and nitrite.

82 Yeast metabolites such as acetaldehyde and pyruvate participate in the formation o

83 ikely as a result of increased production of acetaldehyde and reactive oxygen species and mitochondri

84 Formaldehyde, acetaldehyde and Sb migration increased with sunlight ex

85 Acetaldehyde and, to a lesser degree, ethyl oleate produ

86 opment of alcoholic pancreatitis, oxidative (acetaldehyde) and nonoxidative metabolites (ethyl palmit

87 1) surface is intrinsically selective toward acetaldehyde, and a strong inverse correlation between c

88 s OVOCs, including acetic acid, formic acid, acetaldehyde, and acetone were observed during photodegr

89 Formaldehyde, acetaldehyde, and butyraldehyde were the most significan

90 including acetate, reactive oxygen species, acetaldehyde, and epigenetic changes, that can induce in

91 ADH-mediated interconversions of acetyl-CoA, acetaldehyde, and ethanol but seemed to be poised toward

92 production was observed for glycolaldehyde, acetaldehyde, and formaldehyde only at elevated temperat

93 s well as increases in emissions of ethanol, acetaldehyde, and formaldehyde.

94 se 2 (ALDH2) is a key enzyme that eliminates acetaldehyde, and impairment of ALDH2 increases the risk

95 TRPV1 by histamine, its metabolite imidazole acetaldehyde, and supernatants from biopsy specimens was

96 e of buffer and air to produce formaldehyde, acetaldehyde, and the aldehydes corresponding to the bre

97 ips between glycolaldehyde and beta-mercapto-acetaldehyde, and the corresponding proteinogenic amino

98 enyl lactone of the southern portion with an acetaldehyde appendage on the cyclobutane of the norther

99 recombinative desorption of enolate and H as acetaldehyde are in good agreement with previously repor

100 Loss and toxic effects of acetaldehyde are minimized by accelerating its consumpti

101 Formaldehyde and acetaldehyde are the dominant HAP concentration and canc

102 raphy for liquid phase analysis, we identify acetaldehyde as a minor product and key intermediate in

103 se-like protein (Ald2) to form 2-(methylthio)acetaldehyde as an intermediate.

104 ommonly produce ethanol from acetyl-CoA with acetaldehyde as intermediate and play a key role in anae

105 ncluding the sulfur-containing compounds and acetaldehyde, as well as lipid oxidation derived odorant

106 BRCA2-null cells for the ethanol metabolite, acetaldehyde, associated with widespread chromosomal bre

107 The present study employs exogenous acetaldehyde at relatively low and high treatment concen

108 ross-coupling of methanol with formaldehyde, acetaldehyde, benzaldehyde and benzeneacetaldehyde to me

109 thought to retain the volatile intermediate acetaldehyde but allow diffusion of the much larger cofa

110 -Phe substitution increases turnover rate of acetaldehyde but decreases turnover rate of larger aldeh

111 ted chemical species, dominantly ammonia and acetaldehyde, but also two new species previously not re

112 2-deficient DT40 cells are hypersensitive to acetaldehyde, but not to acrolein, crotonaldehyde, glyox

113 Furthermore, oxidation of indole-3-acetaldehyde by AOs is likely to represent one route to

114 Conversion of Thr to Gly and acetaldehyde by Thr aldolase (EC 4.1.2.5) was only recen

115 Our results show that the acetaldehyde-catabolising enzyme Aldh2 is essential for

116 Nevertheless, acetaldehyde-catabolism-competent mothers (Aldh2(+/-)) c

117 converted glucose to ethanol via acetate and acetaldehyde, catalyzed by the host-encoded aldehyde fer

118 Ethanol and acetaldehyde caused a rapid and synergistic elevation of

119 which are identified as the enolate form of acetaldehyde (CH(2)CHO ).

120 H(3)COO(-)(ad)), acyl species (CH(3)CO(ad)), acetaldehyde (CH(3)CHO(ad)), CO(2), and H(2)O.

121 erature-dependent adsorption and reaction of acetaldehyde (CH(3)CHO) on a fully oxidized and a highly

122 y that interstellar aldehydes and enols like acetaldehyde (CH3CHO) and vinyl alcohol (C2H3OH) act as

123 (H2O2), ozone (O3), formaldehyde (HCHO), and acetaldehyde (CH3CHO).

124 of these proteins was AdhE, a bi-functional acetaldehyde-CoA dehydrogenase and alcohol dehydrogenase

125 e is primarily due to a mutated bifunctional acetaldehyde-CoA/alcohol dehydrogenase gene (adhE), hypo

126 increases in oxygenates such as ethanol and acetaldehyde compared to E0 and E10 fuels.

127 s1 mutant to exogenous acetate, ethanol, and acetaldehyde compared to wild-type plants.

128 The aims of this study were to measure acetaldehyde concentration in different beverages consum

129 Comparison of initial acetaldehyde concentration with that after enzymatic oxi

130 ethanol emissions lead to higher atmospheric acetaldehyde concentrations (by up to 14% during winter

131 The model underestimation of acetaldehyde concentrations all year round implies a con

132 ements in urban areas may have overestimated acetaldehyde concentrations at times due to this interfe

133 Underestimation of acetaldehyde concentrations is responsible for the bulk

134 nexpectedly, to hypothermia, increased blood acetaldehyde concentrations, and enhanced lethality.

135 roacetamide under typical monochloramine and acetaldehyde concentrations.

136 ctors couples the three reactions and drives acetaldehyde consumption.

137 tive for the aerobic oxidation of ethanol to acetaldehyde (conversion 100%; yield approximately 95%).

138 rds representing different carbonyl classes, acetaldehyde could be ionized only after labeling and MS

139 h showed excellent catalytic activity in the acetaldehyde cyclotrimerization reaction.

140 on employs a commercially available reagent, acetaldehyde-d4, to label the amine groups on the monoam

141 Among these, the bifunctional alcohol/acetaldehyde dehydrogenase (ADH1), highly homologous to

142 ator LasR and redox-regulated activities for acetaldehyde dehydrogenase ExaC, arginine deiminase ArcA

143 Deletion of ALD6 coding for acetaldehyde dehydrogenase not only prevented acetate ac

144 ter has two important functions: detoxifying acetaldehyde derived from dietary ethanol [11] and detox

145 Notably, levels of acetaldehyde-derived DNA damage represented by N(2)-ethy

146 preventive role of oesophageal ALDH2 against acetaldehyde-derived DNA damage.

147 s-links arising from the crotonaldehyde- and acetaldehyde-derived R- and S-alpha-CH3-gamma-OH-1,N2-pr

148 The synthesis of the acetaldehyde-derived tris(trimethylsilyl)silyl (super si

149 were somewhat more efficient than ozone for acetaldehyde destruction, ozone was more efficient for a

150 coni anaemia pathway-mediated DNA repair and acetaldehyde detoxification.

151 he formation of the major flavour compounds (acetaldehyde, diacetyl, acetoin, and 2-butanone) followe

152 At 30 degrees C, acetaldehyde did not increase but ethanol increased rapi

153 etaldehyde direction), increased rapidly but acetaldehyde did not rise because of its oxidation to ac

154 t 30 degrees C, the ADH activity (ethanol to acetaldehyde direction), increased rapidly but acetaldeh

155 Carbonyls such as formaldehyde and acetaldehyde dominated VOC emissions, making up approxim

156 Acetaldehyde donors proceed with yields up to 77% and en

157 Acetaldehyde emissions exhibited sharp increases with hi

158 y for the gas-phase SN2 reaction between the acetaldehyde enolate anion and methyl fluoride, for both

159 nthetic equivalent of the alpha-arylation of acetaldehyde enolate.

160 l, glyoxal, glycolaldehyde, ethylene glycol, acetaldehyde, ethane, and methanol).

161 tory quotient 1.5 (DCA-RQ 1.5) increased the acetaldehyde, ethanol and ethyl acetate concentration, r

162 n for the study were ethyl acetate, acetone, acetaldehyde, ethanol, ethylene glycol, dimethylsilanedi

163 harboring putative genes for a bifunctional acetaldehyde/ethanol dehydrogenase (Aad), serine/threoni

164 cid and all of the major volatiles excepting acetaldehyde, ethyl acetate and acetoine, whereas the ap

165 ction of seed tannins, exhibited the highest acetaldehyde, ethyl acetate and C6-compounds levels, and

166 octanoate, butyrolactone, isoamyl alcohols, acetaldehyde, ethyl acetate, 2,3-butanediol, acetoin and

167 of ethanol to its carbonyl compounds, namely acetaldehyde, ethyl acetate, acetic acid, and ketene, oc

168 higher amounts, with increased citronellol, acetaldehyde, ethyl acetate, dicarboxylic acids esters,

169 nation of six toxic compounds (formaldehyde, acetaldehyde, ethyl carbamate, furan, furfural and acrol

170 The ethanol metabolites acetaldehyde, ethyl palmitate, and ethyl oleate perturb

171 The ethanol metabolites acetaldehyde, ethyl palmitate, and ethyl oleate reduced

172 Two central enzymes convert ethanolamine to acetaldehyde (EutBC) and then to acetyl-CoA (EutE).

173 One of our mixture components, phenyl acetaldehyde, evoked significant levels of nonlinear inp

174 a crucial role in stabilizing and activating acetaldehyde for coupling reactions.

175 ycles for nonmethane organic gases, ethanol, acetaldehyde, formaldehyde, acetone, nitrous oxide, nitr

176 nature; the same trend of increased ethanol, acetaldehyde, formaldehyde, and CH4 emissions and decrea

177 For example, E85 resulted in high acetaldehyde, formaldehyde, ethanol, ethene, and acetyle

178 , total hydrocarbons (THC), methane, ethene, acetaldehyde, formaldehyde, ethanol, N2O, and NH3 from a

179 nalysis was applied for the combined VOCs of acetaldehyde, formaldehyde, hydrogen sulphide, and methy

180 the fuel, the tailpipe emissions of ethanol, acetaldehyde, formaldehyde, methane, and ammonia increas

181 The GSH reduces browning and acetaldehyde formation for up to 12months.

182 ddition of EDTA to samples prevented de novo acetaldehyde formation from ethanol oxidation.

183 Reaction products of (-)-epicatechin with acetaldehyde formed in model solution were selected for

184 We show that acetaldehyde forms at low steady-state concentrations, a

185 onal enzyme DmpFG channels its intermediate, acetaldehyde, from one active site to the next using a b

186 Acetaldehyde (GC-FID) and pyruvic acid (Y15 enzymatic au

187 four different chemical input signals (NADH, acetaldehyde, glucose, and oxygen).

188 ncluding nicotine, nicotyrine, formaldehyde, acetaldehyde, glycidol, acrolein, acetol, and diacetyl.

189 Furthermore, acetaldehyde had the greatest odor activity value of up

190 idium-mediated dehydrogenation of ethanol to acetaldehyde has led to the development of an ethanol-to

191 the following 7 VOCs, acetone, formaldehyde, acetaldehyde, hexanoic acid, hydrogen sulphide, hydrogen

192 often treated with oxygen in order to yield acetaldehyde, however this approach can lead to unintend

193 r billion (ppb) or 8 ppb gas-phase MG and/or acetaldehyde in an aerosol reaction chamber for up to 5

194 asymmetric direct crossed-aldol reaction of acetaldehyde in aqueous media using brine.

195 s and consumer guidance may be necessary for acetaldehyde in beverages.

196 Ethanol is metabolized into acetaldehyde in most tissues.

197 e ERK-dependent pressor effect of ethanol or acetaldehyde in normotensive rats.

198 c fibrosis through increased accumulation of acetaldehyde in the liver.

199 show measurements of acetone, methanol, and acetaldehyde in the tropical remote marine boundary laye

200 To temporarily increase metabolism of acetaldehyde in vivo, we describe an approach in which a

201 At 10 degrees C, acetaldehyde increased rapidly and then declined, while

202 Ethanol and acetaldehyde induced a rapid and synergistic increase in

203 In vitro experiments revealed that acetaldehyde induced ALDH2 production in both mouse and

204 Linoleic acid, but not ethanol or acetaldehyde, induced ALOX15 expression in Hepa-1c1c7 ce

205 TA or Ca(2+)-free medium blocked ethanol and acetaldehyde-induced barrier dysfunction and tight junct

206 eine and cyclosporine A, blocked ethanol and acetaldehyde-induced barrier dysfunction and tight junct

207 -specific activator, Alda-1, Alda-89 reduced acetaldehyde-induced behavioral impairment by causing a

208 EGF-mediated prevention of acetaldehyde-induced decrease in transepithelial electri

209 aI and attenuated EGF-mediated prevention of acetaldehyde-induced disruption of tight junctions.

210 nconi anaemia DNA repair pathway counteracts acetaldehyde-induced genotoxicity in mice.

211 ptides attenuated EGF-mediated prevention of acetaldehyde-induced increase in inulin permeability and

212 -mediated protection of tight junctions from acetaldehyde-induced insult.

213 e intestinal epithelial tight junctions from acetaldehyde-induced insult.

214 Moreover, P2X7R silencing prevented ATP- and acetaldehyde-induced renin release.

215 aV1.3 channels, by shRNA blocked ethanol and acetaldehyde-induced tight junction disruption and barri

216 n, whereas aldehyde dehydrogenase attenuated acetaldehyde-induced tight junction disruption.

217 cathepsin S to obtain a novel (2-arylphenoxy)acetaldehyde inhibitor, 2, with a 0.49 microM Ki value.

218 n important role in controlling the entry of acetaldehyde into the DmpF active site.

219 rnal coenzyme B12 and injecting its product, acetaldehyde, into the lumen, where it is degraded by th

220 h as glycine or serine, as aldol donors, and acetaldehyde is a coproduct.

221 Acetaldehyde is a naturally-occurring carcinogenic compo

222 Acetaldehyde is an ethanol-derived definite carcinogen t

223 w steady-state concentrations, and that free acetaldehyde is difficult to detect in alkaline solution

224 The formation of acetaldehyde is due to the deprotonation of ethoxy, whic

225 Acetaldehyde is produced with a Faradaic efficiency of a

226 2-(Methylthio)acetaldehyde is reduced to 2-(methylthio)ethanol, which

227 Ethanol, which is metabolised to acetaldehyde, is both carcinogenic and teratogenic in hu

228 e smaller organic compounds such as acetone, acetaldehyde, isoprene, or cysteamine can be detected in

229 ospheric trace gases, methylglyoxal (MG) and acetaldehyde, known to be surface-active, can enhance ae

230 using a rapid reduction in blood ethanol and acetaldehyde levels after acute ethanol intoxication in

231 ucrose, and increasing fructose, glucose and acetaldehyde levels, which are potential contributors to

232 stion-induced elevation in plasma and tissue acetaldehyde levels.

233 Acetaldehyde, like ethanol, promoted fusion between ZGs

234 -) mice had higher levels of malondialdehyde-acetaldehyde (MAA) adduct and greater hepatic inflammati

235 These data strongly support the notion that acetaldehyde may be an essential contributor to the chro

236 ompounds (VOCs) such as ethylmercaptan (EM), acetaldehyde (MeCHO) and methyl ethyl ketone (MEK) among

237 Acetaldehyde-mediated DNA damage may critically contribu

238 function, but it dose-dependently increased acetaldehyde-mediated tight junction disruption and barr

239 m alcohol-treated mice had a greater rate of acetaldehyde metabolism and respiration when treated wit

240 onse to alcohol, suggesting that the greater acetaldehyde metabolism by isolated mitochondria from al

241 This study examined the impact of altered acetaldehyde metabolism through systemic transgenic over

242 D(+), the rate-limiting substrate in alcohol/acetaldehyde metabolism.

243 Total acetaldehyde, Mn, Cu/Fe, blue and red pigments and galli

244 ing ligands oxidized LDL and malondialdehyde-acetaldehyde-modified LDL.

245 ling of the carbonyl O and the acyl C of two acetaldehyde molecules.

246 Facile acetylation of dimethylamine by acetaldehyde occurs with high selectivity on oxygen-cove

247 ange (IQR) increases in prenatal exposure to acetaldehyde [odds ratio (OR) = 2.30; 95% CI: 1.44, 3.67

248 ential cofactor to catalyze the formation of acetaldehyde on the pathway of ethanol synthesis.

249 sion of CO and H2 into methane, ethanol, and acetaldehyde on the Rh (211) and (111) surfaces, chosen

250 igated the synergistic effect of ethanol and acetaldehyde on the tight junction integrity in Caco-2 c

251 ocked this synergistic effect of ethanol and acetaldehyde on tight junction.

252 nd ethyl acetate formed from condensation of acetaldehyde or acetic acid with excess ethanol.

253 creased supply of peroxyacetyl radicals from acetaldehyde oxidation, and the lower NO(x) emissions fo

254 isomer 2-phenyl-2-(propan-2-ylidenehydrazono)acetaldehyde oxime (7).

255 nserve certain volatile metabolites-CO(2) or acetaldehyde-perhaps by providing a low-pH compartment.

256 (ALDH2) is the major enzyme that metabolizes acetaldehyde produced from alcohol metabolism.

257 The addition of a coupling reaction removing acetaldehyde produced from the alcohol dehydrogenase (AD

258 at H4IIEC3 cells, respectively, dependent on acetaldehyde production, oxidative stress, and zinc rele

259 The saturated aldehydes (acetaldehyde, propionaldehyde, and butyraldehyde) in cig

260 us direct identification and quantitation of acetaldehyde, pyruvic acid, acetoin, methylglyoxal, and

261 unds mainly responsible for trapping SO2 are acetaldehyde, pyruvic acid, and 2-oxoglutaric acid.

262 8), alcoholic strength (r(2)=97.2; RPD=6.0), acetaldehyde (r(2)=98.2; RPD=7.5) and fusel alcohols (r(

263 Acetaldehyde Reactive Polyphenols (ARPs) may be key elem

264 The acetaldehyde reacts with 2,4-dinitrophenylhydrazine form

265 rylalkylamines and the formation of aromatic acetaldehydes, respectively.

266 The present work found that acetaldehyde served as a relatively poor precursor for t

267 the CO produced in the 308-nm photolysis of acetaldehyde show clear evidence of two dissociation mec

268 trate that both PD20 and UM are sensitive to acetaldehyde, supporting a role for FANCD2 in repair of

269 These results demonstrate that ethanol and acetaldehyde synergistically disrupt tight junctions by

270 s thaliana and Petroselinum crispum aromatic acetaldehyde synthases primarily converts the enzymes ac

271 metabolism and respiration when treated with acetaldehyde than control.

272 er amounts of ethanol, but larger amounts of acetaldehyde, than biofilms formed by the parent and rev

273 ated activation barriers for the coupling of acetaldehyde, the decomposition of the dimer state, and

274 Acetaldehyde, the first metabolite of ethanol, can upreg

275 Acetaldehyde, the first metabolite of ethanol, reacts wi

276 zed ethanol dehydrogenation to acetaldehyde, acetaldehyde to acetone conversion via a complex pathway

277 ring enzyme that catalyses the conversion of acetaldehyde to ethanol during fermentation.

278 D (acetyl-CoA to acetyl phosphate) and EutG (acetaldehyde to ethanol).

279 otein that is required for the conversion of acetaldehyde to ethanol.

280 catalyze the key aldol coupling reaction of acetaldehyde to exclusively yield the C4 coupling produc

281 e on the si face of the aldehyde carbonyl of acetaldehyde to form 4(S)-hydroxy-2-oxopentanoate.

282 ay be attributed to the adsorption of MG and acetaldehyde to the gas-aerosol interface, leading to su

283 n, epicatechin, caffeic acid, coumaric acid, acetaldehyde, total and reduced glutathione.

284 ursors, require Aldh2 for protection against acetaldehyde toxicity.

285 The high acetaldehyde treatment significantly increased polymeric

286 NOX4 promoter was induced in HSC by acetaldehyde treatment, and NOX4 has significantly incre

287 addition of (E)-2-butenyltrimethylsilane to acetaldehyde under electrophilic (BF3, H3O(+)) and nucle

288 ds of ethane and convert it into ethanol and acetaldehyde using nitrous oxide as the terminal oxidant

289 followed by extrusion of the "C(2)" unit as acetaldehyde, using cobalt complexes as substrates.

290 F-mediated protection of tight junction from acetaldehyde was evaluated in Caco-2 cell monolayers.

291 Acetaldehyde was present in the highest amount in RP-O (

292 Mean mixing ratios of acetone, methanol, and acetaldehyde were 546 +/- 295 pptv, 742 +/- 419 pptv, an

293 , alpha-ketoglutarate, pyruvate, acetoin and acetaldehyde were derivatised with 2,4-dinitrophenylhydr

294 Yields of 1-pentene and acetaldehyde were measured at 62 +/- 7% and 63 +/- 7%, r

295 Formaldehyde and acetaldehyde were the most abundant carbonyl compounds i

296 Annual average concentrations of acetaldehyde were underestimated by a factor of 10, risi

297 However, formaldehyde and acetaldehyde, which are formed in the air photochemicall

298 the two-electron/two-proton hydrogenation of acetaldehyde, which reverses the EtOH photooxidation rea

299 ethyl acetate, proceeds via the coupling of acetaldehyde with excess surface ethoxy.

300 o decline (e.g., benzene) or increase (e.g., acetaldehyde) with ethanol usage.