1 lemented with 0.025% Silwet L-77 and 200 mum acetosyringone.

2 ere strongly induced by the vir gene inducer acetosyringone.

3 ription of a virG-lacZ fusion in response to acetosyringone.

4 pckA gene is induced by acidic pH but not by acetosyringone.

5                           On the other hand, acetosyringone and several other phenolic compounds indu

6                                              Acetosyringone (AS) induced the expression of all known

7 lant-bacterium interface: phenolics, such as acetosyringone (AS), monosaccharides, and acidic pH ( ap

8 TG)-inducible Plac and other virB genes from acetosyringone (AS)-inducible PvirB; (ii) a delta virB o

9 activates VirG in response to high levels of acetosyringone (AS).

10 n of its Ti plasmid virulence (vir) genes by acetosyringone (AS).

11 hese conditions are the usual induction with acetosyringone at pH 5.5 of Ti-plasmid vir genes.

12 with the strongest vir gene inducers such as acetosyringone being demethylated extremely slowly.

13 on induction of the virB and virD operons by acetosyringone but was not dependent upon induction of t

14 ompeting with the inducing phenolic compound acetosyringone for interaction with VirA.

15 regulon) be induced by plant signals such as acetosyringone in an acidic environment.

16 d genes were induced by the vir gene inducer acetosyringone, two of which are conserved in the nopali