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1  had a simpler waveform than the response to acoustic stimulation.
2 ortex changed with the spectral range of the acoustic stimulation.
3 e administration, and moderate-to-high level acoustic stimulation.
4 xcitation indicates a requirement for strong acoustic stimulation.
5 c seizures (AGS) can be triggered by intense acoustic stimulation.
6 throughout the tonotopic field determined by acoustic stimulation.
7 instem after 1 hour of continuous free-field acoustic stimulation.
8  a cochlear ablation, even in the absence of acoustic stimulation.
9 awal (ETX) in rodents can be precipitated by acoustic stimulation.
10 e for modulating auditory nerve responses to acoustic stimulation.
11  of synaptic inputs to the IC in response to acoustic stimulation.
12 e units in response to monaural and binaural acoustic stimulation.
13  nucleus magnocellularis (NM) neurons during acoustic stimulation.
14 lizations are known, and they all respond to acoustic stimulation.
15 excitation that are normally associated with acoustic stimulation.
16  between opsins and less robust overall than acoustic stimulation.
17                                We found that acoustic stimulation alone evokes a change in the freque
18                                As repetitive acoustic stimulation and auditory conditioning do, elect
19 n auditory cortex channels that responded to acoustic stimulation and demonstrated well-defined frequ
20 lvian sulcus (FAES) is largely responsive to acoustic stimulation and its unilateral deactivation res
21 om olivocochlear terminals during high-level acoustic stimulation and suggest that muscarinic antagon
22 n normal animals, expression was produced by acoustic stimulation and was found to be tonotopically d
23 als generally display transient responses to acoustic stimulation, and typically respond to a brief s
24                                              Acoustic stimulation at either 10 kHz or 40 kHz was used
25 eduction of surface AMPA receptors following acoustic stimulation correlated with changes in acoustic
26 x extracellular matrix which, in response to acoustic stimulation, displaces the hair bundles of oute
27 For cochlear implant users, combined electro-acoustic stimulation (EAS) significantly improves the pe
28                        Combined electric and acoustic stimulation has proven to be an effective strat
29  well as entrainment to rhythmic noise-burst acoustic stimulation in 14% of electrodes.
30 thin auditory cortex by combining free-field acoustic stimulation in the frontal azimuthal plane with
31                            Combining dynamic acoustic stimulation in virtual space with extracellular
32 nists in cultured neurons and in response to acoustic stimulation in vivo.
33 or reactivity was observed with tactile (vs. acoustic) stimulation in both TBI and naive rats althoug
34 otility and hair bundle movement, to amplify acoustic stimulations increasing hearing sensitivity and
35                         Moreover, repetitive acoustic stimulation induces robust short-term habituati
36 ly being sensitive to rapid relative to slow acoustic stimulation, insensitive to the difference betw
37 is change in the IC becomes greater when the acoustic stimulation is made behaviorally relevant by pa
38                    Moreover, after 30 min of acoustic stimulation, levels of Kv3.1b immunoreactivity
39                            With increases in acoustic stimulation, males lengthen call duration while
40 ectrical stimulation of a deaf ear can mimic acoustic stimulation of a normal-hearing ear.
41                           We also found that acoustic stimulation of egr-1 and fos differed in the th
42 cts whose hearing is evoked by either normal acoustic stimulation or electric stimulation of the audi
43 to be combined within the same ear (electric-acoustic stimulation, or EAS) and/or across ears (bimoda
44                     For instance, persistent acoustic stimulation produces sensory adaptation, which
45                                  Strikingly, acoustic stimulation promotes Foxo3 nuclear localization
46  of rippling water (SW), and 3) rest without acoustic stimulation (R).
47 cations in perceived tinnitus loudness after acoustic stimulation (residual inhibition) [4], permitti
48 resolved which statistical parameters in the acoustic stimulation spectrum affect frequency-specific
49 range as well as the spectral spacing of the acoustic stimulation spectrum on frequency-specific neur
50                                         Upon acoustic stimulation, the partition responds principally
51 odest epileptiform EEG activity on the first acoustic stimulation to progressively higher amplitude,
52 ed spike fidelity surpassed ChR2 and natural acoustic stimulation to support a superior code for the
53                                              Acoustic stimulation vibrates the cochlear basilar membr
54 s in c-fos mRNA expression in the absence of acoustic stimulation were observed in the superficial do
55                          Neural responses to acoustic stimulation were present during both slow-wave
56 coupled to the overall spectral range of the acoustic stimulation, which suggests that neural adjustm
57 electric ITD tuning is as sharp as found for acoustic stimulation with broadband noise in normal-hear
58 odulated their discharge rate in response to acoustic stimulation with species-specific calls.

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