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1 ignaling after aminoglycoside ototoxicity or acoustic trauma.
2 ey role for ion homeostasis in resistance to acoustic trauma.
3 enerate conditioning-induced protection from acoustic trauma.
4 ding the differential sensitivity of ANFs to acoustic trauma.
5 s conditioned protection of the cochlea from acoustic trauma.
6 cochlea, and organ of Corti, all targets for acoustic trauma.
7 o accumulate in the murine cochlea following acoustic trauma.
8 y input and protection of the inner ear from acoustic trauma.
9 nction and ribbon synapse regeneration after acoustic trauma.
10                                  Second, the acoustic trauma altered dendritic morphology and decreas
11 imuli known to cause hair cell loss, such as acoustic trauma and aminoglycoside administration.
12  cochlear pericytes are markedly affected by acoustic trauma and display an abnormal morphology.
13  are vulnerable to a variety of insults like acoustic trauma and ototoxic drugs.
14 d tip links of hair cells are susceptible to acoustic trauma and ototoxic drugs.
15                          We exposed flies to acoustic trauma and quantified physiological and anatomi
16  million individuals, [2] often results from acoustic trauma and, [3] is very often exacerbated under
17 ured cochlea during the first week following acoustic trauma, and further BMDC accumulation was seen
18 ed by others in the auditory nerve following acoustic trauma, and suggest that the map alterations ha
19  in the auditory nerve following less severe acoustic trauma, and thus would seem to have a periphera
20                                              Acoustic trauma (AT, loud sounds) slow AMPAR-EPSC decay
21                    Damaging levels of sound (acoustic trauma, AT) diminish peripheral synapses, but w
22 ed to resist cochlear damage associated with acoustic trauma by exposure to a variety of "conditionin
23                                 We find that acoustic trauma causes activation of PVM/Ms and physical
24                     Thus, Drosophila exhibit acoustic trauma effects resembling those found in verteb
25 s in the modulation of cochlear responses to acoustic trauma in rats.
26                             We simulated the acoustic trauma-induced tip link damage or stereociliary
27                        The primary effect of acoustic trauma is manifested as damage to the delicate
28 cochlear sensory cell degeneration following acoustic trauma is unknown.
29            This study explored the effect of acoustic trauma on cochlear pericytes.
30                                              Acoustic trauma, one of the leading causes of sensorineu
31 lls of the inner ear undergo apoptosis after acoustic trauma or aminoglycoside antibiotic treatment,
32 ccurring during hair cell regeneration after acoustic trauma or aminoglycoside treatment.
33                 Sensory hair cells die after acoustic trauma or ototoxic insults, but the signal tran
34  the cochlea in response to injury caused by acoustic trauma or ototoxicity, but the nature of the in
35                         Hair cells die after acoustic trauma, ototoxic drugs or aging diseases, leadi
36 ude surgical ablation of the organ of Corti, acoustic trauma, ototoxic drugs, and hereditary deafness
37           The susceptibility of tip links to acoustic trauma raises questions as to whether these fra
38 icient in PLZF have hearing and responses to acoustic trauma similar to their wild type littermates b
39                    When birds are exposed to acoustic trauma, the normal pattern and number of nerve
40 VM/Ms and endothelial barrier breakdown from acoustic trauma to the mouse ear.
41 r synergistic protection of the cochlea from acoustic trauma when given together with DFO and mannito
42 sceptible to glutamate excitotoxicity and to acoustic trauma, with potentially adverse consequences t

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