ライフサイエンスコーパス: acid reflux

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1 Three patients exclusively had weakly acid reflux.

2 PIs even in the absence of abnormal rates of acid reflux.

3 vation that arise from early-life esophageal acid reflux.

4 agus, size of hiatus hernia, and severity of acid reflux.

5 of nausea were accompanied by an episode of acid reflux.

6 ral diaphragm, esophageal common cavity, and acid reflux.

7 Esophagitis, whether caused by acid reflux, allergic responses, graft-versus-host disea

8 She has past medical history significant for acid reflux and Clostridium difficile colitis.

9 Gastroesophageal acid reflux and human papillomavirus infection were rule

10 ad persistently pathological intraesophageal acid reflux and no change in villin or PCNA expression.

11 It is clear that acid reflux as identified by a conventional pH electrode

12 Gastroesophageal acid reflux at each of the pH sensors extending 5.5 cm p

13 Besides acid reflux, bile acid reflux may also play an important

14 Symptoms are more common with acid reflux but are also produced by nonacid reflux.

15 and acid taste were more commonly linked to acid reflux but were also produced by nonacid reflux.

16 Chronic acid reflux by itself is not likely to play a role in re

17 Ambulatory impedance testing underestimates acid reflux compared to esophageal acid exposure by disc

18 ce, which lack squamous epithelia, may model acid-reflux damage.

19 Percentage of acid reflux decreased (from 45% to 3%, P = 0.02) and non

20 tal hernia, and frequency of reflux, but not acid reflux, differed between CRs and ICRs.

21 The in vitro data suggest that acid reflux enhances the indirect alloresponse to proces

22 rolonged period of mucosal contact with each acid reflux episode, particularly in the recumbent posit

23 episodes (except the 30 mg dose) and weakly acid reflux episodes (all doses) significantly, relative

24 dose-dependently reduced the mean number of acid reflux episodes (except the 30 mg dose) and weakly

25 d more studies as abnormal than MII-detected acid reflux episodes [42 vs 34 % (p < 0.01)].

26 centage of time with a pH < 4, the number of acid reflux episodes and the percentage of proximal esop

27 ophagus, we found belt compression increased acid reflux following a meal.

28 The second step involves acid reflux (HCl or HNO(3)) to remove the residual growt

29 IGN, SETTING, AND PARTICIPANTS: The Study of Acid Reflux in Children With Asthma, a randomized, maske

30 gm inhibition, esophageal common cavity, and acid reflux in normal subjects.

31 Acid reflux-induced oesophagitis and the multilayered ep

32 d GERD; 4) in 50% of the patients with GERD, acid refluxed into the proximal esophagus.

33 Acid reflux is directly related to lower levels of LESP.

34 The mechanisms whereby bile acid reflux may accelerate the progression from Barrett'

35 The mechanisms whereby acid reflux may accelerate the progression from BE to EA

36 Besides acid reflux, bile acid reflux may also play an important role in the progr

37 Acid reflux may be a contributing factor in the pathogen

38 While patients with acid reflux may have a few eosinophils, patients with EE

39 less sensitive than pH-testing in detecting acid reflux off therapy as a result of discounting durat

40 It is possible that bile acid reflux present in patients with BE may increase rea

41 ur eyes to the trafficking of much more than acid reflux through the esophageal lumen.

42 Abnormal acid reflux was found to be the cause of intractable nau

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