ライフサイエンスコーパス: acetyl group

1 to assign unambiguously the position of the acetyl group.

2 ant hydroxylamine was then protected with an acetyl group.

3 degradation through their N(alpha)-terminal acetyl group.

4 ghtly higher than that for the removal of an acetyl group.

5 ccommodate L-carnitine as an acceptor of the acetyl group.

6 ly of phospholipases A2 that remove the sn-2 acetyl group.

7 ate is not due to steric interference by the acetyl group.

8 stent ~3.1 degrees C decrease in Tm per 2'-O-acetyl group.

9 ction other than the biologically relevant N-acetyl group.

10 ay result from Ile-313 packing against the N-acetyl group.

11 omers due to intramolecular migration of the acetyl group.

12 (dG-C8-AAF) adducts that differ by a single acetyl group.

13 bunit is on average substituted with three O-acetyl groups.

14 nitro, trifluoromethyl, fluoro, chloro, and acetyl groups.

15 tion, alpha1 and alpha2 contained N-terminal acetyl groups.

16 e labeled methyl and carbonyl carbons of the acetyl groups.

17 ey were substituted with from zero to four O-acetyl groups.

18 chiometrically substituted with up to four O-acetyl groups.

19 starch more hydrophobic by the insertion of acetyl groups.

20 2 promotes p53 degradation by removing these acetyl groups.

21 and each mutant produced alginate lacking O-acetyl groups.

22 A PS were specific for epitopes involving O-acetyl groups.

23 hat have the capacity to modify core histone acetyl groups.

24 serotyped, and had marked reduction in GXM O-acetyl groups.

25 thylation may lead to the decomposition of O-acetyl groups.

26 ncluding variations such as N-glycolyl and O-acetyl groups.

27 hydrolyze long-chain fatty acyl groups over acetyl groups.

28 esterase (NanS) to confirm the presence of O-acetyl groups.

29 V-1 integrase inhibitor containing aryl di-O-acetyl groups (Acetylated-Inhibitor).

30 Subsequent to the transfer of the acetyl group, acetylated aminoglycoside is released prio

31 r in the cytoplasm, (ii) the export of the O-acetyl group across the cytoplasmic membrane, and (iii)

32 Thus, a 9-O-acetyl group added to the side chain of cell surface sia

33 some cell types such Sia residues can have O-acetyl groups added to them.

34 ndeed, enzymatic removal of cell surface 9-O-acetyl groups alters the tissue distribution of intraven

35 assembles the thiazole functionality from an acetyl group and l-cysteine via condensation, cyclizatio

36 imetry titration, indicating that both the N-acetyl group and sugar type may be essential for donor s

37 intramolecular hydrogen bonds between the N-acetyl group and the protein backbone are an important i

38 generated antibodies to both backbone and O-acetyl groups and (ii) O-acetylated isolates were opsoni

39 Binding was enhanced by removal of 9-O-acetyl groups and attenuated by truncation of the glycer

40 (HDACs) are a family of enzymes that remove acetyl groups and control the acetylation status of hist

41 There were fewer O-acetyl groups and more phosphoethanolamine and sialic ac

42 ions between the degree of substitution with acetyl groups and properties of the modified preparation

43 d histone deacetylases (HDACs), which remove acetyl groups and silence transcription.

44 n isolated native oligomers with 13C labeled acetyl groups and the extraction of orientational constr

45 cally, the STM was used to cleave individual acetyl groups and to form Au-S bonds by manipulating sin

46 e, where d18:1 is sphingosine and C2:O is an acetyl group) and d18:1-C6:0 (C6-ceramide, where C6:O is

47 ygen to the Pd center, (4) hydrolysis of the acetyl group, and (5) reductive elimination of Pd(0), wh

48 ungal genes encoding enzymes that metabolise acetyl groups, and with genes for other hydrolyases such

49 orrelated with protection against disease, O-acetyl groups appear to be important in protection.

50 Although N-acetyl groups are common in nature, N-glycolyl groups ar

51 d their equatorial orientation but not the N-acetyl groups are critical for IRBC binding.

52 acyl esters, and tetra-acyl sucroses have an acetyl group as one of the acyl chains.

53 phosphates, (ii) the release of small excess acetyl groups as acetylcarnitine and ketone bodies, and

54 ation of the biaryl axis and the presence of acetyl groups as important structural elements for the b

55 it contains a carbonyl oxygen instead of an acetyl group at C17 on the steroid D ring.

56 The acetyl group at C17, but not that at C3, was essential f

57 troduction of small alkyl substituents or an acetyl group at C3 is also tolerated.

58 To accommodate the additional N-acetyl group at the C2 position of the sugar, the side c

59 The 2'-O-acetyl group at the ligation junction of the product RNA

60 O-acetylated ManNAc residues that contained acetyl groups at O-3, with some species acetylated at O-

61 orm the L-guluronates and by the addition of acetyl groups at the 0-2 and 0-3 positions.

62 indicate that GlcNAc residues on WTAs and O-acetyl groups at the 6-position of muramic acid residues

63 xylose has a hydroxyl group (instead of an N-acetyl group) at C2 and lacks the CH(2)OH group at C5; t

64 ed of a trisaccharide repeat unit with one O-acetyl group attached to each repeat unit.

65 has also been established that PDC flux and acetyl group availability limits aerobic ATP re-synthesi

66 pyruvate dehydrogenase complex activation or acetyl group availability occurred during the first 20 s

67 urately predicting which proteins receive an acetyl group based on their protein sequence is expected

68 -acylthio substituent, the benzoyl group and acetyl groups being removed 50 and 5 times as fast as th

69 and catalyzes the reversible transfer of an acetyl group between the alpha-amino groups of ornithine

70 tor-binding site on beta4Gal-T1, while the N-acetyl group-binding pocket in beta4Gal-T1 adjusts to ma

71 sugar-acceptor binding site, a hydrophobic N-acetyl group-binding pocket is found, formed by residues

72 ed-Control) that also contained an aryl di-O-acetyl group but did not inhibit HIV-1 integrase.

73 A that gets subsequently converted into a 3-acetyl group by 3-vinyl bacteriochlorophyllide a hydrata

74 lkyl group; (ii) catalyze the removal of the acetyl group by cleaving an amide bond; (iii) increase t

75 deacetylase enzymes catalyze removal of the acetyl group by hydrolysis or by an NAD(+)-dependent rea

76 ides that results in the exchange of the 4-O-acetyl group by the glacial acetic acid that serves as e

77 OPV structures and can be converted to the S-acetyl group by treatment with sodium thiomethoxide and

78 lly active chromatin, whereas the removal of acetyl groups by histone deacetylases (HDACs) correlates

79 The presence of hydrophobic acetyl groups, carbonyl and carboxyl groups caused a par

80 thin 3.9 A of five aromatic residues and its acetyl group close to residues C187/188 of the principle

81 tural changes arise from rotations of the C3-acetyl group conformation and hydrogen bonding.

82 Ethynyl --> acetyl group conversion was also studied on ethynylated

83 ted, for the first time, the existence of an acetyl group deficit at the onset of contraction and has

84 d (P < 0.01), respectively, and overcame the acetyl group deficit at the onset of contraction.

85 ism by which dichloroacetate overcomes this "acetyl group deficit".

86 P re-synthesis at the onset of exercise (the acetyl group deficit).

87 ally decline (indicating the existence of an acetyl group deficit).

88 east in part attributable to a limitation in acetyl group delivery/availability at the onset of exerc

89 We examined the role of acetyl group-dependent and -independent epitopes on the

90 uents such as the fluoro, nitro, chloro, and acetyl groups do not.

91 losamine, utilizing acetyl-coenzyme A as the acetyl group donor.

92 these compounds are possible candidates for acetyl group-donors of 2AP, predominately in the form of

93 that develops on the carbonyl oxygen of the acetyl group during both the formation of the acyl-enzym

94 to encode acetyltransferases, which transfer acetyl groups either to internal lysine residues or to t

95 rgo two distinct steps, beginning with the N-acetyl group expelling water from the donor.

96 protease cathepsin L (CTSL) that remove the acetyl group first and then the unacetylated lysine grou

97 ethods, this synthetic approach introduces O-acetyl groups first, followed by N-acylation.

98 ha-azido may be inherently preferable to the acetyl group for N-terminal protection.

99 pecificity, we suggest that the bulky serine acetyl group forces a realignment of the omega end of th

100 Pure, acetyl group free oligonucleotides were isolated after d

101 the activated complex is the transfer of the acetyl group from acetyl CoA to the acceptor lysine resi

102 ic enzymes that catalyze the transfer of the acetyl group from acetyl coenzyme A (AcCoA) to the free

103 ric enzyme that catalyzes the transfer of an acetyl group from acetyl coenzyme A to polyamines such a

104 se gene product catalyzes the transfer of an acetyl group from acetyl coenzyme A to the C3 hydroxyl m

105 .3.1.8) catalyzes reversible transfer of the acetyl group from acetyl phosphate to coenzyme A (CoA),

106 .8) catalyzes the reversible transfer of the acetyl group from acetyl phosphate to coenzyme A (CoA),

107 .8) catalyzes the reversible transfer of the acetyl group from acetyl phosphate to coenzyme A (CoA):

108 re enzymes that catalyse the transfer of the acetyl group from acetyl-CoA to a primary amine.

109 that catalyze the reversible transfer of an acetyl group from acetyl-CoA to choline and L-carnitine,

110 Acetyltransferase enzymes transfer the acetyl group from acetyl-CoA to lysine residues, while d

111 GCN5 catalyzes the transfer of an acetyl group from acetyl-CoA to the epsilon-amino of lys

112 ferase (EaDAcT) catalyzes the transfer of an acetyl group from acetyl-CoA to the sn-3 position of dia

113 yme was shown to catalyse the transfer of an acetyl group from acetyl-CoA to the terminal nitrogen of

114 ve been shown to catalyze the transfer of an acetyl group from acetyl-coenzyme A (Ac-CoA) to the amin

115 nal helix, such as removal of the N-terminal acetyl group from AcTM1aZip or striated muscle alpha-TM,

116 tylase 6 (HDAC6) catalyzes the removal of an acetyl group from lysine residues of several non-histone

117 le to catalyse the reversible transfer of an acetyl group from N-acetylornithine to glutamate, but no

118 show that LsrF catalyzes the transfer of an acetyl group from P-HPD to coenzyme A yielding dihydroxy

119 fied two enzyme activities that transfer the acetyl group from platelet-activating factor (PAF) in a

120 e deacetylases catalyze the hydrolysis of an acetyl group from post-translationally modified acetyl-l

121 ent enzymes catalyze a reaction in which the acetyl group from substrate is transferred to the ADP-ri

122 ccurs first, followed by the transfer of the acetyl group from the donor substrate to the ADP-ribose

123 ent protein/histone deacetylation, where the acetyl group from the lysine epsilon-amino group is tran

124 cetylase (HDAC) catalyzes the removal of the acetyl group from the lysine residues in the N-terminal

125 ent in membranes of F. novicida, removes the acetyl group from undecaprenyl phosphate-N-acetylgalacto

126 llular function by catalyzing the removal of acetyl groups from -N-acetylated lysine residues of vari

127 T) are two families responsible for removing acetyl groups from acetylated proteins.

128 transferase (SSAT) catalyzes the transfer of acetyl groups from acetylcoenzyme A to spermidine and sp

129 deacetylases (HDACs) catalyze hydrolysis of acetyl groups from acetyllysine side chains and are targ

130 (HDACs) are known to catalyze the removal of acetyl groups from both histones and non-histone protein

131 mily of enzymes that catalyze the removal of acetyl groups from core histones, resulting in a compact

132 gene expression by catalyzing the removal of acetyl groups from core histones.

133 r the enzyme is unknown, HerE hydrolyzes the acetyl groups from heroin to yield morphine and from phe

134 press gene expression through the removal of acetyl groups from histone tails.

135 one deacetylases (HDACs) are known to remove acetyl groups from histone tails.

136 e of biological processes through removal of acetyl groups from histones as well as non-histone prote

137 By removal of acetyl groups from histones, HDACs create a non-permissi

138 press gene expression through the removal of acetyl groups from histones.

139 A second enzyme that removes acetyl groups from lysine residues in E. coli been disco

140 They regulate gene expression by removing acetyl groups from lysine residues in histone tails, res

141 Cs) are enzymes that catalyze the removal of acetyl groups from lysine residues of histone and nonhis

142 Histone deacetylases (HDACs) remove acetyl groups from lysine residues on histone tails, pro

143 atively regulate gene expression by removing acetyl groups from lysine residues present in histones a

144 nzymatic activity, preventing the removal of acetyl groups from lysine residues within histone tails.

145 as nonhistones by catalyzing the removal of acetyl groups from lysine residues.

146 eins (84% identity) that catalyze release of acetyl groups from modified N-terminal lysines of core h

147 tained by selective enzymatic removal of 9-O-acetyl groups from sialic acids.

148 Neisseria gonorrhoeae functions to release O-acetyl groups from the C-6 position of muramoyl residues

149 Indeed, following selective removal of 9-O-acetyl groups from the cell surface by a specific estera

150 Gram-negative bacteria, the translocation of acetyl groups from the cytoplasm is performed by an inte

151 deacetylases (HDACs) catalyse the removal of acetyl groups from the N-terminal tails of histones.

152 harbors the activity that cleaves side chain acetyl groups from xylan-like substrates, and the bindin

153 malonyl and succinyl groups, rather than an acetyl group, from lysine residues.

154 tant strain that produced alginate lacking O-acetyl groups gave an amide II signal approximately five

155 tion of the GD3, de-N-acetylation of the 5-N-acetyl group (giving de-N-Ac-GD3).

156 The absence of the NodL-determined acetyl group greatly reduced the induction of calcium in

157 tide deformylase for the N-formyl over the N-acetyl group has been studied with N-alpha-fluoroacetyl

158 MBPS-protein conjugate vaccines, in which N-acetyl groups have been replaced by propionyl groups (N-

159 hough the steady-state abundances of histone acetyl groups have been reported, the rate at which hist

160 The presence of the sn-3 acetyl group imparts acTAGs with different physical and

161 and 1,250 cm(-1) (C---O stretching of the O-acetyl group in alginate), as compared to biofilms of no

162 and O-acetyl groups in EPS-1 and a single O-acetyl group in EPS-2.

163 bel from water to the carbonyl oxygen of the acetyl group in OAADPr is consistent with water addition

164 duction by p53 inactivates p53 by removal of acetyl group in p53 molecule, thus functioning as an aut

165 ed acyl migration-resistant analogs using an acetyl group in place of the free hydroxyl group in orde

166 were traced to the absence of the N-terminal acetyl group in rWT that participates in an H-bond to th

167 t of native N-acetyl groups with 13C-labeled acetyl groups in a simple disaccharide derivative, (GlcN

168 substituent groups: 1-phosphoglycerol and O-acetyl groups in EPS-1 and a single O-acetyl group in EP

169 The method relies on the replacement of acetyl groups in isolated native oligomers with 13C labe

170 ffect, indicating the critical role of the N-acetyl groups in the stimulation of proinflammatory cyto

171 The extent of the introduction of acetyl groups increased with increasing iodine levels as

172 emonstrated directly that the absence of the acetyl group increases the susceptibility of succinoglyc

173 immunogenicity associated with removal of O-acetyl groups indicates that O acetylation is essential

174 The assignment of the two acetyl groups introduced is based on a novel combination

175 tional role of this modification and how the acetyl group is added to Lys-290 was unclear.

176 , but in all structures the binding of the N-acetyl group is conserved.

177 tiply acetylated residues, but in which each acetyl group is isotopically labeled, permit studies of

178 The 3-acetyl group is required to form LH1 in both bacteria, a

179 as expressed with a native level (>90%) of N-acetyl groups (MAbs F628 and F630).

180 ide group in this class of compounds with an acetyl group maintained the in vitro binding affinity an

181 An efficient intramolecular acetyl group migration from the C7 to C9-position of the

182 Nod-factors made by the nodL mutant lack an acetyl group; mutation of nodF causes the nitrogen (N)-l

183 white matter yielded metabolite ratios of N-acetyl groups (NA), choline, and creatine.

184 by positioning the carbonyl oxygen of the N-acetyl group near to the C1 atom.

185 ation of an active taxol conformation by the acetyl group nor the formation of an important taxol con

186 ied enzyme catalyzed transacetylation of the acetyl group not only from PAF to lysoplasmalogen formin

187 rphyrin IX with strongly electron-polarizing acetyl groups not only leads to much lower meso-carbon r

188 proteins found in chromatin, by addition of acetyl groups occurs to a greater degree when the histon

189 The N-acetyl group of 4 was removed by hydrolysis with ethanol

190 s a ping-pong kinetic mechanism in which the acetyl group of acetyl-CoA is initially transferred to a

191 ct nucleophilic attack on the si-face of the acetyl group of acetyl-CoA.

192 uggested significant interaction between the acetyl group of acetylcholine and the alphaY93 residue.

193 In this study the N-acetyl group of BANA 113 was replaced with a 2-pyrrolidi

194 itates a keto-enol transition of the leaving acetyl group of EDTA.

195 gs suggests that Ser942 interacts with the 7-acetyl group of forskolin.

196 dditional hydrogen bond formed between the N-acetyl group of GalNAc moiety with the Tyr-289 side chai

197 s various N-acyl groups in addition to the N-acetyl group of GlcNAc.

198 de-N-acetylase capable of hydrolyzing the N-acetyl group of Lc(3)Cer was detected in bovine brain ex

199 ctive site lysine and the elimination of the acetyl group of O-acetyl-L-serine (OAS) to form the alph

200 CysM catalyzes the replacement of the acetyl group of O-acetylserine by CysO thiocarboxylate t

201 n 199 serves to hydrogen bond with the C2' N-acetyl group of sugar D, thus preventing the formation o

202 hevamine, the carbonyl oxygen atom of the N-acetyl group of the -1 sugar residue has rotated away fr

203 p of reactive Lys12 for direct attack of the acetyl group of the cofactor.

204 The acetyl group of the muscarinic agonist aceclidine 4 was

205 e regioisomer-selective interaction with the acetyl group of the stabilized regioisomers.

206 TL2)) forms no direct interaction with the N-acetyl group of the UDP-N-acetylhexosamine.

207 iosynthesis, catalyzing the removal of the N-acetyl group of UDP-3-O-(R-3-hydroxymyristoyl)-N-acetylg

208 ysis of the benzoyl group of cocaine and the acetyl groups of 4-methylumbelliferyl acetate, heroin, a

209 ng region displayed modes indicating that 3C-acetyl groups of BChl a are all involved in molecular in

210 To determine the role of the acetyl groups of low molecular mass hyaluronan in stimul

211 have an effect on the conformation of the C3-acetyl groups of the B820 molecules.

212 Acetyl groups of unsaturated heparin disaccharides are w

213 n-391 in UGT3A1 is able to accommodate the N-acetyl group on C2 of UDP-GlcNAc so that the anomeric ca

214 of C1 and C3 configuration adjacent to the N-acetyl group on coupling behavior.

215 Mg(2+) on tRNA dynamics with and without an acetyl group on the A-site tRNA.

216 undertaken to determine the effect of the N-acetyl group on the chemistry of such compounds.

217 perties of the carbonyl moiety in the 17beta-acetyl group on the D-ring of the anesthetic steroids (3

218 ural occurrences of galactosamine lacking an acetyl group on the nitrogen, has been identified as a p

219 The presence of the acetyl group on the phenolic oxygen and the use of silve

220 inone ring was constructed in place of the N-acetyl group on the sialic acid.

221 we show that mitochondria provide 50% of the acetyl groups on H4-K16.

222 uctural features including the location of O-acetyl groups on sialic acid (SA) moieties.

223 deacetylases (HDACs) catalyze the removal of acetyl groups on the amino-terminal lysine residues of c

224 Substitution of N-propionyl (N-Pr) for N-acetyl groups on the meningococcal B polysaccharide, and

225 one acetylation by catalyzing the removal of acetyl groups on the NH(2)-terminal lysine residues of t

226 to further investigate the effect of 2'/3'-O-acetyl groups on the stability of RNA duplex structure,

227 etyl-CoA), and catalyses the transfer of the acetyl group onto a specific lysine residue (K238).

228 d the dynamic incorporation of (13)C-labeled acetyl groups onto specific histone lysines with quantit

229 ture, being different only in their relative acetyl group orientations, cis (gamma' approximately 180

230 The presence of O-acetyl groups plays an important role in the ability of

231 , i.e. whether a limitation in mitochondrial acetyl group provision exists.

232 -methyl groups, whereas abstraction from the acetyl group represents a minor pathway, in line with th

233 The N-acetyl group slows down N-O bond cleavage by a factor of

234 rom oxidation of the methyl group and the N6-acetyl group stems from the methyl group and the adjacen

235 rationalized by the greater capacity of the acetyl group than the benzoyl group to conjugate with th

236 contributing >50% of the fatty acid-derived acetyl groups that end up in malonyl-CoA.

237 Although G-AF and G-AAF differ by only an acetyl group, they exert different effects on DNA replic

238 uished only by the presence or absence of an acetyl group, they have profoundly different effects on

239 ch are metabolized into acetyl-coA, labeling acetyl groups through subsequent incorporation into prot

240 ic membrane, and (iii) the transfer of the O-acetyl group to a periplasmic protein or to alginate.

241 CH(3) bond and the transfer of the resulting acetyl group to CoA.

242 from acetate and ATP and the transfer of the acetyl group to CoA.

243 idine acetyltransferase, which transfers the acetyl group to either the N-1 or N-8 position.

244 silon-amino group on AcCoA, transferring the acetyl group to H3 lysine residues.

245 the antibody (K(Ab)) on even addition of an acetyl group to P(1) of the peptide, the antibody fails

246 e carbonyl carbon of AcCoA, transferring the acetyl group to the acceptor peptide substrate (rate-lim

247 acyl carrier protein domain, transfer of the acetyl group to the acyl carrier protein domain is suppr

248 and undergo light-responsive addition of an acetyl group to the alpha-amino group of the amino-termi

249 Addition of an acetyl group to the amino terminus of peptide 145-159 al

250 rocess that can be blocked by addition of an acetyl group to the amino terminus of the peptide.

251 wn to transfer an 18O label from the peptide acetyl group to the ribose 1'-position of OAADPr, provid

252 I) triggers metal-mediated hydrolysis of the acetyl groups to afford a large, rapid, and zinc-induced

253 known to be essential for the addition of O-acetyl groups to alginate.

254 cetylase (HDAC) proteins that add and remove acetyl groups to and from target lysine residues within

255 strategy employs "light" and "heavy" labeled acetyl groups to block both N-termini and lysine residue

256 groups to DNA cytosine bases, of methyl and acetyl groups to proteins (histones) around which DNA is

257 YopJ catalyzes the transfer of acetyl groups to serine, threonine, and lysine residues

258 Covalent modification adding acetyl groups to the C terminus of the p53 protein has b

259 acetyltranferase involved in the transfer of acetyl groups to the glycopeptide core.

260 The importance of O-acetyl groups to the immunogenicity of Neisseria meningi

261 t the polymer level after the transport of O-acetyl groups to the periplasm.

262 mine N-acetyltransferases (NATs) catalyze an acetyl group transfer from AcCoA to primary arylamines,

263 mine N-acetyltransferases (NATs) catalyze an acetyl group transfer from acetyl coenzyme A (AcCoA) to

264 d with the lipoyl domains and intramolecular acetyl group transfer in the mechanism of active-site co

265 cotinamide-ribosyl cleavage is distinct from acetyl group transfer to the 2'-OH ribose.

266 des methyl group transfer, CO insertion, and acetyl group transfer, fitting to experimental traces re

267 chanism of nicotinamide-ribosyl cleavage and acetyl group transfer.

268 eactions, one of which involves catalysis of acetyl group transfer.

269 stone acetyltransferases (HATs) catalyze the acetyl-group transfer from acetyl-CoA to the epsilon-ami

270 A rhodium-catalyzed intramolecular acetyl-group transfer has been achieved through a "cut a

271 Decarboxylation precedes acetyl-group transfer, leading to acetyl-ACP(L) as the k

272 te in malonyl-CoA decarboxylation but not in acetyl-group transfer.

273 minal adapter domain was shown to facilitate acetyl-group transfer.

274 eavage of an amide bond, and transfer of the acetyl group ultimately to the 2'-ribose hydroxyl of ADP

275 Deprotection of the acetyl groups under basic conditions furnishes the free

276 TIPS-ethynyl group (via TIPS-acetylene), or acetyl group (via tributyl(1-ethoxyvinyl)tin, followed b

277 ersion of the trichloroacetyl group to the N-acetyl group was achieved under mild conditions, fully c

278 n acetate leaving group with or without an N-acetyl group, was cytotoxic and displayed significant in

279 The carboxyl, carbonyl, acetyl groups were determined in modified starches.

280 Antibodies to both backbone and O-acetyl groups were found to be opsonic against S. aureus

281 d possessing one specific internal 2'- or 3'-acetyl group, were used as synthetic standards in a rece

282 ith mice also showed an essential role for O-acetyl groups, where serum bactericidal titers following

283 s stems from steric hindrance effects of the acetyl group which significantly diminish the adduct-bas

284 reased hydrophobicity associated with adding acetyl groups, while the specific effects are a conseque

285 Replacement of the transmitter's ester acetyl group with a hydroxyl (ACh-->choline) results in

286 de, a modified mannosamine that replaced the acetyl group with a levulinate group (ManLev).

287 alyzed exchange of the methyl protons of the acetyl group with solvent; exchange is dependent on the

288 binds in the S1 site, predominantly via the acetyl group with the oxygen and acetamide nitrogen hydr

289 ecies undergoes reductive elimination of the acetyl group with the simultaneous release of two, low p

290 ts thereof, one obtained by substituting the acetyl group with the sulfonic amino acid taurine (Tau-L

291 n presented involves replacement of native N-acetyl groups with 13C-labeled acetyl groups in a simple

292 Replacement of the N-acetyl groups with bulky N-acyl or N-benzoyl substituent