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1 roton flux in tissues that maintain systemic acid-base balance.
2  no adverse changes in serum electrolytes or acid-base balance.
3 tion and is involved in Na(+) absorption and acid-base balance.
4 kaline-forming properties mediate the body's acid-base balance.
5 maintaining adequate fluid, electrolyte, and acid-base balance.
6 retion of acids and partially restore normal acid-base balance.
7 urine output, osmolality, salt excretion, or acid-base balance.
8 limit gross perturbations of electrolyte and acid-base balance.
9 ssibly because of their beneficial effect on acid-base balance.
10  ketone-body transport is needed to maintain acid-base balance.
11 duced ability of Oxgr1(-/-) mice to maintain acid-base balance.
12 nt composition, 4) micronutrient density, 5) acid-base balance, 6) sodium-potassium ratio, and 7) fib
13 tine, and that lack of the exchanger impairs acid-base balance and Na+-fluid volume homeostasis.
14 Hemodynamic resuscitation and maintenance of acid-base balance and tissue oxygen availability were pr
15      Volume resuscitation and maintenance of acid/base balance and tissue oxygen availability were pr
16 With the addition of acetazolamide, however, acid-base balance as well as ENaC subunit abundance and
17 cleic acid synthesis, and the maintenance of acid-base balance by urinary buffering.
18  used to examine changes in gas exchange and acid-base balance during exposure to therapeutic hyperca
19 triving to typify compensations for abnormal acid-base balance have reported their findings in terms
20   The topics examined included alteration in acid-base balance, hemodynamics, femoral venous flow, an
21               Atp6i-/- mice exhibit a normal acid-base balance in blood and urine.
22                      Dietary contribution to acid-base balance in early life may influence subsequent
23  precursors may have opposing effects on the acid-base balance in the body and therefore on bone loss
24 blood sample could be used to assess central acid-base balance in the early stage of arrest and cardi
25             The optimal approach to managing acid-base balance is less well defined for patients rece
26                    Local cerebral changes of acid-base balance may interfere with neuronal communicat
27 at regulates cell volume, ion transport, and acid-base balance; mice knocked out for ClC-2 are blind
28         Normoxic cells remotely regulate the acid-base balance of cells at the hypoxic core of connex
29 source, calcium intake, weight loss, and the acid/base balance of the diet.
30 uate the severity of in vivo electrolyte and acid-base balance perturbations.
31          a) To determine the relationship of acid-base balance (pH, PCO2) of blood samples from the i
32 ppear healthy and are normal with respect to acid-base balance, plasma electrolyte concentrations, se
33  on long-term NaCl reabsorption, BP, K+, and acid-base balance, processes that all are disrupted in F
34 lated cells, which maintain sodium/water and acid/base balance, respectively, but the epigenetic cont
35 ntifying the three predominant influences on acid-base balance: sodium chloride, albumin, and unmeasu
36 egulate calcium levels and the regulation of acid base balance via regulation of carbon dioxide level
37 , 67 (26%) had a different interpretation of acid base balance when the Fencl method was used compare

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