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1 organic compounds, VOCs (e.g., methanol and acetaldehyde).
2 2-knockdown human keratinocytes treated with acetaldehyde.
3 ing concentrations of free and sulfite-bound acetaldehyde.
4 tly decreased with the addition of exogenous acetaldehyde.
5 molecule that enables ALDH3A1 to metabolize acetaldehyde.
6 poised toward the production of ethanol from acetaldehyde.
7 es is detailed using the example of indole-3-acetaldehyde.
8 o our great surprise) restricts diffusion of acetaldehyde.
9 or acetone but a net source for methanol and acetaldehyde.
10 ting it to ammonia, inorganic phosphate, and acetaldehyde.
11 ic effects of ethanol-an exogenous source of acetaldehyde.
12 hough still with a 3-fold underestimation in acetaldehyde.
13 ct CH(2) horizontal lineCHOH tautomerizes to acetaldehyde.
14 oducts were 1-hexene, CO, vinyl alcohol, and acetaldehyde.
15 dative deamination of histamine to imidazole acetaldehyde.
16 boligation reaction between benzaldehyde and acetaldehyde.
17 ic mechanisms of ethanol and its metabolite, acetaldehyde.
18 thanol metabolism and was likely mediated by acetaldehyde.
19 ndicating the effect was in part mediated by acetaldehyde.
20 catalyst) or the aldol process starting from acetaldehyde.
21 thanol metabolism and was likely mediated by acetaldehyde.
22 lyzes the reversible oxidation of alcohol to acetaldehyde.
23 oacetaldehyde was oxidized more rapidly than acetaldehyde.
24 ction between L-cysteine and 5-hydroxyindole acetaldehyde.
25 uld be reproduced by histamine and imidazole acetaldehyde.
26 1) surface is intrinsically selective toward acetaldehyde.
27 ological conditions, preventing a buildup of acetaldehyde.
28 c agent recruited another ALDH to metabolize acetaldehyde.
29 d us to describe reaction of epicatechin and acetaldehyde.
30 s of nitrogen oxides (NOx), formaldehyde, or acetaldehyde.
31 5, usually interpreted as solely coming from acetaldehyde.
32 s, particularly nonaromatic amino acids, and acetaldehyde.
33 choline to produce trimethylamine (TMA) and acetaldehyde.
34 In this process, (tert-butyldimethylsilyloxy)acetaldehyde 1 was successfully utilized in two ways: as
35 y of the Julia-Kocienski olefination between acetaldehyde (1) and ethyl 1-phenyl-1H-tetrazol-5-yl sul
38 d VOCs such as methanol (5.39 pptv/ppbv CO), acetaldehyde (3.93 pptv/ppbv CO), acetone (3.59 pptv/ppb
39 sual AAAD enzyme products including indole-3-acetaldehyde, 4-hydroxyphenylacetaldehyde, and phenyleth
42 ate, and rapid analysis of ethanol (Eth) and acetaldehyde (AA) in a wide variety of beverages and foo
43 ed the effect of ethanol and its metabolite, acetaldehyde (AcAld), on total and ureagenic respiration
44 ans carry an inactive ALDH2 gene and exhibit acetaldehyde accumulation after alcohol consumption.
45 ic site-catalyzed ethanol dehydrogenation to acetaldehyde, acetaldehyde to acetone conversion via a c
46 as alpha-aryl-alpha,alpha-difluoroketones, -acetaldehydes, -acetates, and acetic acids, and difluoro
49 s was applied for a combination of six VOCs (acetaldehyde, acetone, acetic acid, hexanoic acid, hydro
51 e enhancement of HCV replication by ethanol, acetaldehyde, acetone, as well as acetate, whereas inhib
52 etermined over selected spectral regions for acetaldehyde, acetonitrile, ethanol, water, methanol, am
54 2, CO, total particulate mass, formaldehyde, acetaldehyde, acrolein, and several polycyclic aromatic
56 nant OSE adducts termed MAA (malondialdehyde-acetaldehyde-adducts), which are found on apoptotic cell
60 Hansenula sp. which quantitatively produces acetaldehyde after reaction for 120 min at 40 degrees C
62 erefore speculate that the second pathway in acetaldehyde also occurs via a roaming mechanism in the
72 surfaces has been derived from the study of acetaldehyde and dimethylamine in combination with previ
78 icting the alcoholic strength, the methanol, acetaldehyde and fusel alcohols content of grape-derived
79 ese findings suggest that alcohol, by way of acetaldehyde and its associated adducts, stimulates hepa
80 ence of ethanol, S. pneumoniae AdhE produced acetaldehyde and NADH, which subsequently led Rex (redox
83 ikely as a result of increased production of acetaldehyde and reactive oxygen species and mitochondri
86 opment of alcoholic pancreatitis, oxidative (acetaldehyde) and nonoxidative metabolites (ethyl palmit
87 1) surface is intrinsically selective toward acetaldehyde, and a strong inverse correlation between c
88 s OVOCs, including acetic acid, formic acid, acetaldehyde, and acetone were observed during photodegr
90 including acetate, reactive oxygen species, acetaldehyde, and epigenetic changes, that can induce in
91 ADH-mediated interconversions of acetyl-CoA, acetaldehyde, and ethanol but seemed to be poised toward
92 production was observed for glycolaldehyde, acetaldehyde, and formaldehyde only at elevated temperat
94 se 2 (ALDH2) is a key enzyme that eliminates acetaldehyde, and impairment of ALDH2 increases the risk
95 TRPV1 by histamine, its metabolite imidazole acetaldehyde, and supernatants from biopsy specimens was
96 e of buffer and air to produce formaldehyde, acetaldehyde, and the aldehydes corresponding to the bre
97 ips between glycolaldehyde and beta-mercapto-acetaldehyde, and the corresponding proteinogenic amino
98 enyl lactone of the southern portion with an acetaldehyde appendage on the cyclobutane of the norther
99 recombinative desorption of enolate and H as acetaldehyde are in good agreement with previously repor
102 raphy for liquid phase analysis, we identify acetaldehyde as a minor product and key intermediate in
104 ommonly produce ethanol from acetyl-CoA with acetaldehyde as intermediate and play a key role in anae
105 ncluding the sulfur-containing compounds and acetaldehyde, as well as lipid oxidation derived odorant
106 BRCA2-null cells for the ethanol metabolite, acetaldehyde, associated with widespread chromosomal bre
108 ross-coupling of methanol with formaldehyde, acetaldehyde, benzaldehyde and benzeneacetaldehyde to me
109 thought to retain the volatile intermediate acetaldehyde but allow diffusion of the much larger cofa
110 -Phe substitution increases turnover rate of acetaldehyde but decreases turnover rate of larger aldeh
111 ted chemical species, dominantly ammonia and acetaldehyde, but also two new species previously not re
112 2-deficient DT40 cells are hypersensitive to acetaldehyde, but not to acrolein, crotonaldehyde, glyox
117 converted glucose to ethanol via acetate and acetaldehyde, catalyzed by the host-encoded aldehyde fer
121 erature-dependent adsorption and reaction of acetaldehyde (CH(3)CHO) on a fully oxidized and a highly
122 y that interstellar aldehydes and enols like acetaldehyde (CH3CHO) and vinyl alcohol (C2H3OH) act as
124 of these proteins was AdhE, a bi-functional acetaldehyde-CoA dehydrogenase and alcohol dehydrogenase
125 e is primarily due to a mutated bifunctional acetaldehyde-CoA/alcohol dehydrogenase gene (adhE), hypo
130 ethanol emissions lead to higher atmospheric acetaldehyde concentrations (by up to 14% during winter
132 ements in urban areas may have overestimated acetaldehyde concentrations at times due to this interfe
134 nexpectedly, to hypothermia, increased blood acetaldehyde concentrations, and enhanced lethality.
137 tive for the aerobic oxidation of ethanol to acetaldehyde (conversion 100%; yield approximately 95%).
138 rds representing different carbonyl classes, acetaldehyde could be ionized only after labeling and MS
140 on employs a commercially available reagent, acetaldehyde-d4, to label the amine groups on the monoam
142 ator LasR and redox-regulated activities for acetaldehyde dehydrogenase ExaC, arginine deiminase ArcA
144 ter has two important functions: detoxifying acetaldehyde derived from dietary ethanol [11] and detox
147 s-links arising from the crotonaldehyde- and acetaldehyde-derived R- and S-alpha-CH3-gamma-OH-1,N2-pr
149 were somewhat more efficient than ozone for acetaldehyde destruction, ozone was more efficient for a
151 he formation of the major flavour compounds (acetaldehyde, diacetyl, acetoin, and 2-butanone) followe
153 etaldehyde direction), increased rapidly but acetaldehyde did not rise because of its oxidation to ac
154 t 30 degrees C, the ADH activity (ethanol to acetaldehyde direction), increased rapidly but acetaldeh
158 y for the gas-phase SN2 reaction between the acetaldehyde enolate anion and methyl fluoride, for both
161 tory quotient 1.5 (DCA-RQ 1.5) increased the acetaldehyde, ethanol and ethyl acetate concentration, r
162 n for the study were ethyl acetate, acetone, acetaldehyde, ethanol, ethylene glycol, dimethylsilanedi
163 harboring putative genes for a bifunctional acetaldehyde/ethanol dehydrogenase (Aad), serine/threoni
164 cid and all of the major volatiles excepting acetaldehyde, ethyl acetate and acetoine, whereas the ap
165 ction of seed tannins, exhibited the highest acetaldehyde, ethyl acetate and C6-compounds levels, and
166 octanoate, butyrolactone, isoamyl alcohols, acetaldehyde, ethyl acetate, 2,3-butanediol, acetoin and
167 of ethanol to its carbonyl compounds, namely acetaldehyde, ethyl acetate, acetic acid, and ketene, oc
168 higher amounts, with increased citronellol, acetaldehyde, ethyl acetate, dicarboxylic acids esters,
169 nation of six toxic compounds (formaldehyde, acetaldehyde, ethyl carbamate, furan, furfural and acrol
175 ycles for nonmethane organic gases, ethanol, acetaldehyde, formaldehyde, acetone, nitrous oxide, nitr
176 nature; the same trend of increased ethanol, acetaldehyde, formaldehyde, and CH4 emissions and decrea
178 , total hydrocarbons (THC), methane, ethene, acetaldehyde, formaldehyde, ethanol, N2O, and NH3 from a
179 nalysis was applied for the combined VOCs of acetaldehyde, formaldehyde, hydrogen sulphide, and methy
180 the fuel, the tailpipe emissions of ethanol, acetaldehyde, formaldehyde, methane, and ammonia increas
183 Reaction products of (-)-epicatechin with acetaldehyde formed in model solution were selected for
185 onal enzyme DmpFG channels its intermediate, acetaldehyde, from one active site to the next using a b
188 ncluding nicotine, nicotyrine, formaldehyde, acetaldehyde, glycidol, acrolein, acetol, and diacetyl.
190 idium-mediated dehydrogenation of ethanol to acetaldehyde has led to the development of an ethanol-to
191 the following 7 VOCs, acetone, formaldehyde, acetaldehyde, hexanoic acid, hydrogen sulphide, hydrogen
192 often treated with oxygen in order to yield acetaldehyde, however this approach can lead to unintend
193 r billion (ppb) or 8 ppb gas-phase MG and/or acetaldehyde in an aerosol reaction chamber for up to 5
199 show measurements of acetone, methanol, and acetaldehyde in the tropical remote marine boundary laye
205 TA or Ca(2+)-free medium blocked ethanol and acetaldehyde-induced barrier dysfunction and tight junct
206 eine and cyclosporine A, blocked ethanol and acetaldehyde-induced barrier dysfunction and tight junct
207 -specific activator, Alda-1, Alda-89 reduced acetaldehyde-induced behavioral impairment by causing a
209 aI and attenuated EGF-mediated prevention of acetaldehyde-induced disruption of tight junctions.
211 ptides attenuated EGF-mediated prevention of acetaldehyde-induced increase in inulin permeability and
215 aV1.3 channels, by shRNA blocked ethanol and acetaldehyde-induced tight junction disruption and barri
217 cathepsin S to obtain a novel (2-arylphenoxy)acetaldehyde inhibitor, 2, with a 0.49 microM Ki value.
219 rnal coenzyme B12 and injecting its product, acetaldehyde, into the lumen, where it is degraded by th
223 w steady-state concentrations, and that free acetaldehyde is difficult to detect in alkaline solution
228 e smaller organic compounds such as acetone, acetaldehyde, isoprene, or cysteamine can be detected in
229 ospheric trace gases, methylglyoxal (MG) and acetaldehyde, known to be surface-active, can enhance ae
230 using a rapid reduction in blood ethanol and acetaldehyde levels after acute ethanol intoxication in
231 ucrose, and increasing fructose, glucose and acetaldehyde levels, which are potential contributors to
234 -) mice had higher levels of malondialdehyde-acetaldehyde (MAA) adduct and greater hepatic inflammati
235 These data strongly support the notion that acetaldehyde may be an essential contributor to the chro
236 ompounds (VOCs) such as ethylmercaptan (EM), acetaldehyde (MeCHO) and methyl ethyl ketone (MEK) among
238 function, but it dose-dependently increased acetaldehyde-mediated tight junction disruption and barr
239 m alcohol-treated mice had a greater rate of acetaldehyde metabolism and respiration when treated wit
240 onse to alcohol, suggesting that the greater acetaldehyde metabolism by isolated mitochondria from al
241 This study examined the impact of altered acetaldehyde metabolism through systemic transgenic over
247 ange (IQR) increases in prenatal exposure to acetaldehyde [odds ratio (OR) = 2.30; 95% CI: 1.44, 3.67
249 sion of CO and H2 into methane, ethanol, and acetaldehyde on the Rh (211) and (111) surfaces, chosen
250 igated the synergistic effect of ethanol and acetaldehyde on the tight junction integrity in Caco-2 c
253 creased supply of peroxyacetyl radicals from acetaldehyde oxidation, and the lower NO(x) emissions fo
255 nserve certain volatile metabolites-CO(2) or acetaldehyde-perhaps by providing a low-pH compartment.
257 The addition of a coupling reaction removing acetaldehyde produced from the alcohol dehydrogenase (AD
258 at H4IIEC3 cells, respectively, dependent on acetaldehyde production, oxidative stress, and zinc rele
260 us direct identification and quantitation of acetaldehyde, pyruvic acid, acetoin, methylglyoxal, and
261 unds mainly responsible for trapping SO2 are acetaldehyde, pyruvic acid, and 2-oxoglutaric acid.
262 8), alcoholic strength (r(2)=97.2; RPD=6.0), acetaldehyde (r(2)=98.2; RPD=7.5) and fusel alcohols (r(
267 the CO produced in the 308-nm photolysis of acetaldehyde show clear evidence of two dissociation mec
268 trate that both PD20 and UM are sensitive to acetaldehyde, supporting a role for FANCD2 in repair of
269 These results demonstrate that ethanol and acetaldehyde synergistically disrupt tight junctions by
270 s thaliana and Petroselinum crispum aromatic acetaldehyde synthases primarily converts the enzymes ac
272 er amounts of ethanol, but larger amounts of acetaldehyde, than biofilms formed by the parent and rev
273 ated activation barriers for the coupling of acetaldehyde, the decomposition of the dimer state, and
276 zed ethanol dehydrogenation to acetaldehyde, acetaldehyde to acetone conversion via a complex pathway
280 catalyze the key aldol coupling reaction of acetaldehyde to exclusively yield the C4 coupling produc
282 ay be attributed to the adsorption of MG and acetaldehyde to the gas-aerosol interface, leading to su
287 addition of (E)-2-butenyltrimethylsilane to acetaldehyde under electrophilic (BF3, H3O(+)) and nucle
288 ds of ethane and convert it into ethanol and acetaldehyde using nitrous oxide as the terminal oxidant
290 F-mediated protection of tight junction from acetaldehyde was evaluated in Caco-2 cell monolayers.
292 Mean mixing ratios of acetone, methanol, and acetaldehyde were 546 +/- 295 pptv, 742 +/- 419 pptv, an
293 , alpha-ketoglutarate, pyruvate, acetoin and acetaldehyde were derivatised with 2,4-dinitrophenylhydr
298 the two-electron/two-proton hydrogenation of acetaldehyde, which reverses the EtOH photooxidation rea
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