ライフサイエンスコーパス: acme

1 ge of the arginine catabolic mobile element (ACME).

2 y island, arginine catabolic mobile element (ACME).

3 us of the arginine catabolic mobile element (ACME).

4 olates tested, regardless of the presence of ACME.

5 mecA, and arginine catabolic mobile element [ACME]).

6 To mitigate this, ACME also encodes SpeG, a polyamine-resistance enzyme th

7 300) differing in the presence or absence of ACME and a USA300 wild-type/ACME deletion mutant pair we

8 data underscore the functional modularity of ACME and its contribution to the success of USA300 CA-MR

9 the presence or absence of the SCCmec motif, ACME and the unique signature pattern for the prophage i

10 IVa, the arginine catabolic mobile element (ACME), and a specific mutation in capsular polysaccharid

11 ec types, arginine catabolic mobile element (ACME), and PVL-carrying prophage, PhiSa2 or PhiSa2-like

12 L) genes, arginine catabolic mobile element (ACME), and staphylococcal cassette chromosome mec typing

13 PVL), the arginine catabolic mobile element (ACME), and the staphylococcal cassette chromosome mec (S

14 A or VRSA isolates that carried PVL genes or ACME, and most strains (69.8%) were staphylococcal casse

15 tive for the presence of SCCmec type IV, the ACME, and the PVL toxin gene and matched the t008 or t12

16 ulent when evaluated prospectively, and PVL, ACME, and type IV SCCmec are associated with these absce

17 isolates, isolates positive for USA300, PVL, ACME, and type IV SCCmec were significantly associated w

18 Consequently, the ACME-Arc system drives excessive production of host poly

19 Nor was the presence of ACME associated with increased dermonecrosis in a model

20 s for the arginine catabolic mobile element (ACME), characteristic of USA300 MRSA.

21 ce or absence of ACME and a USA300 wild-type/ACME deletion mutant pair were analyzed for in vitro exp

22 ved) mutants, suggesting that the native and ACME-derived ADIs are compensatory in S. epidermidis.

23 ltaarcA2 (arginine catabolic mobile element [ACME]-derived) mutants, suggesting that the native and A

24 uring infection; however, the acquisition of ACME encoded speG allows USA-300 clones to circumvent po

25 We show that the constitutive ACME-encoded arginine-deiminase system (Arc) allows USA3

26 -specific arginine catabolic mobile element (ACME) encoding a Spm/Spd N-acetyltransferase that is nec

27 se data are consistent with a model in which ACME enhances growth and survival of USA300, allowing fo

28 c mutants containing deletions of SCCmec and ACME in a USA300 clinical isolate to determine the role

29 quence analysis confirmed the integration of ACME in orfX.

30 We noted a high prevalence of ACME in S epidermidis, suggesting not only that ACME tra

31 on of the arginine catabolic mobile element (ACME) in North American isolates and a novel copper and

32 dentified arginine catabolic mobile element (ACME) in the USA300 genome has been advocated as one pos

33 t, termed arginine catabolic mobile element (ACME), in other S aureus strains.

34 Unlike classical extensometers, ACME is mounted on a confocal microscope and uses confoc

35 We conclude that ACME is not necessary for virulence in rodent models of

36 Additionally, ACME is suitable for measuring the mechanical responses

37 bored an arginine catabolism mobile element (ACME) locus.

38 ants among ACME-positive isolates than among ACME-negative isolates, but there was no significant dif

39 ACME-negative strains were mainly isolated from patients

40 s around the world, and (3) co-occur with an acme of prasinophyte algae 'disaster taxa' also dominant

41 with the arginine catabolic mobile element (ACME), Panton-Valentine leukocidin (PVL), and other toxi

42 (PVL) and arginine catabolic mobile element (ACME) positive, and genetically related.

43 lpha-hemolysin in culture supernatants among ACME-positive isolates than among ACME-negative isolates

44 b server and tutorial can be found at http://acmes.rnet.missouri.edu.

45 A standard ACME screen uses the technologies previously applied to

46 ect competitive fitness, whereas deletion of ACME significantly attenuated the pathogenicity or fitne

47 Physical linkage between SCCmec and ACME suggests that selection for antibiotic resistance a

48 ed to as arrayed cellular microenvironments (ACMEs), that allows for a high-throughput examination of

49 However, the contribution of ACME to USA300's success in SSTIs is not completely unde

50 By using our software (ACME) to study somite formation, we were able to segment

51 E in S epidermidis, suggesting not only that ACME transfers into USA300 from S epidermidis, but also

52 apy in the Angioplasty Compared to Medicine (ACME) trial.

53 The presence of ACME was not associated with increased virulence in a ra

54 nt an automated confocal micro-extensometer (ACME), which greatly expands the scope of existing metho