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1 ignaling after aminoglycoside ototoxicity or acoustic trauma.
2 ey role for ion homeostasis in resistance to acoustic trauma.
3 enerate conditioning-induced protection from acoustic trauma.
4 ding the differential sensitivity of ANFs to acoustic trauma.
5 s conditioned protection of the cochlea from acoustic trauma.
6 cochlea, and organ of Corti, all targets for acoustic trauma.
7 o accumulate in the murine cochlea following acoustic trauma.
8 y input and protection of the inner ear from acoustic trauma.
9 nction and ribbon synapse regeneration after acoustic trauma.
16 million individuals, [2] often results from acoustic trauma and, [3] is very often exacerbated under
17 ured cochlea during the first week following acoustic trauma, and further BMDC accumulation was seen
18 ed by others in the auditory nerve following acoustic trauma, and suggest that the map alterations ha
19 in the auditory nerve following less severe acoustic trauma, and thus would seem to have a periphera
22 ed to resist cochlear damage associated with acoustic trauma by exposure to a variety of "conditionin
31 lls of the inner ear undergo apoptosis after acoustic trauma or aminoglycoside antibiotic treatment,
34 the cochlea in response to injury caused by acoustic trauma or ototoxicity, but the nature of the in
36 ude surgical ablation of the organ of Corti, acoustic trauma, ototoxic drugs, and hereditary deafness
38 icient in PLZF have hearing and responses to acoustic trauma similar to their wild type littermates b
41 r synergistic protection of the cochlea from acoustic trauma when given together with DFO and mannito
42 sceptible to glutamate excitotoxicity and to acoustic trauma, with potentially adverse consequences t
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