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1 iated disease; 14 [37%] of 38 with community-acquired disease).
2 patients (five with congenital and five with acquired disease).
3 ion SEPs (two with congenital and three with acquired disease).
4 pendent phosphorylation in human genetic and acquired disease.
5 patterns, including patients with community-acquired disease.
6 atients, including 4 patients with community-acquired disease.
7 ngly recognized as a model for inherited and acquired disease.
8 after myocardial infarction (MI), a model of acquired disease.
9 than those individuals with non-transfusion-acquired disease.
10 n their function underlie both inherited and acquired disease.
11 ic diseases, tumors, vascular anomalies, and acquired diseases.
12 rapies and research models for inherited and acquired diseases.
13 the genome and may manifest as inherited or acquired diseases.
14 inical trials to treat genetic disorders and acquired diseases.
15 n the clinical treatment of many genetic and acquired diseases.
16 target for gene therapies for inherited and acquired diseases.
17 n the management of several gene defects and acquired diseases.
18 therapeutic strategy for many inherited and acquired diseases.
19 is vector for the treatment of inherited and acquired diseases.
20 gical calcium signaling, both in genetic and acquired diseases.
21 the relevance of these findings in naturally acquired disease, a composite influenza A signature buil
22 channel that is important in hereditary and acquired diseases affecting urine-concentrating ability.
23 allow us to address the epigenetic state of acquired disease and whether original states, regenerati
24 lly treating a large array of hereditary and acquired diseases, and stands as the paradigm for stem c
29 tellation that develops secondary to various acquired diseases, especially intrathoracic neoplasm.
30 field of gene therapy for both inherited and acquired diseases, especially with regard to respiratory
34 nitially knew little about the diseases, but acquired disease information and increased knowledge of
35 s for phenotypic expression of heritable and acquired diseases involving abnormality in the ABCC6 gen
42 ly curative treatment for both inherited and acquired diseases of the hematopoietic compartment; howe
45 tive differences in gene-for-gene, basal and acquired disease resistance and other aspects of plant i
49 ients with congenital disease and those with acquired disease, suggesting that factors additional to
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