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1 nergic receptor (hbeta2AR) and M2 muscarinic acetylcholine receptor.
2 elicit protean agonism at the muscarinic M2 acetylcholine receptor.
3 allosteric modulator at the alpha7 nicotinic acetylcholine receptor.
4 mutation in RIC3; a chaperone for nicotinic acetylcholine receptors.
5 n glycolysis and by activation of muscarinic acetylcholine receptors.
6 eting the cannabinoid 1 and alpha7 nicotinic acetylcholine receptors.
7 by GABAA receptors in addition to nicotinic acetylcholine receptors.
8 ercome imidacloprid binding to the nicotinic acetylcholine receptors.
9 an be rescued by an antagonist of muscarinic acetylcholine receptors.
10 tein-coupled receptors, including muscarinic acetylcholine receptors.
11 line acting on both nicotinic and muscarinic acetylcholine receptors.
12 responses that result from poorly clustered acetylcholine receptors.
13 most frequent (78%), followed by ganglionic acetylcholine receptor (20%), voltage-gated Kv1 potassiu
14 hat the Chrm4 transcript encoding muscarinic acetylcholine receptor 4 (M4) is excessively translated,
15 aminergic D3 and D4 receptors and muscarinic acetylcholine receptor 4, the receptor tyrosine kinases
16 e address these topics for the M1 muscarinic acetylcholine receptor, a key molecular target for novel
17 at the neuromuscular junction, primarily the acetylcholine receptor (AChR) and the muscle-specific ki
18 ys (CBAs) were shown to improve detection of acetylcholine receptor (AChR) antibodies in patients wit
20 protein at synapse), which is essential for acetylcholine receptor (AChR) clustering and NMJ (neurom
21 hift mutation in the C2C12 cell line reduced acetylcholine receptor (AChR) clustering during myotube
22 iating MuSK/Dok-7/Crk/CrkL complex, regulate acetylcholine receptor (AChR) clustering in vitro, and a
23 s recently found to control the stability of acetylcholine receptor (AChR) clusters on the surface of
24 rior and a decrease in the expression of the acetylcholine receptor (AChR) epsilon subunit gene mRNA
25 thout detectable antibodies to the nicotinic acetylcholine receptor (AChR) or to muscle-specific tyro
26 xamined expression of genes involved in anti-acetylcholine receptor (AChR) response in MG, MHC class
28 -7, myogenin, E2-ubiquitin ligase UBE2Q1 and acetylcholine receptor (AchR), but not of MyoD, and of t
29 ons to this linker of the human adult-muscle acetylcholine receptor (AChR), the alpha3beta4 AChR and
30 currents recorded from the muscle nicotinic acetylcholine receptor (AChR), we have recently hypothes
32 er they produce autoantibodies targeting the acetylcholine receptor (AChR-MG) or muscle specific kina
34 uromuscular junction (NMJ) traps and anchors acetylcholine receptors (AChRs) at high density at the s
35 NT: Rapsyn is required for the clustering of acetylcholine receptors (AChRs) at postsynaptic sites.
36 d protein, is required for the clustering of acetylcholine receptors (AChRs) at synaptic sites betwee
38 ing the timing of expression of postsynaptic acetylcholine receptors (AChRs) impacts presynaptic rele
39 for the proper distribution of extrasynaptic acetylcholine receptors (AChRs) in Caenorhabditis elegan
40 f ALS have revealed defects in expression of acetylcholine receptors (AChRs) in skeletal muscle that
41 scular junction (NMJ) is the high density of acetylcholine receptors (AChRs) in the postsynaptic musc
44 ted autoimmune responses to muscle nicotinic acetylcholine receptors (AChRs) that impair neuromuscula
45 BLA principal neurons through activation of acetylcholine receptors (AChRs), (2) enhances glutamater
50 propose a new mechanism by which muscarinic acetylcholine receptors affect cognition and suggest tha
51 cline is a novel, selective alpha7 nicotinic acetylcholine receptor agonist in development for treati
52 ipts of several targets, including nicotinic acetylcholine receptor alpha 1 and alpha 2 subunit, the
53 g of rye reveals that it encodes a nicotinic acetylcholine receptor alpha subunit required for Drosop
56 regulation of splenic cholinergic nicotinic acetylcholine receptor alpha7 (nAChRa7) signaling in the
57 tory cytokines, adenosine, and the nicotinic acetylcholine receptor alpha7 subunit (alpha7nAChR).
59 stably overexpressing the alpha-7 nicotinic acetylcholine receptor (alpha7-nAChR), a potential thera
60 ormal and cancer cells, the alpha7 nicotinic acetylcholine receptor (alpha7-nAChR), was more highly e
61 ce suggests that activating alpha7 nicotinic acetylcholine receptors (alpha7 nAChR) may facilitate th
63 istinct, and human-specific alpha7-nicotinic acetylcholine receptor (alpha7nAChR) gene [CHRNA7 (gene-
64 ypothesized that agonism on alpha7 nicotinic acetylcholine receptor (alpha7nAChR) in fetal microglia
65 show that ILC2s express the alpha7-nicotinic acetylcholine receptor (alpha7nAChR), which is thought t
66 linergic fibers through the alpha7-nicotinic acetylcholine receptor (alpha7nAChR), whose activation d
70 ation of neural signals and alpha7 nicotinic acetylcholine receptors (alpha7nAChRs) on splenic macrop
71 ncreasing activation of the alpha7-nicotinic acetylcholine receptor, alter the development of behavio
72 llular domain of the native alpha7 nicotinic acetylcholine receptor and acetylcholine binding protein
73 s, RV inflammation, and RV alpha-7 nicotinic acetylcholine receptor and muscarinic acetylcholine type
75 sults from the combined action of ionotropic acetylcholine receptors and associated calcium-activated
76 ring culture, myobundles maintain functional acetylcholine receptors and structurally and functionall
78 Expression of nicotinic (alpha-7 nicotinic acetylcholine receptor) and muscarinic (muscarinic acety
80 is (neural cell adhesion molecule, nicotinic acetylcholine receptor, and p75 neurotrophin receptor),
81 by sensory nerve fibers expressing nicotinic acetylcholine receptors, and intraurethral application o
84 that scopolamine, a nonselective muscarinic acetylcholine receptor antagonist, produces rapid antide
85 that scopolamine, a nonselective muscarinic acetylcholine receptor antagonist, produces rapid antide
86 ergic input because application of nicotinic acetylcholine receptor antagonists impairs this firing p
87 alcium transients were blocked by muscarinic acetylcholine receptor antagonists, whereas during gluta
89 duals from North America diagnosed as having acetylcholine receptor antibody-positive myasthenia grav
90 eaningful improvements in patients with anti-acetylcholine receptor antibody-positive refractory gene
96 these, we identified CHRM3, a M3R muscarinic acetylcholine receptor, as being restricted to oligodend
98 cur within seconds of blocking or unblocking acetylcholine receptors at the mouse neuromuscular junct
99 of response to rituximab in the treatment of acetylcholine receptor autoantibody-positive (AChR+) gen
101 ough deletion of beta2 subunits of nicotinic acetylcholine receptors (beta2-nAChRs) selectively from
103 om molecular defects in the muscle nicotinic acetylcholine receptor, but they can also be caused by m
104 ed in reward, whereas blockade of muscarinic acetylcholine receptors by scopolamine suppresses intake
105 gene [CHRNA7 (gene-encoding alpha7-nicotinic acetylcholine receptor)] called CHRFAM7A (gene-encoding
108 that alters function of the alpha5 nicotinic acetylcholine receptor (CHRNA5) and noncoding SNPs that
109 ination/retraction, vesicle accumulation and acetylcholine receptor clustering and acetylcholinestera
112 ase in human and rat monocytes via nicotinic acetylcholine receptors containing subunits alpha7, alph
113 This work suggests that overstimulation of acetylcholine receptors could disrupt neuronal processin
114 identify two heteroallelic mutations in the acetylcholine receptor delta-subunit from a patient with
115 SS), or secondhand smoke, promoted nicotinic acetylcholine receptor-dependent exacerbation of AA and
116 in, but not imidacloprid, causes a nicotinic acetylcholine receptor-dependent rapid mitochondrial dep
119 ntiinflammatory effects via alpha7 nicotinic acetylcholine receptor-expressing splenic macrophages.
120 uated the increased M2 subtype of muscarinic acetylcholine receptor expression and Galphai coupling a
121 es are matched to a pattern of M2 muscarinic acetylcholine receptor expression at fixed locations of
122 upling and enhanced M3 subtype of muscarinic acetylcholine receptor expression in association with en
123 mary outcome variables) as well as nicotinic acetylcholine receptor expression, muscle mass, and hist
124 onsmokers following stimulation of nicotinic acetylcholine receptors (familywise error-corrected P <
125 myoblast differentiation and aggregation of acetylcholine receptors for the establishment of neuromu
126 bind to the binding pocket of the muscarinic acetylcholine receptor formed by transmembrane alpha-hel
127 rons receive cholinergic input via nicotinic acetylcholine receptors from the Kenyon cells; knocking
128 at most synaptic sites, induction of "fetal" acetylcholine receptor gamma subunit (AChRgamma), reduct
131 t of this hypothesis (local blockade of SubC acetylcholine receptors) has not been rigorously perform
133 homimetic Dok-7 mutants aggregated nicotinic acetylcholine receptors in C2C12 myotubes with significa
134 predict alcohol, and suggest that nicotinic acetylcholine receptors in the NAc are critical for this
135 h can also activate, potentiate, and inhibit acetylcholine receptors, including neuronal nicotinic re
137 rug scopolamine was used to block muscarinic acetylcholine receptors involved in working memory.
138 sts, long-lived open-channel blockers of the acetylcholine receptor ion channel, and adrenergic agoni
139 ies conducted on the extracellular domain of acetylcholine receptors, ion channels from prokaryote ho
141 unctions, clustering of levamisole-sensitive acetylcholine receptors (L-AChRs) requires the muscle-se
142 teric modulators (PAMs) of the M1 muscarinic acetylcholine receptor (M1 mAChR) are a promising strate
143 or to detect activation of the M1 muscarinic acetylcholine receptor (M1 mAChR) in vitro and in vivo M
144 ne receptors, particularly the M1 muscarinic acetylcholine receptor (M1 mAChR), which was previously
145 itulated with blockade of M1-type muscarinic acetylcholine receptors (M1-AChR); however, the cellular
146 upled receptors, including the M1 muscarinic acetylcholine receptor (M1R), and opposes the Src tyrosi
147 with beta-arrestin binding to M1 muscarinic acetylcholine receptors (M1Rs) in two different binding
149 cini preferentially expressed the muscarinic acetylcholine receptor M3 and maintained physiological r
151 tory cytokines and decreased M3R (Muscarinic Acetylcholine receptor M3) and AQP5 (Aquaporin 5) protei
153 e previously reported that type 3 muscarinic acetylcholine receptors (M3-Rs) physically interact with
154 uction of antinuclear and anti-M3 muscarinic acetylcholine receptor (M3R) autoantibodies and impairme
155 stibular afferents is mediated by muscarinic acetylcholine receptor (mAChR) activation and the subseq
159 e allosteric modulators of the M5 muscarinic acetylcholine receptor (mAChR) have been described, but
160 in signaling of the M1 subtype of muscarinic acetylcholine receptor (mAChR) in the prefrontal cortex
161 extracellular vestibule of the M2 muscarinic acetylcholine receptor (mAChR) is targeted for structure
162 topic hybrids, based on the M1/M4 muscarinic acetylcholine receptor (mAChR) orthosteric agonist xanom
163 are selective agonists of the M1 muscarinic acetylcholine receptor (mAChR) that may gain their selec
164 Selective activation of the M1 muscarinic acetylcholine receptor (mAChR) via a positive allosteric
165 tylcholine receptors (nAChRs) and muscarinic acetylcholine receptors (mAChRs) are expressed in glomer
166 ion mediated, for example, by the muscarinic acetylcholine receptors (mAChRs) in relevant brain struc
167 innervation from the septum, and muscarinic acetylcholine receptors (mAChRs) share some signaling pa
171 d HIF-alpha isoform imbalance via muscarinic acetylcholine receptor-mediated Ca(2+) influx, and the r
172 pecific mechanisms of alpha3beta4*-nicotinic acetylcholine receptor modulation by the prototoxin lynx
173 ve processes that are modulated by nicotinic acetylcholine receptor (nAChR) activation by cholinergic
174 ials suggest that drugs that alter nicotinic acetylcholine receptor (nAChR) activity can affect behav
175 ending plasma levels of the alpha7-nicotinic acetylcholine receptor (nAChR) agonist 3-(2,4-dimethoxyb
177 icotine or ABT-418, an alpha4beta2 nicotinic acetylcholine receptor (nAChR) agonist, normalized MSO t
179 RNA5-CHRNB4 cluster coding for the nicotinic acetylcholine receptor (nAChR) alpha3, alpha5 and beta4
180 GeXIVA inhibits the alpha9alpha10 nicotinic acetylcholine receptor (nAChR) and is analgesic in anima
182 These effects were blocked by the nicotinic acetylcholine receptor (nAChR) antagonist mecamylamine.
187 a desensitized state of the alpha7 nicotinic acetylcholine receptor (nAChR) have been associated with
188 with this soluble surrogate of the nicotinic acetylcholine receptor (nAChR) in a cooperative fashion,
194 al role in the characterization of nicotinic acetylcholine receptor (nAChR) structure and function an
195 an antagonist of the alpha9alpha10 nicotinic acetylcholine receptor (nAChR) subtype and an inhibitor
196 Activation of the alpha3beta4 nicotinic acetylcholine receptor (nAChR) subtype has recently been
199 (ECD) of the human neuronal alpha2 nicotinic acetylcholine receptor (nAChR) subunit in complex with t
200 al are separate processes and that nicotinic acetylcholine receptor (nAChR) upregulation underlies ch
202 A7, the gene coding for the alpha7 nicotinic acetylcholine receptor (nAChR), and manifest a variable
203 CHRNA7, encoding for the alpha7 nicotinic acetylcholine receptor (nAChR), has been suggested as a
205 ve allosteric modulators (PAMs) of nicotinic acetylcholine receptors (nAChR) are important therapeuti
206 ulation and synaptic clustering of nicotinic acetylcholine receptors (nAChR) during neurotrophic fact
207 sociations between variants in the nicotinic acetylcholine receptors (nAChR) subunits and nicotine de
209 ed TGFBR2 and the nicotinic antiinflammatory acetylcholine receptor nAChRa7 as murine and human miR-2
210 ivation of 42 genes, including the nicotinic acetylcholine receptors nAChRalpha1 and nAChRalpha3, in
211 human alpha3beta2 and alpha6beta2 nicotinic acetylcholine receptor (nAChRs) and not the closely rela
224 ficant cholinergic innervation and nicotinic acetylcholine receptors (nAChRs) contribute greatly to t
225 t widely co-abused substances, and nicotinic acetylcholine receptors (nAChRs) contribute to the behav
228 hances nicotine-induced changes in nicotinic acetylcholine receptors (nAChRs) expressed on midbrain D
229 d shows high binding affinity with nicotinic acetylcholine receptors (nAChRs) expressed on the surfac
230 l localisation and function of the nicotinic acetylcholine receptors (nAChRs) formed by the subunits
232 There is much interest in alpha7 nicotinic acetylcholine receptors (nAChRs) in CNS function since t
235 a suggest a complex interaction of nicotinic acetylcholine receptors (nAChRs) in regulating vestibula
237 induces functional upregulation of nicotinic acetylcholine receptors (nAChRs) in the mesocorticolimbi
238 wing that the assembled functional nicotinic acetylcholine receptors (nAChRs) included the duplicated
239 dy demonstrates that activation of nicotinic acetylcholine receptors (nAChRs) increases excitatory ne
241 beta2 subunit-containing (beta2*) nicotinic acetylcholine receptors (nAChRs) is implicated in severa
247 but very little is known about how nicotinic acetylcholine receptors (nAChRs) regulate LHb activity.
248 ression and clustering of neuronal nicotinic acetylcholine receptors (nAChRs) remain poorly defined.
250 we show that SSS also antagonizes nicotinic acetylcholine receptors (nAChRs) to reduce synaptic tran
251 t of aging on presynaptic neuronal nicotinic acetylcholine receptors (nAChRs) within the circuitry of
252 G-PG interactions are modulated by nicotinic acetylcholine receptors (nAChRs), and our data suggest t
253 s of the ligand-binding domains of nicotinic acetylcholine receptors (nAChRs), and they reproduce som
262 dependence and stimulation of the nicotinic acetylcholine receptor on the ability to interpret valen
263 nd acute effects of stimulation of nicotinic acetylcholine receptors on behavioral and neural signatu
265 re, we report that the lateral motion of the acetylcholine receptors on live muscle cell membranes do
266 CHRFAM7A (gene-encoding dup-alpha7-nicotinic acetylcholine receptor) on a locus of chromosome 15 asso
267 ates") and their utility as alpha7 nicotinic acetylcholine receptor partial agonists are described.
268 strategy is to selectively target muscarinic acetylcholine receptors, particularly the M1 muscarinic
269 r in animals through inhibition of nicotinic acetylcholine receptors present in the central nervous s
270 t activation of the M4 subtype of muscarinic acetylcholine receptor reduces transmission at corticost
273 on of the ER-resident chaperone of nicotinic acetylcholine receptors, RIC-3, leads to increased muscl
274 CRs, but selectively inhibited M3 muscarinic acetylcholine receptor signaling ( approximately 50%) an
275 otinic acetylcholine receptors, they disturb acetylcholine receptor signaling leading to neurotoxicit
277 ously used for the preparation of muscarinic acetylcholine receptor subtype 1 positive allosteric mod
278 igra pars reticulata (SNr) act on muscarinic acetylcholine receptor subtype 4 (M4) to oppose cAMP-dep
280 ty in patients with PV toward the muscarinic acetylcholine receptor subtypes 3, 4, and 5 as well as t
281 ants near the apolipoprotein E and nicotinic acetylcholine receptor subunit alpha 5 genes are associa
283 of expression and function of the nicotinic acetylcholine receptor subunit cluster of alpha3, alpha5
284 ommon and rare variants of several nicotinic acetylcholine receptor subunits are associated with nico
285 species from all five subtypes of muscarinic acetylcholine receptors, suggesting allosteric binding.
287 h ion channels of the ligand-gated nicotinic acetylcholine receptor superfamily (namely alpha-amino-3
288 coupled receptors, such as the m1 muscarinic acetylcholine receptor, suppresses the M-current and ind
289 e of neuron, expressing non-alpha7 nicotinic acetylcholine receptors, that directly drives inhibition
290 nformation, and in skeletal muscle nicotinic acetylcholine receptors there is an exponential relation
295 -inflammatory alpha7-nAChR (alpha7-nicotinic acetylcholine receptor) was similar in young SHR and WKY
296 Lynx1, an endogenous inhibitor of nicotinic acetylcholine receptors, was previously shown to increas
297 probe the ion channel pore of the nicotinic acetylcholine receptor, which is a prototypical Cys-loop
298 ll types expressing alpha8 subunit nicotinic acetylcholine receptor, while SPO and cOv are characteri
299 the alpha and beta subunits of the nicotinic acetylcholine receptors with weak interaction, (b) dishe
300 t binding sites on the homopentameric alpha7-acetylcholine receptor, yet the number of bound alpha-Bt
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