ライフサイエンスコーパス: acetylated LDL

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1 on (10-55%) compared with nonaggregated 125I-acetylated LDL that did not enter SCC.

2 However, similar to 125I-acetylated LDL degradation, 125I-VxLDL degradation occur

3 ated the binding of opsonized native LDL and acetylated LDL (acLDL) to the complement receptor 1 (CR1

4 ipoprotein (LDL), including oxidized LDL and acetylated LDL, but not native LDL.

5 cts, also increases SMC uptake of Ox-LDL and acetylated LDL, with either effect blocked by an excess

6 in (HDL), low density lipoprotein (LDL), and acetylated LDL (AcLDL).

7 elect-HA(TM), heparin, dermatan sulfate, and acetylated LDL stimulated dose-dependent HARE-mediated N

8 ry analysis for von Willebrand factor and by acetylated LDL uptake.

9 ial cell numbers as defined by day 4 culture acetylated LDL/lectin assay in either of these patient g

10 lls, which were shown to be >98% pure by diI-acetylated LDL uptake and nuclear counterstaining.

11 ulating ligands including HA, heparin (Hep), acetylated LDL (AcLDL), dermatan sulfate (DS), apoptotic

12 ssociation and degradation of (125)I-labeled acetylated LDL, a well characterized ligand for the SR-A

13 f lipoproteins, native LDL and modified LDL (acetylated LDL (AcLDL) and OxLDL) on the expression of C

14 ct of lipoproteins, native and modified LDL (acetylated LDL (AcLDL) and OxLDL) on the expression of t

15 , FLIP was not down-regulated by native LDL, acetylated LDL, LPC-C12:0, cholesterol, or 7-ketocholest

16 n in cells incubated with beta-VLDL, but not acetylated LDL.

17 thelial nitric oxide synthase and capable of acetylated LDL uptake).

18 s and were accompanied by functional loss of acetylated LDL-uptake and migratory capacity, and acquis

19 The majority of acetylated LDL(+)ulex-lectin(+) cells are derived from m

20 their phenotypes were confirmed by uptake of acetylated LDL and binding of ulex-lectin.

21 , CD31, CD14, and CD34 expression, uptake of acetylated LDL and secretion of type IV collagen.

22 ion of LDL, 25OH-cholesterol, oleic acid, or acetylated LDL to SW872 cells increased CETP secretion (

23 th oxidized low density lipoprotein (LDL) or acetylated LDL increased bile salt-stimulated cholestero

24 ritoneal macrophages with either oxidized or acetylated LDL significantly inhibits the growth of C pn

25 s modified forms of LDL, such as oxidized or acetylated LDL, and promotes ingestion by macrophages in

26 locked by an excess of either cold Ox-LDL or acetylated-LDL, and by fucoidin, an SR competitor.

27 ated these cells in medium containing oxLDL, acetylated LDL (acLDL), or native LDL, or on surfaces co

28 Our findings suggest that acetylated LDL(+)ulex-lectin(+) cells, commonly referred

29 d atheronals in a manner highly analogous to acetylated LDL rather than oxidized LDL.

30 flk-1, flt-1, tie-2, the capacity to take up acetylated LDL and the presence of cytoplasmic Weibel-Pa

31 those from control rats when incubated with acetylated LDL.

32 e loaded with cholesterol by incubation with acetylated LDL, labeled with 3H-cholesterol, and then in

33 holesterol deposition during incubation with acetylated LDL.

34 was further increased after stimulation with acetylated LDL.

35 THP-1 macrophages incubated with or without acetylated LDL (acLDL) +/- acyl-CoA:cholesterol O-acyltr

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