ライフサイエンスコーパス: acidotic

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1 n diets, and none of the uremic animals were acidotic.

2 blood analysis revealed that they are mildly acidotic.

3 In muscle of acidotic adrenalectomized rats receiving dexamethasone,

4 renal cortex of rats that were made acutely acidotic also exhibit a similar increase in binding to t

5 arbonate reabsorption capacity was higher in acidotic and NHE3 null animals.

6 In OMCDs from acidotic animals, and in OMCDs incubated in acid in vitr

7 ad an unusually mild clinical course without acidotic attacks.

8 crease UT-A protein in liver, rats were made acidotic, but not uremic, by feeding them HCl.

9 dosis would explain the predominance of this acidotic change; however, no increase in plasma volume o

10 I3-K/Akt pathway was evaluated in muscles of acidotic, CKD and pair-fed control rats under physiologi

11 n was greater in brush border membranes from acidotic compared with control rats.

12 urrent (INa) amplitude compared to WT; under acidotic conditions (pH 7.0) typically found with hypoxi

13 sine concentrations, in particular under the acidotic conditions associated with ischemia.

14 e of the IP(3) receptor or under nonspecific acidotic conditions.

15 roximately half of the inhibition was due to acidotic effects of NMDA-mediated currents, as demonstra

16 ral corticosteroids to all patients with non-acidotic exacerbations of COPD requiring hospital admiss

17 We recruited patients with non-acidotic exacerbations of COPD who were randomly assigne

18 bioenergetically more active, hypoxemic, and acidotic femoral circulation (P<0.05 versus cerebral).

19 = 0 hr), and at death (7.0 +/- 0.3 hrs) were acidotic, hypocapnic, and hypothermic (rectal temperatur

20 cosal hypoperfusion does not account for the acidotic mucosal tonometric pH in endotoxic shock.

21 he inner stripes of kidneys from control and acidotic (NH4Cl-fed for 3 d) rabbits.

22 bicarbonate reabsorption but are only mildly acidotic owing to reduced glomerular filtration rate and

23 ignificantly more hypercarbic (p < 0.01) and acidotic (p < 0.01) than those undergoing conventional l

24 Acidotic patients had worse hemodynamic indicators, with

25 Acidotic patients received fluid resuscitation with eith

26 e early use of NIV for mildly and moderately acidotic patients with COPD in the general ward setting

27 The initial protection by acidotic pH and glycine during simulated reperfusion was

28 During chemical hypoxia, acidotic pH prevented cell death.

29 Glycine and acidotic pH prevented cell killing after reperfusion but

30 7.4) and during 45 minutes of perfusion with acidotic (pH 6.0) Tyrode's solution with (n=8) and witho

31 ere was a 61% higher H flux in segments from acidotic rabbits (11.3+/-0.2 vs. 7.0+/-0.2 pmol/min per

32 Tubules from acidotic rabbits showed higher rates of HCO3- absorption

33 larized to total retinal area was smaller in acidotic rats (94%+/-4% versus 96%+/-2%, P < 0.001).

34 ocampus showed a slight increase in NDCBE in acidotic rats (p=0.05).

35 BCKAD E1alpha subunit mRNA in muscle of acidotic rats given dexamethasone was increased 1.89-fol

36 y in brush border membranes from control and acidotic rats is mediated by NHE3 and that metabolic aci

37 Acidotic rats showed growth retardation (final weight 16

38 etinal neovascularization occurred in 36% of acidotic rats versus 5% of controls (P < 0.001).

39 nal cytosolic extracts prepared from acutely acidotic rats.

40 ntly increased by 50% in livers from uremic, acidotic rats; bicarbonate administration prevented the

41 until acid-challenged, when they became more acidotic than +/+ animals.

42 infarct (P < 0.0001), which in turn was more acidotic than hypoperfused tissue that survived (P < 0.0

43 on channel 1 which, when activated under the acidotic tissue conditions found in inflammatory lesions

44 later be withdrawn, but -/- animals remained acidotic with alkaline urine.

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