ライフサイエンスコーパス: acoustic neuroma

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1 than nicotine may confer protection against acoustic neuroma.

2 lf-report have suggested an association with acoustic neuroma.

3 tic hair dye use with glioma, meningioma, or acoustic neuroma.

4 irmed glioma, 197 had meningioma, and 96 had acoustic neuroma.

5 ween occupational exposure to loud noise and acoustic neuroma.

6 r diagnosing auditory nerve disorders due to acoustic neuromas.

7 nt confidence interval, 0.3 to 1.7), 1.4 for acoustic neuroma (95 percent confidence interval, 0.6 to

8 resonance imaging, which is used to look for acoustic neuromas, abnormal labyrinth signal intensity o

9 tatistically significant association between acoustic neuroma and persistent occupational noise expos

10 of mobile phone use with the risks of brain, acoustic neuroma, and parotid gland tumors.

11 ults from studies of loud noise exposure and acoustic neuroma are conflicting.

12 tive patients who underwent radiosurgery for acoustic neuromas between 1987 and 1992 by means of seri

13 e the association between noise exposure and acoustic neuroma by using an objective measure of exposu

14 out hearing protection was more common among acoustic neuroma cases (odds ratio = 1.47, 95% confidenc

15 A total of 793 acoustic neuroma cases aged 21-84 years were identified

16 d nonoccupational loud noise exposure of 146 acoustic neuroma cases and 564 controls.

17 total of 410 glioma, 178 meningioma, and 90 acoustic neuroma cases and 686 controls responded to a s

18 unilateral hearing loss (UHL) patients with acoustic neuromas compared to 24 normal controls.

19 to loud noise without hearing protection and acoustic neuroma, especially among women, we cannot rule

20 to further examine the role of loud noise in acoustic neuroma etiology.

21 association between use of snuff tobacco and acoustic neuroma has not been investigated previously.

22 sly identified as a possible risk factor for acoustic neuroma in only one relatively small (n = 86 ca

23 h glioma (n = 489), meningioma (n = 197), or acoustic neuroma (n = 96) between 1994 and 1998 at three

24 e from any source were at increased risk for acoustic neuroma (odds ratio (OR) = 1.55, 95% confidence

25 among males was not associated with risk of acoustic neuroma (OR = 0.94, 95% CI: 0.57, 1.55).

26 , and/or construction increased the risk for acoustic neuroma (OR = 1.79, 95% CI: 1.11, 2.89), as did

27 ce of an increase in the risk of meningioma, acoustic neuroma, or parotid gland tumors in relation to

28 A population-based case-control study of 451 acoustic neuroma patients and 710 age-, sex-, and region

29 re consistent with previous reports of lower acoustic neuroma risk among current cigarette smokers th

30 nt findings did not demonstrate an increased acoustic neuroma risk related to occupational noise expo

31 udies suggest that cigarette smoking reduces acoustic neuroma risk; however, an association between u

32 ence of an association between snuff use and acoustic neuroma suggests that some constituent of tobac

33 l alternative to microsurgical resection for acoustic neuromas (vestibular schwannomas).

34 The risk of acoustic neuroma was greatly reduced in male current smo

35 adiosurgery can provide long-term control of acoustic neuromas while preserving neurologic function.

36 The findings of an increased risk of acoustic neuroma with loud noise exposure support previo

37 vated odds ratios for glioma, meningioma, or acoustic neuroma with use or prolonged use of permanent,

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